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Kidney International, ISSN 0085-2538, 12/2005, Volume 68, Issue 6, pp. 2473 - 2483
Journal Article
Cell Cycle, ISSN 1538-4101, 11/2015, Volume 14, Issue 22, pp. 3527 - 3532
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 5, p. e19727
When cells experience environmental stresses, global translational arrest is often accompanied by the formation of stress granules (SG) and an increase in the... 
LOCALIZATION | RECRUITMENT | P-BODIES | DECAY | PHOSPHORYLATION | CELL-MIGRATION | MAMMALIAN STRESS GRANULES | BIOLOGY | BINDING PROTEIN | NATURAL-PRODUCT PATEAMINE | SACCHAROMYCES-CEREVISIAE | Arsenites - pharmacology | Exosomes - drug effects | Exosomes - metabolism | Humans | Actins - metabolism | Nocodazole - pharmacology | Microtubule-Organizing Center - drug effects | Peptide Chain Initiation, Translational - drug effects | RNA, Messenger - metabolism | Actins - genetics | Cell Nucleus - metabolism | Epoxy Compounds - pharmacology | Molecular Probes - metabolism | Cytoplasmic Granules - metabolism | RNA Transport - drug effects | Macrolides - pharmacology | Puromycin - pharmacology | Stress, Physiological - drug effects | Microtubule-Organizing Center - metabolism | Cell Survival - drug effects | Cytoplasmic Granules - drug effects | RNA, Messenger - genetics | Sodium Compounds - pharmacology | Cycloheximide - pharmacology | Poly A - metabolism | Cell Line, Tumor | Thiazoles - pharmacology | Cell Nucleus - drug effects | Messenger RNA | Muscle proteins | Actin | Genetic translation | Arsenic compounds | Oxidative stress | Epithelial cells | Trafficking | Hybridization | Puromycin | Proteins | RNA probes | Fibroblasts | Inhibition | Sodium arsenite | Stress response | Localization | Biomedical engineering | Biodegradation | Enzymes | Translation | DNA probes | Translation initiation | Gene expression | Environmental stress | Cycloheximide | Ribonucleic acids | Sodium | Microscopy | Granular materials | Cell lines | Granules | Immunofluorescence | Arsenite | Cytoplasm
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 06/2009, Volume 329, Issue 3, pp. 856 - 864
Because macrophages play a major role in atherosclerotic plaque destabilization, selective removal of macrophages represents a promising approach to stabilize... 
PATHWAYS | PUROMYCIN | SIGNAL | CELLS | CYCLOHEXIMIDE | CAROTID-ARTERY | MAP-KINASES | RIBOSOMES | PHARMACOLOGY & PHARMACY | FATE | EXPRESSION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Tunica Media - cytology | Nitriles - pharmacology | Tunica Media - drug effects | Apoptosis - drug effects | Carotid Stenosis - drug therapy | Extracellular Signal-Regulated MAP Kinases - metabolism | Carotid Arteries - cytology | Tunica Media - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Rabbits | Butadienes - pharmacology | Cells, Cultured | Imidazoles - pharmacology | Carotid Stenosis - pathology | Macrophages - cytology | Anthracenes - pharmacology | Tunica Intima - drug effects | Tunica Intima - metabolism | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Tunica Intima - cytology | Macrophages, Alveolar - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Anisomycin - pharmacology | Macrophages - drug effects | Macrophages, Alveolar - cytology | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Aorta - cytology | Anisomycin - therapeutic use | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 02/2004, Volume 18, Issue 2, pp. 402 - 411
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92003
Our previous in vitro studies suggested that cyclic AMP (cAMP) signaling prevents adriamycin (ADR) and puromycin aminonucleoside (PAN)-induced apoptosis in... 
IN-VITRO | EPITHELIAL-CELLS | PATHWAY | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | RECEPTOR | GLOMERULAR PODOCYTES | STRESS | EXPRESSION | NEPHROPATHY | DAMAGE | Enzyme Activators - pharmacology | Dynamins - metabolism | Phosphorylation | WT1 Proteins - metabolism | Kidney - pathology | Caspase 3 - metabolism | Nephrosis - metabolism | Kidney - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Caspase 3 - genetics | Cyclic AMP - metabolism | WT1 Proteins - genetics | Quinazolinones - pharmacology | Cyclic AMP-Dependent Protein Kinases - metabolism | Puromycin Aminonucleoside - pharmacology | Kidney - drug effects | Nephrosis - pathology | Podocytes - metabolism | Signal Transduction | Colforsin - pharmacology | Gene Expression Regulation | Dynamins - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Nephrosis - genetics | Podocytes - pathology | Nephrosis - chemically induced | Cyclic AMP - pharmacology | Animals | GTP Phosphohydrolases - metabolism | Mitochondrial Dynamics - drug effects | Podocytes - drug effects | GTP Phosphohydrolases - genetics | Arachidonic Acid - pharmacology | Mice | Enzyme Activation | Cell Line, Transformed | Doxorubicin - pharmacology | Apoptosis | Anthracyclines | Cyclic adenylic acid | Cell differentiation | Fatty acids | Protein kinases | Cell proliferation | Protein kinase A | Nephrology | Activation | Arachidonic acid | Kinases | Caspase-3 | Experiments | Fission | Puromycin | Proteins | Adenylate cyclase | Mitochondria | Rodents | Membrane potential | Urine | Cyclic AMP | Caspase | Medicine | Studies | Signaling | Hospitals | Acids | Forskolin | Cytoskeleton | Kidney diseases | Laboratory animals | AMP
Journal Article
Molecular Oncology, ISSN 1574-7891, 08/2016, Volume 10, Issue 7, pp. 1099 - 1117
The synthetic steroid mifepristone blocks the growth of ovarian cancer cells, yet the mechanism driving such effect is not entirely understood. Unbiased... 
Bortezomib | mRNA translation | Chloroquine | Ubiquitin proteasome system | Autophagic flux | ER stress | Mifepristone | Ovarian cancer | Unfolded protein response | BORTEZOMIB TREATMENT | ER-ASSOCIATED DEGRADATION | DEATH | QUALITY-CONTROL | THERAPY | CISPLATIN | ONCOLOGY | ENDOPLASMIC-RETICULUM-STRESS | GLUCOCORTICOID-RECEPTOR-BETA | PATHWAY | GENE-EXPRESSION | Apoptosis - drug effects | Humans | Ovarian Neoplasms - pathology | Gene Expression Profiling | Thiourea - pharmacology | RNA, Messenger - metabolism | Autophagy - drug effects | Ovarian Neoplasms - genetics | Chloroquine - pharmacology | Lysosomes - metabolism | Eukaryotic Initiation Factor-2 - metabolism | Puromycin - pharmacology | Female | Protein Biosynthesis - genetics | Gene Expression Regulation, Neoplastic - drug effects | Platinum - pharmacology | Lysosomes - drug effects | Unfolded Protein Response - drug effects | Proteasome Inhibitors - pharmacology | RNA, Messenger - genetics | Cinnamates - pharmacology | Activating Transcription Factor 4 - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Cell Line, Tumor | Protein Biosynthesis - drug effects | Cell Proliferation - drug effects | Mifepristone - pharmacology | Thiourea - analogs & derivatives | Medical colleges | Oncology, Experimental | Genes | Research | Glucose | Genetic translation | Dextrose | Motor vehicle driving | Messenger RNA | Cell death | Automobile driving | Cancer cells | Tumor proteins | Cancer | Protein binding | Drugs | p53 Protein | CCAAT/enhancer-binding protein | Kinases | Cancer therapies | Autophagy | Cell growth | Protein folding | Platinum | Penicillin | Cell cycle | Deoxyribonucleic acid--DNA | Phenotypes | Translation | Splicing | Tumor cell lines | Proteasome inhibitors | Chemotherapy | Protein synthesis | Cell lines | Proteasomes | Endoplasmic reticulum | Apoptosis | Pharmaceuticals
Journal Article