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Journal of Neuroscience, ISSN 0270-6474, 11/2009, Volume 29, Issue 47, pp. 14734 - 14740
Journal Article
Diabetes, ISSN 0012-1797, 2014, Volume 63, Issue 12, pp. 4186 - 4196
Gut-derived hormones, such as GLP-1, have been proposed to relay information to the brain to regulate appetite. GLP-1 receptor agonists, currently used for the... 
HIGH-CALORIE | GLUCAGON-LIKE PEPTIDE-1 | FOOD-INTAKE | GLUCOSE | EXENATIDE | ENDOCRINOLOGY & METABOLISM | ENERGY-INTAKE | OPEN-LABEL | WEIGHT | ORBITOFRONTAL CORTEX | PROMOTES SATIETY | Amygdala - drug effects | Receptors, Glucagon - antagonists & inhibitors | Putamen - physiology | Humans | Middle Aged | Male | Peptide Fragments - pharmacology | Diabetes Mellitus, Type 2 - metabolism | Brain - physiology | Case-Control Studies | Cerebral Cortex - metabolism | Brain - metabolism | Prefrontal Cortex - drug effects | Adult | Female | Appetite - physiology | Cerebral Cortex - drug effects | Receptors, Glucagon - agonists | Amygdala - metabolism | Appetite - drug effects | Putamen - drug effects | Feeding Behavior - physiology | Feeding Behavior - drug effects | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Peptides - pharmacology | Brain - drug effects | Cross-Over Studies | Magnetic Resonance Imaging | Functional Neuroimaging | Glucagon-Like Peptide-1 Receptor | Prefrontal Cortex - metabolism | Cerebral Cortex - physiology | Aged | Venoms - pharmacology | Reward | Photic Stimulation | Amygdala - physiology | Prefrontal Cortex - physiology | Receptors, Glucagon - physiology | Putamen - metabolism | Type 2 diabetes | Care and treatment | Brain research | Analysis | Appetite depressants | Dosage and administration | Glycemic index | Research
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 11/2011, Volume 339, Issue 2, pp. 530 - 536
Journal Article
Psychopharmacology, ISSN 0033-3158, 10/2016, Volume 233, Issue 19-20, pp. 3527 - 3536
Journal Article
Journal of Neurosurgery, ISSN 0022-3085, 02/2005, Volume 102, Issue 2, pp. 216 - 222
Object. Glial cell line-derived neurotrophic factor (GDNF) has demonstrated significant antiparkinsonian actions in several animal models and in a recent pilot... 
Striatum | Dopaminergic neuron | Core Assessment Program for Surgical Interventional Therapies | Phase II trial | Unified Parkinson's Disease Rating Scale | SURGERY | RAT STRIATUM | FOLLOW-UP | core assessment program for surgical interventional therapies dopaminergic neuron | DOPAMINERGIC-NEURONS | unified Parkinson's disease rating scale | AGED RHESUS-MONKEYS | CLINICAL NEUROLOGY | striatum | PRIMATE MODELS | IN-VIVO | FACTOR INCREASES | CONVECTION-ENHANCED DELIVERY | FACTOR GDNF | BRAIN | Motor Activity - physiology | Humans | Middle Aged | Motor Activity - drug effects | Male | Parkinson Disease - drug therapy | Postural Balance - drug effects | Gait - physiology | Psychomotor Performance - physiology | Dose-Response Relationship, Drug | Dominance, Cerebral - physiology | Putamen - physiopathology | Glial Cell Line-Derived Neurotrophic Factor | Reaction Time - drug effects | Female | Dominance, Cerebral - drug effects | Infusion Pumps, Implantable | Drug Administration Schedule | Nerve Growth Factors - administration & dosage | Treatment Outcome | Psychomotor Performance - drug effects | Reaction Time - physiology | Parkinson Disease - physiopathology | Putamen - drug effects | Gait - drug effects | Parkinson Disease - diagnosis | Aged | Antiparkinson Agents - administration & dosage | Postural Balance - physiology
Journal Article
Translational Psychiatry, ISSN 2158-3188, 05/2015, Volume 5, Issue 4, pp. e549 - e549
Journal Article
JAMA, ISSN 0098-7484, 03/2009, Volume 301, Issue 11, pp. 1148 - 1154
Journal Article
American Journal of Psychiatry, ISSN 0002-953X, 03/2001, Volume 158, Issue 3, pp. 377 - 382
OBJECTIVE: Methamphetamine is a popular and highly addictive drug of abuse that has raised concerns because it has been shown in laboratory animals to be... 
POSITRON-EMISSION-TOMOGRAPHY | PSYCHIATRY | INDUCED NEUROTOXICITY | PET LIGAND | USERS | BINDING | BRAIN | PARKINSONS-DISEASE | C-11 WIN-35,428 | Brain Chemistry - drug effects | Verbal Learning - drug effects | Brain - diagnostic imaging | Methamphetamine - adverse effects | Humans | Cerebellum - drug effects | Male | Carrier Proteins - drug effects | Brain - metabolism | Psychomotor Disorders - metabolism | Cerebellum - diagnostic imaging | Neuropsychological Tests - statistics & numerical data | Adult | Female | Membrane Glycoproteins | Carrier Proteins - analysis | Dopamine - analysis | Substance-Related Disorders - diagnostic imaging | Dopamine - metabolism | Dopamine Plasma Membrane Transport Proteins | Methamphetamine - pharmacology | Substance-Related Disorders - metabolism | Cerebellum - metabolism | Putamen - diagnostic imaging | Nerve Tissue Proteins | Putamen - drug effects | Caudate Nucleus - metabolism | Brain - drug effects | Methamphetamine - metabolism | Psychomotor Disorders - chemically induced | Caudate Nucleus - diagnostic imaging | Carrier Proteins - metabolism | Tomography, Emission-Computed - statistics & numerical data | Membrane Transport Proteins | Psychomotor Disorders - diagnosis | Caudate Nucleus - drug effects | Methylphenidate - metabolism | Carbon Radioisotopes | Putamen - metabolism | Physiological aspects | Drug addicts | Dopamine | Psychomotor disorders | Methamphetamine | Neurotoxic agents
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2004, Volume 24, Issue 13, pp. 3344 - 3354
Repeated exposure to cocaine can induce neuroadaptations in the brain. One mechanism by which persistent changes occur involves alterations in gene expression... 
c-Fos | Signal transduction | Cocaine | Gene expression | Dopamine receptors | Mitogen-activated protein kinases | RAT STRIATUM | cocaine | signal transduction | MAP KINASE | C-FOS INDUCTION | SYNAPTIC PLASTICITY | NEUROSCIENCES | NUCLEUS-ACCUMBENS | MUTANT MICE | dopamine receptors | DELTA-FOSB | BEHAVIORAL-RESPONSES | mitogen-activated protein kinases | PRODYNORPHIN GENE | KINASE CASCADE | gene expression | Brain Chemistry - drug effects | Oligonucleotide Array Sequence Analysis | Proto-Oncogene Proteins c-fos - biosynthesis | Receptors, Dopamine D2 - metabolism | Receptors, Dopamine D1 - metabolism | Brain - metabolism | Cocaine-Related Disorders - metabolism | Mice, Mutant Strains | Receptors, Dopamine D2 - drug effects | Neurons - metabolism | Neurons - drug effects | Intracellular Fluid - metabolism | Receptors, Dopamine D1 - drug effects | Gene Expression Regulation - physiology | Mice, Transgenic | Enzyme Activation - drug effects | Putamen - drug effects | Caudate Nucleus - metabolism | Brain - drug effects | Gene Expression Regulation - drug effects | Cocaine - pharmacology | Animals | Signal Transduction - drug effects | Receptors, Dopamine D3 | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Mice | Caudate Nucleus - drug effects | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinases - metabolism | Putamen - metabolism | Behavioral | Cognitive | Systems
Journal Article
Synapse, ISSN 0887-4476, 06/2010, Volume 64, Issue 6, pp. 432 - 439
Bisphenol A (BPA), an environmental endocrine-disrupting chemical, has been extensively evaluated for reproductive toxicity and carcinogenicity. However,... 
Endocrine disrupter | Bisphenol A | Anxiolytic behaviors | NMDA receptor | Dopamine receptor | Cognitive deficit | cognitive deficit | NEONATAL EXPOSURE | MALE RATS | SOCIAL ANXIETY DISORDER | anxiolytic behaviors | D-2 RECEPTORS | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | dopamine receptor | PREFRONTAL CORTEX | endocrine disrupter | MEMORY | PERINATAL EXPOSURE | DEVELOPMENTAL EXPOSURE | Dopamine Plasma Membrane Transport Proteins - metabolism | Memory Disorders - physiopathology | Frontal Lobe - metabolism | Prenatal Exposure Delayed Effects - psychology | Dopamine Plasma Membrane Transport Proteins - drug effects | Phenols - toxicity | Cognition Disorders - chemically induced | Receptors, N-Methyl-D-Aspartate - metabolism | Hippocampus - drug effects | Anti-Anxiety Agents - toxicity | Receptors, Dopamine D2 - metabolism | Receptors, Dopamine D1 - metabolism | Brain - metabolism | Receptors, Dopamine D2 - drug effects | Behavior, Animal - drug effects | Female | Cognition Disorders - psychology | Disease Models, Animal | Animals, Newborn | Cognition Disorders - physiopathology | Memory Disorders - psychology | Receptors, N-Methyl-D-Aspartate - drug effects | Benzhydryl Compounds | Maze Learning - physiology | Mental Disorders - chemically induced | Receptors, Dopamine D1 - drug effects | Behavior, Animal - physiology | Prenatal Exposure Delayed Effects - physiopathology | Maze Learning - drug effects | Mice, Inbred ICR | Mental Disorders - physiopathology | Brain - drug effects | Mental Disorders - psychology | Pregnancy | Hippocampus - metabolism | Animals | Estrogens, Non-Steroidal - toxicity | Frontal Lobe - drug effects | Mice | Prenatal Exposure Delayed Effects - chemically induced | Memory Disorders - chemically induced | Endocrine disruptors | Glutamic acid receptors (ionotropic) | Toxicity | Memory | Cognitive ability | N-Methyl-D-aspartic acid receptors | Putamen | Anxiolytics | Open field behavior | Locomotion | Dopamine D1 receptors | Dopamine transporter | Anxiety | Carcinogenicity | Prenatal experience | Short term memory | Dopamine D2 receptors | Synapses | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2014, Volume 124, Issue 3, pp. 1340 - 1349
Journal Article
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 03/2017, Volume 140, Issue 5, pp. 728 - 740
Food restricted (FR) or obesogenic (OB) diets are known to oppositely alter behavioral responses to psychostimulants that target the dopamine transporter... 
CFT | food restriction | insulin |