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Neuron (Cambridge, Mass.), ISSN 0896-6273, 03/2012, Volume 73, Issue 6, pp. 1116 - 1126
... of the neural tube and, cells adopt specific fates based upon their level of exposure to the established Shh gradient. At later stages, during development... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Silver Staining - methods | RNA, Small Interfering - genetics | Furans - metabolism | Electric Stimulation | gamma-Aminobutyric Acid - metabolism | Channelrhodopsins | Hedgehog Proteins - metabolism | Dendritic Spines - metabolism | Functional Laterality - genetics | Nerve Net - cytology | Synaptophysin - genetics | Neurons - ultrastructure | Neurons - metabolism | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Synaptophysin - metabolism | Animals, Newborn | Fluorobenzenes - metabolism | Tumor Suppressor Proteins - metabolism | Matrix Attachment Region Binding Proteins - metabolism | Dendritic Spines - physiology | Mice, Transgenic | Synapses - ultrastructure | Electroporation - methods | Mutation - genetics | Corpus Callosum - cytology | Patch-Clamp Techniques | Immunoglobulin G - genetics | Cerebral Cortex - growth & development | Luminescent Proteins - genetics | Corpus Callosum - growth & development | Mice | Immunoglobulin G - metabolism | RNA, Small Interfering - metabolism | Receptors, Cell Surface - genetics | Membrane Potentials - genetics | Age Factors | Gene Expression Regulation, Developmental - genetics | Phosphopyruvate Hydratase - metabolism | Cerebral Cortex - cytology | DNA-Binding Proteins - metabolism | Synapses - metabolism | Hedgehog Proteins - genetics | Pyramidal Tracts - physiology | Stilbamidines - metabolism | Receptors, Cell Surface - metabolism | Nuclear Proteins - metabolism | Transcription Factors - metabolism | Animals | Nerve Net - metabolism | In Vitro Techniques | Luminescent Proteins - metabolism | Stem cell research | Nervous system diseases | Neurons | Stem cells | Collateralized debt obligations | Physiological aspects | Cells | Proteins | Transcription factors | Software | Sucrose | Synaptogenesis | Circuits | Hedgehog protein | Neural networks | Pyramidal cells | Cortex | Synapses | Index Medicus
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 10/2012, Volume 491, Issue 7422, pp. 109 - 113
Journal Article
Scientific reports, ISSN 2045-2322, 09/2015, Volume 5, Issue 1, pp. 14474 - 14474
...1Scientific RepoRts | 5:14474 | DOi: 10.1038/srep14474 www.nature.com/scientificreports Baclofen mediates neuroprotection on hippocampal CA1 pyramidal cells... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Pyramidal Cells - metabolism | Phosphorylation | Apoptosis - drug effects | Brain Ischemia - genetics | Caspase 3 - metabolism | Brain Ischemia - metabolism | Glycogen Synthase Kinase 3 beta | Male | Autophagy - drug effects | CA1 Region, Hippocampal - drug effects | Neuroprotection - drug effects | Cell Membrane - metabolism | Pyramidal Cells - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Connexin 43 - genetics | Disease Models, Animal | Baclofen - pharmacology | Gene Expression | Connexin 43 - metabolism | Connexins - genetics | Rats | Mitochondria - metabolism | Glycogen Synthase Kinase 3 - metabolism | Connexins - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Brain Ischemia - drug therapy | Receptors, GABA-A - genetics | Biomarkers | Receptors, GABA-A - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Neuroprotection | Bax protein | Bcl-2 protein | γ-Aminobutyric acid A receptors | AKT protein | Connexin 43 | Caspase-3 | Autophagy | Atrophy | Mitochondria | Ischemia | Rodents | Down-regulation | Baclofen | γ-Aminobutyric acid B receptors | Neurodegenerative diseases | Extracellular signal-regulated kinase | Caspase | Connexin 36 | Pyramidal cells | Bcl protein | Brain injury | Hippocampus | Phagocytosis | γ-Aminobutyric acid receptors | Apoptosis | Index Medicus
Journal Article
Cell (Cambridge), ISSN 0092-8674, 08/2002, Volume 110, Issue 4, pp. 429 - 441
The adult brain is extremely vulnerable to various insults. The recent discovery of neural progenitors in adult mammals, however, raises the possibility of... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neural Pathways - drug effects | Recovery of Function - drug effects | Nerve Regeneration - physiology | Neuronal Plasticity - drug effects | Male | Dendrites - ultrastructure | Nerve Degeneration - metabolism | Neuronal Plasticity - physiology | Recovery of Function - physiology | Hippocampus - ultrastructure | Pyramidal Cells - drug effects | Organ Culture Techniques | Dendrites - drug effects | Synapses - drug effects | Neural Pathways - growth & development | Rats | Synapses - ultrastructure | Brain Ischemia - physiopathology | Cell Division - drug effects | Stem Cells - ultrastructure | Cell Division - physiology | Cell Movement - drug effects | Brain Ischemia - drug therapy | Nerve Regeneration - drug effects | Growth Substances - pharmacology | Pyramidal Cells - metabolism | Rats, Wistar | Microtubule-Associated Proteins - metabolism | Nerve Degeneration - physiopathology | Brain Ischemia - metabolism | Hippocampus - drug effects | Stem Cells - metabolism | Excitatory Postsynaptic Potentials - drug effects | Cell Movement - physiology | Excitatory Postsynaptic Potentials - physiology | Synapses - metabolism | Up-Regulation - physiology | Hippocampus - growth & development | Pyramidal Cells - ultrastructure | Dendrites - metabolism | Transcription Factors - genetics | Microscopy, Electron | Transcription Factors - metabolism | Up-Regulation - drug effects | Animals | Growth Substances - therapeutic use | Stem Cells - drug effects | Neural Pathways - ultrastructure | Nerve Degeneration - drug therapy | Neural circuitry | Neurons | Growth | Physiological aspects | Hippocampus (Brain) | Developmental neurology | Research | Index Medicus
Journal Article