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Glia, ISSN 0894-1491, 04/2010, Volume 58, Issue 6, pp. 730 - 740
Brain ischemia leading to stroke is a major cause of disability in developed countries. Therapeutic strategies have most commonly focused on protecting neurons... 
white matter | oligodendrocytes | ATP | P2X7 receptor | pannexins | ischemia | Ischemia | Pannexins | White matter | Oligodendrocytes | WHITE-MATTER | NMDA RECEPTORS | MICROGLIAL CELLS | NEUROSCIENCES | GLUTAMATE RECEPTORS | IMPROVES RECOVERY | P2X RECEPTOR | IN-VIVO | SPINAL-CORD-INJURY | ATP RELEASE | BRAIN | L-Lactate Dehydrogenase - metabolism | Optic Nerve - cytology | Action Potentials - genetics | Reactive Oxygen Species - metabolism | Calcium - metabolism | Microscopy, Electron, Transmission - methods | Oligodendroglia - ultrastructure | Patch-Clamp Techniques - methods | Adenosine Triphosphate - pharmacology | Oligodendroglia - drug effects | Oligodendroglia - physiology | Adenosine Triphosphate - metabolism | Cell Death - drug effects | Pyridoxal Phosphate - analogs & derivatives | Action Potentials - drug effects | Animals, Newborn | Pyridoxal Phosphate - pharmacology | Axons - metabolism | Connexins - genetics | Purinergic P2 Receptor Antagonists | Rats | Excitatory Amino Acid Antagonists - pharmacology | Nerve Tissue Proteins - genetics | Adenosine Triphosphate - analogs & derivatives | Optic Neuropathy, Ischemic - metabolism | Connexins - metabolism | Action Potentials - physiology | Nerve Tissue Proteins - metabolism | Receptors, Purinergic P2X7 | Glucose - deficiency | Animals | Optic Neuropathy, Ischemic - pathology | Hypoxia - pathology | Axons - pathology | Receptors, Purinergic P2 - metabolism
Journal Article
Molecular Genetics and Metabolism, ISSN 1096-7192, 11/2011, Volume 104, Issue 3, pp. 362 - 368
We present an 8-year-old boy with folate receptor alpha (FRα) defect and congenital deafness with labyrinthine aplasia, microtia and microdontia (LAMM... 
Leucodystrophy | Cerebral folate deficiency | Labyrinthine aplasia | Epilepsy | FOLR1 gene | FGF3 gene | BIOCHEMICAL FEATURES | MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-FACTOR 3 | FGF3 MUTATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DIHYDROFOLATE-REDUCTASE DEFICIENCY | BETAINE | NEUROLOGIC DISEASE | FOLIC-ACID | INNER-EAR AGENESIS | GENETICS & HEREDITY | MEGALOBLASTIC-ANEMIA | PLASMA HOMOCYSTEINE | Humans | Dihydroxyphenylalanine - analogs & derivatives | Molecular Sequence Data | Male | DNA Primers - genetics | Electroencephalography | Epilepsy - etiology | Tyrosine - analogs & derivatives | Base Sequence | Child | Levodopa - cerebrospinal fluid | Congenital Microtia | Fibroblast Growth Factor 3 - genetics | Pyridoxal Phosphate - therapeutic use | Ear - abnormalities | Sequence Analysis, DNA | Congenital Abnormalities - pathology | Syndrome | Radiography | Dihydroxyphenylalanine - metabolism | Folic Acid Deficiency - pathology | Folate Receptor 1 - genetics | Skull - diagnostic imaging | Ear, Inner - abnormalities | Tooth Abnormalities - pathology | Epilepsy - drug therapy | Ear - pathology | Epilepsy - pathology | Levodopa - metabolism | Codon, Nonsense - genetics | Phosphates | Deafness | Genetic disorders | Child development | Genes | Physiological aspects | Fibroblast growth factors | Seizures (Medicine) | Folic acid
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2011, Volume 108, Issue 49, pp. 19725 - 19730
Acute myocardial infarction (AMI) initiates an intense inflammatory response that promotes cardiac dysfunction, cell death, and ventricular remodeling. The... 
Heart | Myocardial infarction | Granulation tissue | Receptors | Ischemia | Cell death | Myocardium | Small interfering RNA | Renovations | Endothelial cells | NLRP3 | Interleukin-1 | Pyroptosis | NALP3 | EXTRACELLULAR ATP | CELLS | interleukin-1 | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | RECEPTOR | pyroptosis | RESPONSES | DANGER SIGNALS | IN-VIVO | LIPOPOLYSACCHARIDE | Myocardial Infarction - genetics | Inflammasomes - metabolism | Apoptosis - drug effects | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | Myocardial Infarction - diagnostic imaging | Receptors, Purinergic P2X7 - genetics | Ventricular Remodeling | RNA Interference | Cytoskeletal Proteins - metabolism | Pyridoxal Phosphate - analogs & derivatives | Disease Models, Animal | Cell Line | Gene Expression | Echocardiography | Pyridoxal Phosphate - pharmacology | Myocardial Infarction - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Inflammasomes - genetics | Carrier Proteins - genetics | Myocytes, Cardiac - pathology | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Fluorescent Antibody Technique | Lipopolysaccharides - pharmacology | Myocytes, Cardiac - metabolism | Receptors, Purinergic P2X7 - metabolism | Mice | Oligomers | Care and treatment | Heart cells | Physiological aspects | Development and progression | Health aspects | Heart attack | Biological Sciences
Journal Article
ChemBioChem, ISSN 1439-4227, 08/2017, Volume 18, Issue 15, pp. 1482 - 1486
Journal Article
Journal of Neurophysiology, ISSN 0022-3077, 09/2008, Volume 100, Issue 3, pp. 1184 - 1201
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2001, Volume 21, Issue 17, pp. 6522 - 6531
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2004, Volume 381, Issue 3, pp. 769 - 778
Full-length rat HDC (L-histidine decarboxylase) translated in reticulocyte cell lysate reactions is inactive, whereas C-terminally truncated isoforms are... 
α-fluoromethyl histidine | Dimer | Semi-denaturing SDS/PAGE | Histidine methyl ester | L-histidine decarboxylase | MASTOCYTOMA P-815 CELLS | ACID | BIOCHEMISTRY & MOLECULAR BIOLOGY | dimer | alpha-fluorotrethyl histidine | ALPHA-FLUOROMETHYLHISTIDINE | histidine methyl ester | COENZYME-BINDING-SITES | ORNITHINE DECARBOXYLASE | semi-denaturing SDS/PAGE | INACTIVATION | PURIFICATION | ECL CELLS | MICE | HISTAMINE | Oligonucleotides - genetics | Histidine - analogs & derivatives | COS Cells - chemistry | Cercopithecus aethiops | Substrate Specificity - physiology | Histidine - metabolism | Pyridoxal Phosphate - metabolism | COS Cells - metabolism | Histidine Decarboxylase - deficiency | Isoenzymes - metabolism | Methylhistidines - metabolism | Catalysis | Dimerization | Recombinant Proteins - metabolism | Cell Line | Histidine Decarboxylase - antagonists & inhibitors | Peptides - physiology | Histidine Decarboxylase - metabolism | Rats | Alternative Splicing - physiology | Isoenzymes - deficiency | Electrophoretic Mobility Shift Assay - methods | Animals | Histidine Decarboxylase - biosynthesis | Histidine - chemistry | Isoenzymes - biosynthesis | Enzyme Activation - physiology | Isoenzymes - antagonists & inhibitors | Protein Structure, Tertiary - physiology | FL, FLAG | GFP, green fluorescent protein | α-FMH, α-fluoromethylhistidine | PAGE | HA, haemagglutinin | HME, histidine methyl ester | Ni-NTA, Ni2+-nitrilotriacetate | semi-denaturing SDS | PLP, pyridoxal phosphate | HDC, histidine decarboxylase
Journal Article
Acta Crystallographica Section D, ISSN 2059-7983, 08/2018, Volume 74, Issue 8, pp. 748 - 759
Journal Article
Journal Article