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The Journal of Physiology, ISSN 0022-3751, 12/2008, Volume 586, Issue 24, pp. 6049 - 6061
We tested the hypothesis that increasing blood amino acid (AA) availability would counter the physical inactivity-induced reduction in muscle protein... 
DISUSE ATROPHY | PROLONGED INACTIVITY | PHYSIOLOGY | LENGTHENING CONTRACTIONS | GENE-EXPRESSION | HUMAN QUADRICEPS MUSCLE | ATROPHY INVOLVE | BED REST | NEUROSCIENCES | HUMAN SKELETAL-MUSCLE | NUTRITION COUNTERMEASURES | TRANSLATIONAL CONTROL | Protein Kinases - metabolism | Humans | Elongation Factor 2 Kinase - metabolism | Glycogen Synthase Kinase 3 beta | Male | Amino Acids - administration & dosage | Amino Acids, Essential - metabolism | Insulin - blood | Amino Acids - pharmacology | Muscle Proteins - biosynthesis | Dose-Response Relationship, Drug | Young Adult | Amino Acids - metabolism | Ubiquitination - drug effects | Quadriceps Muscle - drug effects | Adult | Female | Immobilization - methods | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Myofibrils - drug effects | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Glycogen Synthase Kinase 3 - metabolism | Myofibrils - physiology | Quadriceps Muscle - physiology | Amino Acids, Essential - blood | Muscle Strength - drug effects | Myofibrils - metabolism | TOR Serine-Threonine Kinases | Infusions, Intravenous | Quadriceps Muscle - metabolism | Muscle Strength - physiology | Physiological aspects | Protein biosynthesis | Amino acids | Index Medicus | Skeletal Muscle and Exercise
Journal Article
Journal of Nutrition, ISSN 0022-3166, 05/2011, Volume 141, Issue 5, pp. 856 - 862
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2015, Volume 309, Issue 3, pp. E224 - E232
Journal Article
The Journal of Physiology, ISSN 0022-3751, 02/2011, Volume 589, Issue 3, pp. 711 - 725
Glucose is stored as glycogen in skeletal muscle. The importance of glycogen as a fuel during exercise has been recognized since the 1960s; however, little is... 
FIBERS | LOCALIZATION | PHYSIOLOGY | PROLONGED EXERCISE | RAT | INTRACELLULAR CALCIUM | FATIGUE | SUBCELLULAR GLYCOGEN | RELEASE | CANE TOAD | NEUROSCIENCES | PHOSPHORYLASE | Calcium - metabolism | Lactic Acid - blood | Sarcoplasmic Reticulum - drug effects | Humans | Male | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Dietary Carbohydrates - metabolism | Myosin Heavy Chains - metabolism | Young Adult | Glycogen - metabolism | Myofibrils - ultrastructure | Muscle Fibers, Skeletal - ultrastructure | Muscle, Skeletal - drug effects | Quadriceps Muscle - drug effects | Adult | Sarcoplasmic Reticulum - metabolism | Muscle Fatigue - physiology | Cresols - pharmacology | Skiing - physiology | Microscopy, Electron, Transmission | Myofibrils - drug effects | Dietary Carbohydrates - pharmacology | Muscle, Skeletal - ultrastructure | Muscle, Skeletal - physiology | Athletes | Quadriceps Muscle - physiology | Myofibrils - metabolism | Biopsy, Needle | Kinetics | Index Medicus | Carbohydrates | Legs | Muscles (fatigue) | Physical training | Glycogen | Muscles (activity) | Muscles (contractions) | Skiing | Fatigue | Glucose | Fuels | Muscle contraction | Recovery | Skeletal muscle | Energy utilization | Transmission electron microscopy | Sarcoplasmic reticulum | Calcium (reticular) | Skeletal Muscle and Exercise
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 11/2017, Volume 123, Issue 5, pp. 1092 - 1100
Loss of muscle mass during periods of disuse likely has negative health consequences for older adults. We have previously shown that... 
Aging | Mitochondria | Exercise | Bed rest | HMB | DISUSE ATROPHY | SPORT SCIENCES | OXIDATIVE STRESS | SIGNALING PATHWAYS | PHYSIOLOGY | PROTEIN-METABOLISM | mitochondria | exercise | SARCOPENIA | BODY-COMPOSITION | PROTEOLYSIS | SUPPLEMENTATION | bed rest | ARGININE | aging | Prospective Studies | Age Factors | Humans | Middle Aged | Mitochondria, Muscle - metabolism | Male | Autophagy - drug effects | Resistance Training | Proteolysis - drug effects | Valerates - therapeutic use | Time Factors | Mitochondrial Proteins - metabolism | Sarcopenia - etiology | Quadriceps Muscle - drug effects | Female | Quadriceps Muscle - pathology | Sarcopenia - metabolism | Sarcopenia - drug therapy | Double-Blind Method | Mitochondria, Muscle - pathology | Treatment Outcome | Bed Rest - adverse effects | Mitochondrial Dynamics - drug effects | Signal Transduction - drug effects | Sarcopenia - pathology | Lipid Metabolism - drug effects | Aged | Mitochondria, Muscle - drug effects | Quadriceps Muscle - metabolism | Energy Metabolism - drug effects | Elderly people | Phosphorylation | Physical training | Immobilization | Lipids | Atrophy | Proteins | Infusion | Fatty acid composition | Older people | Resistance training | Lipid metabolism | Supplementation | Muscles | Histology | Metabolism | Fatty acids | BNIP3 protein | Skeletal muscle | Musculoskeletal system | Molecular modelling | Oxidative phosphorylation | Adults | Rehabilitation | Phagocytosis | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2016, Volume 310, Issue 9, pp. H1164 - H1175
Skeletal muscle metabolic changes are common in patients with chronic heart failure (HF). Previously, we demonstrated a functional skeletal muscle adiponectin... 
Heart failure | Inflammation | Adiponectin resistance | Primary muscle cell cultures | SiAdipoR1 | heart failure | siAdipoR1 | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | MUSCLE-CELL PROLIFERATION | RECEPTOR 1 | SKELETAL-MUSCLE | IN-VITRO | inflammation | FATTY-ACID OXIDATION | PATHWAY | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | adiponectin resistance | ENERGY-METABOLISM | primary muscle cell cultures | Inflammation - pathology | Humans | Middle Aged | Mitochondria, Muscle - metabolism | Receptors, Adiponectin - metabolism | Male | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Case-Control Studies | Organelle Biogenesis | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Quadriceps Muscle - drug effects | Adiponectin - metabolism | Female | Quadriceps Muscle - pathology | Mitochondria, Muscle - pathology | Cells, Cultured | Gene Expression Regulation | Heart Failure - genetics | Heart Failure - metabolism | Heart Failure - pathology | Tumor Necrosis Factor-alpha - pharmacology | Phenotype | Receptors, Adiponectin - deficiency | Muscle Development - drug effects | Signal Transduction - drug effects | Muscle Fibers, Skeletal - pathology | Inflammation - genetics | Cell Proliferation - drug effects | Mitochondria, Muscle - drug effects | Primary Cell Culture | Quadriceps Muscle - metabolism | Receptors, Adiponectin - genetics | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 803 - 810
Fibroblast growth factor 23 (FGF‐23) plays causative roles in the development of several hypophosphatemic rickets/osteomalacia such as X‐linked... 
X‐LINKED HYPOPHOSPHATEMIA | FIBROBLAST GROWTH FACTOR 23 | PHOSPHATONIN | NEUTRALIZING ANTIBODY | neutralizing antibody | Fibroblast growth factor 23 | phosphatonin | X-linked hypophosphatemia | OSTEOMALACIA | X-LINKED HYPOPHOSPHATEMIA | FIBROBLAST-GROWTH-FACTOR-23 | ADJUVANT | PHOSPHATE | GENE | METABOLISM | HYPOPHOSPHATEMIC RICKETS | ENDOCRINOLOGY & METABOLISM | PHYSIOLOGICAL-ROLE | MUTATIONS | FGF23 | Motor Activity - physiology | Familial Hypophosphatemic Rickets - drug therapy | Body Weight - drug effects | Motor Activity - drug effects | Antibodies, Monoclonal - therapeutic use | Male | Skull - chemistry | Femur - diagnostic imaging | Fibroblast Growth Factors - immunology | Tibia - chemistry | Familial Hypophosphatemic Rickets - blood | Mice, Mutant Strains | Phosphates - blood | Quadriceps Muscle - drug effects | Female | Familial Hypophosphatemic Rickets - complications | Genetic Diseases, X-Linked | Skull - drug effects | Antibodies, Monoclonal - immunology | Disease Models, Animal | Muscle Weakness - etiology | Femur - anatomy & histology | Calcification, Physiologic - drug effects | Familial Hypophosphatemic Rickets - physiopathology | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Blood - drug effects | Femur - drug effects | Tibia - drug effects | Vitamin D - blood | Radiography | Animals | Quadriceps Muscle - anatomy & histology | Fibroblast Growth Factors - blood | Muscle Weakness - drug therapy | Vitamin D - analogs & derivatives | Mice | Fibroblast Growth Factors - antagonists & inhibitors | Muscle Weakness - physiopathology | Hand Strength - physiology | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 9, pp. H1479 - H1489
We investigated the influence of aging on the group III/IV muscle afferents in the exercise pressor reflex-mediated cardiovascular response to rhythmic... 
Dynamic exercise | Exercise pressor reflex | Afferent feedback | CARDIAC-OUTPUT | dynamic exercise | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CAROTID BAROREFLEX CONTROL | exercise pressor reflex | afferent feedback | AFFERENTS CONTRIBUTE | METABOLIC-RESPONSE | SKELETAL-MUSCLE | ADVANCING AGE | ARTERIAL-BLOOD PRESSURE | FIBER-TYPE | PERIPHERAL VASCULAR DISEASE | Cardiac Output - physiology | Reflex - physiology | Humans | Analgesics, Opioid - pharmacology | Cardiovascular System - innervation | Reflex - drug effects | Male | Fentanyl - pharmacology | Young Adult | Regional Blood Flow - drug effects | Arterial Pressure - drug effects | Quadriceps Muscle - drug effects | Adult | Cardiovascular Physiological Phenomena - drug effects | Vasodilation - physiology | Autonomic Nervous System - drug effects | Cardiac Output - drug effects | Autonomic Nervous System - physiology | Cardiovascular System - drug effects | Arterial Pressure - physiology | Muscle, Skeletal - physiology | Femoral Artery - drug effects | Aging - physiology | Leg - blood supply | Muscle Contraction - physiology | Quadriceps Muscle - physiology | Aged | Femoral Artery - physiology | Vasodilation - drug effects | Regional Blood Flow - physiology | Complications and side effects | Care and treatment | Exercise | Aging | Influence | Research | Cardiovascular diseases | Health aspects | Knee | Cardiovascular system | Muscular system | Index Medicus | Call for Papers
Journal Article
American Journal of Clinical Nutrition, ISSN 0002-9165, 07/2013, Volume 98, Issue 1, pp. 233 - 245
Background: The major thiol-disulfide couple of reduced glutathione (GSH) and oxidized glutathione is a key regulator of major transcriptional pathways... 
VITAMIN-C | NADPH OXIDASE | OXIDATIVE STRESS | NUTRITION & DIETETICS | PROLONGED EXERCISE | BLOOD REDOX STATUS | GENE-EXPRESSION | INJURY | ACETYLCYSTEINE | NF-KAPPA-B | CYSTEINE PRETREATMENT | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Sulfhydryl Compounds - administration & dosage | Glutathione - metabolism | Humans | Adaptation, Physiological - drug effects | Male | NF-kappa B - metabolism | Young Adult | C-Reactive Protein - metabolism | Inflammation - drug therapy | Quadriceps Muscle - drug effects | Adult | p38 Mitogen-Activated Protein Kinases - metabolism | Acetylcysteine - administration & dosage | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - metabolism | Double-Blind Method | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Biomarkers - blood | Cross-Over Studies | Diet | Signal Transduction - drug effects | Muscle Contraction - drug effects | Ribosomal Protein S6 - metabolism | Quadriceps Muscle - physiology | Antioxidants - administration & dosage | Creatine Kinase - metabolism | Exercise - physiology | Quadriceps Muscle - metabolism | Dietary Supplements | Antioxidants | Muscles | Inflammation | Genetic aspects | Cellular signal transduction | Research | Health aspects | Risk factors | Glutathione | Musculoskeletal system | TNF inhibitors | Exercise | Kinases | Index Medicus | Abridged Index Medicus
Journal Article