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Journal Article
Inflammation, ISSN 0360-3997, 12/2014, Volume 37, Issue 6, pp. 2047 - 2055
Journal Article
Metabolic Brain Disease, ISSN 0885-7490, 12/2018, Volume 33, Issue 6, pp. 1869 - 1875
To investigate the potential neuroprotection of oxymatrine in hypoxic-ischemic injury in rat’s brain and the associated underlying mechanisms, modified... 
Biochemistry, general | Neurology | Neurosciences | Biomedicine | Hypoxic-ischemic injury | Nrf2/HO-1 pathway | Metabolic Diseases | Oncology | Akt/GSK3β pathway | Oxymatrine | Apoptosis | Akt/GSK3 beta pathway | NRF2 | REPERFUSION INJURY | ENDOCRINOLOGY & METABOLISM | NEUROSCIENCES | DAMAGE | PI3K/AKT | Neuroprotective Agents - therapeutic use | Quinolizines - therapeutic use | Heme Oxygenase-1 - metabolism | Apoptosis - drug effects | Oxidative Stress - physiology | Reperfusion Injury - drug therapy | Hypoxia-Ischemia, Brain - pathology | Neuroprotective Agents - pharmacology | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Reperfusion Injury - metabolism | Hypoxia-Ischemia, Brain - metabolism | Animals, Newborn | Alkaloids - pharmacology | Reperfusion Injury - pathology | Hypoxia-Ischemia, Brain - drug therapy | Rats | Quinolizines - pharmacology | Glycogen Synthase Kinase 3 beta - metabolism | Rats, Sprague-Dawley | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Alkaloids - therapeutic use | Signal Transduction - physiology | Apoptosis - physiology | Oxidative Stress - drug effects | Neuroprotection | Brain | Flow cytometry | Oxidative stress | Glycogen | Glycogen synthase kinase 3 | AKT protein | 1-Phosphatidylinositol 3-kinase | Neurological diseases | Signal transduction | Signaling | Ischemia | GA-binding protein | Rodents | Hypoxia | Brain injury | Injuries
Journal Article
Current Pharmaceutical Design, ISSN 1381-6128, 2013, Volume 19, Issue 19, pp. 3476 - 3496
Cystic fibrosis (CF) is a common inherited fatal disease affecting 70,000 people worldwide, with a median predicted age of survival of approximately 38 years.... 
Hsp70 | F508del-CFTR | Heat shock proteins | Chaperones | Cell stress response | Hsc70 | Cold shock/induced proteins | Osmolytes | osmolytes | SMALL-MOLECULE | PHARMACOLOGICAL CHAPERONES | cold shock/induced proteins | TRANSMEMBRANE CONDUCTANCE REGULATOR | HEAT-SHOCK PROTEINS | cell stress response | ORAL L-ARGININE | HUMAN GENOME | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | CYSTIC-FIBROSIS | DIMETHYL-SULFOXIDE | chaperones | heat shock proteins | DELTA-F508 CFTR | Quinolizines - therapeutic use | Phenylbutyrates - therapeutic use | HSP70 Heat-Shock Proteins - antagonists & inhibitors | Humans | Cystic Fibrosis - pathology | Endoplasmic Reticulum - metabolism | Protein Transport - drug effects | Curcumin - administration & dosage | Endoplasmic Reticulum Stress - genetics | Molecular Targeted Therapy | Endoplasmic Reticulum - drug effects | Phenylbutyrates - administration & dosage | Phenylbutyrates - pharmacology | Cell Membrane - metabolism | Protein Interaction Domains and Motifs | Quinolizines - administration & dosage | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Cold Temperature | Alkaloids - pharmacology | Curcumin - therapeutic use | Endoplasmic Reticulum Stress - drug effects | Cystic Fibrosis - metabolism | Curcumin - pharmacology | Models, Molecular | HSP70 Heat-Shock Proteins - genetics | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Quinolizines - pharmacology | Cystic Fibrosis Transmembrane Conductance Regulator - chemistry | HSP70 Heat-Shock Proteins - metabolism | Animals | Cystic Fibrosis - genetics | Alkaloids - administration & dosage | Alkaloids - therapeutic use | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Cystic Fibrosis - drug therapy
Journal Article
Journal Article
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 06/2017, Volume 804, pp. 21 - 30
Journal Article
Anti-Cancer Drugs, ISSN 0959-4973, 10/2014, Volume 25, Issue 9, pp. 1035 - 1043
Journal Article
Journal Article