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Free Radical Biology and Medicine, ISSN 0891-5849, 03/2016, Volume 92, pp. 152 - 162
The effects of physiological oxygen tension on Nuclear Factor-E2-Related Factor 2 (Nrf2)-regulated redox signaling remain poorly understood. We report the... 
NQO1 | Bach1 | Solute Carrier Family 7-anionic amino acid transporter light chain xCT | Coronary artery | HO-1 | Redox signaling | Endothelial cells | Normoxia | GCL | Physiological oxygen tension | Mitochondria | Nuclear Factor-E2-Related Factor 2, Nrf2 | Glutathione reductase GR | Thioredoxin reductase-1 | Sequestosome-1 | Glutathione | Physiologicaloxygentension | NuclearFactor-E2-RelatedFactor2,Nrf2 | Redoxsignaling | transporterlightchainxCT | Solute CarrierFamily7-anionicaminoacid | Thioredoxinreductase-1 | REPRESSOR BACH1 | ENDOCRINOLOGY & METABOLISM | SMOOTH-MUSCLE-CELLS | HEME OXYGENASE-1 | MOLECULAR-BASIS | KEAP1-NRF2 SYSTEM | NQ01 | OXIDATIVE STRESS | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | VASCULAR ENDOTHELIUM | SENSING MECHANISMS | TRANSCRIPTION FACTOR NRF2 | Heme Oxygenase-1 - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Antioxidants - metabolism | Humans | NAD(P)H Dehydrogenase (Quinone) - genetics | Oxygen - metabolism | Coronary Vessels - metabolism | DNA-Binding Proteins - metabolism | Kelch-Like ECH-Associated Protein 1 - genetics | Heme Oxygenase-1 - genetics | Fanconi Anemia Complementation Group Proteins - metabolism | Amino Acid Transport System y+ - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | NF-E2-Related Factor 2 - genetics | Glutamate-Cysteine Ligase - metabolism | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Fanconi Anemia Complementation Group Proteins - genetics | Kelch-Like ECH-Associated Protein 1 - metabolism | Basic-Leucine Zipper Transcription Factors - genetics | DNA-Binding Proteins - genetics | Veins - metabolism | NF-E2-Related Factor 2 - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | Genetic research | Physiological aspects | Genes | Endothelium | Surface active agents | RNA | Amino acids | DNA binding proteins
Journal Article
Drug Metabolism and Disposition, ISSN 0090-9556, 07/2012, Volume 40, Issue 7, pp. 1366 - 1379
The transcription factors aryl hydrocarbon receptor (AhR), constitutive androstane receptor (CAR), pregnane X receptor (PXR), peroxisome proliferator-activated... 
TRANSCRIPTION FACTORS | PRIMARY HUMAN HEPATOCYTES | ANTIOXIDANT RESPONSE ELEMENT | MESSENGER-RNA EXPRESSION | CONSTITUTIVE ANDROSTANE RECEPTOR | BILIARY-EXCRETION | PROLIFERATOR-ACTIVATED RECEPTOR | MOUSE-LIVER | PHARMACOLOGY & PHARMACY | MICROSOMAL-ENZYME INDUCERS | PREGNANE-X-RECEPTOR | Inactivation, Metabolic | Receptors, Steroid - metabolism | Cytochrome P-450 Enzyme System - metabolism | Male | NAD(P)H Dehydrogenase (Quinone) - genetics | Organic Anion Transporters - metabolism | Glucuronosyltransferase - genetics | Organic Anion Transporters - genetics | Glutathione Transferase - genetics | Receptors, Aryl Hydrocarbon - metabolism | Female | NF-E2-Related Factor 2 - genetics | Sulfotransferases - metabolism | Aldehyde Dehydrogenase - metabolism | Sulfotransferases - genetics | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptors, Aryl Hydrocarbon - genetics | Glutathione Transferase - metabolism | Aldehyde Dehydrogenase - genetics | PPAR alpha - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | Mice, Knockout | Gene Expression Regulation, Enzymologic | Transcription Factors - metabolism | Animals | Glucuronosyltransferase - metabolism | Receptors, Steroid - genetics | NF-E2-Related Factor 2 - metabolism | Cytochrome P-450 Enzyme System - genetics | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | PPAR alpha - metabolism | Multidrug Resistance-Associated Proteins | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2011, Volume 301, Issue 2, pp. H363 - H372
Ungvari Z, Bailey-Downs L, Sosnowska D, Gautam T, Koncz P, Losonczy G, Ballabh P, de Cabo R, Sonntag WE, Csiszar A. Vascular oxidative stress in aging: a... 
Senescence | Vascular injury | Oxidative stress resistance | Apoptosis | LIFE-SPAN | KAPPA-B ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | apoptosis | NECROSIS-FACTOR-ALPHA | NAKED MOLE-RAT | ARTERIAL ENDOTHELIAL-CELLS | LONGEST-LIVING RODENT | senescence | vascular injury | TRANSCRIPTION FACTOR NRF2 | oxidative stress resistance | MN SUPEROXIDE-DISMUTASE | NF-E2-RELATED FACTOR-2 | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | Carotid Arteries - metabolism | Age Factors | Oxidative Stress | Rats, Inbred F344 | Homeostasis | Male | NF-kappa B - metabolism | Aorta - metabolism | NAD(P)H Dehydrogenase (Quinone) - genetics | RNA, Messenger - metabolism | Rats, Inbred BN | Aging - genetics | NF-E2-Related Factor 2 - genetics | Glutamate-Cysteine Ligase - metabolism | Heme Oxygenase (Decyclizing) - metabolism | Heme Oxygenase (Decyclizing) - genetics | Interleukin-6 - metabolism | Tissue Culture Techniques | Rats | Aging - pathology | Hydrogen Peroxide - metabolism | Aorta - pathology | Gene Expression Regulation, Enzymologic | Hyperglycemia - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Animals | Analysis of Variance | NF-E2-Related Factor 2 - metabolism | Glucose - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | Glutamate-Cysteine Ligase - genetics | Aging - metabolism | Antioxidants | Oxidative stress | Transcription factors | Physiological aspects | Aging | Research | Health aspects | Vascular Biology and Microcirculation
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 12/2010, Volume 335, Issue 3, pp. 533 - 545
Doxorubicin (DOX) and daunorubicin (DAUN) are effective anticancer drugs; however, considerable interpatient variability exists in their pharmacokinetics. This... 
DIHYDRODIOL DEHYDROGENASE ISOFORMS | QUINONE REDUCTASE | HUMAN CARBONYL REDUCTASE | ENZYMES | ANTHRACYCLINE-INDUCED CARDIOTOXICITY | SUPERFAMILY | PHARMACOLOGY & PHARMACY | HUMAN LIVER | ALDEHYDE REDUCTASE | SINGLE NUCLEOTIDE POLYMORPHISMS | PROSTAGLANDIN-F SYNTHASE | Aldehyde Reductase - genetics | Humans | Polymorphism, Single Nucleotide - physiology | Mitochondrial Proteins - genetics | NAD(P)H Dehydrogenase (Quinone) - genetics | Hydroxyprostaglandin Dehydrogenases - metabolism | Alcohol Oxidoreductases - genetics | Recombinant Proteins - isolation & purification | Mitochondrial Proteins - metabolism | 3-Hydroxysteroid Dehydrogenases - metabolism | Aldehyde Reductase - metabolism | Hydroxysteroid Dehydrogenases - metabolism | Doxorubicin - metabolism | Daunorubicin - metabolism | Glyceraldehyde - metabolism | Phenanthrenes - metabolism | Recombinant Proteins - metabolism | 20-Hydroxysteroid Dehydrogenases - genetics | Biocatalysis | Oxidoreductases - metabolism | Oxidoreductases - genetics | Gene Frequency | Indans - metabolism | Models, Molecular | Alcohol Oxidoreductases - metabolism | Recombinant Proteins - genetics | Hydroxysteroid Dehydrogenases - genetics | 20-Hydroxysteroid Dehydrogenases - metabolism | Hydroxyprostaglandin Dehydrogenases - genetics | Aldo-Keto Reductases | Vitamin K 3 - metabolism | Aldo-Keto Reductase Family 1 Member C3 | NAD(P)H Dehydrogenase (Quinone) - metabolism | Kinetics | 3-Hydroxysteroid Dehydrogenases - genetics
Journal Article
Brain, ISSN 0006-8950, 03/2011, Volume 134, Issue Pt 3, pp. 678 - 692
Inflammation and oxidative stress are thought to promote tissue damage in multiple sclerosis. Thus, novel therapeutics enhancing cellular resistance to free... 
Antigens, Differentiation | Green Fluorescent Proteins | Neuroprotective Agents | Peptide Fragments | Cell Proliferation | Oxidative Stress | Embryo, Mammalian | Humans | Hydrogen Peroxide | Chromatography, High Pressure Liquid | Journal Article | Antioxidants | Multiple Sclerosis | Fumarates | Tandem Mass Spectrometry | Transfection | Time Factors | Mass Spectrometry | Statistics, Nonparametric | Motor Neurons | Female | Nogo Proteins | Disease Models, Animal | Cytokines | Signal Transduction | Astrocytes | 2',3'-Cyclic-Nucleotide Phosphodiesterases | CD3 Complex | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Myelin-Oligodendrocyte Glycoprotein | Aldehyde Reductase | Glycoproteins | NAD(P)H Dehydrogenase (Quinone) | Axons | Encephalomyelitis, Autoimmune, Experimental | Animals | Sleep | Spinal Cord | Research Support, Non-U.S. Gov't | Myelin Proteins | NF-E2-Related Factor 2 | Mice | RNA, Small Interfering | oral therapy | transcription factor | autoimmune demyelination | multiple sclerosis | neuroprotection | astrocyte | oxidative stress | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ENZYME INDUCERS | DIMETHYLFUMARATE | SPINAL-CORD | NEUROSCIENCES | HUMAN ENDOTHELIAL-CELLS | CLINICAL NEUROLOGY | RESPONSE ELEMENT | TRANSCRIPTION FACTOR NRF2 | MULTIPLE-SCLEROSIS | IN-VIVO | Antioxidants - metabolism | Green Fluorescent Proteins - genetics | Neuroprotective Agents - pharmacology | Aldehyde Reductase - metabolism | Sleep - physiology | Tandem Mass Spectrometry - methods | Fumarates - therapeutic use | Glycoproteins - adverse effects | CD3 Complex - metabolism | Motor Neurons - metabolism | Signal Transduction - drug effects | Axons - pathology | Fumarates - pharmacology | NF-E2-Related Factor 2 - metabolism | Encephalomyelitis, Autoimmune, Experimental - prevention & control | Peptide Fragments - adverse effects | Oxidative Stress - drug effects | Myelin Proteins - metabolism | Neuroprotective Agents - therapeutic use | Chromatography, High Pressure Liquid - methods | Encephalomyelitis, Autoimmune, Experimental - etiology | Astrocytes - immunology | Motor Neurons - cytology | Antigens, Differentiation - metabolism | Spinal Cord - cytology | Motor Neurons - drug effects | Multiple Sclerosis - metabolism | Astrocytes - drug effects | Cytokines - metabolism | RNA, Small Interfering - pharmacology | Hydrogen Peroxide - pharmacology | Axons - metabolism | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | Gene Expression Regulation - drug effects | Multiple Sclerosis - pathology | NAD(P)H Dehydrogenase (Quinone) - metabolism | Cell Proliferation - drug effects
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 07/2017, Volume 174, Issue 14, pp. 2161 - 2173
Azoreductases are flavoenzymes that have been characterized in a range of prokaryotes and eukaryotes. Bacterial azoreductases are associated with the... 
NITROAROMATIC COMPOUNDS | NAD(P)H-QUINONE OXIDOREDUCTASE-1 | FMN-DEPENDENT AZOREDUCTASE | DT-DIAPHORASE | CRYSTAL-STRUCTURE | ESCHERICHIA-COLI | PROSTATE-CANCER | PHARMACOLOGY & PHARMACY | OXIDOREDUCTASE 1 NQO1 | 5-AMINOSALICYLIC ACID | BETA-LAPACHONE | Neoplasms - metabolism | Bacteria - metabolism | Quinones - chemistry | Humans | Bacteria - drug effects | Anti-Bacterial Agents - metabolism | Antineoplastic Agents - chemistry | Nitrofurans - pharmacology | Neoplasms - drug therapy | Antineoplastic Agents - metabolism | Azo Compounds - chemistry | Anti-Bacterial Agents - chemistry | Nitrofurans - metabolism | Anti-Bacterial Agents - pharmacology | Antineoplastic Agents - pharmacology | Azo Compounds - metabolism | NADH, NADPH Oxidoreductases - metabolism | Azo Compounds - pharmacology | Quinones - pharmacology | Nitrofurans - chemistry | Quinones - metabolism | Oxidases | Drugs | Drug resistance in microorganisms | Chemotherapy | Gastrointestinal diseases | Quinone | Physiological aspects | Azo compounds | Single nucleotide polymorphisms | Chromosomes | Cancer | Inflammatory bowel diseases | Prokaryotes | Medical treatment | Single-nucleotide polymorphism | Metabolism | Drug development | Substrates | NAD | Quinones | Reduction | Eukaryotes | Antibiotics | Intestine | Medical prognosis | Antibiotic resistance | Azoreductase | Azo | Drug metabolism | Bacteria | Colon | Themed Section | Review
Journal Article
PLoS Genetics, ISSN 1553-7390, 01/2012, Volume 8, Issue 1, p. e1002456
Pink1 is a mitochondrial kinase involved in Parkinson's disease, and loss of Pink1 function affects mitochondrial morphology via a pathway involving Parkin and... 
LIFE-SPAN | OXIDATIVE STRESS | DROSOPHILA-PARKIN MUTANTS | ALTERNATIVE OXIDASE | QUINONE OXIDOREDUCTASE | NDI1 GENE | HUMAN-CELLS | INCREASED SENSITIVITY | GENETICS & HEREDITY | MITOCHONDRIAL-DYSFUNCTION | RESERVE POOL | Electron Transport Complex III - genetics | Electron Transport Complex III - metabolism | Cytoskeletal Proteins - genetics | Saccharomyces cerevisiae - genetics | Humans | Male | Drosophila Proteins - metabolism | Ciona intestinalis - genetics | Drosophila melanogaster - genetics | Electron Transport Complex I - metabolism | GTP-Binding Proteins - genetics | Electron Transport Complex IV - metabolism | Drosophila melanogaster - metabolism | Mitochondria - genetics | Electron Transport Complex I - genetics | Mitochondrial Proteins - metabolism | Cytoskeletal Proteins - metabolism | Membrane Proteins - metabolism | Plant Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Oxidoreductases - metabolism | Membrane Proteins - genetics | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Ubiquitin-Protein Ligases - metabolism | Mitochondria - metabolism | Electron Transport Complex IV - genetics | Parkinson Disease - genetics | Saccharomyces cerevisiae Proteins - genetics | Animals, Genetically Modified - metabolism | Animals | Animals, Genetically Modified - genetics | Saccharomyces cerevisiae Proteins - metabolism | Drosophila Proteins - genetics | Mutation | Ubiquitin-Protein Ligases - genetics | GTP-Binding Proteins - metabolism | Parkinson's disease | Physiological aspects | NADH dehydrogenase | Genetic aspects | Mitochondrial DNA | Research | Electron transport | Risk factors | Enzymes | Mitochondria | Yeast | Insects | Parkinsons disease | Genetic engineering | Grants | Experiments | Evacuations & rescues | Deoxyribonucleic acid--DNA | Defects | Deoxyribonucleic acid | DNA
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 44, pp. 18203 - 18216