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Cell cycle (Georgetown, Tex.), ISSN 1551-4005, 10/2014, Volume 10, Issue 10, pp. 1571 - 1581
Journal Article
Journal of polymer science. Part A, Polymer chemistry, ISSN 0887-624X, 03/2013, Volume 51, Issue 5, pp. 1137 - 1146
Aluminum‐based salen and salan complexes mediate the ring‐opening polymerization (ROP) of rac‐β‐butyrolactone (β‐BL), rac‐lactide, and ε‐caprolactone. Al‐salen... 
copolymerization | ring‐opening polymerization | rac‐lactide | biodegradable | rac‐β‐butyrolactone | rac-β-butyrolactone | rac-lactide | ring-opening polymerization | Polymer Science | Physical Sciences | Science & Technology
Journal Article
Journal of organometallic chemistry, ISSN 0022-328X, 11/2013, Volume 745-746, pp. 335 - 340
Three aluminium salophen and two aluminium salen complexes were synthesised, characterised and screened in the ring-opening polymerisation (ROP) of rac-lactide... 
Salen | Salophen | Poly(3-hydroxybutyrate) | Poly(lactic acid) | rac-Lactide | rac-β-Butyrolactone | Poly(3-hydroxybutyrate) rac-Lactide rac-β-Butyrolactone
Journal Article
Oncogene, ISSN 0950-9232, 04/2009, Volume 28, Issue 13, pp. 1570 - 1583
The motility of cancer cells in 3D matrices is of two types: mesenchymal motility, in which the cells are elongated and amoeboid motility, in which the cells... 
Amoeboid | Rac | Migration | Invasion | Cancer | Mesenchymal | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Humans | rac GTP-Binding Proteins - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - physiology | Cell Shape - genetics | rhoA GTP-Binding Protein - genetics | Cell Culture Techniques - methods | Cell Movement - genetics | Cell Movement - physiology | Neoplasms - genetics | rac GTP-Binding Proteins - genetics | Collagen - pharmacology | Tumor Cells, Cultured | rac GTP-Binding Proteins - physiology | Cell Adhesion - genetics | Neoplasm Invasiveness | rho GTP-Binding Proteins - genetics | Mutant Proteins - metabolism | Signal Transduction - genetics | Mutant Proteins - physiology | rho GTP-Binding Proteins - physiology | rac1 GTP-Binding Protein - physiology | Cell Adhesion - physiology | rho GTP-Binding Proteins - metabolism | Signal Transduction - physiology | Cell Shape - physiology | Neoplasms - pathology | rac1 GTP-Binding Protein - metabolism | Gels - pharmacology | rac1 GTP-Binding Protein - genetics | Physiological aspects | Cellular signal transduction | Motility | Research | Cells | Cancer cells | Signal transduction | Genetics | Substrates | Cell adhesion & migration | Index Medicus
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 11/2008, Volume 28, Issue 46, pp. 12039 - 12051
Reactive oxygen species (ROS) modulate intracellular signaling but are also responsible for neuronal damage in pathological states. Microglia, the resident CNS... 
Interleukin-1 | NADPH oxidase | Nitric oxide | Nox | Neuroinflammation | LPS | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nitrites - metabolism | rac GTP-Binding Proteins - metabolism | Male | Gliosis - enzymology | Gliosis - physiopathology | Cytochrome b Group - metabolism | Zymosan - metabolism | Encephalitis - physiopathology | Inflammation Mediators - pharmacology | rac GTP-Binding Proteins - genetics | NADPH Oxidases - genetics | Female | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | Corpus Striatum - enzymology | NADH, NADPH Oxidoreductases - genetics | Encephalitis - enzymology | Corpus Striatum - physiopathology | Microglia - drug effects | Mice, Inbred C57BL | Microglia - enzymology | Neuropeptides - metabolism | Cytochrome b Group - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Neurotoxins - pharmacology | Proteins - genetics | Animals | Proteins - metabolism | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Oxidative Stress - drug effects | rac1 GTP-Binding Protein | Index Medicus | Reactive Oxygen Species | Oxidative Stress | Nitrites | Neuropeptides | Lipopolysaccharides | Proteins | Life Sciences | Immunology | NADH, NADPH Oxidoreductases | Membrane Glycoproteins | Zymosan | rac GTP-Binding Proteins | Encephalitis | Neurotoxins | Inflammation Mediators | Cytochrome b Group | Gliosis | Corpus Striatum | NADPH Oxidase | nitric oxide | interleukin-1 | neuroinflammation | microglia
Journal Article
Cardiovascular research, ISSN 1755-3245, 01/2013, Volume 97, Issue 1, pp. 77 - 87
Doxorubicin causes damage to the heart, often leading to irreversible cardiomyopathy, which is fatal. Reactive oxygen species (ROS) or oxidative stress is... 
Cardiotoxicity | Rac1 | Doxorubicin | HDAC | p53 | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Free Radical Scavengers - pharmacology | Phosphorylation | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Caspase 3 - metabolism | NADPH Oxidases - metabolism | rac GTP-Binding Proteins - deficiency | rac GTP-Binding Proteins - metabolism | Heart Diseases - prevention & control | rac GTP-Binding Proteins - antagonists & inhibitors | Transfection | rac GTP-Binding Proteins - genetics | DNA Fragmentation | Acetylation | Neuropeptides - genetics | Heart Diseases - chemically induced | Disease Models, Animal | Neuropeptides - deficiency | Cell Line | Enzyme Inhibitors - pharmacology | Tumor Suppressor Protein p53 - metabolism | Rats | Histone Deacetylases - metabolism | Neuropeptides - metabolism | Doxorubicin - toxicity | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Myocytes, Cardiac - metabolism | Histone Deacetylase Inhibitors - pharmacology | Mice | Enzyme Activation | Histones - metabolism | Oxidative Stress - drug effects | Heart Diseases - genetics | Heart Diseases - pathology | rac1 GTP-Binding Protein - metabolism | Apoptosis | Neuropeptides - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article