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1. Full Text OX40 Ligand and CD30 Ligand Are Expressed on Adult but Not Neonatal CD4+CD3- Inducer Cells: Evidence That IL-7 Signals Regulate CD30 Ligand but Not OX40 Ligand Expression
by Kim, Mi-Yeon and Anderson, Graham and White, Andrea and Jenkinson, Eric and Arlt, Wiebke and Martensson, Inga-Lill and Erlandsson, Lena and Lane, Peter J. L
The Journal of Immunology, ISSN 0022-1767, 06/2005, Volume 174, Issue 11, pp. 6686 - 6691
In this report, we have examined the expression of the T cell survival signals, OX40 ligand (OX40L) and CD30 ligand (CD30L) on CD4(+)CD3(-)CD11c(-)B220(-)IL-7R... 
LYMPH-NODE DEVELOPMENT | CD4 | ROR-GAMMA | FOLLICULAR DENDRITIC CELLS | MICE | T-CELL | TISSUES | IMMUNOLOGY | TNF | RECEPTOR-ALPHA | INTERLEUKIN-7 | Animals, Newborn - genetics | Receptor Activator of Nuclear Factor-kappa B | Membrane Glycoproteins - metabolism | Membrane Glycoproteins - biosynthesis | Tumor Necrosis Factors | Interleukin-7 - physiology | RANK Ligand | Receptors, OX40 | CD3 Complex - biosynthesis | Interleukin-7 - genetics | Signal Transduction - immunology | Cellular Senescence - immunology | CD3 Complex - genetics | T-Lymphocytes, Helper-Inducer - immunology | Histocompatibility Antigens Class I - biosynthesis | Receptors, Tumor Necrosis Factor - deficiency | Histocompatibility Antigens Class II - physiology | Receptors, Tumor Necrosis Factor - metabolism | Cellular Senescence - genetics | T-Lymphocytes, Helper-Inducer - metabolism | Interleukin-7 - deficiency | Carrier Proteins - biosynthesis | Mice, Inbred C57BL | Cells, Cultured | Histocompatibility Antigens Class I - physiology | CD30 Ligand | Histocompatibility Antigens Class II - biosynthesis | Mice, Transgenic | Signal Transduction - genetics | Ki-1 Antigen - metabolism | Mice, Knockout | Receptors, Interleukin-7 - physiology | Animals | Ligands | Mice | Interleukin Receptor Common gamma Subunit | Animals, Newborn - immunology
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2. Full Text RANK ligand as a potential target for breast cancer prevention in BRCA1-mutation carriers
by Nolan, Emma and Vaillant, François and Branstetter, Daniel and Pal, Bhupinder and Giner, Göknur and Whitehead, Lachlan and Lok, Sheau W and Mann, Gregory B and Rohrbach, Kathy and Huang, Li-Ya and Soriano, Rosalia and Smyth, Gordon K and Dougall, William C and Visvader, Jane E and Lindeman, Geoffrey J and Kathleen Cuningham Fdn Consortium and Kathleen Cuningham Foundation Consortium for Research into Familial Breast Cancer (kConFab)
Nature Medicine, ISSN 1078-8956, 08/2016, Volume 22, Issue 8, pp. 933 - 939
Individuals who have mutations in the breast-cancer-susceptibility gene BRCA1 (hereafter referred to as BRCA1-mutation carriers) frequently undergo... 
BRCA2 MUTATION CARRIERS | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | OVARIAN-CANCER | CELL BIOLOGY | RISK-REDUCING SURGERY | STEM-CELL | MICE | DIFFERENTIAL EXPRESSION ANALYSIS | GLAND | MAMMARY EPITHELIAL-CELLS | Immunohistochemistry | Neoplasm Transplantation | RANK Ligand - metabolism | Humans | Molecular Targeted Therapy | Prophylactic Mastectomy | Stem Cells - metabolism | Breast - metabolism | Breast Neoplasms - metabolism | Denosumab - therapeutic use | Flow Cytometry | Organoids - metabolism | Female | Receptor Activator of Nuclear Factor-kappa B - metabolism | Disease Models, Animal | Carcinogenesis - genetics | Bone Density Conservation Agents - therapeutic use | Reverse Transcriptase Polymerase Chain Reaction | Bone Density Conservation Agents - pharmacology | Breast Neoplasms - prevention & control | Carcinogenesis - drug effects | Xenograft Model Antitumor Assays | BRCA1 Protein - genetics | Animals | Breast Neoplasms - genetics | RANK Ligand - antagonists & inhibitors | Breast - drug effects | DNA Repair | Organoids - drug effects | Pilocarpine - analogs & derivatives | Stem Cells - drug effects | Heterozygote | Cell Proliferation - drug effects | Mice | Mutation | Denosumab - pharmacology | Tumor Suppressor Proteins | Care and treatment | Cytokines | BRCA mutations | Development and progression | Breast cancer | Genetic aspects | Health aspects | Ligands | Genes
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3. Full Text Over‐expression of forkhead box P3 and its association with receptor activator of nuclear factor‐κ B ligand, interleukin (IL) ‐17, IL‐10 and transforming growth factor‐β during the progression of chronic periodontitis
by Dutzan, Nicolás and Gamonal, Jorge and Silva, Augusto and Sanz, Mariano and Vernal, Rolando
Journal of Clinical Periodontology, ISSN 0303-6979, 05/2009, Volume 36, Issue 5, pp. 396 - 403
Aim: T regulatory (Treg) cells have been detected in periodontitis lesions, and forkhead box P3 (Foxp3) expression has been negatively correlated to receptor... 
regulatory T cells | CTLA‐4 | RANKL | periodontitis | Treg | CD25 | Foxp3 | IL‐17 | RORC2 | Periodontitis | IL-17 | CTLA-4 | Regulatory T cells | CYTOKINES | TGF-BETA | SUBPOPULATIONS | MATRIX METALLOPROTEINASE-13 | DENDRITIC CELLS | LYMPHOCYTE | INFLAMMATION | DENTISTRY, ORAL SURGERY & MEDICINE | REGULATORY T-CELLS | PARADIGM | Forkhead Transcription Factors - immunology | GATA3 Transcription Factor - immunology | Humans | Interleukin-17 - analysis | Interleukin-17 - immunology | DNA-Binding Proteins - analysis | T-Box Domain Proteins - immunology | Th2 Cells - immunology | Th1 Cells - immunology | Lymphotoxin-alpha - immunology | T-Lymphocytes, Regulatory - immunology | RANK Ligand - immunology | Transforming Growth Factor beta1 - immunology | T-Lymphocytes, Helper-Inducer - immunology | Receptors, Retinoic Acid - immunology | Receptors, Retinoic Acid - analysis | Chronic Periodontitis - immunology | T-Box Domain Proteins - analysis | GATA3 Transcription Factor - analysis | Transforming Growth Factor beta1 - analysis | Interleukin-10 - analysis | Receptors, Thyroid Hormone - immunology | Cytokines - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 | Interleukin-1beta - analysis | DNA-Binding Proteins - immunology | Gene Expression Regulation - genetics | Interleukin-1beta - immunology | RANK Ligand - analysis | Reverse Transcriptase Polymerase Chain Reaction | Disease Progression | Alveolar Bone Loss - immunology | Interferon-gamma - analysis | Receptors, Thyroid Hormone - analysis | Lymphotoxin-alpha - analysis | Down-Regulation - immunology | Interferon-gamma - immunology | Forkhead Transcription Factors - analysis | Interleukin-10 - immunology
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4. Full Text RANK ligand
by Blair, J.M and Zheng, Y and Dunstan, C.R
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 2007, Volume 39, Issue 6, pp. 1077 - 1081
RANK ligand (RANKL), a key mediator of bone resorption in normal and pathological states, is expressed as membrane-bound or soluble forms by tissues as diverse... 
Bone resorption | Chemoattraction | Osteoprotegerin | MULTIPLE-MYELOMA | ACTIVATION | METASTASIS | OSTEOPROTEGERIN-LIGAND | BIOCHEMISTRY & MOLECULAR BIOLOGY | bone resorption | CYTOKINE | CANCER | chemoattraction | CELL BIOLOGY | DENDRITIC-CELL | TRANCE | TNF FAMILY-MEMBER | osteoprotegerin | OSTEOCLAST DIFFERENTIATION | Amino Acid Sequence | RANK Ligand - metabolism | Humans | Molecular Sequence Data | Gene Expression Profiling | Bone Resorption - prevention & control | RANK Ligand - genetics | Sequence Homology, Amino Acid | Animals | RANK Ligand - antagonists & inhibitors | Models, Biological | Bone Resorption - metabolism | Osteoprotegerin - metabolism
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5. Full Text RANK ligand mediates progestin-induced mammary epithelial proliferation and carcinogenesis
by Gonzalez-Suarez, Eva and Jacob, Allison P and Jones, Jon and Miller, Robert and Roudier-Meyer, Martine P and Erwert, Ryan and Pinkas, Jan and Branstetter, Dan and Dougall, William C
Nature, ISSN 0028-0836, 11/2010, Volume 468, Issue 7320, pp. 103 - 107
RANK ligand (RANKL), a TNF-related molecule, is essential for osteoclast formation, function and survival through interaction with its receptor RANK(1,2).... 
CELL FUNCTION | OSTEOPROTEGERIN-LIGAND | MULTIDISCIPLINARY SCIENCES | MEDROXYPROGESTERONE ACETATE | RECEPTOR | HORMONE REPLACEMENT THERAPY | HUMAN BREAST-TUMORS | OSTEOCLAST DIFFERENTIATION | EXPRESSION | GLAND DEVELOPMENT | TRANSGENIC MICE | Epithelial Cells - metabolism | RANK Ligand - metabolism | Progesterone - administration & dosage | Epithelial Cells - drug effects | Humans | Progestins - adverse effects | Cell Transformation, Neoplastic - chemically induced | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Lung Neoplasms - secondary | Mammary Neoplasms, Experimental - pathology | Medroxyprogesterone Acetate - adverse effects | Female | Precancerous Conditions - pathology | Receptor Activator of Nuclear Factor-kappa B - metabolism | Disease Models, Animal | 9,10-Dimethyl-1,2-benzanthracene - administration & dosage | Neoplasm Invasiveness | Mice, Inbred C57BL | Epithelial Cells - pathology | Mice, Transgenic | Progesterone - adverse effects | Mammary Neoplasms, Experimental - chemically induced | RANK Ligand - genetics | Animals | RANK Ligand - antagonists & inhibitors | Breast Neoplasms - pathology | Progestins - administration & dosage | Receptor Activator of Nuclear Factor-kappa B - genetics | Cell Proliferation - drug effects | Medroxyprogesterone Acetate - administration & dosage | Mice | Cell Transformation, Neoplastic - drug effects | Precancerous Conditions - prevention & control | Cell Transformation, Neoplastic - pathology | Mammary Tumor Virus, Mouse - genetics | Mammary Tumor Virus, Mouse - physiology | 9,10-Dimethyl-1,2-benzanthracene - adverse effects | Immunohistochemistry | Care and treatment | Usage | Physiological aspects | Breast cancer | Genetic aspects | Research | Progesterone | Tumor proteins | Health aspects | Risk factors | Proteins | Hormone replacement therapy | Rodents | Apoptosis | Index Medicus
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6. Full Text CXC chemokine ligand 2 induced by receptor activator of NF-κB ligand enhances osteoclastogenesis
by Ha, Jeongim and Choi, Hyo-Sun and Lee, Youngkyun and Kwon, Hyung-Joo and Song, Yeong Wook and Kim, Hong-Hee
Journal of Immunology, ISSN 0022-1767, 05/2010, Volume 184, Issue 9, pp. 4717 - 4724
CXCL2 has been known to regulate immune functions mainly by chemo-attracting neutrophils. In this study, we show that CXCL2 can be induced by receptor... 
CCR1 | COLLAGEN-INDUCED ARTHRITIS | ADHESION | CELLS | MEMBERS | MOUSE | MACROPHAGE INFLAMMATORY PROTEIN-2 | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | ACTS | Stem Cells - immunology | Humans | Chemokine CXCL2 - deficiency | Stem Cells - metabolism | Cell Movement - immunology | Osteoclasts - immunology | Stem Cells - enzymology | RANK Ligand - physiology | Bone Resorption - metabolism | Cell Line | Osteoclasts - pathology | Cells, Cultured | Bone Resorption - immunology | Chemokine CXCL2 - physiology | Osteoclasts - metabolism | Mice, Inbred ICR | Bone Resorption - enzymology | Cell Adhesion - immunology | Cell Differentiation - immunology | Animals | Bone Resorption - pathology | Stem Cells - pathology | Chemokine CXCL2 - biosynthesis | Mice | Osteoclasts - enzymology
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7. Full Text Osteoprotegerin and Denosumab Stimulate Human Beta Cell Proliferation through Inhibition of the Receptor Activator of NF-κB Ligand Pathway
by Kondegowda, Nagesha Guthalu and Fenutria, Rafael and Pollack, Ilana R and Orthofer, Michael and Garcia-Ocaña, Adolfo and Penninger, Josef M and Vasavada, Rupangi C
Cell Metabolism, ISSN 1550-4131, 07/2015, Volume 22, Issue 1, pp. 77 - 85
Diabetes results from a reduction of pancreatic β-cells. Stimulating replication could normalize β-cell mass. However, adult human β-cells are recalcitrant to... 
DENSITY | HUMAN ISLETS | PROTEIN | MASS | ENDOCRINOLOGY & METABOLISM | PLACENTAL-LACTOGEN | MICE | DEATH | EXPRESSION | BONE-RESORPTION | PREGNANCY | CELL BIOLOGY | Cell Line | Diabetes Mellitus, Experimental - drug therapy | Humans | Mice, Inbred C57BL | Rats | Male | NF-kappa B - metabolism | Mice, SCID | Bone Density Conservation Agents - pharmacology | Animals | Insulin-Secreting Cells - drug effects | RANK Ligand - antagonists & inhibitors | Signal Transduction - drug effects | Cell Proliferation - drug effects | Insulin-Secreting Cells - cytology | Mice | Osteoprotegerin - metabolism | Denosumab - pharmacology | Diabetes Mellitus, Experimental - metabolism | human islet transplant | lactogens | Denosumab | diabetes | human pancreatic β-cell proliferation | Receptor activator of NF-κB ligand | Osteoprotegerin
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8. Full Text RANK Ligand and Osteoprotegerin: Paracrine Regulators of Bone Metabolism and Vascular Function
by Schoppet, Michael and Preissner, Klaus T and Hofbauer, Lorenz C
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 04/2002, Volume 22, Issue 4, pp. 549 - 553
In 1997, investigators isolated a secreted glycoprotein that blocked osteoclast differentiation from precursor cells, prevented osteoporosis (decreased bone... 
Osteoporosis | RANK ligand | Dendritic cells | Arterial calcification | Osteoprotegerin | NECROSIS-FACTOR RECEPTOR | TNF FAMILY MEMBER | DIFFERENTIATION FACTOR | FIBROBLAST-GROWTH-FACTOR | OSTEOBLAST-LIKE CELLS | osteoporosis | dendritic cells | DENDRITIC-CELL | MESSENGER-RNA | arterial calcification | OSTEOCLASTOGENESIS-INHIBITORY FACTOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | osteoprotegerin | NF-KAPPA-B | MARROW STROMAL CELLS | Carrier Proteins - physiology | Glycoproteins - genetics | Bone Resorption | Receptor Activator of Nuclear Factor-kappa B | Vascular Diseases - etiology | Humans | RANK Ligand | Receptors, Cytoplasmic and Nuclear - physiology | Bone Remodeling - physiology | Osteopetrosis - etiology | Receptors, Cytoplasmic and Nuclear - genetics | Glycoproteins - physiology | Osteoporosis - etiology | Arteries | Membrane Glycoproteins - physiology | Animals | Calcinosis - etiology | Receptors, Tumor Necrosis Factor | Bone and Bones - metabolism | Mice
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9. Full Text The RANKL‐OPG system in clinical periodontology
by Belibasakis, Georgios N and Bostanci, Nagihan
Journal of Clinical Periodontology, ISSN 0303-6979, 03/2012, Volume 39, Issue 3, pp. 239 - 248
Background and Objectives The receptor activator of NF‐κB ligand‐osteoprotegerin (RANKL‐OPG) bi‐molecular system is the “bottle‐neck” regulator of... 
gingival crevicular fluid | receptor activator of NF‐κB ligand | diagnostics | receptor activator of NF‐κB ligand/osteoprotegerin ratio | osteoprotegerin | periodontal disease | receptor activator of NF-ÎB ligand/osteoprotegerin ratio | receptor activator of NF-ÎB ligand | receptor activator of NF-?B ligand | DIFFERENTIAL EXPRESSION | osteoprotegerin ratio | NECROSIS-FACTOR-ALPHA | TRANSCRIPTION FACTOR NFATC1 | MESSENGER-RNA | DENTISTRY, ORAL SURGERY & MEDICINE | OSTEOPROTEGERIN LIGAND | RECEPTOR ACTIVATOR | OSTEOCLAST DIFFERENTIATION | KAPPA-B LIGAND | ALVEOLAR BONE DESTRUCTION | Alveolar Bone Loss - metabolism | Biomarkers - metabolism | Lymphocytes - metabolism | Alveolar Bone Loss - therapy | RANK Ligand - metabolism | Humans | Periodontitis - therapy | Diabetes Mellitus - metabolism | Periodontitis - blood | Gingival Crevicular Fluid - chemistry | Biomarkers - blood | Saliva - chemistry | Periodontal Ligament - metabolism | Periodontitis - metabolism | Dental Plaque - microbiology | Smoking - metabolism | RANK Ligand - blood | Animals | Gingiva - chemistry | Osteoporosis - metabolism | Osteoprotegerin - blood | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Alveolar Bone Loss - blood | Oral hygiene | Immune system | Diabetes mellitus | biomarkers | Blood | Osteoprotegerin | Bone resorption | Osteoclastogenesis | Periodontal diseases | NF- Kappa B protein | Molecular modelling | Periodontitis | Gingiva | TRANCE protein | Saliva | Smoking
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10. Full Text Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss
by Onal, Melda and Xiong, Jinhu and Chen, Xinrong and Thostenson, Jeff D and Almeida, Maria and Manolagas, Stavros C and O'Brien, Charles A
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 35, pp. 29851 - 29860
Production of the cytokine receptor activator of NF kappa B ligand (RANKL) by lymphocytes has been proposed as a mechanism by which sex steroid deficiency... 
TUMOR-NECROSIS-FACTOR | LYMPHOPOIESIS | MARROW | MESSENGER-RNA | OSTEOCLAST FORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESTROGEN DEFICIENCY | MICE | OSTEOPROTEGERIN | ACCELERATED OSTEOCLASTOGENESIS | T-CELLS | Bone Remodeling - immunology | Bone Density - immunology | RANK Ligand - metabolism | Humans | Bone Density - genetics | RANK Ligand - immunology | Osteoclasts - immunology | Osteoporosis - metabolism | T-Lymphocytes - metabolism | Estrogens - genetics | Female | Osteoporosis - genetics | Estrogens - metabolism | B-Lymphocytes - metabolism | Osteoporosis - immunology | Ovariectomy | Mice, Transgenic | Osteoclasts - metabolism | RANK Ligand - genetics | Animals | B-Lymphocytes - immunology | Alleles | Estrogens - immunology | T-Lymphocytes - immunology | Mice | Molecular Bases of Disease | Osteoporosis | RANKL | Estrogen | Osteoclast | Bone | Lymphocyte
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11. Full Text Cooperation of TNF family members CD40 ligand, receptor activator of NF-κB ligand, and TNF-α in the activation of dendritic cells and the expansion of viral specific CD8 + T cell memory responses in HIV-1-infected and HIV-1-uninfected individuals
by Yu, Qigui and Gu, Jenny X and Kovacs, Colin and Freedman, John and Thomas, Elaine K and Ostrowski, Mario A
Journal of Immunology, ISSN 0022-1767, 02/2003, Volume 170, Issue 4, pp. 1797 - 1805
Members of the TNF superfamily have been shown to be instrumental in enhancing cell-mediated immune responses, primarily through their interactions with... 
HELP | PLASMA VIREMIA | TUMOR-NECROSIS-FACTOR | VIRUS-INFECTION | HIV-INFECTION | INTERFERON-GAMMA | TRANCE | IMMUNOLOGY | MHC CLASS-II | DISEASE PROGRESSION | CUTTING EDGE | CD40 Ligand - physiology | Receptor Activator of Nuclear Factor-kappa B | Monocytes - cytology | Coculture Techniques | Dendritic Cells - immunology | Humans | Glycoproteins - metabolism | Culture Media, Conditioned - pharmacology | Monocytes - metabolism | NF-kappa B - metabolism | RANK Ligand | Monocytes - immunology | Lymphocyte Depletion | Interleukin-15 - biosynthesis | Osteoprotegerin | CD4-Positive T-Lymphocytes - immunology | HIV Infections - immunology | Membrane Glycoproteins - physiology | Cell Division - immunology | Leukocytes, Mononuclear - immunology | Epitopes, T-Lymphocyte - immunology | Dendritic Cells - metabolism | Interleukin-12 - biosynthesis | Carrier Proteins - physiology | T-Lymphocytes, Cytotoxic - immunology | CD4-Positive T-Lymphocytes - cytology | HIV Infections - virology | Cells, Cultured | Herpesvirus 4, Human - immunology | Immunophenotyping | Cell Differentiation - immunology | HIV-1 - immunology | Receptors, Tumor Necrosis Factor | CD8-Positive T-Lymphocytes - virology | Tumor Necrosis Factor-alpha - physiology | Immunologic Memory | Interleukin-10 - biosynthesis | Leukocytes, Mononuclear - cytology | Dendritic Cells - cytology | CD8-Positive T-Lymphocytes - immunology | T-Lymphocytes, Cytotoxic - cytology | Drug Combinations | Receptors, Cytoplasmic and Nuclear - metabolism
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12. Full Text Quantification of Ligand PET Studies using a Reference Region with a Displaceable Fraction: Application to Occupancy Studies with [11C]-DASB as an Example
by Turkheimer, Federico E and Selvaraj, Sudhakar and Hinz, Rainer and Murthy, Venkatesha and Bhagwagar, Zubin and Grasby, Paul and Howes, Oliver and Rosso, Lula and Bose, Subrata K
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 1/2012, Volume 32, Issue 1, pp. 70 - 80
This paper aims to build novel methodology for the use of a reference region with specific binding for the quantification of brain studies with radioligands... 
rank-shaping spectral analysis | [C-11]-DASB | SEROTONIN TRANSPORTER | HUMAN BRAIN | PROTEIN | occupancy | CITALOPRAM | spectral analysis | NEUROSCIENCES | kinetic modelling | POSITRON-EMISSION-TOMOGRAPHY | IN-VIVO | ENDOCRINOLOGY & METABOLISM | CEREBELLUM | OPTIMIZATION | positron emission tomography | SPECTRAL-ANALYSIS | HEMATOLOGY | reference region | RECEPTOR-BINDING | Binding, Competitive | Serotonin Uptake Inhibitors - pharmacokinetics | Reproducibility of Results | Radiopharmaceuticals - pharmacokinetics | Cerebellum - metabolism | Humans | Middle Aged | Linear Models | Male | Reference Values | Aniline Compounds - pharmacokinetics | Positron-Emission Tomography - methods | Serotonin Plasma Membrane Transport Proteins - metabolism | Tissue Distribution | Sulfides - pharmacokinetics | Cerebellum - diagnostic imaging | Models, Biological | Protein Binding | Adult | Citalopram - pharmacokinetics | Ligands | Radioligand Assay | Carbon Radioisotopes | [11C]-DASB | Original
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