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Nature, ISSN 0028-0836, 11/2010, Volume 468, Issue 7320, pp. 103 - 107
RANK ligand (RANKL), a TNF-related molecule, is essential for osteoclast formation, function and survival through interaction with its receptor RANK. Mammary... 
CELL FUNCTION | OSTEOPROTEGERIN-LIGAND | MULTIDISCIPLINARY SCIENCES | MEDROXYPROGESTERONE ACETATE | RECEPTOR | HORMONE REPLACEMENT THERAPY | HUMAN BREAST-TUMORS | OSTEOCLAST DIFFERENTIATION | EXPRESSION | GLAND DEVELOPMENT | TRANSGENIC MICE | Epithelial Cells - metabolism | RANK Ligand - metabolism | Progesterone - administration & dosage | Epithelial Cells - drug effects | Humans | Progestins - adverse effects | Cell Transformation, Neoplastic - chemically induced | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Lung Neoplasms - secondary | Mammary Neoplasms, Experimental - pathology | Medroxyprogesterone Acetate - adverse effects | Female | Precancerous Conditions - pathology | Receptor Activator of Nuclear Factor-kappa B - metabolism | Disease Models, Animal | 9,10-Dimethyl-1,2-benzanthracene - administration & dosage | Neoplasm Invasiveness | Mice, Inbred C57BL | Epithelial Cells - pathology | Mice, Transgenic | Progesterone - adverse effects | Mammary Neoplasms, Experimental - chemically induced | RANK Ligand - genetics | Animals | RANK Ligand - antagonists & inhibitors | Breast Neoplasms - pathology | Progestins - administration & dosage | Receptor Activator of Nuclear Factor-kappa B - genetics | Cell Proliferation - drug effects | Medroxyprogesterone Acetate - administration & dosage | Mice | Cell Transformation, Neoplastic - drug effects | Precancerous Conditions - prevention & control | Cell Transformation, Neoplastic - pathology | Mammary Tumor Virus, Mouse - genetics | Mammary Tumor Virus, Mouse - physiology | 9,10-Dimethyl-1,2-benzanthracene - adverse effects | Immunohistochemistry | Care and treatment | Usage | Physiological aspects | Breast cancer | Genetic aspects | Research | Progesterone | Tumor proteins | Health aspects | Risk factors | Proteins | Hormone replacement therapy | Rodents | Apoptosis | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 866 - 873
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, pp. e0154680 - e0154680
The receptor-activator of nuclear kappaB ligand (RANKL) signaling pathway plays an important role in the regulation of bone growth and mediates the formation... 
OSTEOCLASTOGENESIS | GIANT-CELL TUMOR | DENOSUMAB | MULTIDISCIPLINARY SCIENCES | Osteosarcoma - drug therapy | RANK Ligand - metabolism | Humans | Male | Antineoplastic Agents - therapeutic use | Fibrous Dysplasia of Bone - drug therapy | RNA, Messenger - metabolism | Bone Neoplasms - pathology | Giant Cell Tumor of Bone - pathology | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Bone Cysts, Aneurysmal - metabolism | Bone Neoplasms - genetics | Fibrous Dysplasia of Bone - genetics | Chondroma - drug therapy | Osteoclasts - pathology | Bone Cysts, Aneurysmal - pathology | Multiple Myeloma - genetics | Osteosarcoma - pathology | Bone Cysts, Aneurysmal - drug therapy | Chondroma - genetics | Gene Expression Regulation, Neoplastic | Chondroma - metabolism | Chondroma - pathology | Bone Neoplasms - metabolism | Denosumab - therapeutic use | Multiple Myeloma - drug therapy | Giant Cell Tumor of Bone - drug therapy | RNA, Messenger - antagonists & inhibitors | Female | Bone Neoplasms - drug therapy | Giant Cell Tumor of Bone - genetics | Osteosarcoma - metabolism | Fibrous Dysplasia of Bone - pathology | RNA, Messenger - genetics | Multiple Myeloma - metabolism | Organ Specificity | Chondrosarcoma - drug therapy | Fibrous Dysplasia of Bone - metabolism | Osteoclasts - metabolism | Bone Resorption - prevention & control | RANK Ligand - genetics | Multiple Myeloma - pathology | Giant Cell Tumor of Bone - metabolism | RANK Ligand - antagonists & inhibitors | Chondrosarcoma - metabolism | Osteosarcoma - genetics | Osteoclasts - drug effects | Bone Cysts, Aneurysmal - genetics | Bone resorption | Bones | Dosage and administration | Denosumab | Growth | Analysis | Immunohistochemistry | Chondrosarcoma | Multiple myeloma | Aneurysm | Bone tumors | Metastasis | Bone surgery | Metastases | Osteoprotegerin | Signal transduction | Bone growth | Biomedical materials | Osteosarcoma | Fibrous dysplasia | Bone dysplasia | Biocompatibility | Lesions | TRANCE protein | University graduates | Dysplasia | Statistical analysis | Gene expression | Osteoclastogenesis | Signaling | Bone cancer | Hospitals | Biopsy | Stromal cells | Orthopedics | Monoclonal antibodies | Ligands | Osteoclasts | Bone | Prostate cancer | Tumors | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2012, Volume 188, Issue 1, pp. 163 - 169
SHIP is an SH2-containing inositol-5-phosphatase expressed in hematopoietic cells. It hydrolyzes the PI3K product PI(3,4,5)P-3 and blunts the PI3K-initiated... 
SURVIVAL | ACTIVATION | COLONY-STIMULATING FACTOR | T-CELLS C1 | NUCLEAR-FACTOR | PHOSPHATIDYLINOSITOL 3-KINASE | GLYCOGEN-SYNTHASE KINASE-3 | SHIP | IMMUNOLOGY | FACTOR RECEPTOR | NEGATIVE REGULATOR | Cell Nucleus - immunology | RANK Ligand - metabolism | Inositol Polyphosphate 5-Phosphatases | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | Glycogen Synthase Kinase 3 - immunology | Osteoclasts - cytology | Enzyme Activation - immunology | Phosphatidylinositol 3-Kinases - immunology | Proto-Oncogene Proteins c-akt - genetics | RANK Ligand - immunology | Cell Differentiation - genetics | Signal Transduction - immunology | Phosphorylation - genetics | Cell Nucleus - metabolism | NFATC Transcription Factors - immunology | Osteoclasts - immunology | Phosphoric Monoester Hydrolases - immunology | Bone Marrow Cells - immunology | Phosphorylation - immunology | Active Transport, Cell Nucleus | Proto-Oncogene Proteins c-akt - metabolism | Phosphoric Monoester Hydrolases - genetics | NFATC Transcription Factors - metabolism | Signal Transduction - genetics | Glycogen Synthase Kinase 3 - metabolism | Osteoclasts - metabolism | Mice, Knockout | Phosphatidylinositol 3-Kinases - genetics | RANK Ligand - genetics | Cell Differentiation - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Cell Nucleus - genetics | Glycogen Synthase Kinase 3 - genetics | Mice | Enzyme Activation - genetics | Phosphoric Monoester Hydrolases - metabolism | Bone Marrow Cells - metabolism | NFATC Transcription Factors - genetics | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e74211 - e74211
The osteoprotegerin (OPG) and receptor activator of nuclear factor-kappa B ligand (RANKL) cytokine system, not only controls bone homeostasis, but has been... 
APOPTOSIS-INDUCING LIGAND | OSTEOBLAST DIFFERENTIATION | MATRIX GLA PROTEIN | MULTIDISCIPLINARY SCIENCES | RECEPTOR ACTIVATOR | OSTEOCLASTIC DIFFERENTIATION | SMOOTH-MUSCLE-CELLS | CHONDROCYTE-LIKE CELLS | CORONARY-ARTERY-DISEASE | KAPPA-B LIGAND | SERUM OSTEOPROTEGERIN LEVELS | Plaque, Atherosclerotic - genetics | RANK Ligand - metabolism | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Atherosclerosis - genetics | Humans | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Aorta - metabolism | RNA, Messenger - metabolism | Brachiocephalic Trunk - pathology | Collagen Type II - metabolism | Chondrocytes - drug effects | Vascular Calcification - metabolism | TNF-Related Apoptosis-Inducing Ligand - pharmacology | Brachiocephalic Trunk - metabolism | Diet, High-Fat | Osteoprotegerin - genetics | TNF-Related Apoptosis-Inducing Ligand - deficiency | Myocytes, Smooth Muscle - drug effects | Plaque, Atherosclerotic - pathology | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | Muscle, Smooth, Vascular - drug effects | Chondrocytes - pathology | Atherosclerosis - pathology | Calcium - pharmacology | Plaque, Atherosclerotic - metabolism | Signal Transduction | RNA, Messenger - genetics | Cells, Cultured | Gene Expression Regulation | Vascular Calcification - pathology | Atherosclerosis - metabolism | Collagen Type II - genetics | Mice, Knockout | Aorta - pathology | RANK Ligand - genetics | Muscle, Smooth, Vascular - pathology | Animals | Apolipoproteins E - genetics | Mice | Osteoprotegerin - metabolism | TNF-Related Apoptosis-Inducing Ligand - genetics | Calcification | Cytokines | RNA | Collagen | Apoptosis | Calcification (ectopic) | Calcium | Collagen (type II) | Homeostasis | Smooth muscle | Arthritis | Arteries | High fat diet | Osteoprotegerin | Proteins | Ethics | Osteoporosis | Biomedical materials | Apolipoprotein E | Atherosclerosis | Gangrene | Bone morphogenetic proteins | Tumor necrosis factor-TNF | Biocompatibility | Aorta | Bioindicators | Inhibition | Lesions | TRANCE protein | Cardiology | Bone morphogenetic protein 2 | Muscles | Inflammation | Bone turnover | IL-1β | Metabolism | Gene expression | Studies | Metaplasia | Tumor necrosis factor | Arteriosclerosis | Ligands | TRAIL protein | In vivo methods and tests | Peroxisome proliferator-activated receptors | Bone | Veins & arteries | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 11/2010, Volume 468, Issue 7320, pp. 98 - 102
Breast cancer is one of the most common cancers in humans and will on average affect up to one in eight women in their lifetime in the United States and... 
EPITHELIAL-CELLS | OSTEOPROTEGERIN-LIGAND | MULTIDISCIPLINARY SCIENCES | MEDROXYPROGESTERONE ACETATE | RECEPTOR ACTIVATOR | HORMONE-REPLACEMENT THERAPY | STEM-CELL | BONE METASTASIS | POSTMENOPAUSAL WOMEN | GLAND DEVELOPMENT | ESTROGEN-PLUS-PROGESTIN | Phosphoproteins - immunology | Gamma Rays | Epithelial Cells - metabolism | RANK Ligand - metabolism | Apoptosis - radiation effects | Epithelial Cells - drug effects | Progestins - adverse effects | NF-kappa B - metabolism | Stem Cells - cytology | Mammary Neoplasms, Experimental - metabolism | Osteoclasts - cytology | Stem Cells - metabolism | Mammary Neoplasms, Experimental - genetics | Receptor Activator of Nuclear Factor-kappa B - deficiency | Phosphoproteins - analysis | Mammary Neoplasms, Experimental - pathology | Medroxyprogesterone Acetate - adverse effects | Female | Cell Differentiation | Receptor Activator of Nuclear Factor-kappa B - metabolism | Epithelial Cells - cytology | Epithelial Cells - radiation effects | Signal Transduction | Mammary Neoplasms, Experimental - chemically induced | RANK Ligand - genetics | Integrin alpha6 - metabolism | Animals | Progestins - administration & dosage | Receptor Activator of Nuclear Factor-kappa B - genetics | Stem Cells - drug effects | RANK Ligand - deficiency | Cell Proliferation - drug effects | Medroxyprogesterone Acetate - administration & dosage | Mice | DNA Damage | Prevention | Complications and side effects | Care and treatment | Hormone therapy | Medroxyprogesterone | Development and progression | Breast cancer | Research | Cell differentiation | Health aspects | Risk factors | Cancer | Proteins | Womens health | Rodents | Cell cycle | Mammography | Females | Apoptosis | Index Medicus
Journal Article
BBA - General Subjects, ISSN 0304-4165, 06/2013, Volume 1830, Issue 6, pp. 3756 - 3766
Journal Article
Journal of Vascular Research, ISSN 1018-1172, 06/2014, Volume 51, Issue 2, pp. 118 - 131
Journal Article