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Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0173916 - e0173916
Damaged lung grafts obtained after circulatory death (DCD lungs) and warm ischemia may be at high risk of reperfusion injury after transplantation. Such lungs... 
OXIDATIVE STRESS | ACTIVATION | ALVEOLAR EPITHELIAL-CELLS | CIRCULATORY DEATH | MULTIDISCIPLINARY SCIENCES | PRIMARY GRAFT DYSFUNCTION | REPERFUSION INJURY | IN-VIVO | FACTOR-KAPPA-B | INTERLEUKIN-6 | TRANSPLANTATION | Bronchoalveolar Lavage Fluid - immunology | Pyrrolidines - administration & dosage | Male | Transplantation Immunology | Disease Models, Animal | Reperfusion Injury - rehabilitation | NF-kappa B - antagonists & inhibitors | Thiocarbamates - administration & dosage | Cytokines - metabolism | Immunity, Innate - drug effects | Lung Injury - rehabilitation | Rats | Tissue and Organ Procurement | Lung - physiopathology | Lung Transplantation - adverse effects | Rats, Sprague-Dawley | Lung Injury - physiopathology | Animals | Perfusion | Warm Ischemia - adverse effects | Reperfusion Injury - immunology | Antioxidants - administration & dosage | Reperfusion Injury - physiopathology | Lung Transplantation - methods | Tissue Donors | In Vitro Techniques | Lung Injury - immunology | Lung - immunology | Complications and side effects | Care and treatment | Lungs | Dosage and administration | Transplantation | Research | Chemical compounds | Reperfusion injury | Risk factors | Phosphorylation | Thoracic surgery | Intensive care | Transplants & implants | Lung | Lung transplantation | Biology | Activation | Kinases | Interleukin 6 | Proteins | Respiratory tract | Reperfusion | Ischemia | Bronchoalveolar lavage | Rodents | Surgery | Tumor necrosis factor-TNF | Damage | Alveoli | Pyrrolidine | Injuries | Edema | Ventilators | NF-κB protein | Immune response | Cytokines | Blood & organ donations | Pulmonary arteries | Weight gain measurement | Health risks | Bronchus | Pharmacology | Permeability | Tumor necrosis factor-α | Medicine | Hypotheses | Hospitals | Inhibitors | Grafts | In vivo methods and tests | Rehabilitation | Laboratory animals | Oxygenation | Pyrrolidine dithiocarbamate | Index Medicus
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2012, Volume 84, Issue 1, pp. 238 - 243
Purpose Angiotensin-converting enzyme (ACE) inhibitors have been shown to mitigate radiation-induced lung injury in preclinical models. The aim of this study... 
Radiology | Hematology, Oncology and Palliative Medicine | Radiation-induced lung injury | ACE inhibitors | Pulmonary toxicity | Lung cancer | Radiation pneumonitis | FIBROSIS | INDUCED PNEUMONOPATHY | PREVENTION | INJURY | MODEL | COMPUTED-TOMOGRAPHY | RAT LUNG | CELL LUNG-CANCER | RECEPTOR BLOCKER | ONCOLOGY | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Carcinoma, Non-Small-Cell Lung - pathology | Carcinoma, Non-Small-Cell Lung - radiotherapy | Lung Neoplasms - drug therapy | Radiation Pneumonitis - etiology | Radiation Pneumonitis - pathology | Humans | Middle Aged | Lung Neoplasms - radiotherapy | Lung Neoplasms - pathology | Male | Tumor Burden | Radiation Pneumonitis - epidemiology | Radiation Pneumonitis - prevention & control | Small Cell Lung Carcinoma - drug therapy | Incidence | Captopril - therapeutic use | Small Cell Lung Carcinoma - pathology | Lisinopril - therapeutic use | Small Cell Lung Carcinoma - radiotherapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Lung - radiation effects | Aged | Retrospective Studies | Carcinoma, Non-Small-Cell Lung - drug therapy | Enzymes | Medical colleges | Pneumonia | Nuclear radiation | Oncology, Experimental | Bacterial pneumonia | Research | Radiotherapy | Cancer | Index Medicus | Angiotensin-converting enzyme inhibitors | Corticoids | Antiinflammatory agents | Toxicity | Radiation | Thorax | Angiotensin receptors | Chemotherapy | Pneumonitis | Peptidyl-dipeptidase A | statins | Models | Injuries | PATIENTS | NEOPLASMS | ELDERLY PEOPLE | ENZYME INHIBITORS | LUNGS | RADIATION DOSES | TOXICITY | ANGIOTENSIN | CHEMOTHERAPY | RADIATION INJURIES | IRRADIATION | HEALTH HAZARDS | ENZYMES | INFLAMMATION | RADIOLOGY AND NUCLEAR MEDICINE | PNEUMONITIS | RADIOTHERAPY | RECEPTORS
Journal Article
Magnetic Resonance in Medicine, ISSN 0740-3194, 12/2017, Volume 78, Issue 6, pp. 2106 - 2115
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 01/2015, Volume 308, Issue 1, pp. L33 - L47
Mutation of threonine for isoleucine at codon 73 (I73T) in the human surfactant protein C (hSP-C) gene (SFTPC) accounts for a significant portion of SFTPC... 
Surfactant protein | Pulmonary fibrosis | Alveolar epithelium | Autophagy | Amphisome | alveolar epithelium | autophagy | amphisome | surfactant protein | pulmonary fibrosis | DOMAIN | PHYSIOLOGY | SURFACTANT PROTEIN-C | AGGRESOME FORMATION | ENDOPLASMIC-RETICULUM STRESS | RAT TYPE-II | LYSOSOMAL STORAGE DISORDERS | EPITHELIAL-CELLS | MULTIVESICULAR BODIES | RESPIRATORY SYSTEM | PULMONARY-FIBROSIS | Proteostasis Deficiencies - metabolism | Lung Diseases, Interstitial - metabolism | Vacuoles - ultrastructure | Lung Diseases, Interstitial - pathology | Microtubule-Associated Proteins - genetics | Sequestosome-1 Protein | Humans | Lysosomes - genetics | Genetic Diseases, Inborn - genetics | Infant | rab GTP-Binding Proteins - genetics | Genetic Diseases, Inborn - pathology | Mutation, Missense | Mitochondria - ultrastructure | Lysosomes - metabolism | ATP-Binding Cassette Transporters - genetics | Mitochondria - genetics | HEK293 Cells | ATP-Binding Cassette Transporters - metabolism | Membrane Potential, Mitochondrial - genetics | Female | Genetic Diseases, Inborn - metabolism | Microfilament Proteins - genetics | Vacuoles - genetics | Gene Expression Regulation - genetics | Proteostasis Deficiencies - pathology | Pulmonary Surfactant-Associated Protein C - metabolism | Mitochondria - metabolism | Microtubule-Associated Proteins - biosynthesis | rab GTP-Binding Proteins - biosynthesis | Ubiquitin-Protein Ligases - biosynthesis | Lysosomes - ultrastructure | Autophagy-Related Protein 8 Family | Proteostasis Deficiencies - genetics | Microfilament Proteins - biosynthesis | Lung Diseases, Interstitial - genetics | Adaptor Proteins, Signal Transducing - genetics | Pulmonary Surfactant-Associated Protein C - genetics | Vacuoles - metabolism | Adaptor Proteins, Signal Transducing - biosynthesis | Ubiquitin-Protein Ligases - genetics | Amino Acid Substitution | Autophagy (Cytology) | Usage | Genetic aspects | Epithelium | Health aspects | Lung diseases | Proteins | ABC transporters | Mutation | Biomass | Surfactants | Cells | Index Medicus | Call for Papers
Journal Article
Developmental Biology, ISSN 0012-1606, 2003, Volume 256, Issue 1, pp. 62 - 73
T1α, a differentiation gene of lung alveolar epithelial type I cells, is developmentally regulated and encodes an apical membrane protein of unknown function.... 
Lung | Development | Proliferation | Epithelium | Alveoli | Type I cell | Differentiation | Knockout | knockout | CATALYTIC SUBUNIT | development | differentiation | alveoli | proliferation | PULMONARY EPITHELIAL PROLIFERATION | MEMBRANE-PROTEIN | DEVELOPMENTAL BIOLOGY | PLASMA-MEMBRANE | RAT LUNG | MUCIN-TYPE GLYCOPROTEIN | lung | TARGETED DISRUPTION | type I cell | epithelium | GROWTH-FACTOR | TRANSCRIPTION FACTOR | MOUSE LUNG
Journal Article