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PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 4, p. e10025
Background: Cells from individuals with Friedreich's ataxia (FRDA) show reduced activities of antioxidant enzymes and cannot up-regulate their expression when... 
OXIDATIVE STRESS | TRIPLET REPEAT | PROTEIN | PGC-1-ALPHA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | YEAST FRATAXIN | PROMOTES MITOCHONDRIAL BIOGENESIS | MOUSE MODELS | IRON ACCUMULATION | Friedreich Ataxia - metabolism | Superoxide Dismutase - analysis | Oxidative Stress | Friedreich Ataxia - pathology | Antioxidants - metabolism | Humans | Cells, Cultured | Hydrogen Peroxide - pharmacology | Transcription Factors - genetics | PPAR gamma | Case-Control Studies | Down-Regulation - genetics | Mice, Knockout | Heat-Shock Proteins - genetics | Animals | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Mice | Cyclic AMP-Dependent Protein Kinases | Antioxidants | Enzymes | Ataxia | Superoxide | Hydrogen peroxide | Protein kinases | Cerebellum | Protein kinase A | Oxidative stress | Spinal cord | Animal models | Yeast | Transcription | Pathogenesis | Superoxide dismutase | Biosynthesis | Mitochondrial DNA | Kinases | Proteins | Mitochondria | Control | Hyperglycemia | Bioenergetics | Rodents | Down-regulation | Fibroblasts | Stress response | Deoxyribonucleic acid--DNA | Hydrogen ion concentration | Pioglitazone | siRNA | Pharmacology | Metabolism | Patients | Friedreich's ataxia | Musculoskeletal system | Protein kinase | Diabetes | Mutation | Molecular biology | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53765
Despite improvements in therapeutic approaches for head and neck squamous cell carcinomas (HNSCC), clinical outcome has remained disappointing, with 5-year... 
HEAD | DOMAIN | ACTIVATION | GROWTH-FACTOR-RECEPTOR | GENE | RAS | MULTIDISCIPLINARY SCIENCES | IDENTIFICATION | EXPRESSION | CANCER | SOMATIC MUTATIONS | Cell Proliferation | Prognosis | Carcinoma, Squamous Cell - genetics | Down-Regulation | Humans | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - genetics | Cell Movement - genetics | Disease Progression | Squamous Cell Carcinoma of Head and Neck | Animals | Carcinoma, Squamous Cell - mortality | Models, Biological | Cell Line, Tumor | Female | Head and Neck Neoplasms - genetics | Mice | MicroRNAs - genetics | Head and Neck Neoplasms - mortality | Genes, Tumor Suppressor | Neurofibromin 1 - genetics | Medical research | Squamous cell carcinoma | MicroRNA | Luciferase | Medicine, Experimental | B cells | Health aspects | Tumors | Cell proliferation | Target recognition | Transcription | Genomics | Neurofibromin 1 | Hepatocellular carcinoma | Kinases | Cancer therapies | Hovering | Ethics | Epidermal growth factor | Health care networks | Cell cycle | Head and neck | Head and neck carcinoma | Head | Cell survival | MiRNA | Extracellular signal-regulated kinase | Biophysics | Radiation therapy | Gene expression | Ribonucleic acid--RNA | Patients | Survival | MicroRNAs | Cell lines | Head and neck cancer | Mutation | Cell migration | Cancer | RNA | Ribonucleic acid
Journal Article
PLoS Biology, ISSN 1544-9173, 03/2011, Volume 9, Issue 3, p. e1000598
Journal Article
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e30771
Background: Hypoxia-induced renal tubular cell epithelial-mesenchymal transition (EMT) is an important event leading to renal fibrosis. MicroRNAs (miRNAs) are... 
HEPATOCELLULAR-CARCINOMA | REPRESSION | MULTIDISCIPLINARY SCIENCES | GROWTH | TRANSCRIPTION | E-CADHERIN | MESENCHYMAL TRANSITION | EXPRESSION | CANCER | RENAL FIBROGENESIS | FAMILY | Epithelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Molecular Sequence Data | Epithelial-Mesenchymal Transition - genetics | RNA, Messenger - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Kidney Failure, Chronic - pathology | Serrate-Jagged Proteins | Base Sequence | Membrane Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Cell Line | Kidney Tubules, Proximal - cytology | Membrane Proteins - genetics | RNA, Messenger - genetics | Intercellular Signaling Peptides and Proteins - genetics | Epithelial Cells - pathology | Signal Transduction - genetics | Down-Regulation - genetics | Kidney Failure, Chronic - genetics | MicroRNAs - genetics | Cell Hypoxia - genetics | Calcium-Binding Proteins - genetics | RNA, Small Interfering - metabolism | Infection | MicroRNA | Intermediate filament proteins | Analysis | Genes | Stem cells | Vimentin | Nephrology | Mesenchyme | Epithelial cells | Arthritis | mRNA | E-cadherin | Signal transduction | Reporter gene | Transfection | Rodents | Tumorigenesis | Jagged1 protein | Growth factors | Kidneys | MiRNA | Melanoma | siRNA | Gene expression | Polymerase chain reaction | Ribonucleic acids | Fibrosis | Hypoxia | Ligands | Notch protein | Non-coding RNA | Clear cell-type renal cell carcinoma | Cancer
Journal Article