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receptor-induced apoptosis (132) 132
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apoptosis (94) 94
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nf-kappa-b (67) 67
cell biology (50) 50
index medicus (49) 49
biochemistry & molecular biology (43) 43
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trail (14) 14
apoptosis - genetics (13) 13
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casp8 and fadd-like apoptosis regulating protein (12) 12
cell death (12) 12
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article (11) 11
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fadd (11) 11
fas receptor - genetics (11) 11
in-vivo (11) 11
inhibition (11) 11
jurkat cells (11) 11
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analysis (10) 10
cd95-mediated apoptosis (10) 10
flip (10) 10
gene expression (10) 10
models, biological (10) 10
nf-kappa b - metabolism (10) 10
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apoptosis-inducing ligand (9) 9
b-lymphocytes - immunology (9) 9
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domain-containing protein (9) 9
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health aspects (8) 8
hela cells (8) 8
immunohistochemistry (8) 8
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PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35345
Despite the importance of microRNAs (miRs) for regulation of the delicate balance between cell proliferation and death, evidence for their specific involvement... 
BLADDER-CANCER | MESSENGER-RNAS | INHIBITORY MOLECULE | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | SQUAMOUS-CELL CARCINOMA | TUMOR-SUPPRESSIVE MICRORNAS | GLUTATHIONE-S-TRANSFERASE | NF-KAPPA-B | PROTEIN-SYNTHESIS | Prostatic Neoplasms - pathology | Adenocarcinoma - pathology | Apoptosis - drug effects | Humans | Apoptosis - genetics | Male | MicroRNAs - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Glutathione Transferase - genetics | Prostatic Neoplasms - genetics | Transfection | Apoptosis Regulatory Proteins - genetics | Adenocarcinoma - genetics | MicroRNAs - genetics | HeLa Cells | Tumor Cells, Cultured | TNF-Related Apoptosis-Inducing Ligand - genetics | Receptors, Death Domain - genetics | Infection | MicroRNA | Tumor necrosis factor | Glutathione transferase | Analysis | Genes | B cells | Prostate cancer | Apoptosis | Cell proliferation | Cell culture | Transformation | Homeostasis | Amino acids | Biology | Infections | Kinases | Urology | Proteins | Cell growth | Immunology | Tumor necrosis factor-TNF | Tumorigenesis | Glutathione | CD95 antigen | Mortality | MiRNA | Tumor necrosis factor-α | Gene expression | Bladder cancer | Musculoskeletal system | Ribonucleic acids | Cell death | MicroRNAs | Proteomics | Ligands | TRAIL protein | Prostate | Cervical cancer | Tumors | Cancer
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 12/2004, Volume 10, Issue 24, pp. 8284 - 8292
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2008, Volume 14, Issue 10, pp. 3168 - 3176
Journal Article
Molecular Systems Biology, ISSN 1744-4292, 2011, Volume 7, Issue 1, pp. 553 - n/a
Receptor‐mediated apoptosis proceeds via two pathways: one requiring only a cascade of initiator and effector caspases (type I behavior) and the second... 
apoptosis | dynamical systems analysis | XIAP | caspases | kinetic modeling | SIGNALING PATHWAYS | LIGASE ACTIVITY | X-LINKED INHIBITOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR-INDUCED APOPTOSIS | CELL-DEATH | SURVIVING APOPTOSIS | CD95 TYPE-I | PROTEIN XIAP | CASPASE ACTIVATION | XIAP INHIBITION | Phenotype | Ubiquitination | Humans | Caspase 3 - metabolism | X-Linked Inhibitor of Apoptosis Protein - metabolism | Cell Line, Tumor | Mitochondria - metabolism | Apoptosis - physiology | TNF-Related Apoptosis-Inducing Ligand - metabolism | TNF-Related Apoptosis-Inducing Ligand - genetics | X-Linked Inhibitor of Apoptosis Protein - genetics | Mitochondrial Membranes - metabolism | Cell culture | Regulators | Liver | Homologous recombination | Immunoblotting | Systems analysis | Proteins | Genotype & phenotype | Signal transduction | Mitochondria | Genetic modification | Deoxyribonucleic acid--DNA | Binding | Computer simulation | Mortality | Liapunov exponents | Mammals | Hepatocytes | Computer applications | Predictions | Ligands | TRAIL protein | Mutation | Flow mapping | Drugs | Flow cytometry | Biotechnology | Phase diagrams | Variability | Colorectal carcinoma | Antibodies | Homology | Mammalian cells | Cell fate | Lymphocytes | Tumor necrosis factor-TNF | Mathematical models | Ubiquitin-protein ligase | Phenotypes | Cell survival | Caspase | Breast cancer | Tumor cell lines | Lymphoma | Lymphocytes B | Cell death | Protein expression | Death receptors | Major outer membrane protein | Apoptosis | Cancer | Tumors
Journal Article
Oncogene, ISSN 0950-9232, 02/2006, Volume 25, Issue 6, pp. 838 - 848
FLIP inhibits caspase 8 activation and apoptosis mediated by death receptors such as Fas and DR5. We studied the effect of c-FLIP on the apoptotic response to... 
5-fluorouracil | Irinotecan | Oxaliplatin | c-FLIP | Apoptosis | CARCINOMA-CELLS | 1ST-LINE TREATMENT | LIGAND-INDEPENDENT ACTIVATION | MULTICENTER RANDOMIZED-TRIAL | irinotecan | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | RECEPTOR-INDUCED APOPTOSIS | FLUOROURACIL | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | RESISTANCE | oxaliplatin | FAS | UP-REGULATION | Tumor Necrosis Factor-alpha - metabolism | Caspase 8 | Membrane Glycoproteins - metabolism | Humans | Intracellular Signaling Peptides and Proteins - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand | fas Receptor - metabolism | Apoptosis Regulatory Proteins - drug effects | Tumor Suppressor Protein p53 - genetics | Caspases - metabolism | TNF-Related Apoptosis-Inducing Ligand | Colorectal Neoplasms - drug therapy | fas Receptor - genetics | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Tumor Cells, Cultured | Intracellular Signaling Peptides and Proteins - genetics | Colorectal Neoplasms - metabolism | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Down-Regulation | Protein Splicing | Tumor Suppressor Protein p53 - metabolism | Caspase Inhibitors | Tumor Suppressor Protein p53 - drug effects | Apoptosis Regulatory Proteins - metabolism | Tumor Necrosis Factor-alpha - drug effects | Cell Death - physiology | fas Receptor - drug effects | Membrane Glycoproteins - drug effects | CASP8 and FADD-Like Apoptosis Regulating Protein | RNA, Small Interfering | Apoptosis - physiology | Mutation | Colorectal Neoplasms - pathology | Receptors, Tumor Necrosis Factor - drug effects
Journal Article