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Journal Article
Nature Neuroscience, ISSN 1097-6256, 08/2017, Volume 20, Issue 8, pp. 1074 - 1084
Aberrant epidermal growth factor receptor (EGFR) signaling is widespread in cancer, making the EGFR an important target for therapy. EGFR gene amplification... 
CANCER-CELLS | GROWTH-FACTOR RECEPTOR | LUNG-CANCER | PHASE-II TRIAL | TARGETED THERAPIES | FEEDBACK ACTIVATION | NEWLY-DIAGNOSED GLIOBLASTOMA | RECURRENT GLIOBLASTOMA | KINASE INHIBITORS | TYROSINE KINASES | NEUROSCIENCES | Cell Survival - drug effects | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Extracellular Signal-Regulated MAP Kinases - drug effects | Humans | ErbB Receptors - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Brain Neoplasms - drug therapy | Brain Neoplasms - metabolism | Signal Transduction - drug effects | Cell Line, Tumor | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Glioblastoma - drug therapy | Epidermal growth factor | Tumor necrosis factor | Extracellular signal-regulated kinases | Cellular signal transduction | Genetic aspects | Research | Gene expression | Glioblastoma multiforme | Tyrosine | Therapy | Transcription factors | Cell survival | Epidermal growth factor receptors | Secretion | Brain tumors | Glioblastoma | c-Jun protein | Extracellular signal-regulated kinase | Kinases | Axl protein | Gene amplification | Glioma cells | Protein-tyrosine kinase receptors | Tumor necrosis factor-TNF | Inhibition | Aberration | Protein-tyrosine kinase
Journal Article
Molecular Cancer, ISSN 1476-4598, 06/2010, Volume 9, Issue 1, pp. 159 - 159
Background: Glioblastomas are characterized by rapid cell growth, aggressive CNS infiltration, and are resistant to all known anticancer regimens. Recent... 
SURVIVAL | APOPTOSIS | ACTIVATION | METABOLISM | ONCOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH-FACTORS | DNA-DAMAGE | RECEPTOR | TUMOR-CELLS | RECURRENT MALIGNANT GLIOMAS | EXPRESSION
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2018, Volume 24, Issue 9, pp. 1449 - 1458
Journal Article
Journal of Cancer Research and Clinical Oncology, ISSN 0171-5216, 4/2014, Volume 140, Issue 4, pp. 573 - 582
Journal Article
Brain, ISSN 0006-8950, 05/2018, Volume 141, Issue 5, pp. 1390 - 1403
Reactive oxygen species are hypothesised to play a critical role in the development of epilepsy. Shekh-Ahmad et al. report that decreasing reactive oxygen... 
epilepsy | mitochondrial dysfunction | Nrf2-KEAP1 pathway | epileptogenesis | oxidative stress | ACTIVATION | STATUS EPILEPTICUS | KAINIC ACID | NEUROSCIENCES | RAT MODEL | DAMAGE | CLINICAL NEUROLOGY | NRF2 | CUL3-BASED E3 LIGASE | ADAPTER | MITOCHONDRIAL BIOENERGETICS | SPONTANEOUS RECURRENT SEIZURES
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 05/2012, Volume 56, Issue 5, pp. 2696 - 2704
Classifications Services AAC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
BROAD-SPECTRUM | ANTI-BIOFILM | MUTANT LIBRARY | ESCHERICHIA-COLI | ANTIBIOTIC-RESISTANCE | GENE-EXPRESSION | ANTIBACTERIAL | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | ANTIMICROBIAL PEPTIDES | PSEUDOMONAS-AERUGINOSA BIOFILMS | VIRULENCE | Biofilms - drug effects | Antimicrobial Cationic Peptides - chemical synthesis | Gene Expression - drug effects | Humans | Antimicrobial Cationic Peptides - pharmacology | Biofilms - growth & development | Burkholderia cenocepacia - genetics | Gene Expression Profiling | Listeria monocytogenes - growth & development | Pseudomonas aeruginosa - drug effects | Bacterial Translocation - drug effects | Anti-Bacterial Agents - chemical synthesis | Microbial Sensitivity Tests | Pseudomonas aeruginosa - genetics | Burkholderia cenocepacia - drug effects | Burkholderia cenocepacia - metabolism | Pseudomonas aeruginosa - metabolism | Listeria monocytogenes - genetics | Anti-Bacterial Agents - pharmacology | Microscopy, Fluorescence | Listeria monocytogenes - drug effects | Pathogens | Antimicrobial activity | Motility | Recurrent infection | Cationic peptides | Multidrug resistance | Chronic infection | Fluorescence | Amino acids | Data processing | Biomass | Twitching | Swarming | Biofilms | Cell death | Minimum inhibitory concentration | Swimming | Microscopes | Experimental Therapeutics
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 03/2017, Volume 23, Issue 5, pp. 1286 - 1298
Purpose: The PI3K-AKT-mTOR signaling pathway is frequently activated in glioblastoma and offers several druggable targets. However, clinical efficacy of... 
Journal Article | MAMMALIAN TARGET | INITIATING CELLS | ONCOLOGY | RECURRENT GLIOBLASTOMA | IN-VIVO | BRAIN PENETRATION | RAPAMYCIN INHIBITOR | ANTITUMOR-ACTIVITY | PHOSPHATIDYLINOSITOL 3-KINASE/MAMMALIAN TARGET | CANCER RESISTANCE PROTEIN | P-GLYCOPROTEIN | Humans | Morpholines - administration & dosage | ATP Binding Cassette Transporter, Subfamily G, Member 2 - genetics | Imidazoles - administration & dosage | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Quinolines - administration & dosage | Blood-Brain Barrier - drug effects | ATP Binding Cassette Transporter, Subfamily B, Member 1 - genetics | Mice, Knockout | Glioma - genetics | Phosphatidylinositol 3-Kinases - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Sirolimus - administration & dosage | Animals | TOR Serine-Threonine Kinases - genetics | Signal Transduction - drug effects | Triazines - administration & dosage | Glioma - pathology | Female | Mice | Glioma - drug therapy | TOR protein | Drugs | Brain | Animal models | Drug delivery systems | Brain tumors | Glioblastoma | INK4a protein | Transgenic | AKT protein | Drug delivery | Drug development | Immunosuppressive agents | Anticancer properties | MDR1 protein | Signal transduction | Blood-brain barrier | Rodents | ABC transporters | P-Glycoprotein | Efflux | Effectiveness | Glycoprotein | p16 Protein | Substrate inhibition | Pharmacology | Rapamycin | Survival | Substrates | 1-Phosphatidylinositol 3-kinase | Signaling | Penetration | Inhibitors | Glioma | Experimental design | Antitumor activity | Genetic engineering | Transporter | PTEN protein | Tumors
Journal Article
Journal of Neuro-Oncology, ISSN 0167-594X, 11/2018, Volume 140, Issue 2, pp. 261 - 268
Journal Article