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Arthritis & rheumatology (Hoboken, N.J.), ISSN 2326-5191, 7/2017, Volume 69, Issue 7, pp. 1418 - 1428
Journal Article
Cellular and Molecular Immunology, ISSN 1672-7681, 12/2018, Volume 15, Issue 12, pp. 1085 - 1087
Journal Article
Journal of Virology, ISSN 0022-538X, 09/2018, Volume 92, Issue 17
Journal Article
Medicine & Science in Sports & Exercise, ISSN 0195-9131, 07/2017, Volume 49, Issue 12, pp. 2394 - 2403
Journal Article
FEBS Letters, ISSN 0014-5793, 09/2015, Volume 589, Issue 19, pp. 2859 - 2865
REDD1 is induced by various cellular stresses; however, its expression in response to lipopolysaccharide (LPS) has not been clearly elucidated in immune cells.... 
Protein kinase A | Regulated in development and DNA damage response 1 | Mitogen- and stress-activated protein kinase 1 | Cyclooxygenase-2 | CREB activation | db-cAMP | dibutyryl cAMP | regulated in development and DNA damage response 1 | MSK1 | ChIP | REDD1 | mitogen- and stress-activated protein kinase 1 | chromatin immunoprecipitation | db-cAMP dibutyryl cAMP | Abbreviations REDD1 regulated in development and DNA damage response 1 | ChIP chromatin immunoprecipitation | MSK1 mitogen- and stress-activated protein kinase 1 | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INVOLVEMENT | IDENTIFICATION | CELL BIOLOGY | INHIBITION | BIOPHYSICS | GENE | RTP801 | STRESS | NF-KAPPA-B | UP-REGULATION | EXPRESSION | Cyclic AMP-Dependent Protein Kinases - metabolism | Dinoprostone - biosynthesis | Male | Transcription Factors - genetics | Enzyme Activation - drug effects | Macrophages, Peritoneal - cytology | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Animals | Signal Transduction - drug effects | Cyclic AMP Response Element-Binding Protein - metabolism | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Bucladesine - pharmacology | Mice | Mice, Inbred BALB C | p38 Mitogen-Activated Protein Kinases - metabolism | Macrophages, Peritoneal - metabolism | Macrophages, Peritoneal - drug effects | Macrophages | Mitogens | Index Medicus
Journal Article
TRANSLATIONAL ONCOLOGY, ISSN 1936-5233, 02/2019, Volume 12, Issue 2, pp. 336 - 349
FLT3-ITD and FLT3-TKD are the most frequent tyrosine kinase mutations in acute myeloid leukemia (AML), with the former conferring a poor prognosis. We have... 
ACTIVATION | ONCOLOGY | FLT3 | ACUTE MYELOGENOUS LEUKEMIA | THERAPEUTIC TARGET | DEGRADATION | BORTEZOMIB | MUTATIONS | REDD1 | MTOR | TRANSLATIONAL CONTROL
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2018, Volume 32, Issue 12, pp. 6883 - 6897
The role of dyslipidemia in the development of retinal dysfunction remains poorly understood. Using an animal model of diet-induced obesity/pre-type 2... 
Type 2 diabetes | Sphingolipids | Retinopathy | High-fat diet | Apoptosis | DIABETIC-RETINOPATHY | MECHANISM | sphingolipids | INDUCED APOPTOSIS | INSULIN ACTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | AKT | KINASE-B | CELL BIOLOGY | SKELETAL-MUSCLE | type 2 diabetes | INHIBITION | retinopathy | REPRESSOR REDD1 | high-fat diet | BIOLOGY | EXPRESSION
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2016, Volume 24, Issue 9, pp. 1639 - 1647
Summary Objective Aging is a main risk factor for the development of osteoarthritis (OA) and the molecular mechanisms underlying the aging-related changes in... 
Rheumatology | mTOR | Cartilage | REDD1 | Osteoarthritis | Autophagy | MAMMALIAN TARGET | RAPAMYCIN | SURVIVAL | RHEUMATOLOGY | FOXO TRANSCRIPTION FACTORS | HYPOXIA | INHIBITION | GENE | ARTICULAR-CARTILAGE | RTP801 | ORTHOPEDICS | EXPRESSION | Animals | Signal Transduction | Humans | Cartilage, Articular | Cells, Cultured | Chondrocytes | Mice | Medical colleges | Immunohistochemistry | Analysis | Index Medicus | autophagy | osteoarthritis | cartilage
Journal Article
FRONTIERS IN MOLECULAR NEUROSCIENCE, ISSN 1662-5099, 01/2018, Volume 10, pp. 430 - 430
Journal Article
Experimental Cell Research, ISSN 0014-4827, 11/2018, Volume 372, Issue 2, pp. 188 - 197
Regulated in development and DNA damage response 1 (REDD1) is an evolutionarily conserved, ubiquitous protein that responds to various cell stresses. Studies... 
Serum deprivation | Intervertebral disc degeneration | Hypoxia | REDD1 | ROS | ACTIVATION | MATRIX DEGRADATION | ROS GENERATION | CCAAT/ENHANCER-BINDING PROTEIN | CELL BIOLOGY | IN-VITRO | NUCLEUS PULPOSUS CELLS | GENE | ONCOLOGY | EXPRESSION | Medical colleges | DNA damage | Analysis | DNA | Sulfates | Osteoarthritis | DNA repair
Journal Article
Cancer Science, ISSN 1347-9032, 08/2019, Volume 110, Issue 8, pp. 2368 - 2377
Macrophages are essential inflammatory cells which regulate the features of immune reactions within tumors. Many studies have reported their regulatory roles... 
mTOR | macrophage | gastric cancer | REDD | miR‐30c | miR-30c | ONCOLOGY | MUSCLE | METABOLIC-REGULATION | INHIBITOR | EXPRESSION | REDD1 | REDD-1 | Glucose metabolism | MicroRNA | Genetic transcription | Macrophages | Stomach cancer | Cancer | Index Medicus | REDD‐1 | Original
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 446 - 14
Hepatocellular carcinoma (HCC) cells are metabolically distinct from normal hepatocytes by expressing the high-affinity hexokinase (HK2) and suppressing... 
C-13-METABOLIC FLUX ANALYSIS | METABOLISM | MULTIDISCIPLINARY SCIENCES | GROWTH | PARALLEL LABELING EXPERIMENTS | MASS ISOTOPOMER DISTRIBUTIONS | REDD1 | EXPRESSION | CANCER-THERAPY | MTOR | DELETION | Niacinamide - analogs & derivatives | Metformin - therapeutic use | Humans | Male | Antineoplastic Agents - therapeutic use | Metabolic Flux Analysis | Molecular Targeted Therapy | Carcinogenesis | Carcinoma, Hepatocellular - drug therapy | Antineoplastic Agents - pharmacology | Liver Neoplasms - enzymology | Hypoglycemic Agents - therapeutic use | Hexokinase - antagonists & inhibitors | Metformin - pharmacology | Liver Neoplasms - drug therapy | Oxidative Phosphorylation | Niacinamide - therapeutic use | Phenylurea Compounds - therapeutic use | Mechanistic Target of Rapamycin Complex 1 - antagonists & inhibitors | Carcinoma, Hepatocellular - enzymology | Mechanistic Target of Rapamycin Complex 1 - metabolism | Hep G2 Cells | Hypoglycemic Agents - pharmacology | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Sorafenib | Glycolysis | Phenylurea Compounds - pharmacology | Niacinamide - pharmacology | Hexokinase - metabolism | Phosphorylation | Secretion | Serine | Pyruvic acid | Hepatocellular carcinoma | Fluxes | Glycine | Hexokinase | Liver cancer | Mitochondria | Depletion | Clonal deletion | Oxidative phosphorylation | Hepatocytes | Cell death | Rodents | Glucokinase | Deletion | Tumorigenesis | Lactic acid | Metformin | Inhibition | Apoptosis | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 05/2011, Volume 30, Issue 21, pp. 2463 - 2474
MicroRNAs (miRNAs) are involved in tumorigenecity by regulating specific oncogenes and tumor suppressor genes, and their roles in breast cancer stem cells... 
hypoxia resistance | miR-495 | E-cadherin | E12/E47 | REDD1 | breast cancer stem cell | ACTIVATION | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SELF-RENEWAL | PROLIFERATION | MTOR | CELL BIOLOGY | INDUCIBLE FACTORS | ONCOLOGY | GROWTH | GENES | GENETICS & HEREDITY | EXPRESSION | MICRORNA | Cadherins - metabolism | Humans | Molecular Sequence Data | Immunoblotting | Transplantation, Heterologous | Gene Expression Profiling | Mammary Neoplasms, Experimental - metabolism | Promoter Regions, Genetic - genetics | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Cell Hypoxia | Neoplastic Stem Cells - metabolism | Base Sequence | HEK293 Cells | Mammary Neoplasms, Experimental - pathology | Transcription Factor 3 - metabolism | Female | Cadherins - genetics | Cell Line | Down-Regulation | Transcription Factors - genetics | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Transcription Factor 3 - genetics | Cell Line, Tumor | Protein Binding | Mice, Inbred NOD | Mice | MicroRNAs - genetics | Care and treatment | MicroRNA | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Genetic regulation | Oncology | Hypoxia | Cellular biology | Ribonucleic acid--RNA | Stem cells | Index Medicus
Journal Article