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index medicus (89) 89
pregnancy (85) 85
female (80) 80
animals (71) 71
reduced uterine perfusion (68) 68
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hypertension (61) 61
rats (48) 48
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rats, sprague-dawley (34) 34
pre-eclampsia - physiopathology (29) 29
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placental ischemia (24) 24
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physiology (22) 22
necrosis-factor-alpha (19) 19
placenta - blood supply (19) 19
disease models, animal (18) 18
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placenta (13) 13
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hypertension, pregnancy-induced - physiopathology (10) 10
intrauterine growth restriction (10) 10
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ischemia - physiopathology (10) 10
pathogenesis (10) 10
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adult (9) 9
angiotensin-ii (9) 9
blood pressure - physiology (9) 9
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sflt-1 (9) 9
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placenta - pathology (8) 8
placenta - physiopathology (8) 8
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heme oxygenase-1 (7) 7
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women (7) 7
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insulin-resistance (6) 6
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Journal Article
Brazilian Journal of Medical and Biological Research, ISSN 0100-879X, 2018, Volume 51, Issue 8, p. e6921
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 11/2019
Journal Article
Reproductive and Developmental Medicine, ISSN 2096-2924, 01/2019, Volume 3, Issue 1, pp. 36 - 41
Objective: Preeclampsia (PE) is a serious complication of pregnancy. Placental ischemia could be an initiating event, but the molecular mechanisms underlying... 
Lin28B; Placenta; Preeclampsia; Reduced Uterine Perfusion Pressure
Journal Article
Clinical Science, ISSN 0143-5221, 07/2012, Volume 123, Issue 2, pp. 53 - 72
Pre-eclampsia is increasingly recognized as more than an isolated disease of pregnancy. Women who have had a pregnancy complicated by pre-eclampsia have a... 
Pregnancy | Offspring | Cardiovascular disease | Blood pressure | Pre-eclampsia | In utero | Vascular function | offspring | INTIMA-MEDIA THICKNESS | CORTICOTROPIN-RELEASING HORMONE | ADULT VASCULAR DYSFUNCTION | MEDICINE, RESEARCH & EXPERIMENTAL | blood pressure | cardiovascular disease | vascular function | in utero | pregnancy | FETAL HYPOXIA | pre-eclampsia | PLACENTAL INSUFFICIENCY LEADS | INCREASES BLOOD-PRESSURE | INTRAUTERINE GROWTH RESTRICTION | UTERINE PERFUSION-PRESSURE | UMBILICAL-CORD BLOOD | EXPERIMENTAL TOXEMIA | Cardiovascular Diseases - etiology | Disease Susceptibility | Humans | Rats | Hypoxia - complications | Sex Characteristics | Pre-Eclampsia - physiopathology | Endothelium, Vascular - physiology | Inflammation - complications | Animals | Female | Uterus - blood supply | Vascular Endothelial Growth Factor A - physiology | IUGR, intra-uterine growth restriction | PKCϵ, protein kinase Cϵ | IMT, intima-media thickness | SBP, systolic BP | Review | R, ischaemia | PlGF, placental growth factor | BMI, body mass index | TGF, transforming growth factor | BP, blood pressure | ROS, reactive oxygen species | TIMP, tissue inhibitor of metalloproteinases | CYP, cytochrome P450 | reperfusion | L-NAME, NG-nitro-L-arginine methyl ester | HUVEC, human umbilical vein endothelial cell | VEGF, vascular endothelial growth factor | eNOS, endothelial NO synthase | RUPP, reduced uterine artery perfusion | sEng, soluble endoglin | LV, left ventricular | sFlt-1, soluble fms-like tyrosine kinase-1 | MMP, matrix metalloproteinase | LVH, LV hypertrophy
Journal Article
Physiology (Bethesda, Md.), ISSN 1548-9213, 05/2019, Volume 34, Issue 3, pp. 178 - 188
One of the mechanisms responsible for blood pressure (BP) regulation is thought to be oxidative stress. In this review, we highlight preclinical studies that... 
II-MEDIATED HYPERTENSION | VITAMIN-C | PHYSIOLOGY | NITRIC-OXIDE | RATS | REDUCED UTERINE PERFUSION | ANGIOTENSIN-II | THROMBOXANE A2 RECEPTOR | SUPEROXIDE-DISMUTASE | BLOOD-PRESSURE | T-LYMPHOCYTES
Journal Article