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Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2015, Volume 6, p. 393
Basophils are potent effector cells of innate immunity and also play a role in T helper 2 (Th2)-mediated allergic responses. But, although their in vitro... 
DENDRITIC CELLS | CHRONIC IDIOPATHIC URTICARIA | MAST-CELLS | REGULATES IMMUNITY | contact dermatitis | CD4(+) T-CELLS | basophil | HELPER TYPE-2 RESPONSE | IL-4 PRODUCTION | IMMUNOLOGY | BETA-CATENIN | IgE-CAI | IN-VIVO | ANTIGEN-PRESENTING CELLS | dendritic cell | Th2 | Contact dermatitis | th2
Journal Article
Trends in Molecular Medicine, ISSN 1471-4914, 2013, Volume 19, Issue 8, pp. 487 - 500
Journal Article
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2490
The high cardiovascular mortality associated with chronic kidney disease (CKD) is caused in part by the CKD-mineral bone disorder (CKD-MBD) syndrome. The... 
Osteoblast | Vascular calcification | Activin A | Osteoclast | CKD-MBD | DNA-BINDING | REGULATES OSTEOCLAST DIFFERENTIATION | FSH-RELEASING PROTEIN | TGF-BETA | OSTEOBLAST DIFFERENTIATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | vascular calcification | BINDING-PROTEIN | osteoblast | CHEMISTRY, MULTIDISCIPLINARY | activin A | SMOOTH-MUSCLE-CELLS | osteoclast | TYPE-2 DIABETES-MELLITUS | LACKING C-FOS | Bone and Bones - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - pathology | RANK Ligand - metabolism | Kidney - pathology | Humans | Activins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Activin Receptors - genetics | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Parathyroid Hormone - genetics | Kidney - metabolism | Osteoporosis - metabolism | Bone Resorption - genetics | Parathyroid Hormone - metabolism | Bone and Bones - metabolism | Bone Resorption - metabolism | Osteoporosis - genetics | Osteogenesis - genetics | Disease Models, Animal | Signal Transduction | Gene Expression Regulation | Smad Proteins - genetics | RANK Ligand - genetics | Activin Receptors - metabolism | Animals | Osteoporosis - pathology | Bone Resorption - pathology | Activins - genetics | Mice | Bone Remodeling | Smad Proteins - metabolism | Animal models | Target recognition | Medical services | Activation | Minerals | Bone diseases | Osteoblasts | Bone resorption | Osteoporosis | Receptors | Bone growth | Biomedical materials | Biocompatibility | TRANCE protein | Growth factors | Parathyroid | Public health | Osteopenia | Bone remodelling | Diabetes mellitus | Bone turnover | Metabolism | Activin receptors | Osteoclastogenesis | Signaling | Molecular modelling | Osteodystrophy | Parathyroid hormone | Ligands | Osteoclasts | Kidney diseases | Receptor mechanisms | Cardiovascular diseases | Activin | Osteogenesis | Phosphatase | Proteins | Signal transduction | Mineralization | Bone marrow | Cytokines | Mortality | Gene expression | Calcification | Phosphorylation | Homeostasis | Kinases | Tumor necrosis factor-TNF | Diabetes
Journal Article
Cytokine and Growth Factor Reviews, ISSN 1359-6101, 2014, Volume 25, Issue 6, pp. 681 - 697
Journal Article