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The Journal of physiology, ISSN 0022-3751, 07/2016, Volume 594, Issue 13, pp. 3521 - 3531
Journal Article
Glia, ISSN 0894-1491, 12/2017, Volume 65, Issue 12, pp. 2087 - 2098
The regeneration of oligodendrocytes is a crucial step in recovery from demyelination, as surviving oligodendrocytes exhibit limited structural plasticity and... 
oligodendrocyte progenitor cells | demyelination | multiple sclerosis | gray matter | remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Age Factors | RNA, Untranslated - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Hippocampus - drug effects | Cell Lineage - drug effects | RNA, Untranslated - genetics | Cell Differentiation - genetics | Oligodendrocyte Precursor Cells - drug effects | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Cell Lineage - genetics | Disease Models, Animal | Cuprizone - toxicity | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Mice, Inbred C57BL | Bacterial Proteins - genetics | Corpus Callosum - drug effects | Mice, Transgenic | Hippocampus - pathology | Monoamine Oxidase Inhibitors - toxicity | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Corpus Callosum - pathology | Remyelination - physiology | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Remyelination - drug effects | Luminescent Proteins - metabolism | Medicine, Experimental | Medical research | Multiple sclerosis | Platelet-derived growth factor | Cortex (cingulate) | Myelin | Central nervous system | Axonal plasticity | Substantia alba | Substantia grisea | Glial stem cells | Corpus callosum | Atrophy | Regeneration | Myelination | Cuprizone | Sheaths | Demyelination | Oligodendrocytes | Cells (biology) | In vivo methods and tests | Differentiation | Cortex (temporal) | Index Medicus
Journal Article
Acta neuropathologica, ISSN 0001-6322, 9/2017, Volume 134, Issue 3, pp. 423 - 440
Journal Article
Glia, ISSN 1098-1136, 02/2019, Volume 67, Issue 7, pp. 1320 - 1332
Oligodendrocytes (OLs) are the myelinating glia of the central nervous system. Injury to OLs causes myelin loss. In demyelinating diseases, such as multiple... 
myelin | differentiation | oligodendrocyte | MEK inhibitor | MAPK | oligodendrocyte progenitor cell | ERK | remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | MAP Kinase Signaling System - physiology | Recovery of Function - drug effects | Coculture Techniques | Oligodendroglia - drug effects | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Recovery of Function - physiology | Female | Demyelinating Diseases - chemically induced | Cerebral Cortex - drug effects | Demyelinating Diseases - drug therapy | Cuprizone - toxicity | Cerebral Cortex - enzymology | Demyelinating Diseases - enzymology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Enzyme Inhibitors - therapeutic use | Animals | Encephalomyelitis, Autoimmune, Experimental - enzymology | MAP Kinase Signaling System - drug effects | Remyelination - physiology | Oligodendroglia - enzymology | Mice | Remyelination - drug effects | Multiple sclerosis | Analysis | Myelin proteins | Cell culture | Central nervous system | Kinases | Glial stem cells | Myelin basic protein | Time dependence | Cuprizone | Sheaths | Demyelination | Oligodendrocytes | Inducers | Demyelinating diseases | Myelin | MEK inhibitors | Extracellular signal-regulated kinase | MAP kinase | Progenitor cells | Experimental allergic encephalomyelitis | Diseases | Regeneration | Myelination | Cells (biology) | Neural stem cells | Autoimmune diseases | Differentiation | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 12/2018, Volume 115, Issue 50, pp. E11807 - E11816
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 2006, Volume 26, Issue 29, pp. 7665 - 7673
Previous reports, including transplantation experiments using dominant-negative inhibition of β1-integrin signaling in oligodendrocyte progenitor cells,... 
CNS | Myelin | Survival | Remyelination | Oligodendrocytes | Integrins | myelin | integrins | oligodendrocytes | survival | remyelination
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 12/2017, Volume 96, Issue 5, pp. 1003 - 1012.e7
Blood-brain barrier (BBB) disruption alters the composition of the brain microenvironment by allowing blood proteins into the CNS. However, whether... 
myelin | vasculature | ancrod | stem/progenitor cells | neuroinflammation | regeneration | cell fate | NG2 cells | fibrin | neonatal brain injury | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Lysophosphatidylcholines - pharmacology | Blood Vessels - pathology | Mice, Inbred C57BL | Activin Receptors, Type I - metabolism | Activin Receptors, Type I - genetics | Fibrinogen - antagonists & inhibitors | Activin Receptors, Type I - drug effects | Mice, Knockout | Myelin Sheath - metabolism | Bone Morphogenetic Proteins - metabolism | Animals | Oligodendrocyte Precursor Cells - drug effects | Signal Transduction - drug effects | Microarray Analysis | Blood Vessels - drug effects | Plasmids - genetics | Fibrinogen - pharmacology | Mice | Remyelination - drug effects | Oligodendrocyte Precursor Cells - metabolism | Brain | Fibrin | Bone morphogenetic proteins | Nervous system diseases | Injuries | Cells | Phosphorylation | Smad protein | Multiple sclerosis | Genes | Central nervous system | Homology | Genomes | Glial stem cells | Proteins | Signal transduction | Blood-brain barrier | Rodents | Oligodendrocytes | Fibrinogen | Lesions | CRISPR | Antigens | Stroke | Permeability | Hemopoiesis | Signaling | Myelination | Cells (biology) | Protein expression | Brain damage | Activin | Brain injury | Index Medicus
Journal Article