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Journal of Immunology, ISSN 0022-1767, 04/2011, Volume 186, Issue 7, pp. 4388 - 4395
Journal Article
Circulation, ISSN 0009-7322, 11/2014, Volume 130, Issue 21, pp. 1847 - 1858
BACKGROUND—Growth differentiation factor 15 (GDF-15), high-sensitivity troponin, and N-terminal pro-brain natriuretic peptide levels are predictive of death... 
risk assessment | Brain | atrial fibrillation | cardiovascular physiological processes | troponin | Natriuretic peptide | biological markers | growth differentiation factor 15 | natriuretic peptide, brain | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | RENAL-FUNCTION | PREVENTION | RANDOMIZED EVALUATION | ACUTE CORONARY SYNDROME | ANTICOAGULATION | INTEGRIN ACTIVATION | WARFARIN | INHIBITORY CYTOKINE-1 | PERIPHERAL VASCULAR DISEASE | STRATIFICATION | Pyrazoles - therapeutic use | Follow-Up Studies | Humans | Middle Aged | Oxidative Stress - physiology | Stroke - diagnosis | Male | Inflammation - blood | Thromboembolism - diagnosis | Inflammation - drug therapy | Female | Thromboembolism - drug therapy | Factor Xa Inhibitors - pharmacology | Inflammation - diagnosis | Atrial Fibrillation - diagnosis | Pyrazoles - pharmacology | Double-Blind Method | Risk Assessment | Atrial Fibrillation - drug therapy | Stroke - drug therapy | Biomarkers - blood | Atrial Fibrillation - blood | Stroke - blood | Factor Xa Inhibitors - therapeutic use | Growth Differentiation Factor 15 - blood | Thromboembolism - blood | Aged | Pyridones - therapeutic use | Oxidative Stress - drug effects | Pyridones - pharmacology | Complications and side effects | Oxidative stress | Usage | Atrial fibrillation | Apixaban | Clinical trials | Inflammation | Dosage and administration | Research | Biological markers | Risk factors | Medical and Health Sciences | Medicin och hälsovetenskap | natriuretic peptide | Klinisk medicin | brain | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems
Journal Article
Nature Reviews Microbiology, ISSN 1740-1526, 02/2009, Volume 7, Issue 2, pp. 99 - 109
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0169024
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2013, Volume 169, Issue 3, pp. 477 - 492
Recent advances have improved our understanding of the renin‐angiotensin system (RAS). These have included the recognition that angiotensin (Ang)‐(1‐7) is a... 
angiotensin‐(1‐7) | renal diseases | fibrosis | Mas receptor | inflammation | angiotensin-(1-7) | CONVERTING ENZYME 2 | EXPERIMENTAL AUTOIMMUNE MYOCARDITIS | SPONTANEOUSLY HYPERTENSIVE-RATS | II TYPE-1 RECEPTOR | PROXIMAL TUBULAR CELLS | TRANSFORMING GROWTH FACTOR-BETA | END-ORGAN PROTECTION | INTERACTIONS IN-VIVO | INDUCED CARDIAC DYSFUNCTION | PHARMACOLOGY & PHARMACY | TGF-BETA ACTIVATION | Leukocytes - pathology | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Humans | Kidney - immunology | Angiotensin I - therapeutic use | Nephritis - pathology | Receptors, G-Protein-Coupled - agonists | Leukocytes - immunology | Peptidyl-Dipeptidase A - genetics | Kidney - metabolism | Nephritis - immunology | Proto-Oncogene Proteins - agonists | Peptidyl-Dipeptidase A - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Peptide Fragments - metabolism | Kidney - drug effects | Angiotensin I - metabolism | Animals | Peptidyl-Dipeptidase A - chemistry | Signal Transduction - drug effects | Models, Biological | Nephritis - therapy | Fibrosis | Receptors, G-Protein-Coupled - antagonists & inhibitors | Leukocytes - drug effects | Nephritis - metabolism | Cell Proliferation - drug effects | Peptide Fragments - therapeutic use | Leukocytes - metabolism | Inflammation | Angiotensin | Reviews
Journal Article