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Kidney International, ISSN 0085-2538, 07/2012, Volume 82, Issue 2, pp. 172 - 183
Chronic kidney disease (CKD) remains one of the leading causes of death in the developed world, and acute kidney injury (AKI) is now recognized as a major risk... 
nephron | renal cell biology | chronic kidney disease | acute kidney injury | proliferation | ISCHEMIA/REPERFUSION INJURY | COLLAGEN | RAT | ACUTE-RENAL-FAILURE | EPITHELIAL-CELLS | DISEASE | UROLOGY & NEPHROLOGY | ABLATION | Cell Proliferation | Epithelial Cells - metabolism | Acute Kidney Injury - genetics | Green Fluorescent Proteins - genetics | Mice, 129 Strain | Glomerulonephritis - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Time Factors | Acute Kidney Injury - complications | Acute Kidney Injury - immunology | Glomerulonephritis - etiology | Glomerulonephritis - physiopathology | Disease Models, Animal | Kidney Tubules, Proximal - physiopathology | Green Fluorescent Proteins - metabolism | Kidney Tubules, Proximal - immunology | Kidney Tubules, Proximal - pathology | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Glomerulonephritis - genetics | Intercellular Signaling Peptides and Proteins - genetics | Epithelial Cells - pathology | Glomerulonephritis - immunology | Mice, Transgenic | Acute Kidney Injury - physiopathology | Disease Progression | Heparin-binding EGF-like Growth Factor | Regeneration | Animals | Epithelial Cells - immunology | Glomerulonephritis - pathology | Fibrosis | Kidney Tubules, Proximal - metabolism | Mice | Acute Kidney Injury - metabolism | Apoptosis | Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 05/2012, Volume 302, Issue 10, pp. 1252 - 1264
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 39, pp. 14193 - 14198
Lupus nephritis (LN) is an autoimmune disease that occurs when autoantibodies complex with self-antigen and form immune complexes that accumulate in the... 
Urine | Kidneys | Fibrosis | Fibroblasts | Inflammation | Physical trauma | Mathematical models | Myofibroblasts | Macrophages | Modeling | Renal fibrosis | Tubulointerstitial inflammation | Math modeling | GLOMERULONEPHRITIS | TGF-BETA | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | math modeling | renal fibrosis | EPITHELIAL-CELL DEATH | CARDIAC FIBROBLASTS | tubulointerstitial inflammation | IN-VITRO | LUPUS NEPHRITIS | FIBROTIC RESPONSE | KIDNEY | Transforming Growth Factor beta - urine | Biomarkers - urine | Kidney - pathology | Cell Count | Humans | Extracellular Matrix - metabolism | Mathematical Concepts | Lupus Nephritis - etiology | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Lupus Nephritis - metabolism | Kidney Tubules - pathology | Lupus Nephritis - pathology | Myofibroblasts - pathology | Macrophages - pathology | Epithelial Cells - pathology | Disease Progression | Tissue Inhibitor of Metalloproteinases - metabolism | Platelet-Derived Growth Factor - metabolism | Models, Biological | Nephritis, Interstitial - etiology | Matrix Metalloproteinases - metabolism | Chemokine CCL2 | Usage | Models | Kidney diseases | Antigens | Immunology | Autoimmune diseases | Inflammatory diseases | Index Medicus | Biological Sciences | Physical Sciences
Journal Article
Kidney International, ISSN 0085-2538, 08/2010, Volume 78, Issue 3, pp. 257 - 268
Journal Article
by Li, SL and Wang, YP and Wang, ZJ and Liu, GD and Zuo, BJ and Liu, CX and Sun, D
JOURNAL OF MOLECULAR MEDICINE-JMM, ISSN 0946-2716, 06/2019, Volume 97, Issue 6, pp. 777 - 791
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 02/2014, Volume 29, Issue 2, pp. 313 - 324
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 07/2014, Volume 86, Issue 1, pp. 75 - 85
Fibrosis is a hallmark of chronic kidney disease, for which there is currently no effective cure. The hormone relaxin is emerging as an effective antifibrotic... 
Smad2 | kidney disease | TGF-β | fibrosis | relaxin | AT2 receptor | KIDNEY-DISEASE | NITRIC-OXIDE SYNTHASE | PROTEIN-KINASE | RESONANCE ENERGY-TRANSFER | FIBROBLASTS | TGF-beta | GROWTH-FACTOR-BETA | AT RECEPTORS | IN-VIVO | UROLOGY & NEPHROLOGY | HYPERTENSIVE-RATS | HORMONE RELAXIN | Relaxin - metabolism | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Humans | Protein Multimerization | Male | Receptors, Peptide - chemistry | Angiotensin II Type 2 Receptor Blockers - pharmacology | Relaxin - pharmacology | Renal Insufficiency, Chronic - drug therapy | Myofibroblasts - metabolism | Renal Insufficiency, Chronic - metabolism | Kidney - metabolism | Myofibroblasts - pathology | Receptor, Angiotensin, Type 2 - genetics | Kidney - drug effects | Cells, Cultured | Rats | Imidazoles - pharmacology | Recombinant Proteins - pharmacology | Myofibroblasts - drug effects | Rats, Sprague-Dawley | Disease Progression | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | MAP Kinase Signaling System - drug effects | Receptor, Angiotensin, Type 2 - metabolism | Signal Transduction - drug effects | Fibrosis | Mice | Pyridines - pharmacology | Receptor, Angiotensin, Type 2 - deficiency | Receptors, G-Protein-Coupled - chemistry | Receptors, Peptide - metabolism | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2010, Volume 120, Issue 11, pp. 4040 - 4054
Chronic kidney disease is a leading cause of death in the United States. Tubulointerstitial fibrosis (TIF) is considered the final common pathway leading to... 
MEDICINE, RESEARCH & EXPERIMENTAL | RENAL FIBROSIS | TO-MESENCHYMAL TRANSITION | TARGETED DISRUPTION | TGF-BETA | GROWTH ARREST | IN-VIVO | INJURY | DIABETIC-NEPHROPATHY | URETERAL OBSTRUCTION | RBP-J | Transcription Factor HES-1 | Cell Proliferation | Epithelial Cells - metabolism | Kidney - pathology | Homeodomain Proteins - metabolism | Humans | Male | Intercellular Signaling Peptides and Proteins - metabolism | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Kidney Failure, Chronic - pathology | Serrate-Jagged Proteins | Membrane Proteins - metabolism | Epithelial Cells - cytology | Calcium-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Mice, Transgenic | Kidney - cytology | Receptor, Notch1 - metabolism | Homeodomain Proteins - genetics | Kidney Failure, Chronic - metabolism | Kidney Tubules - cytology | Animals | Signal Transduction - physiology | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Fibrosis - pathology | Receptor, Notch1 - genetics | Calcium-Binding Proteins - genetics | Complications and side effects | Chronic kidney failure | Fibrosis | Physiological aspects | Development and progression | Genetic aspects | Cellular signal transduction | Research | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 10/2009, Volume 297, Issue 4, pp. 996 - 1005
Journal Article