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Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1 - 11
textabstractBesides its role in homologous recombination, the tumor suppressor BRCA2 protects stalled replication forks from nucleolytic degradation. Defective... 
RAD52 | ZRANB3 | MULTIDISCIPLINARY SCIENCES | STRAND EXCHANGE | DNA-DAMAGE | DYNAMICS | HOMOLOGOUS RECOMBINATION | STRESS | BRCA2 | PROTEINS | PROMOTES | BRCA2 protein | Stability | Deactivation | Homologous recombination | Homology | Breast cancer | Inactivation | Remodeling | Intermediates | Replication forks | Degradation | MRE11 protein | Breakage | Tumor suppressor genes | Replication | Deoxyribonucleic acid--DNA | Rad52 protein | Tumors
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 06/2018, Volume 25, Issue 6, pp. 446 - 453
Journal Article
Nature Reviews Molecular Cell Biology, ISSN 1471-0072, 03/2010, Volume 11, Issue 3, pp. 208 - 219
Journal Article
Nature, ISSN 0028-0836, 07/2016, Volume 535, Issue 7612, pp. 382 - 387
Cells deficient in the Brca1 and Brca2 genes have reduced capacity to repair DNA double-strand breaks by homologous recombination and consequently are... 
BREAST-CANCER | RECOMBINATION | LYSINE-4 METHYLTRANSFERASE COMPLEX | MUTANT-CELLS | 53BP1 | MULTIDISCIPLINARY SCIENCES | RESISTANCE | HOMOLOGY-DIRECTED REPAIR | CONDITIONAL MOUSE MODEL | END RESECTION | DNA-REPAIR | Embryonic Stem Cells - metabolism | DNA Replication - drug effects | Homologous Recombination | DNA Repair - genetics | DNA Breaks, Double-Stranded | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | Neoplasms - genetics | Gene Deletion | Nuclear Proteins - deficiency | DNA Repair Enzymes - metabolism | DNA Damage - genetics | Genes, BRCA2 | DNA - biosynthesis | Female | DNA Repair Enzymes - antagonists & inhibitors | Genes, BRCA1 | Nuclear Proteins - genetics | DNA Helicases - genetics | DNA Damage - drug effects | DNA Repair - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Cisplatin - pharmacology | DNA - metabolism | Carrier Proteins - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Poly(ADP-ribose) Polymerases - genetics | DNA Replication - physiology | Embryonic Stem Cells - drug effects | Cell Line, Tumor | Mice | Poly (ADP-Ribose) Polymerase-1 | Neoplasms - pathology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Chemotherapy | Replication fork | B cells | DNA damage | Cancer | Proteins | Lymphocytes | Stem cells | Breast cancer | Drug resistance | Deoxyribonucleic acid--DNA | Tumors
Journal Article