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Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 05/2013, Volume 169, Issue 2, pp. 449 - 461
Background and Purpose We have evaluated the influence of calcium-activated potassium channels (KCa) activation on cGMP-mediated relaxation in human penile... 
NS1619 | PDE5 | human corpus cavernosum | human penile resistance arteries | calcium‐activated potassium channels | diabetes | sildenafil | erectile dysfunction | LARGE-CONDUCTANCE | POTASSIUM CHANNELS | SMOOTH-MUSCLE TONE | SILDENAFIL CITRATE | BKCA CHANNEL | calcium-activated potassium channels | NEUROGENIC CONTRACTIONS | NS-8 | ENDOTHELIAL-CELLS | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | INTERMEDIATE-CONDUCTANCE | Piperazines - administration & dosage | Erectile Dysfunction - physiopathology | Humans | Middle Aged | Male | Purines - administration & dosage | Sulfones - pharmacology | Dose-Response Relationship, Drug | Sildenafil Citrate | Penis - drug effects | Erectile Dysfunction - drug therapy | Potassium Channels, Calcium-Activated - metabolism | Adult | Diabetes Mellitus, Experimental - complications | Phosphodiesterase 5 Inhibitors - pharmacology | Penis - blood supply | Vasodilator Agents - pharmacology | Purines - pharmacology | Phosphodiesterase 5 Inhibitors - administration & dosage | Rats | Erectile Dysfunction - etiology | Piperazines - pharmacology | Rats, Sprague-Dawley | Tadalafil | Animals | Cyclic GMP - metabolism | Electric Stimulation Therapy - methods | Penis - metabolism | Benzimidazoles - pharmacology | Aged | Vasodilation - drug effects | Nitric Oxide - metabolism | Vasodilator Agents - administration & dosage | Carbolines - pharmacology | Sulfones - administration & dosage | Diabetics | Diabetes | Impotence | Vasodilators | Arteries | Research Papers
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2009, Volume 296, Issue 5, pp. 1274 - 1280
The free radical form of nitric oxide (NO·) is a well-known mediator of vascular tone. What is not so well recognized is that NO· exists in several different... 
Nitroxyl | channels | Nitric oxide | Hyperpolarization | Soluble guanylate cyclase | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | POTASSIUM CHANNELS | NITRIC-OXIDE SYNTHASE | nitroxyl | NITROGEN-OXIDE | MESENTERIC-ARTERY | PULMONARY-ARTERY | soluble guanylate cyclase | VASCULAR SMOOTH-MUSCLE | ANGELIS SALT | SOLUBLE GUANYLYL CYCLASE | NITROXYL ANION | GATED K+ CHANNELS | K+ channels | PERIPHERAL VASCULAR DISEASE | hyperpolarization | Free Radical Scavengers - pharmacology | Muscle, Smooth, Vascular - metabolism | Male | Guanylate Cyclase - antagonists & inhibitors | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Potassium Channels, Voltage-Gated - metabolism | Oxadiazoles - pharmacology | Cysteine - pharmacology | Vascular Resistance - drug effects | Nitrogen Oxides - metabolism | 4-Aminopyridine - pharmacology | Potassium Channel Blockers - pharmacology | Muscle, Smooth, Vascular - drug effects | Vasodilator Agents - pharmacology | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Guanylate Cyclase - metabolism | Potassium Channels, Voltage-Gated - antagonists & inhibitors | Soluble Guanylyl Cyclase | Nitrites - pharmacology | Rats, Sprague-Dawley | Animals | Mesenteric Artery, Superior - metabolism | Cyclic GMP - metabolism | Membrane Potentials | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Vasodilation - drug effects | In Vitro Techniques | Nitric Oxide - metabolism | Muscle, Smooth, Vascular - enzymology | Receptors, Cytoplasmic and Nuclear - metabolism | Biochemistry | Pharmacology | Free radicals | Potassium | Veins & arteries
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2017, Volume 37, Issue 12, pp. 2370 - 2378
OBJECTIVE—Excess deposition of fat within and around vital organs and nonadipose tissues is hypothesized to contribute to cardiovascular disease (CVD) risk. We... 
Vascular calcification | Body mass index | Muscle | Adipose tissue | Prevalence | Risk factors | risk factors | CARDIOMETABOLIC RISK | METABOLIC SYNDROME | body mass index | FATTY LIVER-DISEASE | vascular calcification | adipose tissue | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | BODY-COMPOSITION | ABDOMINAL OBESITY | prevalence | HEART-DISEASE | ATHEROSCLEROSIS MESA | INSULIN-RESISTANCE | muscle | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Multivariate Analysis | United States - epidemiology | Intra-Abdominal Fat - physiopathology | Age Factors | Humans | Middle Aged | Vascular Calcification - diagnostic imaging | Male | Asymptomatic Diseases | Young Adult | Adiposity | Coronary Angiography - methods | Coronary Artery Disease - physiopathology | Pericardium - physiopathology | Vascular Calcification - physiopathology | Adult | Female | Pericardium - diagnostic imaging | Odds Ratio | Abdominal Muscles - diagnostic imaging | Multidetector Computed Tomography | Cross-Sectional Studies | Risk Factors | Logistic Models | Abdominal Muscles - physiopathology | Computed Tomography Angiography | Intra-Abdominal Fat - diagnostic imaging | Coronary Artery Disease - diagnostic imaging | Adolescent | Vascular Calcification - epidemiology | Coronary Artery Disease - epidemiology | computed tomography | Obesity | Atherosclerosis | Computerized Tomography (CT) | coronary artery calcification | Epidemiology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2005, Volume 25, Issue 10, pp. 2106 - 2113
OBJECTIVE—Angiotensin (Ang) II-induced vascular damage may be partially mediated by reactive oxygen species generation and inflammation. Homozygous... 
Hypertension | Macrophages | Reactive oxygen species | KAPPA-B ACTIVATION | SPONTANEOUSLY HYPERTENSIVE-RATS | CHRONIC GRANULOMATOUS-DISEASE | CONVERTING-ENZYME-INHIBITION | BLOOD-PRESSURE | macrophages | NAD(P)H OXIDASE | reactive oxygen species | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SUPEROXIDE PRODUCTION | GROWTH-FACTOR | HEMATOLOGY | hypertension | SMOOTH-MUSCLE CELLS | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | Vasculitis - pathology | NADPH Oxidases - metabolism | Mesenteric Arteries - immunology | Mesenteric Arteries - pathology | Aorta - metabolism | Vasculitis - immunology | Mice, Mutant Strains | Inflammation Mediators - metabolism | Macrophage Colony-Stimulating Factor - genetics | Macrophages - immunology | Endothelium, Vascular - immunology | Inflammation Mediators - immunology | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Osteopetrosis - pathology | Vascular Resistance | Macrophages - pathology | Aorta - immunology | Osteopetrosis - metabolism | Macrophage Colony-Stimulating Factor - metabolism | Mesenteric Arteries - metabolism | Mice, Inbred Strains | Osteopetrosis - immunology | Aorta - pathology | Phenotype | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mice | Oxidative Stress - drug effects
Journal Article