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1. Full Text Molecular pathways: Oncologic pathways and their role in T-cell exclusion and immune evasion - A new role for the AXL receptor tyrosine kinase
by Aguilera, Todd A and Giaccia, Amato J
Clinical cancer research, ISSN 1078-0432, 06/2017, Volume 23, Issue 12, pp. 2928 - 2933
Life Sciences & Biomedicine | Oncology | Science & Technology | Humans | Antibodies, Monoclonal - therapeutic use | CTLA-4 Antigen - immunology | B7-H1 Antigen - immunology | Signal Transduction - immunology | Neoplasms - therapy | Neoplasms - immunology | T-Lymphocytes - drug effects | Immunotherapy | T-Lymphocytes - immunology | Programmed Cell Death 1 Receptor - immunology | Neoplasms - pathology | Antibodies, Monoclonal - immunology | PD-1 protein | Leukemia | Clinical trials | Lymphocytes T | Axl protein | CTLA-4 protein | Pathways | Protein-tyrosine kinase receptors | Protein-tyrosine kinase | Tyrosine | Medical research | NF-κB protein | Immune response | Kidneys | Myeloid leukemia | Stat3 protein | Inflammation | Radiation therapy | Chemotherapy | Immune checkpoint | PD-L1 protein | Antitumor activity | Solid tumors | Acute myeloid leukemia | PTEN protein | Clear cell-type renal cell carcinoma | Cancer | Tumors | Index Medicus
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2. Full Text ARE-mRNA degradation requires the 5′-3′ decay pathway
by Mayo, Thomas and Anderson, Paul and Stoecklin, Georg
EMBO reports, ISSN 1469-221X, 01/2006, Volume 7, Issue 1, pp. 72 - 77
AU‐rich element | Xrn1 | decapping | exosome | Lsm1 | Decapping | Exosome | AU-rich element | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins - metabolism | Cell Line | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Response Elements | Humans | Gene Expression Regulation | RNA Caps | Proto-Oncogene Proteins - genetics | RNA Stability | RNA, Messenger - metabolism | Animals | 3' Untranslated Regions | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Index Medicus | Scientific Report
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3. Full Text Wnt/β-catenin pathway mediates (−)-Epigallocatechin-3-gallate (EGCG) inhibition of lung cancer stem cells
by Zhu, Jianyun and Jiang, Ye and Yang, Xue and Wang, Shijia and Xie, Chunfeng and Li, Xiaoting and Li, Yuan and Chen, Yue and Wang, Xiaoqian and Meng, Yu and Zhu, Mingming and Wu, Rui and Huang, Cong and Ma, Xiao and Geng, Shanshan and Wu, Jieshu and Zhong, Caiyun
Biochemical and biophysical research communications, ISSN 0006-291X, 01/2017, Volume 482, Issue 1, pp. 15 - 21
Wnt/β-catenin pathway | Inhibition | Lung cancer stem cells | EGCG | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | A549 Cells | Cell Survival - drug effects | Lung Neoplasms - drug therapy | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Dose-Response Relationship, Drug | Wnt Signaling Pathway - drug effects | Neoplastic Stem Cells - metabolism | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Catechin - analogs & derivatives | Catechin - pharmacology | Index Medicus | STEM CELLS | INHIBITION | NEOPLASMS | 60 APPLIED LIFE SCIENCES | LUNGS
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4. Full Text XBP1 promotes triple-negative breast cancer by controlling the HIF1α pathway
by Chen, Xi and Iliopoulos, Dimitrios and Zhang, Qing and Tang, Qianzi and Greenblatt, Matthew B and Hatziapostolou, Maria and Lim, Elgene and Tam, Wai Leong and Ni, Min and Chen, Yiwen and Mai, Junhua and Shen, Haifa and Hu, Dorothy Z and Adoro, Stanley and Hu, Bella and Song, Minkyung and Tan, Chen and Landis, Melissa D and Ferrari, Mauro and Shin, Sandra J and Brown, Myles and Chang, Jenny C and Liu, X. Shirley and Glimcher, Laurie H
Nature (London), ISSN 0028-0836, 2014, Volume 508, Issue 1, pp. 103 - 107
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cell Proliferation | Prognosis | CD24 Antigen - metabolism | Humans | Transcription Factors - deficiency | Gene Expression Regulation, Neoplastic | RNA Polymerase II - metabolism | Gene Regulatory Networks | DNA-Binding Proteins - deficiency | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hyaluronan Receptors - metabolism | Triple Negative Breast Neoplasms - pathology | Female | Transcription, Genetic | Neoplasm Invasiveness | Gene Silencing | Neoplasm Recurrence, Local | Triple Negative Breast Neoplasms - blood supply | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Unfolded Protein Response | Regulatory Factor X Transcription Factors | Disease Progression | Transcription Factors - metabolism | Animals | Triple Negative Breast Neoplasms - genetics | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Mice | Cell Hypoxia - genetics | Index Medicus
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5. Full Text Hepatitis C Virus Infection Care Pathway—A Report From the American Gastroenterological Association Institute HCV Care Pathway Work Group
by Kanwal, Fasiha and Bacon, Bruce R and Beste, Lauren A and Brill, Joel V and Gifford, Allen L and Gordon, Stuart C and Horberg, Michael A and Manthey, Jacob G and Reau, Nancy and Rustgi, Vinod K and Younossi, Zobair M
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2017, Volume 152, Issue 6, pp. 1588 - 1598
Gastroenterology and Hepatology | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Predictive Value of Tests | Antiviral Agents - therapeutic use | Delivery of Health Care, Integrated | United States | Humans | Decision Support Techniques | Patient Care Team | Treatment Outcome | Hepatitis C, Chronic - diagnosis | Hepatitis C, Chronic - drug therapy | Critical Pathways | Algorithms | Societies, Medical | Antiviral Agents - adverse effects | Gastroenterology | Sustained Virologic Response | Health maintenance organizations | Medical colleges | Societies | Associations, institutions, etc | Hepatitis C virus | Health aspects | Index Medicus | Abridged Index Medicus
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6. Full Text The brain–gut pathway in functional gastrointestinal disorders is bidirectional: a 12-year prospective population-based study
by Koloski, N A and Jones, M and Kalantar, J and Weltman, M and Zaguirre, J and Talley, N J
Gut, ISSN 0017-5749, 09/2012, Volume 61, Issue 9, pp. 1284 - 1290
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Gastroenterology. Liver. Pancreas. Abdomen | Biological and medical sciences | Medical sciences | Prospective Studies | Follow-Up Studies | Brain - physiopathology | Dyspepsia - physiopathology | Humans | Middle Aged | Gastrointestinal Tract - physiopathology | Male | Data Collection | Irritable Bowel Syndrome - physiopathology | Case-Control Studies | Incidence | Depressive Disorder - physiopathology | Adult | Female | Australia | Longitudinal Studies | Anxiety Disorders - physiopathology | Psychological aspects | Research | Risk factors | Irritable bowel syndrome | Cytokines | Nervous system | Inflammation | Permeability | Abdomen | Studies | Hypotheses | Pain | Anxieties | Response rates | Questionnaires | Population | Index Medicus | Abridged Index Medicus
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7. Full Text Sulforaphane Ameliorates 3-Nitropropionic Acid-Induced Striatal Toxicity by Activating the Keap1-Nrf2-ARE Pathway and Inhibiting the MAPKs and NF-κB Pathways
by Jang, Minhee and Jang, Minhee and Cho, Ik-Hyun and Cho, Ik-Hyun
Molecular neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2619 - 2635
Nuclear factor-kappa B | Neurology | Neurosciences | Biomedicine | Sulforaphane | Neurobiology | 3-Nitropropionic acid | Huntington’s disease | Cell Biology | Nuclear factor erythroid 2-related factor 2 | Mitogen-activated protein kinases | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurons - pathology | Male | NF-kappa B - metabolism | Propionates - toxicity | RNA, Messenger - metabolism | Behavior, Animal | Inflammation Mediators - metabolism | Antioxidant Response Elements - genetics | Microglia - pathology | Cell Death - drug effects | Neurons - drug effects | Corpus Striatum - pathology | Isothiocyanates - pharmacology | Microglia - drug effects | Mice, Inbred C57BL | Nitro Compounds - toxicity | RNA, Messenger - genetics | Kelch-Like ECH-Associated Protein 1 - metabolism | Down-Regulation - drug effects | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Succinate Dehydrogenase - metabolism | Oxidative Stress - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
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8. Full Text The cytoprotective role of the Keap1–Nrf2 pathway
by Baird, Liam and Dinkova-Kostova, Albena T
Archives of toxicology, ISSN 0340-5761, 4/2011, Volume 85, Issue 4, pp. 241 - 272
Phase 2 inducer | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | Nrf2 | Keap1 | Cytoprotective enzymes | Pharmacology/Toxicology | Biomedicine general | Life Sciences & Biomedicine | Toxicology | Science & Technology | Biological and medical sciences | Medical sciences | Humans | Gene Expression Regulation | Oxidative Stress - genetics | Alcohol Oxidoreductases - metabolism | Aldehyde Reductase | Intracellular Signaling Peptides and Proteins - metabolism | Cytoprotection - physiology | NAD(P)H Dehydrogenase (Quinone) - genetics | Receptor Cross-Talk - physiology | Alcohol Oxidoreductases - genetics | Aldo-Keto Reductases | Animals | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | NAD(P)H Dehydrogenase (Quinone) - metabolism | NF-E2-Related Factor 2 - genetics | Response Elements - genetics | Adaptor Proteins, Signal Transducing - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Kelch-Like ECH-Associated Protein 1 | Disease Models, Animal | Enzymes | Cysteine | Security software | Analysis | Proteins | Models | Toxicity | Cells | Index Medicus
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