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Journal Article
Journal Article
Chemical Biology & Drug Design, ISSN 1747-0277, 06/2012, Volume 79, Issue 6, pp. 1007 - 1017
Two‐component signal transduction (TCST) is the predominant signaling scheme used in bacteria to sense and respond to environmental changes in order to survive... 
protein–protein interactions | docking | bacterial virulence | protein–ligand interactions | scoring | drug discovery | two‐component signal transduction systems | virtual screening | response regulators | structure‐based drug design | molecular recognition | gram‐negative pathogens | Gram-negative pathogens | Protein-ligand interactions | Structure-based drug design | Bacterial virulence | Molecular recognition | Virtual screening | Docking | Response regulators | Scoring | Drug discovery | Two-component signal transduction systems | Protein-protein interactions | protein-ligand interactions | SYSTEM | structure-based drug design | CHEMISTRY, MEDICINAL | gram-negative pathogens | PERMEABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | EMPIRICAL SCORING FUNCTIONS | DIMERIZATION | COLONIZATION | DISCOVERY | protein-protein interactions | SIGNAL-TRANSDUCTION | BINDING-AFFINITY | two-component signal transduction systems | Protein Structure, Tertiary | Salmonella enterica - drug effects | Bacterial Proteins - antagonists & inhibitors | Signal Transduction - drug effects | Computer Simulation | Bacterial Proteins - metabolism | Electrophoretic Mobility Shift Assay | DNA - metabolism | Salmonella enterica - metabolism | Binding Sites | Dimerization | Protein Interaction Mapping | Research Letters
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e103541
The two-component regulatory system PhoP/PhoQ has been shown to (i) control expression of virulence-associated traits, (ii) confer survival and growth within... 
SURVIVAL | IMMUNE-RESPONSE | INVASIN EXPRESSION | RESPONSE REGULATOR PHOP | OUTER PROTEINS | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | PESTIS | SYSTEMS | HOST | ENTEROCOLITICA | Yersinia pseudotuberculosis Infections - immunology | Mouth Diseases - immunology | Chemokines - blood | Spleen - immunology | Virulence | Female | Cytokines - blood | Disease Models, Animal | Adaptive Immunity | Cell Line | Cell Survival | Bacterial Proteins - genetics | Mouth Diseases - microbiology | Spleen - cytology | Macrophages - cytology | Immunity, Innate | Yersinia pseudotuberculosis Infections - mortality | Yersinia pseudotuberculosis - genetics | Macrophages - metabolism | Animals | Bacterial Proteins - metabolism | Mice | Mice, Inbred BALB C | Yersinia pseudotuberculosis Infections - pathology | Mutation | Mouth Diseases - pathology | Yersinia pseudotuberculosis - pathogenicity | Yersinia pseudotuberculosis - physiology | Infection | Bacteria | B cells | Virulence (Microbiology) | Health aspects | Animal models | Model testing | Pathogenesis | Environmental changes | Infections | Genomes | Biology | Kinases | Macrophages | Cell adhesion & migration | Impact analysis | Pseudotuberculosis | Signal transduction | Immune system | Pathogens | Antigens | Cell survival | Immune response | Immunomodulation | Neutrophils | Gene expression | Oral infection | Colonization
Journal Article
LANCET, ISSN 0140-6736, 04/2007, Volume 369, Issue 9568, pp. 1196 - 1207
Bubonic plague is an often fulminant systemic zoonosis, caused by Yersinia pestis. Conventional microbiology, bacterial population genetics, and genome... 
BUBONIC PLAGUE | MEDICINE, GENERAL & INTERNAL | COMPLETE GENOME SEQUENCE | CAENORHABDITIS-ELEGANS | RESPONSE REGULATOR PHOP | PLASMINOGEN-ACTIVATOR | V-ANTIGEN | YERSINIA-PESTIS INFECTION | EXPERIMENTAL PNEUMONIC PLAGUE | FLEA-BORNE PLAGUE | PASTEURELLA-PESTIS
Journal Article