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1. Full Text GDF-15 enhances intracellular Ca2+by increasing Cav1.3 expression in rat cerebellar granule neurons
by Lu, Jun-Mei and Wang, Chang-Ying and Hu, Changlong and Fang, Yan-Jia and Mei, Yan-Ai
Biochemical Journal, ISSN 0264-6021, 2016, Volume 473, Issue 13, pp. 1895 - 1904
GDF-15 (growth/differentiation factor 15) is a novel member of the TGF (transforming growth factor)-β superfamily that has critical roles in the central and... 
TβRII | Akt/mTOR | 1.3 | GDF-15 | Ca | ERK | Nifedipine - pharmacology | Calcium Channels - metabolism | TOR Serine-Threonine Kinases - metabolism | Calcium - metabolism | Cells, Cultured | Rats | Extracellular Signal-Regulated MAP Kinases - metabolism | Calcium Channel Blockers - pharmacology | Rats, Sprague-Dawley | Animals | Signal Transduction - drug effects | Cerebellum - cytology | Growth Differentiation Factor 15 - pharmacology | Neurons - metabolism | Neurons - drug effects | Oncogene Protein v-akt - metabolism | [Ca2+]i | mTOR | Akt | Cav1.3
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2. Full Text Notch activation on effector T cells increases their sensitivity to Treg cell‐mediated suppression through upregulation of TGF‐βRII expression
by Hue, Sophie and Kared, Hassen and Mehwish, Younas and Mouhamad, Shahul and Balbo, Michelle and Levy, Yves
European Journal of Immunology, ISSN 0014-2980, 07/2012, Volume 42, Issue 7, pp. 1796 - 1803
Notch proteins play an important role in embryonic development and cell‐fate decisions. Notch influences also the activation and differentiation of peripheral... 
Immune regulation | Regulatory T cells | TGF‐βRII | Notch signaling | TGF-βRII | DOMAIN | DELTA-4 | IMMUNOSUPPRESSION | DIFFERENTIATION | LIGAND | IMMUNOLOGY | TGF-ss RII | HEMATOPOIETIC PROGENITORS | Up-Regulation | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - immunology | Humans | Transcriptional Activation | Immunoblotting | RNA - genetics | T-Lymphocytes, Regulatory - immunology | Calcium-Binding Proteins - immunology | Serrate-Jagged Proteins | Leukocytes, Mononuclear - immunology | T-Lymphocytes, Regulatory - cytology | Jagged-1 Protein | Smad Proteins - immunology | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Receptor, Notch1 - immunology | Membrane Proteins - immunology | Smad Proteins - genetics | RNA - chemistry | Reverse Transcriptase Polymerase Chain Reaction | Basic Helix-Loop-Helix Transcription Factors - immunology | Leukocytes, Mononuclear - cytology | Protein-Serine-Threonine Kinases - immunology | Intercellular Signaling Peptides and Proteins - immunology | Life Sciences | Biochemistry, Molecular Biology
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3. Full Text Decreased expression of FcγRII in active Graves' disease patients
by Lu, Xixuan and Peng, Shiqiao and Wang, Xinyi and Shan, Zhongyan and Teng, Weiping
Journal of Clinical Laboratory Analysis, ISSN 0887-8013, 07/2019, Volume 33, Issue 6, pp. e22904 - n/a
Background Graves' disease (GD) is a common autoimmune disease characterized by genetic and environmental factors. Fcγ receptors (FcγRs) are involved in... 
FcγRII | autoimmunity | Graves' disease | CD32B | FcγR | THYROCYTES | IIB | MONOCYTES | IMMUNOGLOBULIN | MECHANISMS | Fc gamma RII | MEMORY B-CELLS | LUPUS | DYSREGULATION | Fc gamma R | INFECTION | MEDICAL LABORATORY TECHNOLOGY | COMPLEMENT RECEPTORS | Flow cytometry | Immunoglobulins | Immune response | Therapeutic applications | Leukocytes (mononuclear) | Environmental factors | CD14 antigen | Western blotting | Monocytes | CD16 antigen | Isotypes | Peripheral blood mononuclear cells | Autoimmune diseases
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4. Full Text Placental expression of microRNA-17 and -19b is down-regulated in early pregnancy loss
by Ventura, Walter and Koide, Keiko and Hori, Kyoko and Yotsumoto, Junko and Sekizawa, Akihiko and Saito, Hiroshi and Okai, Takashi
European Journal of Obstetrics & Gynecology and Reproductive Biology, ISSN 0301-2115, 2013, Volume 169, Issue 1, pp. 28 - 32
Abstract Objective First, to determine if microRNA-17 and -19b are expressed in villous samples at early stages of pregnancy. Second, to determine whether... 
Obstetrics and Gynecology | Villous sample | miRNA-19b | PTEN | CREB-1 | microRNA | Early pregnancy | TGF-β1 | TGFβ-RII | miRNA-17 | TGF beta-RII | TGF-beta 1 | MIR-17-92 | CELL-PROLIFERATION | MISCARRIAGE | CANCER | OBSTETRICS & GYNECOLOGY | INVASION | REPRODUCTIVE BIOLOGY | SPONTANEOUS-ABORTION | POLYCISTRON | DIFFERENTIATION | CLUSTER | Placenta - metabolism | Pregnancy | PTEN Phosphohydrolase - genetics | Abortion, Spontaneous - genetics | Down-Regulation | Humans | Adult | Female | MicroRNAs - biosynthesis | Placentation - genetics | RNA, Messenger - metabolism | Case-Control Studies | Transforming growth factors | Pregnant women | MicroRNA
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5. Full Text Expression of HPV16 E5 down-modulates the TGFbeta signaling pathway
by French, Deborah and Belleudi, Francesca and Mauro, Maria Vittoria and Mazzetta, Francesca and Raffa, Salvatore and Fabiano, Vincenza and Frega, Antonio and Torrisi, Maria Rosaria
Molecular Cancer, ISSN 1476-4598, 05/2013, Volume 12, Issue 1, pp. 38 - 38
Background: Infection with high-risk human papillomavirus (HR-HPV) genotypes, mainly HPV16 and HPV18, is a major risk factor for cervical cancer and... 
Human papillomavirus | SMAD | TGFβ signaling | HPV16 E5 | TGFβRII | ONCOPROTEIN | HOMEOSTASIS | PROTEIN | HUMAN-PAPILLOMAVIRUS TYPE-16 | TGF beta signaling | MESSENGER-RNA EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KERATINOCYTE CELL-LINE | CHROMOSOMAL INSTABILITY | TGF beta RII | ONCOLOGY | NEOPLASIA | GROWTH | INFECTION | Cervical Intraepithelial Neoplasia - pathology | Gene Expression | Signal Transduction | Humans | RNA, Messenger - genetics | Gene Expression Regulation, Neoplastic | Neoplasm Grading | Transforming Growth Factor beta - genetics | Time Factors | Cervical Intraepithelial Neoplasia - metabolism | Female | Oncogene Proteins, Viral - genetics | Cervical Intraepithelial Neoplasia - genetics | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Oncogene Proteins, Viral - metabolism | Viral proteins | RNA | Development and progression | Genetic aspects | Cellular signal transduction | Transforming growth factors | Papillomavirus infections | Health aspects | Risk factors | Cervical cancer | Proteins | Phosphorylation | Epidermal growth factor | Cytokines | Rodents | Homeostasis | Infections | Acquisitions & mergers | Cancer | Oncogene Proteins, Viral | Life Sciences | Smad Proteins | RNA, Messenger | Cervical Intraepithelial Neoplasia | Transforming Growth Factor beta
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6. MiRNA expression profile of vulvar squamous cell carcinoma and identification of the oncogenic role of miR-590-5p
by Yang, Xiuhua and Wu, Xin
Oncology Reports, ISSN 1021-335X, 01/2016, Volume 35, Issue 1, pp. 398 - 408
MicroRNAs (miRNAs), a class of small non-coding RNA molecules, are associated with a variety of human cancers. Currently, little data are available regarding... 
MiR-590-5p | Microarray | Vulvar squamous cell carcinoma | TGFβRII | MiRNA | CANCER CELLS | miR-590-5p | MICRORNAS | vulvar squamous cell carcinoma | TRENDS | microarray | INVASION | TGF beta RII | INHIBITION | ONCOLOGY | PATHWAY | GROWTH | GENES | miRNA | DIFFERENTIATION | DECREASED EXPRESSION | Up-Regulation | Receptors, Transforming Growth Factor beta - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Protein-Serine-Threonine Kinases - genetics | Gene Expression Profiling - methods | Smad Proteins - genetics | Neoplasm Metastasis | Vulvar Neoplasms - pathology | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Vulvar Neoplasms - genetics | Vulvar Neoplasms - metabolism | Aged, 80 and over | Cell Line, Tumor | Female | MicroRNAs - genetics | Protein-Serine-Threonine Kinases - metabolism
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7. Full Text GDF-15 enhances intracellular Ca2+ by increasing Ca(v)1.3 expression in rat cerebellar granule neurons
by Lu, JM and Wang, CY and Hu, C and Fang, YJ and Mei, YA
BIOCHEMICAL JOURNAL, ISSN 0264-6021, 07/2016, Volume 473, pp. 1895 - 1904
GDF-15 (growth/differentiation factor 15) is a novel member of the TGF (transforming growth factor)-beta superfamily that has critical roles in the central and... 
CELLS | SIGNALING PATHWAYS | PROTEIN | [Ca2+](i) | Ca(v)1.3 | T beta RII | BIOCHEMISTRY & MOLECULAR BIOLOGY | OUTWARD K+ CURRENT | TGF-BETA SUPERFAMILY | Akt/mTOR | GATED CALCIUM-CHANNELS | HIPPOCAMPAL-NEURONS | GDF-15 | INHIBITORY CYTOKINE-1 | IN-VIVO | NA+ CURRENT | ERK
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8. Full Text Protein kinase A induces UCP1 expression in specific adipose depots to increase energy expenditure and improve metabolic health
by Dickson, Lorna M and Gandhi, Shriya and Layden, Brian T and Cohen, Ronald N and Wicksteed, Barton
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 07/2016, Volume 311, Issue 1, pp. R79 - R88
Adipose tissue PKA has roles in adipogenesis, lipolysis, and mitochondrial function. PKA transduces the cAMP signal downstream of G protein-coupled receptors,... 
Protein kinase A | Energy expenditure | Adipose | Uncoupling protein 1 | PHYSIOLOGY | GLUCOSE CONTROL | BROWN ADIPOCYTES | TISSUE | RII-BETA-SUBUNIT | CELL-SIZE | adipose | ADULT HUMANS | energy expenditure | uncoupling protein 1 | CYCLIC-AMP | WHITE FAT | OBESITY | GENE-EXPRESSION | protein kinase A | Uncoupling Protein 1 - drug effects | Cyclic AMP-Dependent Protein Kinases - pharmacology | Mice, Inbred C57BL | Insulin Resistance | Adipose Tissue, White - metabolism | Glucose Intolerance | Uncoupling Protein 1 - biosynthesis | Adipose Tissue - metabolism | Insulin-Secreting Cells - metabolism | Animals | Cyclic AMP-Dependent Protein Kinases - genetics | Adiposity | Adiponectin - genetics | Diet, High-Fat | Adiponectin - metabolism | Adipose Tissue, Brown - metabolism | Mice | Energy Metabolism - physiology | Health Status | Weight Gain | Adipose tissues | Physiological aspects | Metabolism | Observations | Protein kinases | Obesity, Diabetes and Energy Homeostasis
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9. Full Text Increased pSmad2 expression and cytoplasmic predominant presence of TGF-βRII in breast cancer tissue are associated with poor prognosis: results from the Shanghai Breast Cancer Study
by Qiu, Qingchao and Qiu, Qingchao and Su, Yinghao and Su, Yinghao and Zheng, Ying and Zheng, Ying and Cai, Hui and Cai, Hui and Wu, Shenghui and Wu, Shenghui and Lu, Wei and Lu, Wei and Zheng, Wei and Zheng, Wei and Shu, Xiao Ou and Shu, Xiao Ou and Cai, Qiuyin and Cai, Qiuyin
Breast Cancer Research and Treatment, ISSN 0167-6806, 1/2015, Volume 149, Issue 2, pp. 467 - 477
Perturbations of transforming growth factor-beta (TGF-β) signaling are pivotal to tumorigenesis and tumor progression through their effects on cell... 
TGF-βRII | Prognosis | Medicine & Public Health | TGF-β signaling | pSmad2 | Oncology | Breast cancer | MOLECULAR CHARACTERIZATION | SMAD2 | CELLS | LOCALIZATION | RECOGNITION | PHOSPHORYLATION | MICROSATELLITE INSTABILITY | TGF-beta RII | INACTIVATION | TGF-beta signaling | ONCOLOGY | II RECEPTOR | KINASE RECEPTOR | Immunohistochemistry | Gene Expression | Receptors, Transforming Growth Factor beta - genetics | Humans | Middle Aged | Risk Factors | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Cytoplasm - metabolism | Tumor Burden | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Neoplasm Grading | Receptors, Transforming Growth Factor beta - metabolism | Breast Neoplasms - pathology | China | Smad2 Protein - genetics | Adult | Breast Neoplasms - mortality | Female | Aged | Neoplasm Staging | Protein-Serine-Threonine Kinases - metabolism
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10. Full Text Neutrophil CD16b crosslinking induces lipid raft-mediated activation of SHP-2 and affects cytokine expression and retarded neutrophil apoptosis
by Yang, H and Jiang, H and Song, Y and Chen, J.H and Chen, D.J and Shen, X.J
Experimental Cell Research, ISSN 0014-4827, 01/2018, Volume 362, Issue 1, pp. 121 - 131
Two different types of FcRs for IgG are constitutively expressed on the surface of human neutrophils, namely, FcγRIIA (CD32a) and FcγRIIIB (CD16b). Unlike... 
Shp-2 | CD16b | Neutrophil | CD32a | SIGNAL-TRANSDUCTION PATHWAYS | RESPIRATORY BURST | PHAGOCYTOSIS | CHEMOKINES | DISTINCT | INVOLVEMENT | RECEPTOR | TYROSINE PHOSPHATASES | CELL BIOLOGY | FC-GAMMA-RII | ONCOLOGY | KINASE ACTIVATION | Phosphorylation - physiology | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Cyclodextrins - metabolism | Cytokines - metabolism | Membrane Microdomains - metabolism | Humans | GPI-Linked Proteins - metabolism | Receptors, IgG - metabolism | Syk Kinase - metabolism | Adult | Signal Transduction - physiology | Apoptosis - physiology | Neutrophils - metabolism
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11. Full Text Simvastatin downregulates expression of TGF-βRII and inhibits proliferation of A549 cells via ERK
by Shang, Li and Jia, Shu-Shan and Jiang, Hai-Ming and Wang, Hua and Xu, Wen-Hua and Lv, Chang-Jun
Tumor Biology, ISSN 1010-4283, 6/2015, Volume 36, Issue 6, pp. 4819 - 4824
Lung cancer is the leading cause of cancer-related death worldwide. Transforming growth factor-β receptor II (TGF-βRII) plays an important role in the... 
Cancer Research | Biomedicine | TGF-βRII | A549 cells | Simvastatin | ERK | APOPTOSIS | ACTIVATION | TGF-beta RII | RECEPTOR | SUPPRESSION | STATINS | FAMILY | ONCOLOGY | PATHWAY | GROWTH | PROMOTES | Lung Neoplasms - genetics | Cell Survival - drug effects | Lung Neoplasms - drug therapy | Receptors, Transforming Growth Factor beta - genetics | Simvastatin - administration & dosage | Humans | Protein-Serine-Threonine Kinases - genetics | Lung Neoplasms - pathology | Sesquiterpenes - administration & dosage | Extracellular Signal-Regulated MAP Kinases - biosynthesis | Extracellular Signal-Regulated MAP Kinases - genetics | Protein-Serine-Threonine Kinases - biosynthesis | Receptors, Transforming Growth Factor beta - biosynthesis | Cell Line, Tumor | Cell Proliferation - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Polyisoprenyl Phosphates - administration & dosage
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12. Full Text Hydatid cyst fluid promotes peri-cystic fibrosis in cystic echinococcosis by suppressing miR-19 expression
by Zhang, Chuanshan and Wang, Limin and Ali, Tuergan and Li, Liang and Bi, Xiaojuan and Wang, Junhua and Lü, Guodong and Shao, Yingmei and Vuitton, Dominique A and Wen, Hao and Lin, Renyong
Parasites and Vectors, ISSN 1756-3305, 05/2016, Volume 9, Issue 1, p. 278
Background: Echinococcus granulosus infection causes cystic echinococcosis (CE); the generation of liver fibrosis around the parasitic larval cyst... 
HSC | miR-19 | TβRII | Liver fibrosis | Echinococcus granulosus | MECHANISM | T beta RII | MICRORNAS | TROPICAL MEDICINE | HEPATIC STELLATE CELLS | GROWTH | MICE | INFECTION | IN-VIVO TREATMENT | PROGRESSION | PARASITOLOGY | Liver - pathology | Humans | Middle Aged | Cells, Cultured | Cyst Fluid - physiology | Hepatic Stellate Cells - metabolism | Cystic Fibrosis - parasitology | Male | Gene Expression Regulation - drug effects | Young Adult | Animals | Echinococcus - physiology | Liver Cirrhosis - parasitology | MicroRNAs - drug effects | Adult | Female | Cell Proliferation - drug effects | MicroRNAs - genetics | Echinococcosis - parasitology | Hepatic Stellate Cells - drug effects | Complications and side effects | Liver diseases | MicroRNA | Fibrosis | Development and progression | Genetic aspects | Properties | Gene expression | Health aspects | Echinococcosis
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13. Full Text Mutual regulation of TGF-β1, TβRII and ErbB receptors expression in human thyroid carcinomas
by Mincione, Gabriella and Tarantelli, Chiara and Vianale, Giovina and Di Marcantonio, Maria Carmela and Cotellese, Roberto and Francomano, Franco and Di Nicola, Marta and Costantini, Erica and Cichella, Annadomenica and Muraro, Raffaella
Experimental Cell Research, ISSN 0014-4827, 09/2014, Volume 327, Issue 1, pp. 24 - 36
The role of EGF and TGF-β1 in thyroid cancer is still not clearly defined. TGF-β1 inhibited the cellular growth and migration of follicular (FTC-133) and... 
TGF-β1 | TβRII | ErbB receptors | Human thyroid carcinoma | EGF | Receptor, Epidermal Growth Factor - genetics | Receptors, Transforming Growth Factor beta - genetics | Cell Proliferation | Epidermal Growth Factor - genetics | Gene Expression - genetics | Humans | Middle Aged | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Male | Transforming Growth Factor beta1 - genetics | Cell Movement - genetics | Young Adult | Thyroid Neoplasms - genetics | Cell Line, Tumor | Adult | Carcinoma - genetics | Female | Aged | Cadherins - genetics | MESSENGER-RNA | PHENOTYPE | THYROID | CELL PROLIFERATION | ANIMAL TISSUES | RECEPTORS | 60 APPLIED LIFE SCIENCES | ADENOMAS | HEMATOXYLIN | PLANT GROWTH
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14. Full Text Vitamin D levels in multiple sclerosis patients: Association with TGF-β2, TGF-βRI, and TGF-βRII expression
by Shirvani-Farsani, Zeinab and Behmanesh, Mehrdad and Mohammadi, Seyed Mahdi and Naser Moghadasi, Abdorreza
Life Sciences, ISSN 0024-3205, 08/2015, Volume 134, Issue 1, pp. 63 - 67
A variety of evidence suggests that vitamin D can prevent the development of multiple sclerosis (MS). TGF-β pathway genes also play important roles in MS.... 
Multiple sclerosis | TGF-β2 | TGF-βRII | Vitamin D | TGF-βRI | Tgf-β2 tgf-βri tgf-βrii multiple sclerosis vitamin D | Humans | Middle Aged | Gene Expression Regulation | Male | Transforming Growth Factor beta2 - biosynthesis | Protein-Serine-Threonine Kinases - biosynthesis | RNA, Messenger - biosynthesis | Receptors, Transforming Growth Factor beta - biosynthesis | Multiple Sclerosis - pathology | Adult | Female | Vitamin D - metabolism | Real-Time Polymerase Chain Reaction | Multiple Sclerosis - metabolism
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15. Full Text Vitamin D levels in multiple sclerosis patients: Association with TGF-beta 2, TGF-beta RI, and TGF-beta RII expression
by Shirvani-Farsani, Z and Behmanesh, M and Mohammadi, SM and Moghadasi, AN
LIFE SCIENCES, ISSN 0024-3205, 08/2015, Volume 134, pp. 63 - 67
Aim: A variety of evidence suggests that vitamin D can prevent the development of multiple sclerosis (MS). TGF-beta pathway genes also play important roles in... 
SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | TGF-beta 2 | Multiple sclerosis | SIGNALING PATHWAYS | INCREASES | TGF-beta RII | DERMAL PAPILLA CELLS | TGF-beta RI | ELEMENT | Vitamin D | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | 1,25-DIHYDROXYVITAMIN D-3 | D-RECEPTOR | DIFFERENTIATION | INHIBITOR
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16. Full Text Uncoupling of Natural IgE Production and CD23 Surface Expression Levels
by Ren, Weicheng and Lagerstedt, Kristina and Grimsholm, Ola and Stern, Anna and Sun, Jia-Bin and Fang, Yu and Xiang, Zou and Mårtensson, Inga-Lill and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Institutionen för biomedicin, avdelningen för mikrobiologi och immunologi and Sahlgrenska akademin and Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning and Institute of Biomedicine, Department of Microbiology and Immunology and Institute of Medicine, Department of Rheumatology and Inflammation Research
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e62851
CD23, the low affinity receptor for immunoglobulin E (IgE), has been proposed to play a critical role in the regulation of IgE production, based on altered IgE... 
B-CELL DEVELOPMENT | GENETIC-VARIATION | MULTIDISCIPLINARY SCIENCES | MOUSE | LYMPHOCYTE RECEPTOR | MICE | 129/SVJ | C57BL/6 | Amyloid Precursor Protein Secretases - genetics | Spleen - immunology | Immunoglobulin E - biosynthesis | Open Reading Frames | Male | Mice, 129 Strain | RNA, Messenger - metabolism | Immunoglobulin E - blood | Female | Membrane Proteins - metabolism | B-Lymphocytes - metabolism | Membrane Proteins - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | ADAM10 Protein | Mice, Transgenic | Spleen - cytology | Receptors, IgE - genetics | Chromosomes, Mammalian | Mice, Knockout | ADAM Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Phenotype | Animals | B-Lymphocytes - immunology | Receptors, IgE - metabolism | Intracellular Space - metabolism | Mice | Mutation | ADAM Proteins - genetics | RNA | Analysis | Genes | Immunoglobulin E | Genetic aspects | B cells | Flow cytometry | Animal models | Immunoglobulins | Transgenic mice | Rheumatology | Inflammation | Gene expression | Blood | Immunology | CD23 antigen | Lymphocytes B | Rodents | Chromosome 8 | Lectins | Laboratory animals | Klinisk medicin | mice | mutagenesis | lymphocyte | Clinical Medicine | mouse | murine cd23 | immunoglobulin-e | receptor | hyper ige | fc-epsilon-rii | b-cell development | genetic-variation
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