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The Journal of Pathology, ISSN 0022-3417, 08/2015, Volume 236, Issue 4, pp. 445 - 456
Anaplastic large cell lymphoma (ALCL) is a rare, aggressive, non‐Hodgkin's lymphoma that is characterized by CD30 expression and disease onset in young... 
IL‐22 | IL‐21 | IL‐10 | ALK kinase | ALCL | cytokines | miR‐155 | miR-155 | IL-22 | IL-10 | IL-21 | NPM-ALK | TYROSINE KINASE | PATHOLOGY | IDENTIFICATION | MICRORNA-155 | PATHOGENESIS | ONCOLOGY | GENE-EXPRESSION | HODGKIN LYMPHOMA | LEUKEMIA | PROMOTER | TRANSGENIC MICE | Translocation, Genetic | Humans | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | Suppressor of Cytokine Signaling 1 Protein | MicroRNAs - metabolism | Case-Control Studies | Chromosomes, Human, Pair 2 | DNA Methylation | Transfection | Anaplastic Lymphoma Kinase | Lymphoma, Large-Cell, Anaplastic - metabolism | STAT3 Transcription Factor - metabolism | Lymphoma, Large-Cell, Anaplastic - genetics | Promoter Regions, Genetic | Argonaute Proteins - metabolism | Lymphoma, Large-Cell, Anaplastic - therapy | Receptor Protein-Tyrosine Kinases - deficiency | Argonaute Proteins - genetics | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Mice, SCID | CCAAT-Enhancer-Binding Protein-beta - metabolism | Chromosomes, Human, Pair 5 | Xenograft Model Antitumor Assays | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Line, Tumor | Mice, Inbred NOD | Lymphoma, Large-Cell, Anaplastic - pathology | MicroRNAs - genetics | Suppressor of Cytokine Signaling Proteins - metabolism | Genetic Therapy - methods | Original Papers
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2793 - 2806
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18671
We previously observed association between variants in the plasmacytoma variant translocation 1 gene (PVT1) and end-stage renal disease (ESRD) attributed to... 
TGF-BETA | HUMAN MESANGIAL CELLS | MESSENGER-RNA | PIMA-INDIANS | MULTIDISCIPLINARY SCIENCES | ANGIOTENSIN-II | HIGH-GLUCOSE | MICRORNAS | KIDNEY | MAMMALIAN-CELLS | STAGE RENAL-DISEASE | Extracellular Matrix - drug effects | Extracellular Matrix Proteins - genetics | Humans | RNA, Messenger - genetics | Transforming Growth Factor beta1 - metabolism | Extracellular Matrix - metabolism | Diabetic Nephropathies - genetics | Glucose - pharmacology | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | RNA, Messenger - metabolism | Gene Knockdown Techniques | Mesangial Cells - drug effects | Gene Expression Regulation - drug effects | Proteins - genetics | Proteins - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Models, Biological | RNA, Long Noncoding | Mesangial Cells - pathology | Plasminogen Activator Inhibitor 1 - genetics | Extracellular Matrix Proteins - metabolism | Type 2 diabetes | Fibronectins | Messenger RNA | Chronic kidney failure | Analysis | Genes | Diabetic nephropathies | Development and progression | Lymphomas | Plasmacytoma | Glucose | Dextrose | End-stage renal disease | Regulators | Genomics | Transforming growth factor-a | mRNA | Kinases | Fibronectin | Proteins | Rodents | Mesangial cells | Extracellular matrix | Collagen (type IV) | Enzyme-linked immunosorbent assay | Translocation | Transforming growth factor-b1 | RNA-mediated interference | Diabetes mellitus | Polyamide-imides | Gene expression | Ribonucleic acid--RNA | Molecular modelling | Nephropathy | Plasminogen activator inhibitors | Kidney diseases | Clear cell-type renal cell carcinoma | Kidney transplantation | RNA | Ribonucleic acid
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2005, Volume 7, Issue 7, pp. 665 - 674
Glyceraldehyde- 3- phosphate dehydrogenase ( GAPDH) influences cytotoxicity, translocating to the nucleus during apoptosis. Here we report a signalling pathway... 
OXIDATIVE-STRESS | ACTIVATION | PROTEIN | HISTONE H2B | GENE | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | SHORT-INTERFERING RNAS | NEURONAL NITRIC-OXIDE | CEREBRAL-ISCHEMIA | MAMMALIAN-CELLS | CELL BIOLOGY | Humans | Ubiquitin - metabolism | Cytoplasm - metabolism | Protein Transport - physiology | Protein Transport - drug effects | Nitric Oxide Synthase - genetics | S-Nitrosothiols - metabolism | Nitric Oxide Synthase Type II | N-Methylaspartate - pharmacology | Cell Nucleus - metabolism | Transfection | Nitric Oxide Synthase Type I | Protein Binding - drug effects | S-Nitrosoglutathione - pharmacology | Cysteine - metabolism | Neurons - metabolism | Neurons - drug effects | Nuclear Proteins - genetics | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - physiology | Cell Line | Cells, Cultured | Rats | Nuclear Proteins - metabolism | Nerve Tissue Proteins - genetics | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - genetics | Mice, Knockout | Ubiquitin-Protein Ligases | Two-Hybrid System Techniques | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Models, Biological | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Macrophages - drug effects | Signal Transduction - physiology | Mice | Apoptosis - physiology | Mutation | Nitric Oxide - metabolism | Microscopy, Fluorescence | Physiological aspects | Research | Oxidoreductases | Translocation (Genetics) | Apoptosis
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 3, pp. 780 - 789
Journal Article
Journal Article