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Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, pp. 6716 - 6716
Journal Article
Cancer Research, ISSN 0008-5472, 08/2011, Volume 71, Issue 15, pp. 5296 - 5306
The switch of tumor cells from an epithelial to a mesenchymal-like phenotype [designated as epithelial-to-mesenchymal transition (EMT)] is known to induce... 
BREAST-CARCINOMA | STEM-CELLS | IN-VITRO | ONCOLOGY | SERUM INTERLEUKIN-8 | PROSTATE-CANCER | METASTATIC PHENOTYPE | MALIGNANT-MELANOMA | TUMOR-GROWTH | EXPRESSION | PROGRESSION | Receptors, Interleukin-8 - genetics | T-Box Domain Proteins - physiology | Breast Neoplasms - secretion | Fibronectins - biosynthesis | Carcinoma - secretion | T-Box Domain Proteins - antagonists & inhibitors | Epithelial-Mesenchymal Transition - physiology | Humans | Neoplasm Proteins - physiology | T-Box Domain Proteins - biosynthesis | Culture Media, Conditioned - pharmacology | Culture Media, Serum-Free | Interleukin-8 - physiology | Neoplasm Proteins - antagonists & inhibitors | Bystander Effect | Tumor Microenvironment - physiology | Recombinant Fusion Proteins - antagonists & inhibitors | Cell Line, Tumor - pathology | Receptors, Interleukin-8 - biosynthesis | Cell Line, Tumor - secretion | Gene Expression Regulation, Neoplastic - drug effects | Intercellular Signaling Peptides and Proteins - secretion | Carcinoma - pathology | Neoplasm Proteins - genetics | Pancreatic Neoplasms - secretion | Promoter Regions, Genetic | Chemokines - secretion | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | Pancreatic Neoplasms - pathology | RNA, Small Interfering - pharmacology | Cytokines - secretion | Fetal Proteins - physiology | Fetal Proteins - antagonists & inhibitors | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Breast Neoplasms - pathology | Fetal Proteins - genetics | Fibronectins - genetics | Cell Movement | Index Medicus | Brachyury | tumor microenvironment | IL-8 | metastasis | EMT
Journal Article
Journal Article
Molecular Cancer, ISSN 1476-4598, 09/2011, Volume 10, Issue 1, pp. 117 - 117
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1049 - 1061
The TOR (target of rapamycin) kinase limits longevity by poorly understood mechanisms. Rapamycin suppresses the mammalian TORC1 complex, which regulates... 
EPITHELIAL-CELL LINE | GROWTH ARREST | DNA-DAMAGE | IN-VIVO | INFLAMMATORY CYTOKINE SECRETION | HUMAN FIBROBLASTS | TUMOR-CELLS | CANCER-THERAPY | REPLICATIVE SENESCENCE | P53 | CELL BIOLOGY | Up-Regulation | Fibroblasts - enzymology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Interleukin-1alpha - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Prostatic Neoplasms - secretion | Prostatic Neoplasms - immunology | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Prostatic Neoplasms - genetics | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Cellular Senescence | Inflammation Mediators - metabolism | Transcription, Genetic | Antineoplastic Agents - pharmacology | Interleukin-1alpha - genetics | Interleukin-6 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Anti-Inflammatory Agents - pharmacology | Interleukin-1alpha - secretion | Sirolimus - metabolism | Tumor Burden | Mice, SCID | Sirolimus - pharmacology | Xenograft Model Antitumor Assays | Phenotype | Animals | Fibroblasts - drug effects | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mitoxantrone - pharmacology | Interleukins | Aging | Genetic aspects | Properties | Phosphotransferases | Carcinogenesis | Health aspects | Cells | Index Medicus
Journal Article
Molecular and Cellular Proteomics, ISSN 1535-9476, 06/2013, Volume 12, Issue 6, pp. 1602 - 1620
Journal Article
Cancer Research, ISSN 0008-5472, 05/2006, Volume 66, Issue 9, pp. 4795 - 4801
The p53 protein responds to stress signals by regulating the transcription of a variety of genes. Some of these genes encode secreted proteins that may be... 
WILD-TYPE P53 | EMBRYO FIBROBLASTS | B-LYMPHOCYTES | ONCOLOGY | VESICLES | GENE-EXPRESSION | MULTIVESICULAR BODY | CELL-DERIVED EXOSOMES | PROTEOMIC ANALYSIS | CANCER | UNIRRADIATED CELLS | Biomarkers, Tumor - secretion | Oncogene Proteins - genetics | Apoptosis - radiation effects | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Cytoplasmic Vesicles - secretion | Oncogene Proteins - physiology | Carcinoma, Non-Small-Cell Lung - secretion | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Protein p53 - physiology | DNA Damage - physiology | Cytoplasmic Vesicles - metabolism | Oncogene Proteins - biosynthesis | Cyclophilin A - secretion | Secretory Rate - radiation effects | Genes, Tumor Suppressor | DNA-Binding Proteins - secretion | Lung Neoplasms - genetics | Phosphopyruvate Hydratase - secretion | Peptide Elongation Factor 1 - secretion | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Serpins - secretion | HSP90 Heat-Shock Proteins - secretion | Tumor Suppressor Protein p53 - deficiency | Mice, Knockout | Lung Neoplasms - secretion | Animals | Alleles | Cell Line, Tumor | Tumor Suppressor Proteins - secretion | Mice | Apoptosis - physiology | Cytoplasmic Vesicles - radiation effects | Phosphoglycerate Kinase - secretion | Index Medicus
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 02/2015, Volume 17, Issue 2, pp. 170 - 182
Journal Article