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1. Full Text Thresholds for thermal damage to normal tissues: An update
by Yarmolenko, Pavel S and Moon, Eui Jung and Landon, Chelsea and Manzoor, Ashley and Hochman, Daryl W and Viglianti, Benjamin L and Dewhirst, Mark W
International Journal of Hyperthermia, ISSN 0265-6736, 6/2011, Volume 27, Issue 4, pp. 320 - 343
The purpose of this review is to summarise a literature survey on thermal thresholds for tissue damage. This review covers published literature for the... 
thermal dosimetry | thermal damage threshold | normal tissue damage | thermal injury | hermal damage threshold | GUIDED FOCUSED ULTRASOUND | SCROTAL HEAT-STRESS | MONOPOLAR RADIOFREQUENCY | GERM-CELL APOPTOSIS | BIPOLAR RADIOFREQUENCY | WHOLE-BODY HYPERTHERMIA | ONCOLOGY | MR THERMOMETRY | IN-VIVO | MONKEYS MACACA-FASCICULARIS | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | RADIOFREQUENCY ABLATION | Hyperthermia, Induced - adverse effects | Humans | Eye Injuries | Male | Time | Blood-Brain Barrier - injuries | Brain Injuries - metabolism | Skin - injuries | Central Nervous System - injuries | Prostate - injuries | Brain - metabolism | Liver - injuries | Testosterone - metabolism | Fertility | Cell Death | Muscles - injuries | Sympathetic Nervous System - injuries | Spermatozoa - pathology | Brain Edema - etiology | Hot Temperature - adverse effects | Intestines - injuries | Cerebrovascular Circulation | Kidney - injuries | Regional Blood Flow | Testis - injuries | Animals | Testis - pathology | Brain - pathology | DNA Damage | Brain Injuries - etiology | Respiration | Dose-Response Relationship, Radiation | Urinary Bladder - injuries
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2. Full Text SIRT3 deacetylates ceramide synthases: Implications for mitochondrial dysfunction and brain injury
by Novgorodov, Sergei A and Riley, Christopher L and Keffler, Jarryd A and Yu, Jin and Kindy, Mark S and Macklin, Wendy B and Lombard, David B and Gudz, Tatyana I
Journal of Biological Chemistry, ISSN 0021-9258, 01/2016, Volume 291, Issue 4, pp. 1957 - 1973
Experimental evidence supports the role of mitochondrial ceramide accumulation as a cause of mitochondrial dysfunction and brain injury after stroke. Herein,... 
PERMEABILITY TRANSITION PORE | SPHINGOLIPID BIOSYNTHESIS | CYCLOPHILIN-D | REACTIVE OXYGEN | MANGANESE SUPEROXIDE-DISMUTASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | OXYGEN SPECIES GENERATION | SERINE PALMITOYLTRANSFERASE | RADIATION-INDUCED APOPTOSIS | CELL-DEATH | Mitochondria - enzymology | Sirtuin 3 - genetics | Ceramides - metabolism | Sirtuin 3 - metabolism | Oxidative Stress | Membrane Proteins - genetics | Humans | Mice, Inbred C57BL | Brain Injuries - enzymology | Male | Mitochondria - metabolism | Brain Injuries - genetics | Brain Injuries - metabolism | Brain Injuries - physiopathology | Mice, Knockout | Animals | Mitochondria - genetics | Sphingosine N-Acyltransferase - genetics | Membrane Proteins - metabolism | Mice | Apoptosis | Sphingosine N-Acyltransferase - metabolism | ceramide synthase | Lipids | sirtuin | brain | sphingolipid | mitochondria
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3. Full Text A combination of Podophyllotoxin and Rutin attenuates radiation induced gastrointestinal injury by negatively regulating NF-κB/p53 signaling in lethally irradiated mice
by Kalita, Bhargab and Ranjan, Rajiv and Singh, Abhinav and Yashavarddhan, M.H and Bajaj, Sania and Gupta, Manju Lata
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0168525 - e0168525
Development of an effective radio protector to minimise radiation-inflicted damages have largely failed owing to inherent toxicity of most of the agents... 
ACTIVE PRINCIPLES | IN-VITRO | ACTIVATION | PROTECTION | HEXANDRUM | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | MOUSE | KAPPA-B | IONIZING-RADIATION | DAMAGE | Radiation-Protective Agents | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Podophyllotoxin - pharmacology | Radiation Injuries - drug therapy | Reactive Oxygen Species - metabolism | Rutin - pharmacology | Lipopolysaccharides - metabolism | Gastrointestinal Tract - radiation effects | Male | NF-kappa B - metabolism | Interleukin-6 - metabolism | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Gastrointestinal Tract - drug effects | Dinoprostone - metabolism | Drug Therapy, Combination - methods | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | Mice | Nitric Oxide - metabolism | Radiation Injuries - metabolism | Nitric Oxide Synthase Type II - metabolism | Mucosa | p53 Protein | Radiation | Cancer therapies | Prostaglandin endoperoxide synthase | Bone marrow | Growth factors | Medical research | NF-κB protein | Radiation effects | Cytokines | Cell division | Exposure | Nitric-oxide synthase | Inflammatory bowel disease | Disease prevention | Oxygenase | Tumor necrosis factor | Nitric oxide | Crypts | Stem cells | Leukocytes (granulocytic) | Radiation damage | Nuclear medicine | Podophyllotoxin | Toxicity | Interleukin | Irradiated | Prostaglandin E2 | Interleukin 6 | Pathways | Granulocyte colony-stimulating factor | Intestine | Rodents | Radiation injuries | Colony-stimulating factor | Injuries | Translocation | Inflammation | Radiation therapy | Rutin | Nuclear transport | Hemopoiesis | Signaling | Cell death | Fatalities | Cyclooxygenase-2 | Apoptosis | Index Medicus
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4. Full Text Delayed increase of astrocytic aquaporin 4 after juvenile traumatic brain injury: Possible role in edema resolution?
by Fukuda, A.M and Pop, V and Spagnoli, D and Ashwal, S and Obenaus, A and Badaut, J
Neuroscience, ISSN 0306-4522, 2012, Volume 222, pp. 366 - 378
Highlights â–º Diffuse edema formation in a rodent model of juvenile traumatic brain injury. â–º Astrocytic AQP4 expression parallels the edema resolution after... 
Neurology | juvenile traumatic brain injury | edema | aquaporin | astrocyte | Edema | Juvenile traumatic brain injury | Astrocyte | Aquaporin | AQP4 EXPRESSION | HEAD-INJURY | ALZHEIMERS-DISEASE | WATER CHANNELS | MODEL | NEUROSCIENCES | CHILDREN | PAIN | MICE | RAT-BRAIN | CEREBRAL-ISCHEMIA | Immunohistochemistry | Brain Edema - metabolism | Rats | Aquaporins - metabolism | Male | Brain Injuries - metabolism | Brain Injuries - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Rats, Sprague-Dawley | Aquaporin 1 - metabolism | Blotting, Western | Magnetic Resonance Imaging | Animals | Brain Edema - physiopathology | Image Processing, Computer-Assisted | Aquaporin 4 - metabolism | Astrocytes - metabolism | Phosphates | Brain | Dropsy | Neurosciences | Nuclear radiation | Neurons | Aquaporins | Albumin | Injuries
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5. Full Text Extracellular ATP enhances radiation-induced brain injury through microglial activation and paracrine signaling via P2X7 receptor
by Xu, Pengfei and Xu, Yongteng and Hu, Bin and Wang, Jue and Pan, Rui and Murugan, Madhuvika and Wu, Long-Jun and Tang, Yamei
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 50, pp. 87 - 100
Highlights • The ATP level in CSF is related to the severity of RBI. • ATP enhances microglial activity and amplifies inflammatory response. • Blockade of... 
Psychiatry | Allergy and Immunology | COX-2 | TNF-α | BBG | Radiation-induced brain injury | Inflammation | Extracellular ATP | P2X7R | IL-6 | Microglia | RHEUMATOID-ARTHRITIS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ANTAGONIST | ADULT HIPPOCAMPAL NEUROGENESIS | IMMUNOLOGY | TNE-alpha | NEUROSCIENCES | INTRACRANIAL VOLUME | IMPROVES RECOVERY | P2X RECEPTOR | DISEASE | SPINAL-CORD-INJURY | EXPRESSION | Microglia - metabolism | Humans | Male | Radiotherapy - adverse effects | Brain Injuries - metabolism | Microglia - radiation effects | Brain - metabolism | Radiation Injuries - cerebrospinal fluid | Inflammation - metabolism | Female | Signal Transduction - radiation effects | Adenosine Triphosphate - adverse effects | Microglia - drug effects | Paracrine Communication | Cells, Cultured | Adenosine Triphosphate - cerebrospinal fluid | Brain - radiation effects | Radiation Injuries - complications | Brain - drug effects | Animals | Brain Injuries - cerebrospinal fluid | Signal Transduction - drug effects | Receptors, Purinergic P2X7 - metabolism | Mice | Brain Injuries - etiology | Radiation Injuries - metabolism | Brain | Injuries | Radiation | COX-2 inhibitors | Interleukins | Neurons | Analysis | Brain damage | Radiotherapy
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6. Full Text Mesenchymal Stem Cell Therapy Protects Lungs from Radiation-Induced Endothelial Cell Loss by Restoring Superoxide Dismutase 1 Expression
by Klein, Diana and Steens, Jennifer and Wiesemann, Alina and Schulz, Florian and Kaschani, Farnusch and Röck, Katharina and Yamaguchi, Masahiro and Wirsdörfer, Florian and Kaiser, Markus and Fischer, Jens W and Stuschke, Martin and Jendrossek, Verena
Antioxidants & Redox Signaling, ISSN 1523-0864, 04/2017, Volume 26, Issue 11, pp. 563 - 582
Aims: Radiation-induced normal tissue toxicity is closely linked to endothelial cell (EC) damage and dysfunction (acute effects). However, the underlying... 
Original Research Communications | radioprotection | mesenchymal stem cell therapy | vascular dysfunction | Radiotherapy | TOTAL-BODY IRRADIATION | PRO-INFLAMMATORY CYTOKINES | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | TISSUE | INJURY | MECHANISMS | ENDOCRINOLOGY & METABOLISM | radiotherapy | IONIZING-RADIATION | UP-REGULATION | PULMONARY-FIBROSIS | Salicylates - pharmacology | Endothelial Cells - ultrastructure | Mesenchymal Stromal Cells - cytology | Radiation Injuries - genetics | Superoxide Dismutase-1 - metabolism | Lung - radiation effects | Lung - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Radiation Injuries - therapy | Gene Expression | Lung - pathology | Endothelial Cells - metabolism | Endothelial Cells - radiation effects | Phenotype | Animals | Fibroblasts - radiation effects | Fibrosis | Superoxide Dismutase-1 - genetics | Radiation Injuries - pathology | Mice | Endothelial Cells - pathology | Radiation Injuries - metabolism | Radiation Injuries, Experimental | Mesenchymal Stem Cell Transplantation | Organometallic Compounds - pharmacology | Superoxide dismutase | Research | Lungs | Gene expression | Health aspects | Cellular therapy | Cell culture | Therapy | Mesenchyme | Toxicity | Radiation | Innovations | Stem cell transplantation | Paracrine signalling | Adoptive transfer | Antioxidants | Reduction | Bone marrow | Radiation injuries | Aorta | Acute effects | Radiation effects | Thorax | Radioprotection | Endothelial cells | Stem cells | Irradiation | Viability | Radiation damage | Index Medicus
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7. Full Text Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury
by Chandrakesan, Parthasarathy and May, Randal and Weygant, Nathaniel and Qu, Dongfeng and Berry, William L and Sureban, Sripathi M and Ali, Naushad and Rao, Chinthalapally and Huycke, Mark and Bronze, Michael S and Houchen, Courtney W
Scientific Reports, ISSN 2045-2322, 11/2016, Volume 6, Issue 1, p. 37667
Crypt epithelial survival and regeneration after injury require highly coordinated complex interplay between resident stem cells and diverse cell types. The... 
BARRIER DYSFUNCTION | APOPTOSIS | REPAIR | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | KINASE | SELF-RENEWAL | MICE | GROWTH-FACTOR | BACTERIAL-INFECTION | KAPPA-B ACTIVITY | Protein-Serine-Threonine Kinases - deficiency | Whole-Body Irradiation | Epithelial Cells - metabolism | Ataxia Telangiectasia Mutated Proteins - metabolism | Apoptosis - radiation effects | Enterocytes - metabolism | Cell Membrane Permeability - radiation effects | Stem Cells - metabolism | Integrases - metabolism | Microfilament Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | Intestines - pathology | Signal Transduction | Cell Survival - radiation effects | Mice, Knockout | Dinoprostone - metabolism | Enterocytes - radiation effects | Phosphorylation - radiation effects | Animals | Cyclooxygenase 2 - metabolism | Survival Analysis | Radiation Injuries - pathology | DNA Damage | Radiation Injuries - metabolism | Cell Proliferation - radiation effects | Signal transduction | Restitution | DNA damage | Deoxyribonucleic acid--DNA | Stem cells
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8. Full Text Light-Induced Rescue of Breathing after Spinal Cord Injury
by Alilain, Warren J and Li, Xiang and Horn, Kevin P and Dhingra, Rishi and Dick, Thomas E and Herlitze, Stefan and Silver, Jerry
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 11862 - 11870
Paralysis is a major consequence of spinal cord injury (SCI). After cervical SCI, respiratory deficits can result through interruption of descending... 
Kindling | Nmda receptor | Plasticity | Motor neuron | Paralysis | Respiration | Spinal cord injury | spinal cord injury | FUNCTIONAL RECOVERY | CELLS | RESPIRATORY RHYTHM | NMDA RECEPTORS | paralysis | plasticity | respiration | SILENT SYNAPSES | SYNAPTIC PLASTICITY | NEURAL CIRCUITRY | NEUROSCIENCES | INTERMITTENT HYPOXIA | PHRENIC MOTONEURONS | motor neuron | OPTICAL INDUCTION | NMDA receptor | kindling | Anterior Horn Cells - metabolism | Spinal Cord - metabolism | Anterior Horn Cells - radiation effects | Receptors, N-Methyl-D-Aspartate - metabolism | Respiratory Insufficiency - etiology | Spinal Cord - radiation effects | Anterior Horn Cells - physiopathology | Paralysis - metabolism | Rhodopsin - metabolism | Rhodopsin - radiation effects | Phototherapy - methods | Neuronal Plasticity - physiology | Neural Pathways - radiation effects | Female | Diaphragm - innervation | Neural Pathways - physiopathology | Spinal Cord Injuries - therapy | Disease Models, Animal | Respiratory Insufficiency - physiopathology | Neuronal Plasticity - radiation effects | Spinal Cord Injuries - metabolism | Periodicity | Rats | Respiration - radiation effects | Treatment Outcome | Respiratory Insufficiency - therapy | Diaphragm - physiology | Rats, Sprague-Dawley | Paralysis - therapy | Animals | Paralysis - physiopathology | Neural Pathways - metabolism | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology
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9. Full Text Comparison of therapeutic effects between pulsed and continuous wave 810-nm wavelength laser irradiation for traumatic brain injury in mice
by Ando, Takahiro and Xuan, Weijun and Xu, Tao and Dai, Tianhong and Sharma, Sulbha K and Kharkwal, Gitika B and Huang, Ying-Ying and Wu, Qiuhe and Whalen, Michael J and Sato, Shunichi and Obara, Minoru and Hamblin, Michael R
PLoS ONE, ISSN 1932-6203, 10/2011, Volume 6, Issue 10, p. e26212
Background and Objective: Transcranial low-level laser therapy (LLLT) using near-infrared light can efficiently penetrate through the scalp and skull and could... 
CLOSED-HEAD INJURY | FUNCTIONAL RECOVERY | PROMOTES REGENERATION | MEMORY PERFORMANCE | CONTROLLED CORTICAL IMPACT | HELIUM-NEON LASER | MULTIDISCIPLINARY SCIENCES | SPINAL-CORD-INJURY | TERM NEUROLOGICAL DEFICITS | ISCHEMIC-STROKE | LEVEL LIGHT THERAPY | Brain - physiopathology | Male | Treatment Outcome | Brain Injuries - metabolism | Brain Injuries - physiopathology | Brain - metabolism | Animals | Depression - surgery | Adenosine Triphosphate - metabolism | Brain - pathology | Brain Injuries - surgery | Depression - complications | Laser Therapy - methods | Mice | Mice, Inbred BALB C | Brain Injuries - pathology | Brain | Antidepressants | Lasers in surgery | Analysis | Brain damage | Injuries | Lasers in medicine | Head | Traumatic brain injury | Body weight | Cortex | I.R. radiation | Continuous radiation | Wavelength | Studies | Head injuries | Lasers | Scalp | Rodents | Irradiation | Infrared radiation
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10. Full Text Dynamic O-(2-18F-fluoroethyl)-L-tyrosine positron emission tomography differentiates brain metastasis recurrence from radiation injury after radiotherapy
by Ceccon, Garry and Lohmann, Philipp and Stoffels, Gabriele and Judov, Natalie and Filss, Christian P and Rapp, Marion and Bauer, Elena and Hamisch, Christina and Ruge, Maximilian I and Kocher, Martin and Kuchelmeister, Klaus and Sellhaus, Bernd and Sabel, Michael and Fink, Gereon R and Shah, Nadim J and Langen, Karl-Josef and Galldiks, Norbert
Neuro-oncology, ISSN 1522-8517, 02/2017, Volume 19, Issue 2, pp. 281 - 288
BACKGROUND: The aim of this study was to investigate the potential of dynamic O-(2-[(18)F]fluoroethyl)-L-tyrosine ((18)F-FET) PET for differentiating local... 
radiation-induced changes | DIAGNOSIS | TUMOR RECURRENCE | radionecrosis | IMAGING CHANGES | kinetic analysis | O-(2-F-18-FLUOROETHYL)-L-TYROSINE PET | stereotactic radiosurgery | UPTAKE KINETICS | F-18-FET PET | NECROSIS | FET PET | LOW-GRADE GLIOMA | Stereotactic radiosurgery | Kinetic analysis | Radiation-induced changes | Radionecrosis | CLINICAL NEUROLOGY | ONCOLOGY | Prognosis | Follow-Up Studies | Brain Neoplasms - diagnostic imaging | Humans | Middle Aged | Radiation Injuries - diagnostic imaging | Male | Radiotherapy - adverse effects | Tyrosine - analogs & derivatives | Brain Neoplasms - metabolism | Neoplasm Recurrence, Local - pathology | Young Adult | Radiopharmaceuticals - metabolism | Brain Neoplasms - secondary | Neoplasm Recurrence, Local - diagnostic imaging | Adult | Female | Diagnosis, Differential | Neoplasm Recurrence, Local - metabolism | Positron-Emission Tomography - methods | Disease Progression | Radiation Injuries - etiology | Tyrosine - metabolism | Adolescent | Aged | Neoplasm Staging | Radiation Injuries - metabolism | Clinical Investigation
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