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International Journal of Hyperthermia, ISSN 0265-6736, 6/2011, Volume 27, Issue 4, pp. 320 - 343
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0168525 - e0168525
Development of an effective radio protector to minimise radiation-inflicted damages have largely failed owing to inherent toxicity of most of the agents... 
ACTIVE PRINCIPLES | IN-VITRO | ACTIVATION | PROTECTION | HEXANDRUM | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | MOUSE | KAPPA-B | IONIZING-RADIATION | DAMAGE | Radiation-Protective Agents | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Podophyllotoxin - pharmacology | Radiation Injuries - drug therapy | Reactive Oxygen Species - metabolism | Rutin - pharmacology | Lipopolysaccharides - metabolism | Gastrointestinal Tract - radiation effects | Male | NF-kappa B - metabolism | Interleukin-6 - metabolism | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Gastrointestinal Tract - drug effects | Dinoprostone - metabolism | Drug Therapy, Combination - methods | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | Mice | Nitric Oxide - metabolism | Radiation Injuries - metabolism | Nitric Oxide Synthase Type II - metabolism | Mucosa | p53 Protein | Radiation | Cancer therapies | Prostaglandin endoperoxide synthase | Bone marrow | Growth factors | Medical research | NF-κB protein | Radiation effects | Cytokines | Cell division | Exposure | Nitric-oxide synthase | Inflammatory bowel disease | Disease prevention | Oxygenase | Tumor necrosis factor | Nitric oxide | Crypts | Stem cells | Leukocytes (granulocytic) | Radiation damage | Nuclear medicine | Podophyllotoxin | Toxicity | Interleukin | Irradiated | Prostaglandin E2 | Interleukin 6 | Pathways | Granulocyte colony-stimulating factor | Intestine | Rodents | Radiation injuries | Colony-stimulating factor | Injuries | Translocation | Inflammation | Radiation therapy | Rutin | Nuclear transport | Hemopoiesis | Signaling | Cell death | Fatalities | Cyclooxygenase-2 | Apoptosis | Index Medicus
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 50, pp. 87 - 100
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 04/2017, Volume 26, Issue 11, pp. 563 - 582
Aims: Radiation-induced normal tissue toxicity is closely linked to endothelial cell (EC) damage and dysfunction (acute effects). However, the underlying... 
Original Research Communications | radioprotection | mesenchymal stem cell therapy | vascular dysfunction | Radiotherapy | TOTAL-BODY IRRADIATION | PRO-INFLAMMATORY CYTOKINES | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | TISSUE | INJURY | MECHANISMS | ENDOCRINOLOGY & METABOLISM | radiotherapy | IONIZING-RADIATION | UP-REGULATION | PULMONARY-FIBROSIS | Salicylates - pharmacology | Endothelial Cells - ultrastructure | Mesenchymal Stromal Cells - cytology | Radiation Injuries - genetics | Superoxide Dismutase-1 - metabolism | Lung - radiation effects | Lung - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Radiation Injuries - therapy | Gene Expression | Lung - pathology | Endothelial Cells - metabolism | Endothelial Cells - radiation effects | Phenotype | Animals | Fibroblasts - radiation effects | Fibrosis | Superoxide Dismutase-1 - genetics | Radiation Injuries - pathology | Mice | Endothelial Cells - pathology | Radiation Injuries - metabolism | Radiation Injuries, Experimental | Mesenchymal Stem Cell Transplantation | Organometallic Compounds - pharmacology | Superoxide dismutase | Research | Lungs | Gene expression | Health aspects | Cellular therapy | Cell culture | Therapy | Mesenchyme | Toxicity | Radiation | Innovations | Stem cell transplantation | Paracrine signalling | Adoptive transfer | Antioxidants | Reduction | Bone marrow | Radiation injuries | Aorta | Acute effects | Radiation effects | Thorax | Radioprotection | Endothelial cells | Stem cells | Irradiation | Viability | Radiation damage | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 11/2016, Volume 6, Issue 1, p. 37667
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 11862 - 11870
Paralysis is a major consequence of spinal cord injury (SCI). After cervical SCI, respiratory deficits can result through interruption of descending... 
Kindling | Nmda receptor | Plasticity | Motor neuron | Paralysis | Respiration | Spinal cord injury | spinal cord injury | FUNCTIONAL RECOVERY | CELLS | RESPIRATORY RHYTHM | NMDA RECEPTORS | paralysis | plasticity | respiration | SILENT SYNAPSES | SYNAPTIC PLASTICITY | NEURAL CIRCUITRY | NEUROSCIENCES | INTERMITTENT HYPOXIA | PHRENIC MOTONEURONS | motor neuron | OPTICAL INDUCTION | NMDA receptor | kindling | Anterior Horn Cells - metabolism | Spinal Cord - metabolism | Anterior Horn Cells - radiation effects | Receptors, N-Methyl-D-Aspartate - metabolism | Respiratory Insufficiency - etiology | Spinal Cord - radiation effects | Anterior Horn Cells - physiopathology | Paralysis - metabolism | Rhodopsin - metabolism | Rhodopsin - radiation effects | Phototherapy - methods | Neuronal Plasticity - physiology | Neural Pathways - radiation effects | Female | Diaphragm - innervation | Neural Pathways - physiopathology | Spinal Cord Injuries - therapy | Disease Models, Animal | Respiratory Insufficiency - physiopathology | Neuronal Plasticity - radiation effects | Spinal Cord Injuries - metabolism | Periodicity | Rats | Respiration - radiation effects | Treatment Outcome | Respiratory Insufficiency - therapy | Diaphragm - physiology | Rats, Sprague-Dawley | Paralysis - therapy | Animals | Paralysis - physiopathology | Neural Pathways - metabolism | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology
Journal Article
Journal Article
Neuro-oncology, ISSN 1522-8517, 02/2017, Volume 19, Issue 2, pp. 281 - 288
Journal Article