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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
We previously reported that endoplasmic reticulum (ER) stress is induced in the subfornical organ (SFO) and the hypothalamic paraventricular nucleus (PVN) of... 
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | heart failure | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | INDUCED PHOSPHORYLATION | MYOCARDIAL-INFARCTION | ER STRESS | subfornical organ | KINASE | RATS | sympathetic activity | KAPPA-B | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus | mitogen-activated protein kinase | UNFOLDED PROTEIN RESPONSE | PERIPHERAL VASCULAR DISEASE | UP-REGULATION | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Cardiovascular Neurohormonal Regulation
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2013, Volume 33, Issue 9, pp. 3953 - 3966
Down syndrome (DS) is associated with neurological complications, including cognitive deficits that lead to impairment in intellectual functioning. Increased... 
Protein Binding - genetics | Electric Stimulation | Cell Count | Male | GABA Modulators - therapeutic use | Learning Disorders - etiology | Hyperkinesis - drug therapy | Neurogenesis - genetics | Long-Term Potentiation - drug effects | Protein Binding - drug effects | Exploratory Behavior - drug effects | Reflex, Startle - drug effects | Imidazoles - therapeutic use | Excitatory Postsynaptic Potentials - genetics | Disease Models, Animal | Mice, Transgenic | Hippocampus - pathology | Imidazoles - pharmacology | Psychomotor Performance - drug effects | Hyperkinesis - etiology | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Analysis of Variance | Seizures - etiology | Glutamate Decarboxylase - metabolism | Mice | Receptors, GABA-A - metabolism | Benzodiazepines - therapeutic use | Tritium - pharmacokinetics | Sensory Gating - drug effects | Reflex - genetics | Reflex - drug effects | Hippocampus - drug effects | Excitatory Postsynaptic Potentials - drug effects | Ki-67 Antigen | Reaction Time - drug effects | GABA Modulators - pharmacology | Neurons - physiology | Down Syndrome - complications | Membrane Proteins - metabolism | Neurogenesis - drug effects | Neurons - drug effects | Cues | Down Syndrome - pathology | Benzodiazepines - pharmacology | Acoustic Stimulation | Mice, Inbred C57BL | Learning Disorders - drug therapy | Long-Term Potentiation - genetics | Biophysics | Maze Learning - drug effects | Rotarod Performance Test | Animals | Carrier Proteins - metabolism | Down Syndrome - drug therapy | Cell Proliferation - drug effects
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 04/2012, Volume 8, Issue 4, pp. 1440 - 1449
.... However, few in vitro studies have been performed to identify the effects of environmental elasticity on the differentiation of MSC into vascular cell types... 
Mesenchymal stem cell | Elasticity | Vascular differentiation | Nanofiber | 3-D matrix | MATERIALS SCIENCE, BIOMATERIALS | STIFFNESS | ENGINEERING, BIOMEDICAL | EXTRACELLULAR-MATRIX | SCAFFOLD | Cross-Linking Reagents - pharmacology | Up-Regulation - radiation effects | Tensile Strength - drug effects | Elastic Modulus - drug effects | Polyethylene Glycols - chemistry | Methacrylates - chemistry | Spectroscopy, Fourier Transform Infrared | Tissue Scaffolds - chemistry | Polymerization - drug effects | Mesenchymal Stromal Cells - cytology | Ultraviolet Rays | Time Factors | Polymerase Chain Reaction | Compressive Strength - drug effects | Compressive Strength - radiation effects | Hydrogel, Polyethylene Glycol Dimethacrylate - pharmacology | Porosity - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Cell Differentiation - radiation effects | Nanofibers - chemistry | Mesenchymal Stromal Cells - drug effects | Nanofibers - ultrastructure | Polymerization - radiation effects | Endothelial Cells - metabolism | Mesenchymal Stromal Cells - metabolism | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Elasticity - drug effects | Endothelial Cells - radiation effects | Materials Testing | Elasticity - radiation effects | Muscle, Smooth, Vascular - cytology | Tensile Strength - radiation effects | Porosity - radiation effects | Elastic Modulus - radiation effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Cell Differentiation - drug effects | Endothelial Cells - cytology | Gene Expression Regulation - radiation effects | Endothelial Cells - drug effects | Actin | Polyethylene glycol | Stem cells | Smooth muscle | Muscle proteins | Polyols | Nanotechnology | Endothelium | mesenchymal stem cell | vascular differentiation | 3D matrix | nanofiber
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 09/2016, Volume 5, Issue 9, p. n/a
...‐density lipoprotein (oxLDL) promotes immune activation and inflammation. We studied the effects of statins... 
atherosclerosis | dendritic cells | statin | immune system | microRNA | oxidized low‐density lipoprotein | T cells | MicroRNA | Dendritic cells | Oxidized low-density lipoprotein | Atherosclerosis | Statin | Immune system | CARDIAC & CARDIOVASCULAR SYSTEMS | METAANALYSIS | INDUCTION | CANCER | ARTERIOSCLEROSIS | HEAT-SHOCK-PROTEIN | THERAPY | INHIBITION | INFLAMMATION | oxidized low-density lipoprotein | ASSOCIATION | LYMPHOCYTES | Interleukin-1beta - drug effects | Extracellular Signal-Regulated MAP Kinases - drug effects | Atorvastatin Calcium - pharmacology | Dendritic Cells - immunology | Humans | Interferon-gamma - drug effects | Interleukin-17 - immunology | Chaperonin 60 - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Simvastatin - pharmacology | Th1 Cells - immunology | Mitochondrial Proteins - drug effects | Endarterectomy, Carotid | Lymphocyte Activation - immunology | Th17 Cells - drug effects | HSP90 Heat-Shock Proteins - immunology | T-Lymphocytes - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Tumor Necrosis Factor-alpha - immunology | Dendritic Cells - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Real-Time Polymerase Chain Reaction | T-Box Domain Proteins - drug effects | Th1 Cells - drug effects | HSP90 Heat-Shock Proteins - drug effects | Interleukin-1beta - immunology | Lipoproteins, LDL - pharmacology | HSP27 Heat-Shock Proteins - immunology | Plaque, Atherosclerotic - immunology | MicroRNAs - immunology | Reverse Transcriptase Polymerase Chain Reaction | HSP27 Heat-Shock Proteins - drug effects | T-Box Domain Proteins - metabolism | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Tumor Necrosis Factor-alpha - drug effects | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Interleukin-6 - immunology | MicroRNAs - drug effects | Th17 Cells - immunology | T-Lymphocytes - immunology | Chaperonin 60 - drug effects | Mitochondrial Proteins - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - drug effects | Proto-Oncogene Proteins c-akt - drug effects
Journal Article
British Journal of Cancer, ISSN 0007-0920, 01/2016, Volume 114, Issue 2, pp. 177 - 187
Background: Oestrogen receptor-negative (ER-) breast cancer is intrinsically sensitive to chemotherapy. However, tumour response is often incomplete, and... 
CELLS | REDUCES TUMOR-GROWTH | STAT1 | DNA-DAMAGE | chemotherapy | IFN | THERAPY | NEOADJUVANT CHEMOTHERAPY | ONCOLOGY | ER-negative breast cancer | GENE-EXPRESSION | RESISTANCE | PATHWAY ANALYSIS | predictive signature | XENOGRAFTS | Caspase 7 - metabolism | Immunohistochemistry | Receptors, Estrogen - metabolism | Cytoskeletal Proteins - genetics | Humans | Intracellular Signaling Peptides and Proteins - drug effects | Caspase 3 - metabolism | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Interferon-gamma - metabolism | Carrier Proteins - drug effects | Mitochondrial Proteins - drug effects | STAT1 Transcription Factor - metabolism | Mitochondrial Proteins - metabolism | Caspase 3 - genetics | Interferon-beta - genetics | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Ubiquitins - metabolism | Caspase 7 - drug effects | Antigens - drug effects | Caspase 7 - genetics | Membrane Proteins - genetics | Myxovirus Resistance Proteins - drug effects | Capecitabine - pharmacology | STAT1 Transcription Factor - genetics | Breast Neoplasms - drug therapy | Blotting, Western | Breast Neoplasms - genetics | Interferon-beta - metabolism | Signal Transduction - drug effects | Mice, Nude | Membrane Proteins - drug effects | Mice | Myxovirus Resistance Proteins - genetics | Ubiquitins - drug effects | Antigens - genetics | Neoplasm Transplantation | Phosphorylation | Ubiquitins - genetics | Gene Expression Regulation, Neoplastic | Interferon-gamma - drug effects | STAT1 Transcription Factor - drug effects | Mitochondrial Proteins - genetics | Interferon-beta - drug effects | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Breast Neoplasms - metabolism | In Situ Hybridization | Caspase 3 - drug effects | Cytoskeletal Proteins - metabolism | Female | Cytoskeletal Proteins - drug effects | Membrane Proteins - metabolism | Interferon-gamma - genetics | Cytokines - metabolism | Antigens - metabolism | Cisplatin - pharmacology | Xenograft Model Antitumor Assays | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cytokines - drug effects | Myxovirus Resistance Proteins - metabolism | Life Sciences | Computer Science | Translational Therapeutics
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 5, pp. 547 - 556
...–response cardiotoxic side effect that can lead to heart failure. At present, it is not possible to predict which patients will be affected by doxorubicin-induced cardiotoxicity (DIC... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | RISK-FACTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEXRAZOXANE | INDUCED CARDIOMYOPATHY | MATURATION | MUSCLE GENE-EXPRESSION | CELL BIOLOGY | CHILDHOOD-CANCER | INHIBITION | ANTHRACYCLINE-INDUCED CARDIOTOXICITY | CONGESTIVE-HEART-FAILURE | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Calcium - metabolism | Humans | Middle Aged | Transcriptome | Antibiotics, Antineoplastic - adverse effects | Mitochondria, Heart - drug effects | Membrane Potential, Mitochondrial - drug effects | Flow Cytometry | Adult | Female | Real-Time Polymerase Chain Reaction | Cardiotoxicity - genetics | DNA Damage - drug effects | Cell Survival - drug effects | Disease Susceptibility | Heart Failure - genetics | Breast Neoplasms - drug therapy | Myocytes, Cardiac - drug effects | Fluorescent Antibody Technique | Myocytes, Cardiac - metabolism | Aged | Polymorphism, Single Nucleotide | Oxidative Stress - drug effects | Induced Pluripotent Stem Cells | Doxorubicin - adverse effects | Doxorubicin - pharmacology | Heart Failure - chemically induced | Complications and side effects | Patient outcomes | Stem cells | Development and progression | Breast cancer | Transplantation | Cardiovascular diseases | Drug therapy | Doxorubicin | Methods | Heart failure | Chemotherapy | Toxicity | Index Medicus
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2003, Volume 348, Issue 6, pp. 538 - 549
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 51, pp. 173 - 183
.... In vitro cell adhesion, alkaline phosphatase activity, collagen secretion, extracellular matrix mineralization, and real-time PCR analyses disclose enhanced adhesion, proliferation, and osteogenic... 
Advanced Basic Science | Dentistry | Polyetheretherketone | Elastic modulus | Tantalum | Osteointegration | Plasma immersion ion implantation | STEM-CELLS | MATERIALS SCIENCE, BIOMATERIALS | ETHER-ETHER-KETONE | HUMAN OSTEOBLASTS | ENGINEERING, BIOMEDICAL | BIOACTIVE MATERIALS | MECHANICAL-PROPERTIES | METALLIC BIOMATERIALS | IN-VITRO | POROUS TANTALUM | IMMERSION ION-IMPLANTATION | NANOCOMPOSITE COATINGS | Mesenchymal Stromal Cells - enzymology | Bone Marrow Cells - enzymology | Alkaline Phosphatase - metabolism | Extracellular Matrix - metabolism | Fluorescent Dyes - metabolism | Elastic Modulus - drug effects | X-Ray Microtomography | Bone and Bones - drug effects | Mesenchymal Stromal Cells - cytology | Bone and Bones - diagnostic imaging | Surface Properties | Tantalum - pharmacology | Bone Marrow Cells - drug effects | Real-Time Polymerase Chain Reaction | Osteogenesis - genetics | Osseointegration - drug effects | Polyethylene Glycols - pharmacology | Ketones - pharmacology | Mesenchymal Stromal Cells - drug effects | Photoelectron Spectroscopy | Bone and Bones - physiology | Calcification, Physiologic - drug effects | Bone Marrow Cells - cytology | Extracellular Matrix - drug effects | Osteogenesis - drug effects | Cells, Cultured | Prostheses and Implants | Rats | Cell Adhesion - drug effects | Collagen - secretion | Gene Expression Regulation - drug effects | Animals | Cell Proliferation - drug effects | Collagen | Analysis | Stem cells | Medical colleges | Phosphatases | Ceramics | Implant dentures | Ceramic materials | Biomedical materials | Dental materials | Biocompatibility | Bones | Modulus of elasticity | Polyetheretherketones
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 2018, Volume 67, Issue 1, pp. 216 - 231
Sorafenib remains the only approved drug for treating patients with advanced hepatocellular carcinoma (HCC... 
CANCER-CELLS | OVEREXPRESSION | ACTIVATION | SIGNALING PATHWAY | TYRO3 | PATTERNS | INHIBITOR | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | RECEPTOR TYROSINE KINASE | INTERLEUKIN-8 | Niacinamide - analogs & derivatives | Humans | Cell Survival - genetics | MicroRNAs - metabolism | Cell Line, Tumor - drug effects | Cell Movement - genetics | Carcinoma, Hepatocellular - drug therapy | Cell Line, Tumor - pathology | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | RNA, Small Interfering - drug effects | Real-Time Polymerase Chain Reaction | Molecular Targeted Therapy - methods | Cell Proliferation - genetics | Liver Neoplasms - drug therapy | Blotting, Western | Cell Movement - drug effects | Drug Resistance, Neoplasm - genetics | Analysis of Variance | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Carcinoma, Hepatocellular - pathology | MicroRNAs - drug effects | Cell Proliferation - drug effects | Phenylurea Compounds - pharmacology | Niacinamide - pharmacology | Drug Resistance, Neoplasm - drug effects | RNA, Small Interfering - metabolism | Tyrosine | Cell proliferation | Invasiveness | Hepatocellular carcinoma | AKT protein | Drug resistance | Kinases | Axl protein | Signal transduction | Liver cancer | MicroRNAs | Protein-tyrosine kinase receptors | Tumor suppressor genes | miRNA | Transduction | Cell migration | Index Medicus
Journal Article
Developmental cell, ISSN 1534-5807, 2016, Volume 39, Issue 3, pp. 302 - 315
Journal Article