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Endocrinology, ISSN 0013-7227, 05/2017, Volume 158, Issue 5, pp. 1359 - 1372
Neointimal hyperplasia is the primary lesion underlying atherosclerosis and restenosis after coronary intervention. We previously described the essential... 
PROGENITOR CELLS | ARTERIAL INJURY | VASCULAR INJURY | ACCELERATES REENDOTHELIALIZATION | NAD(P)H OXIDASE | HYPOTENSIVE PEPTIDE | ENDOCRINOLOGY & METABOLISM | ACTIVATED-RECEPTOR-GAMMA | NEOINTIMAL HYPERPLASIA | NECROSIS-FACTOR-ALPHA | SMOOTH-MUSCLE-CELLS | Femoral Artery - pathology | Vascular System Injuries - genetics | Humans | Mice, Inbred C57BL | Cells, Cultured | Male | Signal Transduction - genetics | Vascular System Injuries - prevention & control | Neointima - metabolism | Adrenomedullin - genetics | Mice, Knockout | Femoral Artery - injuries | Neointima - genetics | Animals | Adrenomedullin - physiology | Neointima - pathology | Femoral Artery - metabolism | Receptor Activity-Modifying Protein 2 - genetics | Cytoprotection - genetics | Mice | Endothelial Cells - physiology | Receptor Activity-Modifying Protein 2 - physiology | Cell proliferation | Oxidative stress | Leukocyte migration | Syngeneic grafts | Hyperplasia | Bone marrow transplantation | Adrenomedullin | Smooth muscle | Macrophages | Receptor activity modifying proteins | Angiogenesis | Vascular diseases | Clonal deletion | Rodents | Atherosclerosis | Bone marrow | Deletion | Injuries | Medical treatment | Muscles | Blood vessels | Transplants | Inflammation | Endothelial cells | Restenosis | Arteriosclerosis | Stem cells | Cells (biology) | Infiltration | Bone | Cell migration
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2013, Volume 182, Issue 6, pp. 2380 - 2390
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0181597
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 29 - 39
Adrenomedullin (AM) is a peptide involved both in the pathogenesis of cardiovascular diseases and in circulatory homeostasis. The high-affinity AM receptor is... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVITY-MODIFYING PROTEINS | ENDOGENOUS ADRENOMEDULLIN | CARDIAC-HYPERTROPHY | ENDOTHELIAL-CELLS | GENE-EXPRESSION | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | KNOCKOUT MICE | INDUCED AIRWAY HYPERRESPONSIVENESS | TRANSGENIC MICE | Capillary Permeability - physiology | Embryonic Stem Cells - metabolism | Edema - genetics | Receptors, Calcitonin - metabolism | Tight Junctions - genetics | Humans | Male | Receptors, Calcitonin - genetics | Calcitonin Receptor-Like Protein | Cardiovascular Diseases - pathology | Cardiovascular Diseases - genetics | Receptors, Peptide - genetics | Arteries - metabolism | Embryo Loss - metabolism | Embryo Loss - pathology | Female | Embryo Loss - genetics | Intracellular Signaling Peptides and Proteins - genetics | Receptor Activity-Modifying Protein 2 | Tight Junctions - metabolism | Cardiovascular Diseases - metabolism | Membrane Proteins - genetics | Cells, Cultured | Receptors, Adrenomedullin | Adrenomedullin - metabolism | Arteries - pathology | Adrenomedullin - genetics | Mice, Knockout | Pregnancy | Edema - metabolism | Neovascularization, Physiologic - physiology | Membrane Proteins - biosynthesis | Animals | Endothelium, Vascular - metabolism | Homeostasis - physiology | Embryonic Stem Cells - pathology | Endothelium, Vascular - pathology | Receptor Activity-Modifying Proteins | Mice | Edema - pathology | Tight Junctions - pathology | Receptors, Peptide - metabolism | Proteins | Physiological aspects | Research | Peptides | Cardiovascular diseases
Journal Article
Journal Article