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1. Full Text Possible Role of Angiotensin-Converting Enzyme 2 and Activation of Angiotensin II Type 2 Receptor by Angiotensin-(1–7) in Improvement of Vascular Remodeling by Angiotensin II Type 1 Receptor Blockade
by Ohshima, Kousei and Mogi, Masaki and Nakaoka, Hirotomo and Iwanami, Jun and Min, Li-Juan and Kanno, Harumi and Tsukuda, Kana and Chisaka, Toshiyuki and Bai, Hui-Yu and Wang, Xiao-Li and Ogimoto, Akiyoshi and Higaki, Jitsuo and Horiuchi, Masatsugu
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 03/2014, Volume 63, Issue 3, pp. e53 - e59
angiotensin | remodeling | angiotensin-converting enzyme 2 | receptors | inflammation | oxidative stress | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Immunohistochemistry | Receptor, Angiotensin, Type 2 - genetics | Vascular Resistance | Vasodilator Agents - pharmacology | Oxidative Stress | Mice, Inbred C57BL | Gene Expression Regulation | Peptide Fragments - pharmacology | Angiotensin II Type 1 Receptor Blockers - pharmacology | RNA - genetics | Peptidyl-Dipeptidase A - genetics | Animals | Receptor, Angiotensin, Type 2 - metabolism | Peptidyl-Dipeptidase A - metabolism | Angiotensin I - pharmacology | Mice | Real-Time Polymerase Chain Reaction | Disease Models, Animal | RNA - metabolism | Index Medicus
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2. Full Text Angiotensin-(1–7) suppresses hepatocellular carcinoma growth and angiogenesis via complex interactions of angiotensin II type 1 receptor, angiotensin II type 2 receptor and mas receptor
by Liu, Yanping and Li, Bin and Wang, Ximing and Li, Guishuang and Shang, Rui and Yang, Jianmin and Wang, Jiali and Zhang, Meng and Chen, Yuguo and Zhang, Yun and Zhang, Cheng and Hao, Panpan
Molecular medicine (Cambridge, Mass.), ISSN 1076-1551, 2015, Volume 21, Issue 1, pp. 626 - 636
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Phosphorylation | Apoptosis - drug effects | Humans | Neovascularization, Pathologic - pathology | Mitogen-Activated Protein Kinase 11 - metabolism | Receptor, Angiotensin, Type 1 - genetics | Carcinoma, Hepatocellular - drug therapy | Receptor, Angiotensin, Type 1 - biosynthesis | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Mitogen-Activated Protein Kinase 11 - genetics | Receptor, Angiotensin, Type 2 - genetics | Liver Neoplasms - genetics | Peptide Fragments - administration & dosage | Receptor, Angiotensin, Type 2 - biosynthesis | Angiotensin I - administration & dosage | Liver Neoplasms - drug therapy | Hep G2 Cells | Xenograft Model Antitumor Assays | Animals | Neovascularization, Pathologic - drug therapy | Carcinoma, Hepatocellular - pathology | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Mice | Index Medicus
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3. Full Text Cardioprotective Effects Mediated by Angiotensin II Type 1 Receptor Blockade and Enhancing Angiotensin 1-7 in Experimental Heart Failure in Angiotensin-Converting Enzyme 2–Null Mice
by Patel, Vaibhav B and Bodiga, Sreedhar and Fan, Dong and Das, Subhash K and Wang, Zuocheng and Wang, Wang and Basu, Ratnadeep and Zhong, JiuChang and Kassiri, Zamaneh and Oudit, Gavin Y
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 06/2012, Volume 59, Issue 6, pp. 1195 - 1203
Heart failure | Signaling | Angiotensin-converting enzyme 2 | receptor | NADPH oxidase | Angiotensin 1-7 | Renin-angiotensin system | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Blood and lymphatic vessels | Antihypertensive Agents - pharmacology | Tetrazoles - pharmacology | Heart Failure - physiopathology | NADPH Oxidases - metabolism | Male | Peptide Fragments - pharmacology | Systole | Angiotensin II Type 1 Receptor Blockers - pharmacology | Heart Failure - prevention & control | Peptidyl-Dipeptidase A - genetics | Biphenyl Compounds - pharmacology | Myocardium - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Female | Angiotensin I - pharmacology | Blood Pressure - drug effects | Peptidyl-Dipeptidase A - deficiency | Heart - physiopathology | Peptide Fragments - metabolism | Mice, Inbred C57BL | Cells, Cultured | Heart Failure - genetics | Myocardium - pathology | Cardiotonic Agents - pharmacology | Angiotensin I - metabolism | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Drug Synergism | Animals | Heart - drug effects | Mice | Index Medicus
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4. Full Text The β-arrestin pathway-selective type 1A angiotensin receptor (AT 1A) agonist [Sar1, Ile4, Ile 8]angiotensin II regulates a robust G protein-independent signaling network
by Kendall, Ryan T and Strungs, Erik G and Rachidi, Saleh M and Lee, Mi-Hye and El-Shewy, Hesham M and Luttrell, Deirdre K and Janech, Michael G and Luttrell, Louis M
The Journal of biological chemistry, ISSN 0021-9258, 06/2011, Volume 286, Issue 22, pp. 19880 - 19891
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Phosphorylation - physiology | Dinoprostone - biosynthesis | Histone Chaperones - genetics | Humans | Histone Chaperones - metabolism | Arrestins - genetics | Glycogen Synthase Kinase 3 beta | Arrestins - metabolism | GTP-Binding Proteins - genetics | Receptor, Angiotensin, Type 1 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Phosphorylation - drug effects | DNA-Binding Proteins | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Cyclooxygenase 1 - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Dinoprostone - genetics | Protein Phosphatase 2 - genetics | Angiotensin II - analogs & derivatives | Transcription Factors - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Glycogen Synthase Kinase 3 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | beta-Arrestin 2 | beta-Arrestin 1 | beta-Arrestins | Receptor, Angiotensin, Type 1 - metabolism | Protein Phosphatase 2 - metabolism | Signal Transduction - physiology | Cyclooxygenase 1 - metabolism | GTP-Binding Proteins - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | Prostaglandins | Signal Transduction | AT1 Receptor | PGES3 | G-protein Coupled Receptors (GPCR) | PP2A | Arrestin | Akt PKB | Biased Agonism | I2PP2A
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5. Full Text Angiotensin II Type 2 Receptor Antagonizes Angiotensin II Type 1 Receptor–Mediated Cardiomyocyte Autophagy
by Porrello, Enzo R and DʼAmore, Angelo and Curl, Claire L and Allen, Andrew M and Harrap, Stephen B and Thomas, Walter G and Delbridge, Lea M.D
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 06/2009, Volume 53, Issue 6, pp. 1032 - 1040
Angiotensin II type 1 receptor | Hypertrophic heart rat | Neonate | Angiotensin II type 2 receptor | Angiotensin II | Autophagy | Hypertrophy | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Blood and lymphatic vessels | Animals, Newborn | Gene Transfer Techniques | Receptor, Angiotensin, Type 2 - genetics | Cells, Cultured | RNA, Messenger - analysis | Rats | Cardiomegaly - pathology | Reference Values | Autophagy - physiology | Random Allocation | Rats, Sprague-Dawley | Myocytes, Cardiac - pathology | Receptor, Angiotensin, Type 1 - genetics | Animals | Receptor, Angiotensin, Type 2 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Sensitivity and Specificity | Adenoviridae - genetics | Myocytes, Cardiac - metabolism | Female | Autophagy - genetics | Cardiomegaly - metabolism | Disease Models, Animal | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
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6. Full Text Mineralocorticoid and angiotensin II type 1 receptors in the subfornical organ mediate angiotensin II – Induced hypothalamic reactive oxygen species and hypertension
by Wang, Hong-Wei and Huang, Bing S and White, Roselyn A and Chen, Aidong and Ahmad, Monir and Leenen, Frans H.H
Neuroscience, ISSN 0306-4522, 2016, Volume 329, pp. 112 - 121
Neurology | mineralocorticoid receptors | angiotensinergic pathways | angiotensin II type 1 receptor | brain | reactive oxygen species | Angiotensin II type 1 receptor | Brain | Reactive oxygen species | Mineralocorticoid receptors | Angiotensinergic pathways | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Receptors, Mineralocorticoid - genetics | Reactive Oxygen Species - metabolism | Angiotensin II - administration & dosage | Angiotensin II - metabolism | Rats, Wistar | Receptors, Mineralocorticoid - metabolism | Medulla Oblongata - metabolism | Male | Random Allocation | Dependovirus | RNA, Messenger - metabolism | Gene Knockdown Techniques | Hypertension - metabolism | Aldosterone - metabolism | Receptor, Angiotensin, Type 1 - genetics | Animals | Hypothalamus - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Heart Rate - physiology | Blood Pressure - physiology | RNA, Small Interfering | Genetic Vectors | Subfornical Organ - metabolism | Hypertension | Corticosteroids | Angiotensin | Steroids | Index Medicus
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7. Full Text Targeting the Degradation of Angiotensin II With Recombinant Angiotensin-Converting Enzyme 2: Prevention of Angiotensin II–Dependent Hypertension
by Wysocki, Jan and Ye, Minghao and Rodriguez, Eva and González-Pacheco, Francisco R and Barrios, Clara and Evora, Karla and Schuster, Manfred and Loibner, Hans and Brosnihan, K Bridget and Ferrario, Carlos M and Penninger, Josef M and Batlle, Daniel
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 01/2010, Volume 55, Issue 1, pp. 90 - 98
ACE2 | Soluble | Angiotensin II | Angiotensin-(1-7) | Recombinant | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Blood and lymphatic vessels | Angiotensin II - administration & dosage | Humans | Angiotensin II - blood | Male | Dose-Response Relationship, Drug | Peptidyl-Dipeptidase A - genetics | Kidney - metabolism | Hypertension - chemically induced | Peptide Fragments - blood | Peptidyl-Dipeptidase A - metabolism | Hypertension - prevention & control | Anesthesia | Blood Pressure - drug effects | Recombinant Proteins - metabolism | Peptide Fragments - metabolism | Kidney - drug effects | Angiotensin II - metabolism | Infusion Pumps | Mice, Inbred C57BL | Peptide Fragments - administration & dosage | Angiotensin I - blood | Angiotensin II - analogs & derivatives | Angiotensin I - metabolism | Angiotensin II Type 1 Receptor Blockers - antagonists & inhibitors | Recombinant Proteins - administration & dosage | Hypertension - metabolism | Mice, Knockout | Animals | Peptidyl-Dipeptidase A - administration & dosage | Mice | Index Medicus
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8. Full Text Type 1 angiotensin receptors on macrophages ameliorate IL-1 receptor-mediated kidney fibrosis
by Zhang, Jian-Dong and Patel, Mehul B and Griffiths, Robert and Dolber, Paul C and Ruiz, Phillip and Sparks, Matthew A and Stegbauer, Johannes and Jin, Huixia and Gomez, Jose A and Buckley, Anne F and Lefler, William S and Chen, Daian and Crowley, Steven D
The Journal of clinical investigation, ISSN 0021-9738, 2014, Volume 124, Issue 5, pp. 2198 - 2203
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Interleukin-1 - genetics | Ureteral Obstruction - complications | Epithelial Cells - metabolism | Kidney - pathology | Receptors, Interleukin-1 - genetics | Ureteral Obstruction - metabolism | Fibrosis - metabolism | Kidney Diseases - genetics | Kidney - metabolism | Receptor, Angiotensin, Type 1 - genetics | Ureteral Obstruction - genetics | Kidney Diseases - metabolism | Interleukin-1 - metabolism | Fibrosis - genetics | Macrophages - pathology | Kidney Diseases - pathology | Epithelial Cells - pathology | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Macrophages - metabolism | Animals | Receptor, Angiotensin, Type 1 - metabolism | Ureteral Obstruction - pathology | Mice | Fibrosis - pathology | Kidney Diseases - etiology | Care and treatment | Receptors | Fibrosis | Angiotensin | Physiological aspects | Kidney diseases | Macrophages | Properties | Index Medicus | Abridged Index Medicus | Brief Report
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9. Full Text Overexpression of angiotensin II type 1 receptor in breast cancer cells induces epithelial–mesenchymal transition and promotes tumor growth and angiogenesis
by Oh, Eunhye and Kim, Ji Young and Cho, Youngkwan and An, Hyunsook and Lee, Nahyun and Jo, Hunho and Ban, Changill and Seo, Jae Hong
Biochimica et biophysica acta. Molecular cell research, ISSN 0167-4889, 06/2016, Volume 1863, Issue 6, pp. 1071 - 1081
AGTR1 | Angiogenesis | Breast cancer | Smad4 | EMT | Losartan | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cadherins - metabolism | Humans | Transplantation, Heterologous | Epithelial-Mesenchymal Transition - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Breast Neoplasms - metabolism | Matrix Metalloproteinase 9 - metabolism | Receptor, Angiotensin, Type 1 - genetics | MCF-7 Cells | Smad4 Protein - genetics | Matrix Metalloproteinase 9 - genetics | Female | Neovascularization, Pathologic - prevention & control | Tumor Burden - genetics | Gene Expression Regulation, Neoplastic - drug effects | Cadherins - genetics | Gene Expression Regulation, Neoplastic - genetics | Cell Proliferation - genetics | Losartan - pharmacology | Breast Neoplasms - blood supply | Reverse Transcriptase Polymerase Chain Reaction | Smad4 Protein - metabolism | Blotting, Western | Microscopy, Confocal | Poly(ADP-ribose) Polymerases - metabolism | Animals | Breast Neoplasms - genetics | Poly(ADP-ribose) Polymerases - genetics | Tumor Burden - drug effects | Mice, Nude | Receptor, Angiotensin, Type 1 - metabolism | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Mice, Inbred BALB C | Neovascularization, Pathologic - metabolism | Development and progression | Growth | Analysis | Stem cells | Angiotensin | Medical colleges | Cancer cells | Sugars | Monosaccharides
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10. Full Text Aerobic Exercise Training–Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7)
by Fernandes, Tiago and Hashimoto, Nara Y and Magalhães, Flávio C and Fernandes, Fernanda B and Casarini, Dulce E and Carmona, Adriana K and Krieger, José E and Phillips, M Ian and Oliveira, Edilamar M
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 08/2011, Volume 58, Issue 2, pp. 182 - 189
microRNAs | ACE2 | Aerobic exercise training | angiotensin II receptors | cardiac hypertrophy | renin angiotensin system | angiotensin (1-7) | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Blood and lymphatic vessels | Peptide Fragments - metabolism | Rats, Wistar | Hypertrophy, Left Ventricular - metabolism | Rats | MicroRNAs - metabolism | Renin-Angiotensin System - physiology | Angiotensin I - metabolism | Physical Conditioning, Animal - physiology | Peptidyl-Dipeptidase A - genetics | Receptor, Angiotensin, Type 1 - genetics | Animals | Hypertrophy, Left Ventricular - genetics | Angiotensin I - genetics | Myocardium - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Peptidyl-Dipeptidase A - metabolism | Female | MicroRNAs - genetics | Hemodynamics | Hypertrophy, Left Ventricular - physiopathology | Peptide Fragments - genetics | Index Medicus
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