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Journal Article
Molecular Medicine, ISSN 1076-1551, 2015, Volume 21, Issue 1, pp. 626 - 636
Journal Article
Nature, ISSN 0028-0836, 04/2017, Volume 544, Issue 7650, pp. 327 - 332
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 02/2011, Volume 57, Issue 2, pp. 308 - 313
Journal Article
Circulation, ISSN 0009-7322, 12/2004, Volume 110, Issue 25, pp. 3849 - 3857
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 02/2009, Volume 296, Issue 2, pp. 208 - 216
The adaptive immune response and, in particular, T cells have been shown to be important in the genesis of hypertension. In the present study, we sought to... 
Superoxide | Cytokines | TNF-α | Electron spin resonance | NADPH oxidase | Adoptive transfer | RHEUMATOID-ARTHRITIS | PHYSIOLOGY | TNF-alpha | VASCULAR DYSFUNCTION | adoptive transfer | FACTOR-KAPPA-B | MECHANISMS | FACTOR-ALPHA | electron spin resonance | TUMOR-NECROSIS-FACTOR | AT RECEPTORS | cytokines | superoxide | CORONARY-ARTERY-DISEASE | CONVERTING-ENZYME | Blood Pressure | Catalase - pharmacology | Renin - metabolism | Tumor Necrosis Factor-alpha - metabolism | Angiotensin II - administration & dosage | Homeodomain Proteins - metabolism | Adoptive Transfer | Angiotensin II Type 1 Receptor Blockers - pharmacology | T-Lymphocytes - transplantation | NADPH Oxidases - deficiency | T-Lymphocytes - metabolism | Time Factors | T-Lymphocytes - drug effects | Peptidyl-Dipeptidase A - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Polyethylene Glycols - pharmacology | Autocrine Communication | Receptor, Angiotensin, Type 2 - genetics | Infusion Pumps, Implantable | Superoxide Dismutase - pharmacology | Angiotensin II - metabolism | Mice, Inbred C57BL | Cells, Cultured | Losartan - pharmacology | Angiotensinogen - metabolism | Imidazoles - pharmacology | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Lymphocyte Activation - drug effects | Receptor, Angiotensin, Type 1 - metabolism | Angiotensin II Type 2 Receptor Blockers | Renin-Angiotensin System - drug effects | T-Lymphocytes - immunology | Mice | Pyridines - pharmacology | Receptor, Angiotensin, Type 2 - deficiency | Call for Papers
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 10/2010, Volume 56, Issue 4, pp. 658 - 666
Females are less sensitive to the hypertensive effects of angiotensin II compared with males, although the molecular mechanisms responsible are unknown. We... 
blood pressure | proteinuria | sex | mas receptor | Ang (1-7) | SHR | renin-angiotensin system | SYSTEM | PERIPHERAL VASCULAR DISEASE | SEX-DIFFERENCES | GENDER-DIFFERENCES | CONTRIBUTES | EXPRESSION | Kidney Cortex - drug effects | Receptors, G-Protein-Coupled - metabolism | Kidney Cortex - metabolism | Angiotensin II - blood | Male | Peptide Fragments - pharmacology | Vasoconstrictor Agents - toxicity | Receptor, Angiotensin, Type 1 - genetics | Hypertension - chemically induced | Female | Blood Pressure - drug effects | Blood Pressure - physiology | Angiotensin II - toxicity | Proto-Oncogene Proteins - metabolism | Rats, Inbred SHR | Proto-Oncogene Proteins - antagonists & inhibitors | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - pharmacology | Peptide Fragments - metabolism | Angiotensin II - metabolism | Rats | Proto-Oncogene Proteins - genetics | Angiotensin II - analogs & derivatives | Angiotensin I - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Hypertension - physiopathology | Animals | Receptor, Angiotensin, Type 2 - metabolism | Vasoconstrictor Agents - blood | Vasoconstrictor Agents - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Sex Factors | Receptors, G-Protein-Coupled - genetics | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 07/2014, Volume 86, Issue 1, pp. 75 - 85
Fibrosis is a hallmark of chronic kidney disease, for which there is currently no effective cure. The hormone relaxin is emerging as an effective antifibrotic... 
Smad2 | kidney disease | TGF-β | fibrosis | relaxin | AT2 receptor | KIDNEY-DISEASE | NITRIC-OXIDE SYNTHASE | PROTEIN-KINASE | RESONANCE ENERGY-TRANSFER | FIBROBLASTS | TGF-beta | GROWTH-FACTOR-BETA | AT RECEPTORS | IN-VIVO | UROLOGY & NEPHROLOGY | HYPERTENSIVE-RATS | HORMONE RELAXIN | Relaxin - metabolism | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Humans | Protein Multimerization | Male | Receptors, Peptide - chemistry | Angiotensin II Type 2 Receptor Blockers - pharmacology | Relaxin - pharmacology | Renal Insufficiency, Chronic - drug therapy | Myofibroblasts - metabolism | Renal Insufficiency, Chronic - metabolism | Kidney - metabolism | Myofibroblasts - pathology | Receptor, Angiotensin, Type 2 - genetics | Kidney - drug effects | Cells, Cultured | Rats | Imidazoles - pharmacology | Recombinant Proteins - pharmacology | Myofibroblasts - drug effects | Rats, Sprague-Dawley | Disease Progression | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | MAP Kinase Signaling System - drug effects | Receptor, Angiotensin, Type 2 - metabolism | Signal Transduction - drug effects | Fibrosis | Mice | Pyridines - pharmacology | Receptor, Angiotensin, Type 2 - deficiency | Receptors, G-Protein-Coupled - chemistry | Receptors, Peptide - metabolism
Journal Article