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Nature Medicine, ISSN 1078-8956, 05/2011, Volume 17, Issue 5, pp. 589 - 595
Hepatitis C virus (HCV) is a major cause of liver disease, but therapeutic options are limited and there are no prevention strategies. Viral entry is the first... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | B TYPE-I | NEUTRALIZING ANTIBODIES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | FACTOR RECEPTOR | CELL BIOLOGY | EPIDERMAL-GROWTH-FACTOR | CELL TRANSMISSION | HUMAN LIVER | INFECTION | ERLOTINIB | Erlotinib Hydrochloride | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Hepatitis C - therapy | Humans | Virus Internalization - drug effects | RNA Interference | Membrane Proteins - physiology | Base Sequence | Hepacivirus - physiology | Hepatitis C - prevention & control | Receptor, Epidermal Growth Factor - physiology | Hepacivirus - drug effects | Cell Line | Antiviral Agents - pharmacology | Receptor, EphA2 - physiology | Tetraspanin 28 | Claudin-1 | Animals | Hepatitis C - virology | Receptor, EphA2 - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Antigens, CD - physiology | Hepatitis C - physiopathology | Ligands | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, EphA2 - antagonists & inhibitors | Quinazolines - pharmacology | Host-Pathogen Interactions - physiology | Ephrin A | Antiviral agents | Care and treatment | Patient outcomes | Physiological aspects | Research | Hepatitis C | Hepatitis | Viruses | Drug therapy | Antiviral drugs | Receptor, EphA2 | Hépatology and Gastroenterology | Antigens, CD81 | Antiviral Agents | Quinazolines | Virus Internalization | Membrane Proteins | Host-Pathogen Interactions | Life Sciences | Human health and pathology | Protein Kinase Inhibitors | RNA, Small Interfering | Hepacivirus | Antigens, CD | Receptor, Epidermal Growth Factor | Liver | HCV escape variants | Cell-cell transmission | genetics | pharmacology | Antiviral | therapy | physiology | drug effects | prevention & control | antagonists & inhibitors | physiopathology | Phosphotyrosine kinase | virology
Journal Article
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 09/2011, Volume 301, Issue 3, pp. F486 - F493
Ledeganck KJ, Boulet GA, Horvath CA, Vinckx M, Bogers JJ, Van Den Bossche R, Verpooten GA, De Winter BY. Expression of renal distal tubule transporters TRPM6... 
Claudin-19 | Renin-angiotensin-aldosterone system | Magnesium | Sodium | Claudin-16 | SECONDARY HYPOCALCEMIA | PHYSIOLOGY | claudin-19 | magnesium | claudin-16 | ANGIOTENSIN-ALDOSTERONE SYSTEM | TRANSPLANT RECIPIENTS | NEPHROPATHY | MAGNESIUM TRANSPORT | TREATED RATS | sodium | renin-angiotensin-aldosterone system | NA+-CL-COTRANSPORTER | EPIDERMAL-GROWTH-FACTOR | UROLOGY & NEPHROLOGY | INHIBITOR | HYPOMAGNESEMIA | Kidney Tubules, Distal - pathology | Rats, Wistar | Cyclosporine - pharmacology | Male | Epidermal Growth Factor - therapeutic use | Nephrocalcinosis - physiopathology | Receptor, Epidermal Growth Factor - metabolism | Claudins - metabolism | Hyponatremia - physiopathology | Models, Animal | Homeostasis - drug effects | Renal Tubular Transport, Inborn Errors - physiopathology | Enzyme Inhibitors - adverse effects | Sodium Chloride Symporters - metabolism | Hypercalciuria - physiopathology | Enzyme Inhibitors - pharmacology | Rats | Epidermal Growth Factor - metabolism | Magnesium - metabolism | Renin-Angiotensin System - physiology | Animals | Kidney Tubules, Distal - drug effects | Cyclosporine - adverse effects | TRPM Cation Channels - metabolism | Epidermal Growth Factor - pharmacology | Kidney Tubules, Distal - metabolism | Physiological aspects | Care and treatment | Epidermal growth factor | Cyclosporine | Kidney diseases | Health aspects | Homeostasis | Kidneys | Gene expression | Rodents
Journal Article
Oncogene, ISSN 0950-9232, 09/2010, Volume 29, Issue 36, pp. 4989 - 5005
Hepatocellular carcinoma (HCC) is a highly prevalent, treatment-resistant malignancy with a multifaceted molecular pathogenesis. Current evidence indicates... 
Signaling | Hepatocellular carcinoma | Multikinase inhibitor | Vascular endothelial growth factor | Sorafenib | Epidermal growth factor receptor | FACTOR-RECEPTOR | signaling | sorafenib | MESSENGER-RNA EXPRESSION | epidermal growth factor receptor | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-MET PROTOONCOGENE | CELL BIOLOGY | CELL LUNG-CANCER | PHASE-II TRIAL | ONCOLOGY | vascular endothelial growth factor | GENETICS & HEREDITY | TYROSINE KINASE INHIBITOR | PROGNOSTIC-SIGNIFICANCE | ACTIVATED PROTEIN-KINASES | multikinase inhibitor | TUMOR-GROWTH | hepatocellular carcinoma | ENDOTHELIAL GROWTH-FACTOR | Receptor, Epidermal Growth Factor - genetics | Receptors, Vascular Endothelial Growth Factor - physiology | MAP Kinase Signaling System - physiology | Humans | Liver Neoplasms - therapy | Liver Neoplasms - etiology | Somatomedins - physiology | Hepatocyte Growth Factor - physiology | Carcinoma, Hepatocellular - genetics | Hepatocyte Growth Factor - genetics | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - etiology | Receptors, Vascular Endothelial Growth Factor - genetics | Receptor, Epidermal Growth Factor - physiology | Liver Neoplasms - genetics | Signal Transduction - genetics | Proto-Oncogene Proteins c-met - genetics | Liver Neoplasms - ethnology | Proto-Oncogene Proteins c-met - physiology | Animals | Models, Biological | Somatomedins - genetics | Carcinoma, Hepatocellular - pathology | Signal Transduction - physiology | Carcinoma, Hepatocellular - therapy | Care and treatment | Gene mutations | Cellular signal transduction | Genetic aspects | Research | Hepatoma | Health aspects | Signal transduction | Genetics | Liver diseases | Mutation | Cancer
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 08/2009, Volume 15, Issue 16, pp. 5267 - 5273
Journal Article
Journal Article
Journal Article