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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2011, Volume 108, Issue 17, pp. 7142 - 7147
Trastuzumab, a monoclonal antibody targeting human epidermal growth factor receptor-2 (HER2/ErbB-2), has become the mainstay of treatment for HER2-positive... 
T lymphocytes | Inoculation | Medical treatment | Antibodies | Cytotoxicity | Breast cancer | Adaptive immunity | Natural killer cells | Tumors | Cancer | Natural killer cell | Innate | Adaptive | Tumor immunology | tumor immunology | TRASTUZUMAB | MECHANISM | MULTIDISCIPLINARY SCIENCES | innate | OVEREXPRESSING HER2 | COMBINATION | HER2-POSITIVE BREAST-CANCER | ADAPTIVE IMMUNITY | adaptive | IMMUNOTHERAPY | MICE | natural killer cell | PHASE-I | ESTABLISHED TUMORS | Receptor, ErbB-2 - genetics | Receptors, Interleukin-1 Type I - immunology | CD8-Positive T-Lymphocytes - transplantation | Humans | Receptors, Interleukin-17 - immunology | Receptors, Interleukin-17 - genetics | Tumor Necrosis Factor Receptor Superfamily, Member 9 - genetics | Adoptive Transfer | Interferon Type I - immunology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Antibodies, Monoclonal, Humanized | Receptor, ErbB-2 - immunology | Interferon-gamma - genetics | Antibodies, Monoclonal - immunology | Mammary Neoplasms, Animal - genetics | Mammary Neoplasms, Animal - immunology | Antibodies, Monoclonal - pharmacology | Tumor Necrosis Factor Receptor Superfamily, Member 9 - immunology | Mice, Transgenic | Antineoplastic Combined Chemotherapy Protocols - immunology | Mice, SCID | Animals | Apoptosis Regulatory Proteins | Interferon-gamma - immunology | Receptors, Interleukin-1 Type I - genetics | Cell Line, Tumor | Interferon Type I - genetics | Programmed Cell Death 1 Receptor | Antigens, Surface | Mammary Neoplasms, Animal - drug therapy | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Trastuzumab | Proteins | Cytokines | Monoclonal antibodies | Interferon | Gene expression | Cells | Index Medicus | Biological Sciences
Journal Article
Oncogene, ISSN 0950-9232, 04/2016, Volume 35, Issue 17, pp. 2208 - 2222
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7691, pp. 189 - 194
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 12/2010, Volume 30, Issue 23, pp. 5456 - 5472
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2015, Volume 17, Issue 5, pp. 627 - 638
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2016, Volume 17, Issue 6, pp. 738 - 746
Journal Article
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
Pancreas, ISSN 0885-3177, 03/2013, Volume 42, Issue 2, pp. 216 - 222
Objectives: The objective of this study was to evaluate genetic alterations of K-ras, p53, c-erbB-2, and deleted in pancreatic cancer, locus 4 (DPC4) genes in... 
c-erbB-2 genes | k-ras genes | pancreas | p53 genes | CANCER SURVIVAL | PROTEIN | PAPILLARY-MUCINOUS NEOPLASM | INTRAEPITHELIAL NEOPLASIA | LESIONS | OVEREXPRESSION | CODON-12 MUTATIONS | PROGNOSTIC-SIGNIFICANCE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CARCINOMA | Immunohistochemistry | Multivariate Analysis | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Prognosis | Receptor, ErbB-2 - genetics | Humans | Middle Aged | Male | Carcinoma, Pancreatic Ductal - surgery | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - genetics | Young Adult | Tumor Suppressor Protein p53 - analysis | Smad4 Protein - genetics | Time Factors | DNA Mutational Analysis | Polymerase Chain Reaction | Adult | Carcinoma, Pancreatic Ductal - mortality | Female | Pancreatic Neoplasms - mortality | Retrospective Studies | CA-19-9 Antigen - blood | Odds Ratio | Republic of Korea - epidemiology | Pancreatic Neoplasms - blood | Genetic Predisposition to Disease | Risk Assessment | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Risk Factors | Kaplan-Meier Estimate | Pancreatic Neoplasms - enzymology | Proportional Hazards Models | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Carcinoma, Pancreatic Ductal - pathology | Biomarkers, Tumor - blood | Carcinoma, Pancreatic Ductal - blood | Carcinoma, Pancreatic Ductal - enzymology | Phenotype | Aged | Biomarkers, Tumor - genetics | Mutation | Receptor, ErbB-2 - analysis | Smad4 Protein - analysis | Index Medicus
Journal Article
Journal Article