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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 34, pp. 14379 - 14384
FGF19 is a hormone that regulates bile acid and glucose homeostasis. Progress has been made in identifying cofactors for receptor activation. However, several... 
Adipose tissues | Molecules | Receptors | Phosphorylation | Bile acids | Liver | Homeostasis | Fibroblast growth factors | Heparin | Adipocytes | Diabetes | FGF21 | BETA-KLOTHO | SIGNAL | HOMEOSTASIS | SPECIFICITY | liver | MULTIDISCIPLINARY SCIENCES | RECEPTOR | FIBROBLAST-GROWTH-FACTOR | SUPPRESSION | DETERMINES | diabetes | FGF23 | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Transcriptional Activation | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Obesity - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Tissue Distribution | Adipose Tissue - metabolism | Fibroblast Growth Factors - metabolism | Membrane Proteins - metabolism | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Cell Line | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Glucose Tolerance Test | Membrane Proteins - genetics | Liver - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Obesity - metabolism | Animals | Fibroblast Growth Factors - pharmacokinetics | Glucose - pharmacokinetics | Glucose - metabolism | Protein Binding | Mice, Obese | Mice | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Glucose metabolism | Hormone receptors | Research | Chemical properties | Signal transduction | Rodents | Glucose | Gene expression | Metabolism | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e104154 - e104154
Increases in fibroblastic growth factor 23 (FGF23 or Fgf23) production by osteocytes result in hypophosphatemia and rickets in the Hyp mouse homologue of... 
SIGNALING PATHWAYS | VITAMIN-D METABOLISM | PHOSPHATE HOMEOSTASIS | X-LINKED HYPOPHOSPHATEMIA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MINERAL METABOLISM | FACTOR RECEPTOR-1 | FIBROBLAST-GROWTH-FACTOR | CHRONIC KIDNEY-DISEASE | HYP-MOUSE OSTEOBLASTS | Transcriptional Activation - genetics | Osteocytes - metabolism | Extracellular Matrix Proteins - genetics | Humans | Fibroblast Growth Factors - genetics | Gene Expression Regulation, Developmental - genetics | Fibroblast Growth Factors - biosynthesis | Hypophosphatemia - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Autocrine Communication - genetics | Mice, Knockout | Hypophosphatemia - genetics | RNA, Messenger - biosynthesis | Animals | Hypophosphatemia - pathology | Female | Osteocytes - pathology | Vitamin D - metabolism | Mice | Phosphates | Calcifediol | Fibroblast growth factors | Alfacalcidol | Vitamin D | RNA | Dentin | Post-transcription | Fibroblast growth factor | Fibroblast growth factor 23 | Pathogenesis | Dmp1 protein | Science | Homeostasis | Paracrine signalling | Osteocytes | Homology | AKT protein | SOST protein | Activation | Kinases | Osteoblasts | Reduction | Rickets | Clonal deletion | Mineralization | Deletion | Fibroblasts | Biocompatibility | Hypophosphatemia | Autocrine signalling | Osteomalacia | Fibroblast growth factor receptor 1 | Growth factors | Fibroblast growth factor 2 | MAP kinase | Bone turnover | Metabolism | Gene expression | Phosphoglycoprotein | Flox | 1-Phosphatidylinositol 3-kinase | Endopeptidase | Kidney diseases | Bone | Mutation | Laboratory animals | Fibroblast growth factor receptors | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2017, Volume 23, Issue 20, pp. 6138 - 6151
Purpose: FGFR1 amplification occurs in approximately 15% of estrogen receptor-positive (ER+) human breast cancers. Weinvestigated mechanisms by which FGFR1... 
AMPLIFICATION | PROTEIN | ONCOLOGY | THERAPEUTIC TARGET | GENES | ENDOCRINE THERAPY | FACTOR RECEPTOR-1 | FIBROBLAST-GROWTH-FACTOR | SIGNALING MODULE | EXPRESSION | NUCLEAR TRANSLOCATION | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Molecular Targeted Therapy | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Breast Neoplasms - metabolism | Fibroblast Growth Factors - metabolism | Estrogen Receptor alpha - metabolism | Female | Transcription, Genetic | Disease Models, Animal | Estrogen Receptor alpha - antagonists & inhibitors | Breast Neoplasms - drug therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Estrogen Receptor Modulators - pharmacology | Protein Transport | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Gene Amplification | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Neoplasm Staging | Cell proliferation | Fibroblast growth factor | Transcription | Genes | Estrogens | Estrogen | Estrogen receptors | Antagonists | Kinases | Nuclei | Fulvestrant | Gene sequencing | E2F protein | Surgery | Xenografts | Inhibition | Fibroblast growth factor receptor 1 | Protein-tyrosine kinase | Deoxyribonucleic acid--DNA | Binding | Tyrosine | Deprivation | Breast cancer | siRNA | Gene expression | Patients | Polymerase chain reaction | Amplification | Ribonucleic acids | Experimental design | Breast | Ligands | Nuclei (cytology) | Tumors | Cancer | Fibroblast growth factor receptors | Index Medicus | breast cancer | drug resistance | fulvestrant | FGFR1
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2012, Volume 32, Issue 22, pp. 7477 - 7492
Adult zebrafish show a remarkable capacity to regenerate their spinal column after injury, an ability that stands in stark contrast to the limited repair that... 
MOTOR-NEURON REGENERATION | RETINAL GANGLION-CELLS | TRANSGENIC ZEBRAFISH | FUNCTIONAL RECOVERY | AXONAL REGENERATION | REACTIVE ASTROCYTES | CULTURED ASTROCYTES | FIBROBLAST-GROWTH-FACTOR | ADULT ZEBRAFISH | ASTROGLIAL CELLS | NEUROSCIENCES | Glial Fibrillary Acidic Protein - genetics | Mitogen-Activated Protein Kinase Kinases - genetics | Nestin | Humans | Nerve Regeneration - physiology | Fibroblast Growth Factor 2 - pharmacology | Motor Activity - drug effects | Ki-67 Antigen - metabolism | Green Fluorescent Proteins - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Cell Movement - genetics | Recovery of Function | Neuroglia - drug effects | Spinal Cord Injuries - pathology | Cell Differentiation - genetics | Mitogen-Activated Protein Kinase Kinases - metabolism | Dextrans | Time Factors | Intermediate Filament Proteins - genetics | Fibroblast Growth Factor 3 - metabolism | Bromodeoxyuridine - metabolism | Fibroblast Growth Factor 8 - pharmacology | Disease Models, Animal | Fibroblast Growth Factor 3 - genetics | Animals, Genetically Modified | Gene Expression Regulation - genetics | Zebrafish Proteins - metabolism | Enzyme Inhibitors - pharmacology | Neuroglia - physiology | Zebrafish | Signal Transduction - genetics | Nerve Tissue Proteins - genetics | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Cell Movement - drug effects | Motor Activity - genetics | Pyrroles - pharmacology | Animals | Analysis of Variance | Signal Transduction - drug effects | Cell Differentiation - drug effects | Rhodamines | Nerve Regeneration - drug effects | Cell Proliferation - drug effects | Spinal Cord Injuries - physiopathology | Zebrafish Proteins - genetics | Intermediate Filament Proteins - metabolism | RNA, Messenger | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 36, pp. 18632 - 18642
Parathyroid hormone (PTH) and FGF23 are the primary hormones regulating acute phosphate homeostasis. Human renal proximal tubule cells (RPTECs) were used to... 
RENAL PROXIMAL TUBULE | PROTEIN-KINASE-A | MDCK CELLS | ACTIVATION | PTH | FGF-RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | I COTRANSPORTERS | FACTOR RECEPTOR GENES | CALCIUM-TRANSPORT | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Receptor, Parathyroid Hormone, Type 1 - metabolism | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Phosphoproteins - genetics | Phosphoproteins - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 4 - biosynthesis | Parathyroid Hormone - genetics | Sodium-Hydrogen Exchangers - metabolism | Sodium-Phosphate Cotransporter Proteins, Type IIa - metabolism | Sodium-Phosphate Cotransporter Proteins, Type IIa - genetics | Fibroblast Growth Factors - metabolism | Phosphates - metabolism | Glucuronidase - biosynthesis | Glucuronidase - genetics | Parathyroid Hormone - metabolism | Receptor, Parathyroid Hormone, Type 1 - genetics | Signal Transduction - physiology | Sodium-Hydrogen Exchangers - genetics | Receptor, Fibroblast Growth Factor, Type 3 - biosynthesis | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Cell Line, Transformed | Index Medicus | parathyroid hormone | NPT2A | klotho | PDZ Protein | fibroblast growth factor receptor (FGFR) | NHERF1 | alternative splicing | G protein-coupled receptor (GPCR) | transport | Cell Biology
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e40164 - e40164
Fibroblast growth factor 21 (FGF21) is a potent metabolic regulator, and pharmacological administration elicits glucose and lipid lowering responses in... 
BETA-KLOTHO | DIABETES-MELLITUS | PPAR-ALPHA | OBESITY | FIBROBLAST-GROWTH-FACTOR-21 | TISSUE | BIOLOGY | RESISTANCE | INSULIN SENSITIVITY | GROWTH-FACTOR 21 | MOUSE MODELS | Adipose Tissue, White - transplantation | Humans | Leptin - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Adipose Tissue, White - metabolism | Male | PPAR gamma - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Fibroblast Growth Factors - metabolism | Fibroblast Growth Factors - administration & dosage | Female | Leptin - administration & dosage | Leptin - pharmacology | Homeostasis - drug effects | Lipodystrophy - therapy | Disease Models, Animal | PPAR gamma - genetics | Signal Transduction | Mice, Transgenic | Fibroblast Growth Factors - pharmacology | Lipodystrophy - metabolism | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Gene Expression Regulation - drug effects | Lipodystrophy - genetics | Animals | Glucose - metabolism | Mice | Adipose Tissue, White - drug effects | Adipose tissues | Leptin | Body weight | Anticholesteremic agents | Triglycerides | Fibroblast growth factors | Glucose | Insulin | Dextrose | Protein binding | Fibroblast growth factor | Adipose tissue | Body fat | Liver | Homeostasis | Lipids | Transplantation | Adipocytes | Kinases | Phosphatase | Body composition | Genotype & phenotype | Transgenic animals | Rodents | Sterol regulatory element-binding protein | Fibroblasts | Fibroblast growth factor receptor 1 | Growth factors | Recombinant | Obesity | Congenital diseases | Diabetes mellitus | Pharmacology | Gene expression | Immunological tolerance | Cholesterol | Glucose tolerance | Signaling | Overexpression | Lipodystrophy | Insulin resistance | Diabetes | Metabolic disorders | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2009, Volume 119, Issue 3, pp. 512 - 523
Pulmonary hypertension (PH) is a progressive, lethal lung disease characterized by pulmonary artery SMC (PA-SMC) hyperplasia. leading to right-sided heart... 
MEDICINE, RESEARCH & EXPERIMENTAL | INHIBITION | SUBENDOTHELIAL EXTRACELLULAR-MATRIX | FACTOR-2 | GENE-EXPRESSION | RATS | MONOCROTALINE | FIBROBLAST-GROWTH-FACTOR | SMOOTH-MUSCLE-CELLS | LUNG-CANCER CELLS | FACTOR RECEPTOR | RNA, Small Interfering - genetics | Humans | Hypertension, Pulmonary - physiopathology | Fibroblast Growth Factor 1 - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Muscle, Smooth, Vascular - physiopathology | Hypertension, Pulmonary - prevention & control | Endothelium, Vascular - physiology | In Situ Hybridization | Cell Division | Fibroblast Growth Factor 1 - physiology | Fibroblast Growth Factor 2 - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | RNA, Messenger - genetics | Cells, Cultured | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Rats | Lung - physiopathology | Pulmonary Artery - physiopathology | Lung - physiology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Disease Progression | Pulmonary Artery - physiology | Animals | Fibroblast Growth Factor 2 - genetics | Hypertension, Pulmonary - pathology | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Pulmonary hypertension | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Science translational medicine, ISSN 1946-6234, 12/2011, Volume 3, Issue 113, pp. 113 - ra126
Journal Article
Cancer Research, ISSN 0008-5472, 03/2010, Volume 70, Issue 5, pp. 2085 - 2094
Journal Article