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Journal Article
Cancer Science, ISSN 1347-9032, 11/2016, Volume 107, Issue 11, pp. 1667 - 1676
Fibroblast growth factor receptor ( FGFR ) gene alterations are relatively frequent in lung squamous cell carcinoma ( LSCC ) and are a potential targets for... 
nintedanib | Copy number gain | FGFR | lung squamous cell cancer | next‐generation sequencing | next-generation sequencing | FGFR1 | CONTROLLED-TRIAL | BLADDER-CANCER | BIBF 1120 | AMPLIFICATION | ONCOLOGY | RANDOMIZED PHASE-II | GASTRIC-CANCER | TRIPLE ANGIOKINASE INHIBITOR | THERAPEUTIC TARGET | DOUBLE-BLIND | MUTATIONS | Lung Neoplasms - genetics | Lung Neoplasms - drug therapy | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Mutant Proteins - genetics | Neoplasm Recurrence, Local | Lung Neoplasms - pathology | DNA Copy Number Variations - genetics | Molecular Targeted Therapy | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Xenograft Model Antitumor Assays | Animals | Carcinoma, Squamous Cell - drug therapy | Survival Analysis | Female | Indoles - pharmacology | Indoles - therapeutic use | Cell Proliferation - drug effects | Mice | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Receptor, Fibroblast Growth Factor, Type 2 - genetics | RNA sequencing | Squamous cell carcinoma | Relapse | Analysis | Genes | Genetic research | Genetic aspects | Nucleotide sequencing | Lung cancer, Non-small cell | DNA sequencing | Cancer | Diseases | Fibroblast growth factor | Copy number | Lung cancer | Clinical trials | Values | Kinases | Signal transduction | Cell growth | Surgery | Fibroblast growth factor receptor 4 | Fibroblasts | Fibroblast growth factor receptor 1 | Vascular endothelial growth factor | Fibroblast growth factor receptor 2 | Medical research | Immunoglobulins | Small cell lung carcinoma | Research funding | Committees | Extracellular signal-regulated kinase | Lung carcinoma | Patients | Gene amplification | Medical prognosis | Cell lines | Antitumor activity | Mutation | University faculty | Laboratory animals | Esophageal cancer | Pharmaceuticals | Fibroblast growth factor receptors | Tumors | Index Medicus | Original
Journal Article
Kidney International, ISSN 0085-2538, 05/2014, Volume 85, Issue 5, pp. 1103 - 1111
Fibroblast growth factor 23 (FGF23) is a phosphate-regulating hormone that acts primarily on the kidney and parathyroid. With declining kidney function there... 
phosphate | chronic kidney disease | Klotho | fibroblast growth factor 23 | vascular calcification | Vascular calcification | Chronic kidney disease | Fibroblast growth factor 23 | Phosphate | VITAMIN-D | AORTIC CALCIFICATION | OSTEOBLAST DIFFERENTIATION | CARDIOVASCULAR EVENTS | FGF RECEPTOR 1 | ENDOTHELIAL-CELLS | ARTERIAL CALCIFICATION | HEMODIALYSIS-PATIENTS | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | Phosphorylation | Muscle, Smooth, Vascular - metabolism | Glucuronidase - metabolism | Myocytes, Smooth Muscle - pathology | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Aorta - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Renal Insufficiency, Chronic - complications | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Aortic Diseases - chemically induced | Aortic Diseases - metabolism | Glucuronidase - deficiency | Dose-Response Relationship, Drug | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Vascular Calcification - metabolism | Transfection | Phosphates - toxicity | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Aorta - drug effects | Osteoblasts - drug effects | Cells, Cultured | Vascular Calcification - pathology | Rats, Sprague-Dawley | Aorta - pathology | Osteoblasts - pathology | Muscle, Smooth, Vascular - pathology | Animals | Glucuronidase - genetics | Signal Transduction - drug effects | Cell Differentiation - drug effects | Vascular Calcification - chemically induced | Recombinant Proteins - toxicity | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Osteoblasts - metabolism | Fibroblast Growth Factors - toxicity | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2007, Volume 117, Issue 10, pp. 2766 - 2777
Tumors produce multiple growth factors, but little is known about the interplay between various angiogenic factors in promoting tumor angiogenesis, growth, and... 
MEDICINE, RESEARCH & EXPERIMENTAL | INHIBITION | THERAPY | ALPHA-RECEPTOR | LYMPHANGIOGENESIS | FIBROBLAST-GROWTH-FACTOR | EMERGING MECHANISMS | CELL-GROWTH | CANCER | LYMPHATIC METASTASIS | BETA-RECEPTOR | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Humans | Fibroblast Growth Factor 2 - pharmacology | Endothelium, Vascular - drug effects | Fibrosarcoma - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Platelet-Derived Growth Factor - genetics | Fibrosarcoma - pathology | Lung Neoplasms - secondary | Pericytes - pathology | Fibroblast Growth Factor 2 - metabolism | Capillaries | Fibrosarcoma - blood | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Signal Transduction | Proto-Oncogene Proteins c-sis | Pericytes - metabolism | Rats | Platelet-Derived Growth Factor - pharmacology | Mice, SCID | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Fibroblast Growth Factor 2 - genetics | Endothelium, Vascular - metabolism | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Cell Movement | Complications and side effects | Causes of | Development and progression | Metastasis | Research | Neovascularization | Growth factors | Index Medicus | Abridged Index Medicus | MEDICIN OCH HÄLSOVETENSKAP | MEDICAL AND HEALTH SCIENCES
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 34, pp. 15140 - 15144
Between the genetic extremes of rare monogenic and common polygenic diseases lie diverse oligogenic disorders involving mutations in more than one locus in... 
Deficiency diseases | Sex hormones | Nervous system diseases | Disease models | Medical genetics | Genetic diseases | Genetic mutation | Kallmann syndrome | Human genetics | Hypogonadism | Rare variant | Digenic | Idiopathic hypogonadotropic hypogonadism | FGFR1 | idiopathic hypogonadotropic hypogonadism | GPR54 | MULTIDISCIPLINARY SCIENCES | CLINICAL HETEROGENEITY | MISSENSE MUTATIONS | PUBERTY | digenic | GENE | BARDET-BIEDL-SYNDROME | LINKED KALLMANN-SYNDROME | FIBROBLAST-GROWTH-FACTOR-RECEPTOR-1 | GNRH RECEPTOR | rare variant | Receptors, LHRH - genetics | Humans | Male | Gene Regulatory Networks | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Case-Control Studies | Receptors, Peptide - genetics | Genetic Variation | DNA Mutational Analysis | Female | Neuropeptides - genetics | Fibroblast Growth Factor 8 - genetics | Kallmann Syndrome - genetics | Extracellular Matrix Proteins - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Hypogonadism - genetics | Homozygote | Phenotype | Pedigree | Gastrointestinal Hormones - genetics | Heterozygote | Models, Genetic | Receptors, G-Protein-Coupled - genetics | Mutation | Receptors, Kisspeptin-1 | Gonadotropin-Releasing Hormone - deficiency | Cohort Studies | Multifactorial diseases | Genetic aspects | Hormones | Gonadotropin | Health aspects | Pathogens | Genetics | Genetic disorders | Pathogenesis | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e57284 - e57284
Fibroblast growth factor receptors (FGFRs) are activated by mutation and overexpressed in bladder cancers (BCs), and FGFR inhibitors are currently being... 
TRANSITIONAL-CELL CARCINOMA | MULTIDISCIPLINARY SCIENCES | MUTATION | KINASE | SENSITIVITY | MICE | INHIBITOR | FGFR3 | EXPRESSION | TUMORS | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Humans | Gene Expression Profiling | DNA Primers | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Receptor, Fibroblast Growth Factor, Type 1 - physiology | Blotting, Western | Cell Division - drug effects | Cell Division - physiology | Receptor, Fibroblast Growth Factor, Type 3 - physiology | Neoplasm Metastasis | Animals | RNA Interference | Mice, Nude | Base Sequence | Urinary Bladder Neoplasms - pathology | Cell Line, Tumor | Female | Mice | Mutation | Real-Time Polymerase Chain Reaction | RNA | Stem cells | Fibroblast growth factors | Genetic aspects | Metastasis | Health aspects | Bladder cancer | Vimentin | Cell proliferation | Fibroblast growth factor | Mesenchyme | Clinical trials | Bladder | Biology | mRNA | Kinases | E-cadherin | Urology | Lymph nodes | Metastases | Heterogeneity | Signal transduction | Receptors | Biological effects | Urinary bladder | Fibroblasts | Fibroblast growth factor receptor 1 | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Tumor cells | RNA-mediated interference | Growth factor receptors | Gene expression | Patients | Studies | Inhibitors | Medical prognosis | Cell lines | Laboratory animals | Cancer | Fibroblast growth factor receptors | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2006, Volume 103, Issue 16, pp. 6281 - 6286
Journal Article
International Journal of Molecular Medicine, ISSN 1107-3756, 11/2017, Volume 40, Issue 5, pp. 1477 - 1485
Fibroblast growth factor 21 (FGF21), as an endocrine factor, is secreted into circulation by injured cardiomyocytes. Endoplasmic reticulum (ER) stress-induced... 
endo plasmic reticulum stress | cardiomyocytes | fibroblast growth factor 21 | apoptosis | extracellular signal-regulated kinase 1/2 | fibroblast growth factor receptor 1 | Cardiomyocytes | Fibroblast growth factor 21 | Fibroblast growth factor receptor 1 | Extracellular signal-regulated kinase 1/2 | Apoptosis | Endoplasmic reticulum stress | INDUCED CELL-DEATH | BETA-KLOTHO | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | MITOCHONDRIAL | ER-STRESS | INJURY | DEPENDENT APOPTOSIS | endo-plasmic reticulum stress | DISEASE | ISCHEMIA-REPERFUSION | EXPRESSION | Cell Line | Gene Expression | Phosphorylation | Endoplasmic Reticulum Stress - drug effects | Signal Transduction | Apoptosis - drug effects | Cell Survival | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Rats | Apoptosis - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Fibroblast Growth Factors - metabolism | Animals | Myocytes, Cardiac - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Tunicamycin - pharmacology | Myocytes, Cardiac - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Care and treatment | Development and progression | Fibroblast growth factors | Cellular signal transduction | Genetic aspects | Gene expression | Cardiovascular diseases | Health aspects | Heart failure | Immunoglobulins | Cardiovascular disease | Kinases | Studies | Proteins | Ischemia | Fibroblasts | Growth factors | Endoplasmic reticulum | extracellular signal-regulated kinase 1
Journal Article
Journal Article