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PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0122961
Background Fibroblast growth factor receptor 4 (FGFR4) polymorphisms are positively correlated with tumor progression in numerous malignant tumors. However,... 
HEPATOCELLULAR-CARCINOMA | HUMAN BREAST | RISK-FACTORS | HEPATITIS | PATHOLOGICAL DEVELOPMENT | GASTRIC-CANCER | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | FGFR4 | GENE POLYMORPHISMS | EXPRESSION | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Liver Neoplasms - complications | Male | Case-Control Studies | alpha-Fetoproteins - analysis | Carcinoma, Hepatocellular - complications | DNA - analysis | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Liver Cirrhosis - genetics | Real-Time Polymerase Chain Reaction | Liver Neoplasms - genetics | Liver Cirrhosis - complications | Risk Factors | Genotype | DNA - isolation & purification | Alleles | Carcinoma, Hepatocellular - pathology | Taiwan | Liver Cirrhosis - pathology | Aged | Polymorphism, Single Nucleotide | Neoplasm Staging | Development and progression | Genetic aspects | Single nucleotide polymorphisms | Research | Hepatoma | Liver cirrhosis | Risk factors | Fibroblast growth factor receptors | Fibroblast growth factor | Liver | Hepatocellular carcinoma | Alcohol | Infections | Emergency medical care | Mitochondrial DNA | Single-nucleotide polymorphism | Metastasis | Kinases | Gene polymorphism | Cancer therapies | Stomach cancer | Liver cancer | Hepatitis | Surgery | Fibroblast growth factor receptor 4 | Fibroblasts | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Risk analysis | Gene expression | α-Fetoprotein | Patients | Medicine | Genetic variance | Cirrhosis | Hospitals | Medical prognosis | Viral infections | Tumors | Polymorphism | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 08/2015, Volume 5, Issue 1, pp. 13462 - 13462
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 12/2006, Volume 45, Issue 12, pp. 934 - 942
Inappropriate fibroblast growth factor (FGF) signaling is involved in most tissue‐specific pathologies including cancer. Previously we showed that... 
growth factors | liver adenoma | hepatoma models | receptor switching | transgenic models | tyrosine kinases | hepatocellular carcinoma | Receptor switching | Transgenic models | Liver adenoma | Hepatoma models | Tyrosine kinases | Hepatocellular carcinoma | Growth factors | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | FIBROBLAST-GROWTH-FACTOR | PROLIFERATION | CANCER | REGULATOR | ONCOLOGY | AUTOCRINE | LIVER | HEPARAN-SULFATE | MICE | RECEPTORS | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Carcinoma, Hepatocellular - chemically induced | Transcriptional Activation | Fibroblast Growth Factors - genetics | Liver Neoplasms - chemically induced | RNA, Messenger - analysis | Hepatocytes - pathology | Cell Transformation, Neoplastic - chemically induced | Fibroblast Growth Factor 1 - genetics | Fibroblast Growth Factor 1 - antagonists & inhibitors | Hepatocytes - metabolism | RNA, Messenger - metabolism | Cell Transformation, Neoplastic - genetics | Carcinoma, Hepatocellular - genetics | Fibroblast Growth Factor 1 - physiology | Liver Neoplasms - pathology | Fibroblast Growth Factor 2 - physiology | Fibroblast Growth Factors - physiology | Gene Targeting | Liver Neoplasms - genetics | Mice, Transgenic | Carbon Tetrachloride - toxicity | Fibroblast Growth Factor 2 - antagonists & inhibitors | Animals | Fibroblast Growth Factor 2 - genetics | Receptor, Fibroblast Growth Factor, Type 4 - agonists | Carcinoma, Hepatocellular - pathology | Mice | Fibroblast Growth Factors - antagonists & inhibitors | Cell Transformation, Neoplastic - pathology
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63695
Fibroblast growth factor receptor 4 (FGFR4) is vital in early development and tissue repair. FGFR4 expression levels are very restricted in adult tissues,... 
HEPATOCELLULAR-CARCINOMA | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | IN-VIVO | T(4/14) MULTIPLE-MYELOMA | GLY388ARG POLYMORPHISM | LUNG ADENOCARCINOMA | FIBROBLAST-GROWTH-FACTOR | CELL-LINES | RECEPTOR 4 | SOMATIC MUTATIONS | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Cell Adhesion - genetics | Cell Proliferation | Colorectal Neoplasms - genetics | Epithelial-Mesenchymal Transition - physiology | Humans | Apoptosis - genetics | Epithelial-Mesenchymal Transition - genetics | Blotting, Western | Animals | Cell Adhesion - physiology | Mice, Nude | Cell Line, Tumor | Polymorphism, Single Nucleotide - genetics | Mice | Apoptosis - physiology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Real-Time Polymerase Chain Reaction | Colorectal Neoplasms - metabolism | Viral antibodies | Stem cells | Colorectal cancer | Antibodies | Fibroblast growth factors | Transforming growth factors | Gene expression | Health aspects | AKT protein | Metastasis | Proteins | Angiogenesis | Signal transduction | Reversion | Animal tissues | Fibroblasts | Tumorigenesis | Growth factors | Polyclonal antibodies | Survival | Ligands | Solid tumors | Mutation | Cell migration | Cell proliferation | Fibroblast growth factor | Biotechnology | Mesenchyme | Laboratories | Colorectal carcinoma | Multiple myeloma | Bladder | Kinases | E-cadherin | Cell adhesion & migration | Colon cancer | Pathways | Rodents | Fibroblast growth factor receptor 4 | Colon | Cell survival | Invasiveness | Extracellular signal-regulated kinase | Tumor cell lines | Medicine | Signaling | Gene amplification | Medical prognosis | Cell lines | Prostate cancer | Chemokines | Tumors | Cancer | Apoptosis | Apoptosi | Proliferació | Cèl·lules
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 02/2016, Volume 310, Issue 4, pp. E289 - E300
Journal Article
Journal Article
Journal Article