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Journal of Hepatology, ISSN 0168-8278, 2010, Volume 52, Issue 4, pp. 550 - 559
Background & Aims IGF signaling has a relevant role in a variety of human malignancies. We analyzed the underlying molecular mechanisms of IGF signaling... 
Gastroenterology and Hepatology | IGF signaling | Hepatocellular carcinoma | A12 | Molecular therapy | miR-100 | SIGNALING PATHWAYS | RECEPTOR | MONOCLONAL-ANTIBODY | PROLIFERATION | FACTOR-II GENE | INSULIN | INHIBITION | GROWTH | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | MICRORNA | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Hepatocytes - pathology | MicroRNAs - metabolism | Liver Neoplasms - therapy | Insulin Receptor Substrate Proteins - metabolism | Receptor, IGF Type 2 - metabolism | Receptor, IGF Type 2 - genetics | Insulin-Like Growth Factor II - genetics | Carcinoma, Hepatocellular - genetics | Insulin Receptor Substrate Proteins - genetics | Hepatocytes - physiology | Insulin-Like Growth Factor Binding Proteins - genetics | Liver Neoplasms - genetics | Antibodies, Monoclonal - pharmacology | Receptor, IGF Type 1 - genetics | Insulin-Like Growth Factor II - metabolism | Hep G2 Cells | Cell Division - physiology | Insulin-Like Growth Factor Binding Proteins - metabolism | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Liver Neoplasms - metabolism | Receptor, IGF Type 1 - immunology | Insulin-Like Growth Factor Binding Protein 3 | Signal Transduction - physiology | Carcinoma, Hepatocellular - therapy | Mice | Mice, Inbred BALB C | Apoptosis - physiology | Carcinoma, Hepatocellular - metabolism | Liver cancer | Analysis | Transplantation of organs, tissues, etc | Hepatoma | Hepatitis C | Vascular endothelial growth factor | Endothelium | Protein binding | molecular therapy | hepatocellular carcinoma
Journal Article
Development, ISSN 0950-1991, 05/2011, Volume 138, Issue 9, pp. 1795 - 1805
Journal Article
Journal Article
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 10/2011, Volume 589, Issue 19, pp. 4709 - 4722
Non‐Technical Summary  Cardiovascular disease is responsible for 30% of deaths worldwide and epidemiological data demonstrate that poor growth before birth is... 
PLACENTAL GROWTH | ADULT DISEASE | HYPERTROPHY | IGF-I | PHYSIOLOGY | FACTOR-II | MESSENGER-RNA | SHEEP FETUS | LATE-GESTATION | GENE-EXPRESSION | NEUROSCIENCES | BLOOD-PRESSURE | Receptor, IGF Type 1 - metabolism | Gene Expression - genetics | Heart - embryology | Fetus - metabolism | Heart Ventricles - embryology | Fetal Growth Retardation - genetics | Insulin-Like Growth Factor I - genetics | RNA, Messenger - metabolism | Receptor, IGF Type 2 - metabolism | Receptor, IGF Type 2 - genetics | DNA Methylation | Insulin-Like Growth Factor II - genetics | Myocardium - metabolism | Insulin-Like Growth Factor II - biosynthesis | Signal Transduction | Heart - growth & development | RNA, Messenger - genetics | Fetal Growth Retardation - metabolism | Receptor, IGF Type 1 - genetics | Insulin-Like Growth Factor II - metabolism | Animals | Heart Ventricles - growth & development | Fetus - embryology | Myocytes, Cardiac - metabolism | Sheep | Heart Ventricles - metabolism | Cardiomegaly - genetics | Insulin-Like Growth Factor I - metabolism | Cardiomegaly - metabolism | Messenger RNA | Genetic research | Physiological aspects | Glucose | Gene expression | Methylation | Coronary heart disease | Dextrose | Heart | Rodents | Fetuses | DNA methylation | Cardiovascular disease | Insulin-like growth factors | Birth weight | Insulin | Cardiovascular
Journal Article
Biochemical Journal, ISSN 0264-6021, 03/2017, Volume 474, Issue 5, pp. 771 - 780
We have recently shown that hypoxia and Akt-induced stem cell factor (HASF) protects the heart from ischemia-induced damage and promotes cardiomyocyte... 
HEART | MORTALITY | HYPERTROPHY | APOPTOSIS | PROTEIN | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | FAILURE | MESENCHYMAL STEM-CELLS | ELDERLY POPULATION | TRANSGENIC MICE | RNA, Small Interfering - genetics | Receptor, IGF Type 1 - metabolism | Heart Ventricles - cytology | Phosphorylation | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Adaptor Proteins, Vesicular Transport - genetics | Adaptor Proteins, Vesicular Transport - metabolism | Receptor, IGF Type 2 - metabolism | Receptor, IGF Type 2 - genetics | Receptor, Insulin - genetics | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Membrane Proteins - metabolism | Binding Sites | Animals, Newborn | Mitogen-Activated Protein Kinase 3 - genetics | Myocytes, Cardiac - cytology | Signal Transduction | Membrane Proteins - genetics | Adaptor Proteins, Vesicular Transport - antagonists & inhibitors | Gene Expression Regulation | Rats | Pyrimidines - pharmacology | Rats, Sprague-Dawley | Receptor, IGF Type 1 - genetics | Receptor, IGF Type 2 - antagonists & inhibitors | Two-Hybrid System Techniques | Pyrroles - pharmacology | Animals | Myocytes, Cardiac - drug effects | Membrane Proteins - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - metabolism | Myocytes, Cardiac - metabolism | Protein Binding | Ligands | Receptor, Insulin - metabolism | Cell Proliferation - drug effects | Heart Ventricles - metabolism | Mice | Primary Cell Culture | Heart Ventricles - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism
Journal Article