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1. Full Text Blockade of Chronic Type I Interferon Signaling to Control Persistent LCMV Infection
by Elizabeth B. Wilson and Douglas H. Yamada and Heidi Elsaesser and Jonathan Herskovitz and Jane Deng and Genhong Cheng and Bruce J. Aronow and Christopher L. Karp and David G. Brooks
Science, ISSN 0036-8075, 4/2013, Volume 340, Issue 6129, pp. 202 - 207
Type I interferons (IFN-I) are critical for antiviral immunity; however, chronic IFN-I signaling is associated with hyperimmune activation and disease... 
T lymphocytes | Isotypes | Lymphocytic choriomeningitis virus | Genes | REPORTS | Blocking antibodies | Antivirals | Antibodies | Viruses | Infections | Immunity | GAMMA | MULTIDISCIPLINARY SCIENCES | CHRONIC HEPATITIS-C | INTERLEUKIN-10 RECEPTOR | HUMAN-IMMUNODEFICIENCY-VIRUS | CHRONIC VIRAL-INFECTION | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | EXPRESSION | T-CELLS | IL-10 | PROGRESSION | Lymphocytic choriomeningitis virus - immunology | Arenaviridae Infections - virology | Oligonucleotide Array Sequence Analysis | Dendritic Cells - immunology | Gene Expression Profiling | Interferon-gamma - metabolism | Interferon Type I - immunology | Receptor, Interferon alpha-beta - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Antibodies - immunology | Interferon Type I - metabolism | Interleukin-10 - metabolism | Lymphocytic choriomeningitis virus - physiology | Receptor, Interferon alpha-beta - genetics | Arenaviridae Infections - immunology | Cytokines - immunology | Receptor, Interferon alpha-beta - metabolism | Cytokines - metabolism | Signal Transduction | Immune Tolerance | B7-H1 Antigen - metabolism | Animals | Virus Replication | Interferon-gamma - immunology | Interferon Type I - genetics | Mice | Signal transduction | Interferon | Viral infections | Immune system | Chronic illnesses | Human | HIV | Organs | Blocking | Hepatitis C virus
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2. Full Text Mapping and Role of the CD8+ T Cell Response During Primary Zika Virus Infection in Mice
by Elong Ngono, Annie and Vizcarra, Edward A and Tang, William W and Sheets, Nicholas and Joo, Yunichel and Kim, Kenneth and Gorman, Matthew J and Diamond, Michael S and Shresta, Sujan
Cell Host & Microbe, ISSN 1931-3128, 01/2017, Volume 21, Issue 1, pp. 35 - 46
CD8 T cells may play a dual role in protection against and pathogenesis of flaviviruses, including Zika virus (ZIKV). We evaluated the CD8 T cell response in... 
LysMCre+IFNARfl/fl | CD8+ T cell | peptide | Zika virus | mouse model | epitope | LysMCre | CD8 | IFNAR | T cell | ANTIBODIES | WESTERN-HEMISPHERE | DENGUE HEMORRHAGIC-FEVER | MICROBIOLOGY | SEXUAL TRANSMISSION | MOUSE MODELS | PATHOGENESIS | INTERFERON | REPLICATION | VIROLOGY | PROTECTIVE ROLE | DISEASE | PARASITOLOGY | Zika Virus - immunology | CD8-Positive T-Lymphocytes - transplantation | Mice, Inbred C57BL | Adoptive Transfer | Receptor, Interferon alpha-beta - antagonists & inhibitors | Mice, Knockout | Zika Virus Infection - virology | Animals | Receptor, Interferon alpha-beta - immunology | Antibodies, Blocking - immunology | Zika Virus Infection - immunology | Mice | Receptor, Interferon alpha-beta - genetics | Epitopes, T-Lymphocyte - immunology | CD8-Positive T-Lymphocytes - immunology | Disease Models, Animal | LysMCre+IFNARfl | CD8+ T cell
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3. Full Text MicroRNA-30c targets the interferon-alpha/beta receptor beta Chain to promote type 2 PRRSV infection
by Liu, Fang and Wang, Honglei and Du, Li and Wei, Zeyu and Zhang, Qiong and Feng, Wen-Hai
Journal of General Virology, ISSN 0022-1317, 12/2018, Volume 99, Issue 12, pp. 1671 - 1680
Porcine reproductive and respiratory syndrome (PRRS) is one of the most important diseases in pigs. MicroRNAs (miRNAs) have emerged as an important regulator... 
IFNAR2 | IFN-I | miR-30c | PRRSV | IMMUNITY | RNA | INNATE | SWINE INFERTILITY | REPLICATION | VIROLOGY | RESPIRATORY SYNDROME VIRUS | GENE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EXPRESSION | INTRACELLULAR DOMAIN | Porcine respiratory and reproductive syndrome virus - growth & development | Macrophages, Alveolar - virology | Porcine respiratory and reproductive syndrome virus - immunology | Cells, Cultured | Gene Expression Regulation | MicroRNAs - metabolism | Swine Diseases | Receptor, Interferon alpha-beta - antagonists & inhibitors | Host-Pathogen Interactions | Animals | Immune Evasion | Swine | Porcine respiratory and reproductive syndrome virus - pathogenicity
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4. Full Text Blocking type I interferon signaling enhances T cell recovery and reduces HIV-1 reservoirs
by Cheng, Liang and Ma, Jianping and Li, Jingyun and Li, Dan and Li, Guangming and Li, Feng and Zhang, Qing and Yu, Haisheng and Yasui, Fumihiko and Ye, Chaobaihui and Tsao, Li-Chung and Hu, Zhiyuan and Su, Lishan and Zhang, Liguo
Journal of Clinical Investigation, ISSN 0021-9738, 01/2017, Volume 127, Issue 1, pp. 269 - 279
Despite the efficient suppression of HIV-1 replication that can be achieved with combined antiretroviral therapy (cART), low levels of type I interferon... 
MEDICINE, RESEARCH & EXPERIMENTAL | LATENT HIV-1 | HEPATITIS-C THERAPY | COINFECTED PATIENTS | VIRUS | PERSISTENT LCMV INFECTION | CD4(+) | ANTIRETROVIRAL THERAPY | SIV INFECTION | RHESUS MACAQUES | HUMANIZED MICE | Recovery of Function - genetics | Recovery of Function - drug effects | HIV Infections - genetics | Humans | Anti-Retroviral Agents - pharmacology | Signal Transduction - genetics | Interferon Type I - immunology | Receptor, Interferon alpha-beta - antagonists & inhibitors | HIV Infections - immunology | Signal Transduction - immunology | HIV-1 - immunology | Recovery of Function - immunology | Animals | HIV Infections - pathology | Receptor, Interferon alpha-beta - immunology | Signal Transduction - drug effects | Mice, Mutant Strains | Interferon Type I - genetics | HIV Infections - drug therapy | Mice | Receptor, Interferon alpha-beta - genetics | Chronic Disease | Disease Models, Animal | Care and treatment | Interferon | T cells | HIV infection | Analysis | Risk factors | Studies | Acquired immune deficiency syndrome--AIDS | Pathogenesis | Lymphocytes | Human immunodeficiency virus--HIV | Software | Infections | Laboratory animals | Gene expression
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5. Full Text Anti-interferon alpha treatment in SLE
by Kirou, Kyriakos A and Gkrouzman, Elena
Clinical Immunology, ISSN 1521-6616, 2013, Volume 148, Issue 3, pp. 303 - 312
Abstract Several studies in the last decade have highlighted the role of the type I interferon (IFN-I) pathway, and particularly interferon alpha (IFNα) in SLE... 
Allergy and Immunology | Clinical trial | Plasmacytoid dendritic cells | Systemic lupus erythematosus | Interferon alpha | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ANTI-INTERFERON | IMMUNOLOGY | I INTERFERON | DISEASE-ACTIVITY | INTERFERON-ALPHA | CLINICAL-MANIFESTATIONS | DOUBLE-BLIND | PHASE-I | IFN-ALPHA | Genetic Predisposition to Disease | Animals | Signal Transduction | Humans | Interferon-alpha - antagonists & inhibitors | Lupus Erythematosus, Systemic - genetics | Lupus Erythematosus, Systemic - drug therapy | Lupus Erythematosus, Systemic - immunology | Dendritic Cells - physiology | Lupus Erythematosus, Systemic - etiology | Receptor, Interferon alpha-beta - antagonists & inhibitors | Interferon-alpha - physiology | Dendritic cells | Drug therapy | Biological response modifiers
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6. Full Text Type I interferon blockade in systemic lupus erythematosus: Where do we stand?
by Lauwerys, Bernard R and Ducreux, Julie and Houssiau, Frédéric A
Rheumatology (United Kingdom), ISSN 1462-0324, 2014, Volume 53, Issue 8, pp. 1369 - 1376
SLE is an autoimmune condition characterized by loss of tolerance to chromatin constituents and the production of ANAs. The majority of SLE patients display... 
Systemic lupus erythematosus | Type I interferon | Interferon-alpha | Sifalimumab | Interferon-alpha kinoid | Interferon blockade | Ontalizumab | ACTIVATION | INDUCIBLE GENE-EXPRESSION | AUTOREACTIVE B-CELLS | IFN-ALPHA INDUCER | RHEUMATOLOGY | sifalimumab | APOPTOTIC U937 CELLS | DISEASE-ACTIVITY | type I interferon | PLASMACYTOID DENDRITIC CELLS | systemic lupus erythematosus | CLINICAL-MANIFESTATIONS | DOUBLE-BLIND | interferon-alpha | rontalizumab | PHASE-I | interferon blockade | interferon-alpha kinoid | Antibodies, Monoclonal - therapeutic use | Humans | Receptor, Interferon alpha-beta - antagonists & inhibitors | Lupus Erythematosus, Systemic - drug therapy | Lupus Erythematosus, Systemic - immunology | Interferon Type I - antagonists & inhibitors
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7. Full Text Parkin and PINK1 mitigate STING-induced inflammation
by Sliter, Danielle A and Martinez, Jennifer and Hao, Ling and Chen, Xi and Sun, Nuo and Fischer, Tara D and Burman, Jonathon L and Li, Yan and Zhang, Zhe and Narendra, Derek P and Cai, Huaibin and Borsche, Max and Klein, Christine and Youle, Richard J
Nature, ISSN 0028-0836, 09/2018, Volume 561, Issue 7722, pp. 258 - 262
Although serum from patients with Parkinson's disease contains elevated levels of numerous pro-inflammatory cytokines including IL-6, TNF, IL-1 beta, and IFN... 
OXIDATIVE STRESS | ACTIVATION | MUTANTS | MULTIDISCIPLINARY SCIENCES | IN-VIVO MITOPHAGY | DISEASE | NEURONS | DEFICIENT MICE | MUTATIONS | MITOCHONDRIAL-DNA | Protein Kinases - metabolism | Protein Kinases - genetics | Humans | Stress, Physiological | Receptor, Interferon alpha-beta - antagonists & inhibitors | Membrane Proteins - deficiency | Inflammation - metabolism | DNA, Mitochondrial - genetics | Membrane Proteins - metabolism | Parkinson Disease - metabolism | Protein Kinases - deficiency | Physical Conditioning, Animal | Membrane Proteins - genetics | Mice, Inbred C57BL | Alarmins - metabolism | Ubiquitin-Protein Ligases - metabolism | Immunity, Innate | DNA, Mitochondrial - blood | Mitochondrial Degradation | Animals | Receptor, Interferon alpha-beta - immunology | Inflammation - genetics | Ubiquitin-Protein Ligases - deficiency | Inflammation - prevention & control | Mice | Ubiquitin-Protein Ligases - genetics | Inflammation | Genetic aspects | Research | Gene mutations | Risk factors | Ubiquitin | Heart | Cell culture | Oxidative stress | Animal models | Parkinson's disease | Substantia nigra | Parkinsons disease | Innate immunity | Mitochondrial DNA | Kinases | Immunity | Interleukin 6 | Mitochondria | Movement disorders | Ubiquitin-protein ligase | Deoxyribonucleic acid--DNA | Dopamine receptors | Enzymes | Phenotypes | Dopamine | Cytokines | Neurodegenerative diseases | Body temperature | Damage patterns | Insects | Tumor necrosis factor | PTEN-induced putative kinase | Proteomics | Interferon | Parkin protein | Mutation | Methods
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8. Full Text Advances in understanding the role of type I interferons in systemic lupus erythematosus
by Crow, Mary K
Current Opinion in Rheumatology, ISSN 1040-8711, 09/2014, Volume 26, Issue 5, pp. 467 - 474
PURPOSE OF REVIEWAdvances in understanding the genetic and molecular basis of innate immune system activation and function have supported the hypothesis that... 
systemic lupus erythematosus | autoimmunity | interferon-α | cytoplasmic sensors | Toll-like receptors | long interspersed nuclear elements | type I interferons | ALPHA MONOCLONAL-ANTIBODY | DNA SENSOR | CYCLIC GMP-AMP | REGULATED GENES | AICARDI-GOUTIERES SYNDROME | TREX1 | RHEUMATOLOGY | ENDOGENOUS RETROELEMENTS | PLASMACYTOID DENDRITIC CELLS | DOUBLE-BLIND | interferon-alpha | PHASE-I | Genetic Association Studies | Humans | Toll-Like Receptors - immunology | Neutrophils - immunology | Autoimmunity - genetics | Lupus Erythematosus, Systemic - etiology | Immunity, Innate | Lupus Erythematosus, Systemic - therapy | Receptor, Interferon alpha-beta - antagonists & inhibitors | Models, Immunological | Animals | Lupus Erythematosus, Systemic - immunology | Toll-Like Receptors - antagonists & inhibitors | Interferon Type I - genetics | Interferon Type I - metabolism | Mice | Interferon Type I - antagonists & inhibitors
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9. Full Text Modeling Arboviral Infection in Mice Lacking the Interferon Alpha/Beta Receptor
by Marín-Lopez, Alejandro and Marín-Lopez, Alejandro and Calvo-Pinilla, Eva and Calvo-Pinilla, Eva and Moreno, Sandra and Moreno, Sandra and Utrilla-Trigo, Sergio and Utrilla-Trigo, Sergio and Nogales, Aitor and Nogales, Aitor and Brun, Alejandro and Brun, Alejandro and Fikrig, Erol and Ortego, Javier and Ortego, Javier
Viruses, ISSN 1999-4915, 01/2019, Volume 11, Issue 1, p. 35
Arboviruses are arthropod-borne viruses that exhibit worldwide distribution and are a constant threat, not only for public health but also for wildlife,... 
IFNAR(−/−) mice model | pathology | interferon | vaccines | arbovirus | WEST-NILE-VIRUS | BLUETONGUE VIRUS | HORSE SICKNESS VIRUS | WHITE-TAILED DEER | IFNAR(-/-) mice model | VIROLOGY | MALE SEXUAL TRANSMISSION | IFNAR(-/-) MICE | MOUSE MODEL | VALLEY FEVER VIRUS | EPIZOOTIC HEMORRHAGIC-DISEASE | ZIKA VIRUS | Interferon-beta - immunology | Humans | Arboviruses - classification | Receptor, Interferon alpha-beta - antagonists & inhibitors | Interferon-alpha - immunology | Mice, Knockout | Animals | Virus Replication | Arboviruses - pathogenicity | Mice | Receptor, Interferon alpha-beta - genetics | Arbovirus Infections - immunology | Disease Models, Animal
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10. Full Text Hemagglutinin of influenza A virus antagonizes type I interferon (IFN) responses by inducing degradation of type I IFN receptor 1
by Xia, Chuan and Vijayan, Madhuvanthi and Pritzl, Curtis J and Fuchs, Serge Y and McDermott, Adrian B and Hahm, Bumsuk
Journal of Virology, ISSN 0022-538X, 2016, Volume 90, Issue 5, pp. 2403 - 2417
Influenza A virus (IAV) employs diverse strategies to circumvent type I interferon (IFN) responses, particularly by inhibiting the synthesis of type I IFNs.... 
NS1 PROTEIN | VIROLOGY | DENDRITIC CELLS | C-PROTEIN | SURFACE EXPRESSION | DOWN-REGULATION | ALPHA RECEPTOR | INFECTED-CELLS | V-PROTEIN | SENSOR RIG-I | SUBUNIT IFNAR1 | Cell Line | Influenza A virus - physiology | Phosphorylation | Humans | Hemagglutinin Glycoproteins, Influenza Virus - metabolism | Receptor, Interferon alpha-beta - antagonists & inhibitors | Host-Pathogen Interactions | Ubiquitination | Animals | Immune Evasion | Proteolysis | Interferon Type I - metabolism | Influenza A virus - immunology | Protein Processing, Post-Translational
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11. Full Text STAT3 inhibition enhances the therapeutic efficacy of immunogenic chemotherapy by stimulating type 1 interferon production by cancer cells
by Yang, Heng and Yamazaki, Takahiro and Pietrocola, Federico and Zhou, Heng and Zitvogel, Laurence and Ma, Yuting and Kroemer, Guido
Cancer Research, ISSN 0008-5472, 09/2015, Volume 75, Issue 18, pp. 3812 - 3822
STAT3 is an oncogenic transcription factor with potent immunosuppressive functions. We found that pharmacologic inhibition of STAT3 or its selective knockout... 
IMMUNITY | RECRUITMENT | ACTIVATION | ONCOLOGY | IMMUNOTHERAPY | RESISTANCE | DEATH | DIFFERENTIATION | TRANSCRIPTION FACTOR STAT3 | TUMORS | T-CELLS | Doxorubicin - therapeutic use | Cyclic S-Oxides - administration & dosage | Antibiotics, Antineoplastic - pharmacology | Chemokine CXCL9 - biosynthesis | Dendritic Cells - immunology | Neoplasm Proteins - physiology | Transcriptional Activation | Fibrosarcoma - immunology | Neoplasm Proteins - antagonists & inhibitors | Receptor, Interferon alpha-beta - antagonists & inhibitors | Interferon Type I - biosynthesis | Chemokine CXCL10 - biosynthesis | Colorectal Neoplasms - therapy | STAT3 Transcription Factor - physiology | Colorectal Neoplasms - drug therapy | Female | Cyclic S-Oxides - pharmacology | Immunocompetence | Receptors, CXCR3 - antagonists & inhibitors | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | STAT3 Transcription Factor - genetics | Doxorubicin - administration & dosage | T-Lymphocytes, Cytotoxic - immunology | Neoplasm Proteins - biosynthesis | Mice, Inbred C57BL | Fibrosarcoma - therapy | Combined Modality Therapy | Chemokine CXCL9 - genetics | Antibiotics, Antineoplastic - administration & dosage | Drug Synergism | Cyclic S-Oxides - therapeutic use | Animals | Colorectal Neoplasms - immunology | Antibiotics, Antineoplastic - therapeutic use | Signal Transduction - drug effects | Chemokine CXCL10 - genetics | Mice, Nude | Receptors, CXCR3 - physiology | Cell Line, Tumor | Interferon Type I - genetics | Mice | Mice, Inbred BALB C | STAT3 Transcription Factor - antagonists & inhibitors | Doxorubicin - pharmacology | Fibrosarcoma - drug therapy | Lymphocytes, Tumor-Infiltrating - immunology
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12. Full Text Endothelial progenitor dysfunction associates with a type I interferon signature in primary antiphospholipid syndrome
by Grenn, Robert C and Yalavarthi, Srilakshmi and Gandhi, Alex A and Kazzaz, Nayef M and Núñez-Álvarez, Carlos and Hernández-Ramírez, Diego and Cabral, Antonio R and McCune, W Joseph and Bockenstedt, Paula L and Knight, Jason S
Annals of the Rheumatic Diseases, ISSN 0003-4967, 02/2017, Volume 76, Issue 2, pp. 450 - 457
ObjectivesPatients with antiphospholipid syndrome (APS) are at risk for subclinical endothelial injury, as well as accelerated atherosclerosis. In the related... 
Cytokines | Antiphospholipid Syndrome | Atherosclerosis | CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | NEUTROPHIL EXTRACELLULAR TRAPS | ANTICARDIOLIPIN ANTIBODIES | AUTOIMMUNE-DISEASES | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | ALPHA | PERIPHERAL-BLOOD | RHEUMATOLOGY | HEPATITIS-C | Endothelial Progenitor Cells - drug effects | Humans | Middle Aged | Antibodies, Neutralizing - pharmacology | Male | Interferon Type I - immunology | Case-Control Studies | Receptor, Interferon alpha-beta - antagonists & inhibitors | Interferon-alpha - immunology | Antiphospholipid Syndrome - immunology | Young Adult | Endothelial Progenitor Cells - physiology | Flow Cytometry | Cell Differentiation - drug effects | Leukocytes, Mononuclear - immunology | Adult | Female | Aged | Cell Differentiation - physiology | Antiphospholipid Syndrome - physiopathology | Complications and side effects | Interferon | Research | Antiphospholipid syndrome | Endothelium | atherosclerosis | endothelial cells | interferons | antiphosholipid syndrome
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