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Science, ISSN 0036-8075, 4/2013, Volume 340, Issue 6129, pp. 202 - 207
Type I interferons (IFN-I) are critical for antiviral immunity; however, chronic IFN-I signaling is associated with hyperimmune activation and disease... 
T lymphocytes | Isotypes | Lymphocytic choriomeningitis virus | Genes | REPORTS | Blocking antibodies | Antivirals | Antibodies | Viruses | Infections | Immunity | GAMMA | MULTIDISCIPLINARY SCIENCES | CHRONIC HEPATITIS-C | INTERLEUKIN-10 RECEPTOR | HUMAN-IMMUNODEFICIENCY-VIRUS | CHRONIC VIRAL-INFECTION | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | EXPRESSION | T-CELLS | IL-10 | PROGRESSION | Lymphocytic choriomeningitis virus - immunology | Arenaviridae Infections - virology | Oligonucleotide Array Sequence Analysis | Dendritic Cells - immunology | Gene Expression Profiling | Interferon-gamma - metabolism | Interferon Type I - immunology | Receptor, Interferon alpha-beta - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Antibodies - immunology | Interferon Type I - metabolism | Interleukin-10 - metabolism | Lymphocytic choriomeningitis virus - physiology | Receptor, Interferon alpha-beta - genetics | Arenaviridae Infections - immunology | Cytokines - immunology | Receptor, Interferon alpha-beta - metabolism | Cytokines - metabolism | Signal Transduction | Immune Tolerance | B7-H1 Antigen - metabolism | Animals | Virus Replication | Interferon-gamma - immunology | Interferon Type I - genetics | Mice | Signal transduction | Interferon | Viral infections | Immune system | Chronic illnesses | Human | HIV | Organs | Blocking | Hepatitis C virus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2017, Volume 127, Issue 1, pp. 269 - 279
Journal Article
Nature, ISSN 0028-0836, 09/2018, Volume 561, Issue 7722, pp. 258 - 262
Although serum from patients with Parkinson's disease contains elevated levels of numerous pro-inflammatory cytokines including IL-6, TNF, IL-1 beta, and IFN... 
OXIDATIVE STRESS | ACTIVATION | MUTANTS | MULTIDISCIPLINARY SCIENCES | IN-VIVO MITOPHAGY | DISEASE | NEURONS | DEFICIENT MICE | MUTATIONS | MITOCHONDRIAL-DNA | Protein Kinases - metabolism | Protein Kinases - genetics | Humans | Stress, Physiological | Receptor, Interferon alpha-beta - antagonists & inhibitors | Membrane Proteins - deficiency | Inflammation - metabolism | DNA, Mitochondrial - genetics | Membrane Proteins - metabolism | Parkinson Disease - metabolism | Protein Kinases - deficiency | Physical Conditioning, Animal | Membrane Proteins - genetics | Mice, Inbred C57BL | Alarmins - metabolism | Ubiquitin-Protein Ligases - metabolism | Immunity, Innate | DNA, Mitochondrial - blood | Mitochondrial Degradation | Animals | Receptor, Interferon alpha-beta - immunology | Inflammation - genetics | Ubiquitin-Protein Ligases - deficiency | Inflammation - prevention & control | Mice | Ubiquitin-Protein Ligases - genetics | Inflammation | Genetic aspects | Research | Gene mutations | Risk factors | Ubiquitin | Heart | Cell culture | Oxidative stress | Animal models | Parkinson's disease | Substantia nigra | Parkinsons disease | Innate immunity | Mitochondrial DNA | Kinases | Immunity | Interleukin 6 | Mitochondria | Movement disorders | Ubiquitin-protein ligase | Deoxyribonucleic acid--DNA | Dopamine receptors | Enzymes | Phenotypes | Dopamine | Cytokines | Neurodegenerative diseases | Body temperature | Damage patterns | Insects | Tumor necrosis factor | PTEN-induced putative kinase | Proteomics | Interferon | Parkin protein | Mutation | Methods
Journal Article
Cancer Research, ISSN 0008-5472, 09/2015, Volume 75, Issue 18, pp. 3812 - 3822
STAT3 is an oncogenic transcription factor with potent immunosuppressive functions. We found that pharmacologic inhibition of STAT3 or its selective knockout... 
IMMUNITY | RECRUITMENT | ACTIVATION | ONCOLOGY | IMMUNOTHERAPY | RESISTANCE | DEATH | DIFFERENTIATION | TRANSCRIPTION FACTOR STAT3 | TUMORS | T-CELLS | Doxorubicin - therapeutic use | Cyclic S-Oxides - administration & dosage | Antibiotics, Antineoplastic - pharmacology | Chemokine CXCL9 - biosynthesis | Dendritic Cells - immunology | Neoplasm Proteins - physiology | Transcriptional Activation | Fibrosarcoma - immunology | Neoplasm Proteins - antagonists & inhibitors | Receptor, Interferon alpha-beta - antagonists & inhibitors | Interferon Type I - biosynthesis | Chemokine CXCL10 - biosynthesis | Colorectal Neoplasms - therapy | STAT3 Transcription Factor - physiology | Colorectal Neoplasms - drug therapy | Female | Cyclic S-Oxides - pharmacology | Immunocompetence | Receptors, CXCR3 - antagonists & inhibitors | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | STAT3 Transcription Factor - genetics | Doxorubicin - administration & dosage | T-Lymphocytes, Cytotoxic - immunology | Neoplasm Proteins - biosynthesis | Mice, Inbred C57BL | Fibrosarcoma - therapy | Combined Modality Therapy | Chemokine CXCL9 - genetics | Antibiotics, Antineoplastic - administration & dosage | Drug Synergism | Cyclic S-Oxides - therapeutic use | Animals | Colorectal Neoplasms - immunology | Antibiotics, Antineoplastic - therapeutic use | Signal Transduction - drug effects | Chemokine CXCL10 - genetics | Mice, Nude | Receptors, CXCR3 - physiology | Cell Line, Tumor | Interferon Type I - genetics | Mice | Mice, Inbred BALB C | STAT3 Transcription Factor - antagonists & inhibitors | Doxorubicin - pharmacology | Fibrosarcoma - drug therapy | Lymphocytes, Tumor-Infiltrating - immunology
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 02/2017, Volume 76, Issue 2, pp. 450 - 457
Journal Article