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Nature medicine, ISSN 1078-8956, 11/2014, Volume 20, Issue 11, pp. 1301 - 1309
Some of the anti-neoplastic effects of anthracyclines in mice originate from the induction of innate and T cell-mediated anticancer immune responses. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE | RIG-I | TUMORS | CELL BIOLOGY | TOLL-LIKE RECEPTOR-3 | BREAST-CANCER | ANTICANCER CHEMOTHERAPY | GENE | IMMUNE-RESPONSES | Doxorubicin - therapeutic use | Breast Neoplasms - immunology | Humans | RNA, Messenger - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Interferon Type I - biosynthesis | Receptors, Pattern Recognition - metabolism | Neoplasm Metastasis | Anthracyclines - pharmacology | Interferon Type I - metabolism | Female | Neoadjuvant Therapy | Gene Expression Regulation, Neoplastic - drug effects | RNA - metabolism | Receptor, Interferon alpha-beta - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Treatment Outcome | Toll-Like Receptor 3 - metabolism | Breast Neoplasms - drug therapy | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Immunocompetence - drug effects | Anthracyclines - therapeutic use | Chemokine CXCL10 - metabolism | Myxovirus Resistance Proteins - metabolism | Doxorubicin - pharmacology | Anthracyclines | Care and treatment | Analysis | Physiological aspects | Interferon | Research | Cancer | Signal transduction | Chemotherapy | Breast cancer | Chemokines | Index Medicus | Life Sciences | Immunology | Immunotherapy
Journal Article
Science, ISSN 0036-8075, 4/2013, Volume 340, Issue 6129, pp. 202 - 207
Type I interferons (IFN-I) are critical for antiviral immunity; however, chronic IFN-I signaling is associated with hyperimmune activation and disease... 
T lymphocytes | Isotypes | Lymphocytic choriomeningitis virus | Genes | REPORTS | Blocking antibodies | Antivirals | Antibodies | Viruses | Infections | Immunity | GAMMA | MULTIDISCIPLINARY SCIENCES | CHRONIC HEPATITIS-C | INTERLEUKIN-10 RECEPTOR | HUMAN-IMMUNODEFICIENCY-VIRUS | CHRONIC VIRAL-INFECTION | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | EXPRESSION | T-CELLS | IL-10 | PROGRESSION | Lymphocytic choriomeningitis virus - immunology | Arenaviridae Infections - virology | Oligonucleotide Array Sequence Analysis | Dendritic Cells - immunology | Gene Expression Profiling | Interferon-gamma - metabolism | Interferon Type I - immunology | Receptor, Interferon alpha-beta - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Antibodies - immunology | Interferon Type I - metabolism | Interleukin-10 - metabolism | Lymphocytic choriomeningitis virus - physiology | Receptor, Interferon alpha-beta - genetics | Arenaviridae Infections - immunology | Cytokines - immunology | Receptor, Interferon alpha-beta - metabolism | Cytokines - metabolism | Signal Transduction | Immune Tolerance | B7-H1 Antigen - metabolism | Animals | Virus Replication | Interferon-gamma - immunology | Interferon Type I - genetics | Mice | Signal transduction | Interferon | Viral infections | Immune system | Chronic illnesses | Index Medicus | Human | HIV | Organs | Blocking | Hepatitis C virus
Journal Article
Science, ISSN 0036-8075, 4/2013, Volume 340, Issue 6129, pp. 207 - 211
Journal Article
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 2017, Volume 23, Issue 12, pp. 1481 - 1487
Interferon regulatory factor 3 (IRF3) and type I interferons (IFNs) protect against infections(1) and cancer(2), but excessive IRF3 activation and type I IFN... 
MEDICINE, RESEARCH & EXPERIMENTAL | CYCLIC GMP-AMP | STEM-CELLS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYTOSOLIC DNA | LARGE GENE LISTS | BIOCONDUCTOR | CELL BIOLOGY | TARGET GENES | PATHWAY | DIFFERENTIAL EXPRESSION ANALYSIS | INNATE IMMUNITY | Severity of Illness Index | Myocardial Infarction - genetics | Myocardial Infarction - mortality | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Receptor, Interferon alpha-beta - physiology | Interferon Regulatory Factor-3 - genetics | Interferon Regulatory Factor-3 - physiology | Mice, Knockout | Inflammation - metabolism | Animals | Myocardial Infarction - pathology | Inflammation - genetics | Interferon Type I - genetics | Interferon Type I - physiology | Mice | Receptor, Interferon alpha-beta - metabolism | Care and treatment | Physiological aspects | Development and progression | Genetic aspects | Interferon | Research | Heart attack | Heart | Myocardial infarction | Heart attacks | Activation | Leukocytes | Macrophages | Vascular diseases | Ischemia | Cyclic GMP | Heart diseases | AMP | Cytokines | Inflammation | Gene expression | Ribonucleic acid--RNA | Survival | White blood cells | Signaling | Interferon regulatory factor 3 | Cell death | Infiltration | Infarction | Ventricle | Interferon regulatory factor | Chemokines | Cancer | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 05/2015, Volume 11, Issue 5, pp. 785 - 797
Journal Article