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Nature Communications, ISSN 2041-1723, 04/2016, Volume 7, Issue 1, p. 11394
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9094
Background: Notch receptors normally play a key role in guiding a variety of cell fate decisions during development and differentiation of metazoan organisms.... 
BREAST-CANCER | GAMMA-SECRETASE INHIBITOR | STRUCTURAL BASIS | MODIFIES NOTCH | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | PROTEOLYTIC ACTIVATION | THERAPEUTIC TARGET | PANCREATIC-CANCER | REGULATORY REGION | MAMMALIAN NOTCH | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - pathology | Humans | Intercellular Signaling Peptides and Proteins - metabolism | Flow Cytometry | Antibody Specificity - immunology | Binding Sites - immunology | Jagged-2 Protein | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Binding, Competitive | Cell Line | Membrane Proteins - genetics | Antibodies, Monoclonal - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch1 - immunology | Binding Sites - genetics | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Animals | Signal Transduction - drug effects | Precursor T-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Cell Line, Tumor | Ligands | Cell Proliferation - drug effects | Mice | Mutation | Receptor, Notch1 - genetics | 3T3 Cells | Monoclonal antibodies | Epidermal growth factor | Gene mutations | T cells | Health aspects | Leukemia | Pediatrics | Biotechnology | Laboratories | Phages | Multiple myeloma | Amino acids | Antagonists | Lymphocytes T | Activation | Vaccines | Metastasis | Angiogenesis | Receptors | Phage display | Reporter gene | Cell fate | Cell cycle | Metalloproteinase | Inhibition | Binding | Enzymes | Lymphatic system | Acute lymphatic leukemia | Breast cancer | Lymphatic leukemia | Tumor cell lines | Gene expression | Pathology | Signaling | Inhibitors | Womens health | Pancreatic cancer | Notch protein | Aberration | Prostate cancer | Secretase | Cancer
Journal Article
European Journal of Immunology, ISSN 0014-2980, 01/2013, Volume 43, Issue 1, pp. 182 - 187
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2015, Volume 45, Issue 9, pp. 2615 - 2627
Delta‐like protein 1 (DLK1) is a noncanonical ligand that inhibits NOTCH1 receptor activity and regulates multiple differentiation processes. In macrophages,... 
NOTCH signaling | TLR | DLK1 | Cytokine | Inflammatory inhibitor | Macrophage | STEM-CELLS | PROTEIN | RECEPTOR | IMMUNOLOGY | REGULATOR | PATHWAY | IMMUNE-RESPONSES | GENE-EXPRESSION | CELL-DIFFERENTIATION | UP-REGULATION | POLARIZATION | Inflammation - pathology | Interferon-beta - immunology | Interleukin-12 - genetics | Humans | Transcription Factor RelB - immunology | Tumor Necrosis Factor-alpha - genetics | Interleukin-23 - genetics | U937 Cells | Tumor Necrosis Factor-alpha - immunology | Interferon-beta - genetics | Cell Differentiation | Toll-Like Receptor 3 - genetics | Macrophages - immunology | Transcription Factor RelB - genetics | Cell Line | Signal Transduction | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Interferon Regulatory Factors - genetics | Receptor, Notch1 - immunology | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - immunology | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Macrophages - cytology | STAT1 Transcription Factor - genetics | Interleukin-23 - immunology | STAT1 Transcription Factor - immunology | Macrophage Activation | Mice, Knockout | Interferon Regulatory Factors - immunology | Animals | Interleukin-12 - immunology | Inflammation - genetics | Mice | Receptor, Notch1 - genetics | Intercellular Signaling Peptides and Proteins - immunology | Proteins | Macrophages | Septic shock | Rodents
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 12/2017, Volume 47, Issue 12, pp. 2090 - 2100
Inhibition of Notch signalling in T cells attenuates the development of experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple sclerosis.... 
Cell priming | Experimental autoimmune encephalomyelitis | Multiple sclerosis | Myeloid cells | Notch signalling | ACTIVATION | DENDRITIC CELLS | MONONUCLEAR-CELLS | IMMUNOLOGY | MULTIPLE-SCLEROSIS | REGULATORY T-CELLS | CENTRAL-NERVOUS-SYSTEM | LIGAND | BRAIN | KAPPA-B ACTIVITY | BLOOD | Spinal Cord - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Interleukin-17 - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Lymphocyte Activation - immunology | Th17 Cells - metabolism | Myeloid Cells - immunology | Gene Expression - immunology | Encephalomyelitis, Autoimmune, Experimental - genetics | Interferon-gamma - genetics | Cytokines - genetics | Cytokines - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Receptor, Notch1 - immunology | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Spinal Cord - immunology | Interleukin-17 - metabolism | Animals | Interferon-gamma - immunology | Myeloid Cells - metabolism | Th17 Cells - immunology | Receptor, Notch1 - genetics | Encephalomyelitis | RNA | T cells | Analysis | Myelin proteins | Spinal cord | Oligodendrocyte-myelin glycoprotein | CD86 antigen | Helper cells | Interleukin 23 | Activation | Lymphocytes T | mRNA | Gene deletion | Interleukin 6 | Cell activation | Clonal deletion | Demyelination | Lymphocytes | Splenocytes | Rodents | Animal tissues | Deletion | Myelin | Glycoprotein | Interleukin 12 | Priming | Lymphoid tissue | Gene expression | Experimental allergic encephalomyelitis | Interleukin 17 | Microglia | CD4 antigen | Signaling | Tumor necrosis factor | Infiltration | Mice | Notch protein
Journal Article
Molecular Immunology, ISSN 0161-5890, 11/2017, Volume 91, pp. 17 - 23
Journal Article
The Journal of Immunology, ISSN 0022-1767, 03/2008, Volume 180, Issue 5, pp. 2796 - 2804
Previous studies have shown that the Notch1 and TGF-beta signaling pathways are mutually re-enforcing. Given recent evidence that regulatory T cell (Treg)... 
GROWTH-FACTOR-BETA | ACTIVATION | DENDRITIC CELLS | IN-VIVO | DIFFERENTIATION | IMMUNOLOGY | BOUND TGF-BETA | GAMMA-SECRETASE | UP-REGULATION | EXPRESSION | CUTTING EDGE | T-Lymphocytes, Regulatory - metabolism | Coculture Techniques | Humans | Interleukin-2 Receptor alpha Subunit - biosynthesis | Intercellular Signaling Peptides and Proteins - biosynthesis | T-Lymphocytes, Regulatory - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Signal Transduction - immunology | Serrate-Jagged Proteins | Transforming Growth Factor beta - antagonists & inhibitors | Female | Growth Inhibitors - biosynthesis | Membrane Proteins - metabolism | Mink | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Cell Line | Immune Sera - pharmacology | Calcium-Binding Proteins - biosynthesis | Jurkat Cells | Membrane Proteins - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Transforming Growth Factor beta - physiology | Receptor, Notch1 - immunology | Receptor, Notch1 - biosynthesis | HT29 Cells | Growth Inhibitors - immunology | Membrane Proteins - biosynthesis | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Cell Line, Tumor | Growth Inhibitors - physiology | Ligands | Mice | Mice, Inbred BALB C | Receptor, Notch1 - antagonists & inhibitors | Growth Inhibitors - antagonists & inhibitors | Receptor, Notch1 - physiology | Calcium-Binding Proteins - genetics
Journal Article
Nature Immunology, ISSN 1529-2908, 08/2010, Volume 11, Issue 8, pp. 717 - 724
Journal Article