X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (2740) 2740
Publication (541) 541
Book Review (38) 38
Book Chapter (26) 26
Conference Proceeding (7) 7
Dissertation (2) 2
Book / eBook (1) 1
Data Set (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (2057) 2057
animals (1851) 1851
humans (1705) 1705
mice (1172) 1172
signal transduction (1082) 1082
receptors, notch - metabolism (932) 932
expression (685) 685
cell biology (683) 683
female (665) 665
notch (566) 566
differentiation (530) 530
male (523) 523
article (506) 506
gene expression (502) 502
oncology (500) 500
cell differentiation (430) 430
cancer (413) 413
receptor, notch1 - metabolism (413) 413
activation (411) 411
receptors, notch - genetics (406) 406
membrane proteins - metabolism (389) 389
stem cells (378) 378
biochemistry & molecular biology (377) 377
research (373) 373
cell proliferation (371) 371
ligands (366) 366
developmental biology (342) 342
cell line, tumor (341) 341
proteins (332) 332
receptor, notch2 - metabolism (326) 326
genetic aspects (321) 321
apoptosis (319) 319
cells, cultured (316) 316
signal transduction - physiology (309) 309
research article (298) 298
receptor, notch1 - genetics (292) 292
analysis (289) 289
immunology (287) 287
mice, inbred c57bl (285) 285
multidisciplinary sciences (283) 283
mutation (281) 281
pathway (260) 260
notch protein (258) 258
membrane proteins - genetics (252) 252
stem-cells (243) 243
genes (242) 242
receptor, notch2 - genetics (236) 236
medicine (235) 235
transcription factors (235) 235
rodents (232) 232
progenitor cells (224) 224
notch signaling (222) 222
mice, knockout (221) 221
physiological aspects (221) 221
serrate-jagged proteins (216) 216
gene-expression (215) 215
signal transduction - drug effects (215) 215
biology (214) 214
mice, transgenic (210) 210
growth (208) 208
proliferation (207) 207
tumors (206) 206
in-vivo (205) 205
gene (202) 202
cells (200) 200
intercellular signaling peptides and proteins - metabolism (197) 197
gene expression regulation, developmental (196) 196
immunohistochemistry (196) 196
signaling (194) 194
science (193) 193
jagged-1 protein (189) 189
middle aged (185) 185
receptor (181) 181
gene expression regulation, neoplastic (180) 180
cell line (176) 176
reverse transcriptase polymerase chain reaction (175) 175
health aspects (173) 173
basic helix-loop-helix transcription factors - metabolism (170) 170
signal transduction - genetics (170) 170
genetics (167) 167
gene expression profiling (166) 166
hematology (164) 164
signaling pathway (163) 163
medicine, research & experimental (162) 162
adult (161) 161
genetics & heredity (161) 161
inhibition (161) 161
neurosciences (160) 160
down-regulation (158) 158
homeodomain proteins - metabolism (158) 158
receptors, notch (157) 157
rats (154) 154
development and progression (152) 152
receptors (146) 146
phenotype (145) 145
gene expression regulation (143) 143
kinases (142) 142
cellular signal transduction (141) 141
endocrinology & metabolism (141) 141
cell differentiation - physiology (140) 140
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 491, Issue 2, pp. 329 - 336
Hyperglycemia plays a crucial role in the pathogenesis of diabetic complications; however, the mechanisms underlying diabetic cardiac fibrosis remain unclear.... 
MicroRNA | EndMT | Notch2 | Fibrosis | High glucose | TARGET | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MICRORNAS | DIABETIC CARDIOMYOPATHY | BIOPHYSICS | ALAGILLE-SYNDROME | EXPRESSION | Dependovirus - genetics | Cadherins - metabolism | Genetic Vectors - administration & dosage | Humans | Aortic Valve - drug effects | Diabetes Mellitus, Experimental - genetics | Male | MicroRNAs - metabolism | Diabetes Mellitus, Experimental - chemically induced | Cadherins - genetics | Endomyocardial Fibrosis - prevention & control | Dependovirus - metabolism | Genetic Vectors - chemistry | Signal Transduction | Endothelial Cells - metabolism | Genetic Vectors - metabolism | Glucose - pharmacology | Heart Ventricles - metabolism | MicroRNAs - genetics | Calcium-Binding Proteins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Streptozocin | Vimentin - metabolism | Actins - metabolism | Receptor, Notch2 - genetics | Actins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Vimentin - genetics | Epithelial-Mesenchymal Transition | Diabetic Cardiomyopathies - prevention & control | Endomyocardial Fibrosis - genetics | Endomyocardial Fibrosis - chemically induced | Endomyocardial Fibrosis - pathology | Diabetes Mellitus, Experimental - metabolism | Heart Ventricles - pathology | Calcium-Binding Proteins - metabolism | Cell Line | Mice, Inbred C57BL | Gene Expression Regulation | Receptor, Notch2 - metabolism | Aortic Valve - cytology | Aortic Valve - metabolism | Fibronectins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Endothelial Cells - cytology | Diabetes Mellitus, Experimental - pathology | Diabetic Cardiomyopathies - pathology | Fibronectins - genetics | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - drug effects | Fibronectins | Hyperglycemia | Stem cells | Glucose | Muscle proteins | Dextrose | Endothelium | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2012, Volume 209, Issue 9, pp. 1537 - 1551
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 2006, Volume 79, Issue 1, pp. 169 - 173
Alagille syndrome (AGS) is caused by mutations in the gene for the Notch signaling pathway ligand Jagged1 ( ), which are found in 94% of patients. To identify... 
HEART | DEFECTS | GENE | ARTERIOHEPATIC DYSPLASIA | GENETICS & HEREDITY | RECEPTOR | LIGAND | EXPRESSION | HUMAN JAGGED1 | Alagille Syndrome - genetics | Signal Transduction | Pedigree | Humans | Receptor, Notch2 - metabolism | Female | Male | Receptor, Notch2 - genetics | Mutation | Genetic aspects | Diagnosis | Research | Gene mutations | Alagille syndrome | Report
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 30, pp. 13438 - 13443
Pancreatic cancer is one of the most fatal malignancies lacking effective therapies. Notch signaling is a key regulator of cell fate specification and... 
Acinar cells | Developmental biology | Cell lines | Pancreatic cells | Mice | Lesions | Carcinogenesis | Pancreatic neoplasms | Tumors | Notch receptors | K-Ras | Genetically engineered mice | Myc | Pancreatic cancer | Notch | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MOUSE | C-MYC | ADULT MICE | CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | pancreatic cancer | SIGNALING PATHWAY | GROWTH | ACINAR-CELLS | EXPRESSION | genetically engineered mice | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Male | Receptor, Notch2 - genetics | Gene Expression Profiling | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | Carcinoma in Situ - genetics | Female | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Signal Transduction | Carcinoma in Situ - pathology | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Receptor, Notch2 - metabolism | Pancreas - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Pancreatic Ductal - pathology | Proto-Oncogene Proteins c-myc - metabolism | Blotting, Western | Disease Progression | Mice, Knockout | Animals | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics | Carcinoma in Situ - metabolism | Receptor, Notch1 - genetics | Biological Sciences
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2011, Volume 121, Issue 3, pp. 1207 - 1216
Journal Article
Journal Article