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Cancer Science, ISSN 1347-9032, 08/2016, Volume 107, Issue 8, pp. 1079 - 1091
Sphere formation in conditioned serum‐free culture medium supplemented with epidermal growth factor and basic fibroblast growth factor (tumorospheres) is... 
Cancer stem cell | epithelial–mesenchymal transition | melanoma | Notch4 protein | Twist1 | Epithelial-mesenchymal transition | Melanoma | VASCULOGENIC MIMICRY | DRUG-RESISTANCE | TUMOR-INITIATING CELLS | BREAST-CANCER | IN-VITRO | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | PLACENTAL GROWTH-FACTOR | LUNG ADENOCARCINOMA | epithelial-mesenchymal transition | FUNCTIONAL-SIGNIFICANCE | Melanoma - metabolism | Proto-Oncogene Proteins - metabolism | Cadherins - metabolism | Receptors, Notch - metabolism | Humans | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Receptors, Notch - genetics | Melanoma - pathology | Proto-Oncogene Proteins - deficiency | Epithelial-Mesenchymal Transition - genetics | Neoplasm Metastasis - genetics | Neoplastic Stem Cells - metabolism | Melanoma - genetics | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Neoplasm Invasiveness - genetics | Receptors, Notch - deficiency | Twist-Related Protein 1 - metabolism | Receptor, Notch4 | Epidermal growth factor | Analysis | Genes | Stem cells | Fibroblast growth factors | B cells | Metastasis | Gene expression | Cancer | Vimentin | Cell culture | Flow cytometry | Fibroblast growth factor | Mesenchyme | Western blotting | NOTCH4 protein | Metastases | Proteins | Angiogenesis | Signal transduction | Transfection | Vascular endothelial growth factor | Fibroblast growth factor 2 | Medical research | Immunoglobulins | Invasiveness | DNA microarrays | Protein expression | Immunofluorescence | Secretase | Tumors | Original
Journal Article
Cancer Letters, ISSN 0304-3835, 2017, Volume 390, pp. 115 - 125
Abstract We previously demonstrated that tamoxifen (TAM)-resistant human breast cancer (TAMR-MCF-7) cells showed increased expression of mesenchymal marker... 
Hematology, Oncology and Palliative Medicine | Notch4 | Breast cancer | Metastasis | Tamoxifen resistance | STAT3 | STEM-CELLS | ACTIVATION | TRANSCRIPTION | NOTCH | MEDIATE | SERINE PHOSPHORYLATION | CROSS-TALK | GROWTH-FACTOR RECEPTOR | ONCOLOGY | EXPRESSION | Cell Proliferation | Receptors, Notch - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Epithelial-Mesenchymal Transition - drug effects | Immunoblotting | Breast Neoplasms - physiopathology | Receptors, Notch - antagonists & inhibitors | Antineoplastic Agents, Hormonal - therapeutic use | Polymerase Chain Reaction | Female | STAT3 Transcription Factor - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Antineoplastic Agents, Hormonal - pharmacology | Breast Neoplasms - drug therapy | Drug Resistance, Neoplasm - genetics | Animals | Signal Transduction - drug effects | Mice, Nude | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Cell Line, Tumor | Protein Binding | Signal Transduction - physiology | Receptor, Notch4 | Proteins | Tyrosine | Medical colleges | Analysis | Pharmacy | Liver | Stem cells | Drugstores | Development and progression | Tamoxifen | Polyols | Genotype & phenotype | Phosphorylation | Immunoglobulins | Genes | Conflicts of interest | Ligands | Fluorides | Kinases | Cell adhesion & migration
Journal Article
Cancer Research, ISSN 0008-5472, 04/2016, Volume 76, Issue 7, pp. 1690 - 1697
The effects of Notch signaling are context-dependent and both oncogenic and tumor-suppressive functions have been described. Notch signaling in melanoma is... 
ACTIVATION | REPRESSION | ONCOLOGY | GROWTH | HES | TWIST1 | E-CADHERIN | RECEPTORS | EXPRESSION | PROGRESSION | Signal Transduction | Melanoma - genetics | Epithelial-Mesenchymal Transition - physiology | Humans | Skin Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Receptors, Notch - genetics | Receptor, Notch4
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2008, Volume 28, Issue 12, pp. 2258 - 2265
OBJECTIVE—Notch signaling pathway controls key functions in vascular and endothelial cells (EC). However, little is known about the role of Notch in... 
Transplant arteriosclerosis | Activation | Endothelial cells | Notch | Apoptosis | transplant arteriosclerosis | endothelial cells | ANGIOGENESIS | apoptosis | MECHANISMS | INDUCTION | ARTERIAL-DISEASE | TUMOR-NECROSIS-FACTOR | INHIBITION | PERIPHERAL VASCULAR DISEASE | notch | DIFFERENTIATION | activation | HEMATOLOGY | EXPRESSION | VASCULOPATHY | SMOOTH-MUSCLE CELLS | RNA, Small Interfering - genetics | Transcription Factor HES-1 | Humans | Rats, Inbred Lew | Heart Transplantation - pathology | Receptors, Notch - genetics | RNA, Messenger - metabolism | Transplantation, Homologous | Receptors, Notch - antagonists & inhibitors | Coronary Artery Disease - physiopathology | Inflammation Mediators - pharmacology | Coronary Artery Disease - pathology | Heart Transplantation - physiology | Inflammation Mediators - metabolism | Endothelial Cells - physiology | Proto-Oncogene Proteins - antagonists & inhibitors | Basic Helix-Loop-Helix Transcription Factors - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Coronary Artery Disease - etiology | Cytokines - metabolism | Signal Transduction | Down-Regulation | RNA, Messenger - genetics | Cells, Cultured | Gene Silencing | Heart Transplantation - adverse effects | Rats | Receptors, Notch - physiology | Proto-Oncogene Proteins - genetics | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Homeodomain Proteins - genetics | Animals | Homeodomain Proteins - antagonists & inhibitors | Proto-Oncogene Proteins - physiology | Apoptosis - physiology | Endothelial Cells - pathology | Homeodomain Proteins - physiology | Cytokines - pharmacology | Receptor, Notch4
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2004, Volume 279, Issue 12, pp. 11657 - 11663
Notch4, a member of the Notch family of transmembrane receptors, is expressed primarily on endothelial cells. Activation of Notch in various cell systems has... 
CELLS | TUMOR-NECROSIS-FACTOR | ANGIOGENESIS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | JUN NH2-TERMINAL KINASE | EXPRESSION | ACTIVATED NOTCH4 | FAMILY
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Journal Article
Journal Article