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Journal Article
Nature, ISSN 0028-0836, 04/2008, Volume 452, Issue 7187, pp. 654 - 658
Journal Article
Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7391, pp. 627 - 631
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2003, Volume 100, Issue 22, pp. 13019 - 13024
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2007, Volume 27, Issue 6, pp. 1456 - 1462
OBJECTIVE—Tissue factor (TF) initiates coagulation and indirectly triggers thrombin-dependent protease activated receptor (PAR) signaling. The TF–VIIa complex... 
Angiogenesis | Thrombin | Protease activated receptors | Coagulation | Tissue factor | FACTOR XA | thrombin | PAR-1 | tissue factor | protease activated receptors | angiogenesis | INDUCTION | coagulation | ENDOTHELIAL PROGENITOR CELLS | PROTEASE-ACTIVATED RECEPTORS | GROWTH | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | THROMBIN RECEPTOR | Receptor, PAR-1 - metabolism | Receptor, PAR-2 - metabolism | Retinal Neovascularization - metabolism | Receptor, PAR-1 - deficiency | Receptor, PAR-2 - deficiency | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Retinal Vessels - pathology | Blood Coagulation Factor Inhibitors - pharmacology | Time Factors | Hyperoxia - metabolism | Disease Models, Animal | Receptor, PAR-2 - genetics | Oxygen | Retinal Vessels - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Hypoxia - complications | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Retinal Neovascularization - etiology | Factor VIIa - metabolism | Hyperoxia - chemically induced | Receptor, PAR-1 - genetics | Mice, Knockout | Hyperoxia - pathology | Animals | Signal Transduction - drug effects | Hypoxia - pathology | Thromboplastin - genetics | Cell Line, Tumor | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Benzamides | Neoplasms, Experimental - metabolism | Thromboplastin - metabolism | Retinal Neovascularization - pathology | Neoplasms, Experimental - blood supply
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 10/2013, Volume 18, Issue 10, pp. 1136 - 1145
Fear memories are acquired through neuronal plasticity, an orchestrated sequence of events regulated at circuit and cellular levels. The conventional model of... 
experience-directed signaling | amygdala | G protein-coupled receptors | PAR-1 | biased agonism | fear | HUMAN BRAIN | TISSUE-PLASMINOGEN ACTIVATOR | KINASE-A | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANXIETY-LIKE BEHAVIOR | SYNAPTIC PLASTICITY | NEUROSCIENCES | MOUSE HIPPOCAMPUS | CORTICOTROPIN-RELEASING-FACTOR | LATERAL AMYGDALA | LONG-TERM POTENTIATION | RECEPTORS | Amygdala - chemistry | Nerve Tissue Proteins - analysis | Fear - drug effects | Male | Nerve Net - drug effects | Receptor, PAR-1 - deficiency | Fear - physiology | Miniature Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - drug effects | Freezing Reaction, Cataleptic - drug effects | Nerve Net - physiology | Recognition (Psychology) - physiology | Excitatory Postsynaptic Potentials - physiology | Exploratory Behavior - physiology | Long-Term Potentiation - drug effects | Exploratory Behavior - drug effects | Long-Term Potentiation - physiology | Miniature Postsynaptic Potentials - physiology | Nerve Tissue Proteins - physiology | Conditioning, Classical - drug effects | Maze Learning - physiology | Receptor, PAR-1 - antagonists & inhibitors | Mice, Inbred C57BL | GTP-Binding Protein alpha Subunits, Gq-G11 - physiology | Maze Learning - drug effects | Conditioning, Classical - physiology | Receptor, PAR-1 - genetics | Mice, Knockout | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Patch-Clamp Techniques | Pyrroles - pharmacology | Animals | Recognition (Psychology) - drug effects | Freezing Reaction, Cataleptic - physiology | Pain Threshold | Mice | Quinazolines - pharmacology | Amygdala - physiology | Receptor, PAR-1 - physiology | Neuroplasticity | Fear | Control | Cell receptors | Physiological aspects | G proteins | Amygdala (Brain)
Journal Article
Glia, ISSN 0894-1491, 09/2013, Volume 61, Issue 9, pp. 1456 - 1470
Kallikrein 6 (KLK6) is a secreted serine protease preferentially expressed by oligodendroglia in CNS white matter. Elevated levels of KLK6 occur in actively... 
myelin | thrombin | protease‐activated receptor | spinal cord | demyelination | oligodendrocyte | Thrombin | Spinal cord | Protease-activated receptor | Oligodendrocyte | Demyelination | Myelin | RAT SPINAL-CORD | CELLS | ASTROCYTES | NEUROSCIENCES | OLIGODENDROCYTES | MULTIPLE-SCLEROSIS | protease-activated receptor | REMYELINATION | SERINE-PROTEASE | PROTEASE-ACTIVATED RECEPTOR-1 | EXPRESSION | BRAIN | Oligodendroglia - metabolism | Receptor, PAR-1 - metabolism | Humans | Receptor, PAR-1 - deficiency | Receptor, PAR-2 - deficiency | Cerebral Cortex - cytology | Spinal Cord Injuries - pathology | Adenosine Triphosphate - pharmacology | Oligodendroglia - drug effects | Thrombin - pharmacology | Myelin Basic Protein | Myelin Proteolipid Protein - metabolism | Disease Models, Animal | Receptor, PAR-2 - genetics | Animals, Newborn | Kallikreins - pharmacology | Spinal Cord Injuries - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Receptor, PAR-1 - agonists | Cells, Cultured | Kallikreins - genetics | Signal Transduction - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Analysis of Variance | Signal Transduction - drug effects | Myelin Proteolipid Protein - genetics | Mice | Kallikreins - metabolism | Kallikrein | Myelin proteins | Medical research | Protease Activated Receptor | Spinal Cord
Journal Article