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FEBS letters, ISSN 0014-5793, 03/2017, Volume 591, Issue 5, pp. 801 - 809
The platelet‐derived growth factor receptors alpha and beta (PDGFRα and PDGFRβ) mark fibroadipogenic progenitor cells/fibroblasts and pericytes in skeletal muscle, respectively... 
platelet‐derived growth factor | skeletal muscle | extracellular matrix | platelet-derived growth factor | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Extracellular Matrix - metabolism | Muscle, Skeletal - metabolism | Gene Expression Profiling | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Hypertrophy - metabolism | Platelet-Derived Growth Factor - genetics | Quinolines - pharmacology | Hypertrophy - physiopathology | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Achilles Tendon - surgery | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Muscle, Skeletal - drug effects | Hypertrophy - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Animals, Newborn | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Mice, Transgenic | Platelet-Derived Growth Factor - metabolism | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Muscle, Skeletal - physiopathology | Benzimidazoles - pharmacology | Weight-Bearing | Mice | Neovascularization, Physiologic | Index Medicus
Journal Article
Stem cells (Dayton, Ohio), ISSN 1066-5099, 03/2012, Volume 30, Issue 3, pp. 548 - 560
...‐derived growth factor receptor (PDGFR) signaling plays a crucial role in specifying mesenchymal stem cell (MSC... 
Marrow stromal stem cells | Adult stem cells | Multipotential differentiation | Mesenchymal stem cells | Cell & Tissue Engineering | Life Sciences & Biomedicine | Hematology | Oncology | Biotechnology & Applied Microbiology | Science & Technology | Cell Biology | Gene Expression - drug effects | Cell Nucleus Size - drug effects | Homeodomain Proteins - metabolism | Humans | Male | Janus Kinases - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Quinolines - pharmacology | Gene Knockdown Techniques | Octamer Transcription Factor-3 - genetics | Young Adult | Receptor, Epidermal Growth Factor - metabolism | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | RNA Interference | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Adult | Female | MAP Kinase Kinase Kinases - antagonists & inhibitors | Mesenchymal Stromal Cells - physiology | STAT3 Transcription Factor - metabolism | Pyrazoles - pharmacology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Proto-Oncogene Proteins c-abl - genetics | Nanog Homeobox Protein | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Cells, Cultured | MAP Kinase Kinase Kinases - metabolism | Actomyosin - metabolism | Homeodomain Proteins - genetics | Cell Shape - drug effects | Janus Kinases - antagonists & inhibitors | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Octamer Transcription Factor-3 - metabolism | Proto-Oncogene Proteins c-abl - metabolism | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Cell Nucleus - drug effects | Indans - pharmacology | Proteins | Medical research | Rodents | Stem cells | Index Medicus | Tissue-Specific Stem Cells | Original Research
Journal Article
Journal of vascular surgery, ISSN 0741-5214, 2014, Volume 59, Issue 5, pp. 1402 - 1409.e4
Background Platelet-derived growth factor C (PDGF-C) has been reported to promote angiogenesis independently of vascular endothelial growth factor (VEGF... 
Surgery | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Antibodies, Neutralizing - administration & dosage | Neuropilin-2 - metabolism | Hindlimb | Platelet-Derived Growth Factor - immunology | Lymphokines - deficiency | Neuropilin-1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Platelet-Derived Growth Factor - genetics | Recovery of Function | Diabetic Angiopathies - physiopathology | Lymphokines - genetics | Time Factors | Lymphokines - antagonists & inhibitors | Lymphokines - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Diabetes Mellitus, Experimental - physiopathology | Muscle, Skeletal - blood supply | Diabetic Angiopathies - metabolism | Gene Transfer Techniques | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - genetics | Ischemia - metabolism | Ischemia - therapy | Platelet-Derived Growth Factor - deficiency | Vascular Endothelial Growth Factor A - immunology | Ischemia - physiopathology | Regional Blood Flow | Mice, Knockout | Diabetic Angiopathies - therapy | Platelet-Derived Growth Factor - metabolism | Animals | Lymphokines - immunology | Mice | Platelet-Derived Growth Factor - antagonists & inhibitors | Neovascularization, Physiologic | Platelet-derived growth factor | Diabetes | Vascular endothelial growth factor | Analysis | Index Medicus
Journal Article
Journal Article
The FEBS journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 10, pp. 2477 - 2489
...‐derived growth factor (PDGF)/PDGF receptor (PDGF‐R) mediated signaling, on the other hand, is highly associated with cancer progression. Specifically, PDGF‐Rα and PDGF‐Rβ... 
migration | breast cancer | PDGF‐R | invasion | heparan sulfate proteoglycans | imatinib (Glivec®) | PDGF-R | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | M Phase Cell Cycle Checkpoints | Phosphorylation | Cell Proliferation | Humans | Neoplasm Invasiveness - prevention & control | Syndecan-4 - genetics | Transcriptome | Glypicans - metabolism | Breast Neoplasms - metabolism | MAP Kinase Signaling System | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | MCF-7 Cells | Neoplasm Invasiveness - pathology | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Female | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Cytostatic Agents - pharmacology | Proto-Oncogene Proteins c-sis - pharmacology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Signal Transduction | Syndecan-4 - metabolism | Pyrimidines - pharmacology | Glypicans - genetics | Imatinib Mesylate | Piperazines - pharmacology | G2 Phase Cell Cycle Checkpoints | Cell Movement - drug effects | Breast Neoplasms - pathology | Ligands | Drug Screening Assays, Antitumor | Antimitotic agents | Evaluation | Platelet-derived growth factor | Analysis | Cancer cells | Development and progression | Breast cancer | Antineoplastic agents | Sulfates | Proteins | Signal transduction | Inhibitor drugs | Cellular biology | Gene expression | Index Medicus
Journal Article
Journal Article
Blood, ISSN 1528-0020, 02/2017, Volume 129, Issue 6, pp. 704 - 714
Molecular diagnostics has generated substantial dividends in dissecting the genetic basis of myeloid neoplasms with eosinophilia. The family of diseases... 
Life Sciences & Biomedicine | Hematology | Science & Technology | Eosinophilia - therapy | Oncogene Proteins, Fusion - metabolism | Leukemia - pathology | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | fms-Like Tyrosine Kinase 3 | Antineoplastic Agents - therapeutic use | Myeloproliferative Disorders - pathology | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Myeloproliferative Disorders - genetics | Transplantation, Homologous | Eosinophilia - pathology | Hypereosinophilic Syndrome - diagnosis | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Janus Kinase 2 - metabolism | Eosinophilia - genetics | Hypereosinophilic Syndrome - pathology | Hypereosinophilic Syndrome - therapy | Eosinophilia - diagnosis | Janus Kinase 2 - antagonists & inhibitors | Leukemia - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Myeloproliferative Disorders - diagnosis | Proto-Oncogene Proteins c-abl - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Myeloproliferative Disorders - therapy | Janus Kinase 2 - genetics | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Leukemia - diagnosis | Proto-Oncogene Proteins c-abl - metabolism | Hypereosinophilic Syndrome - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article