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Journal Article
Nature (London), ISSN 1476-4687, 03/2014, Volume 508, Issue 7494, pp. 118 - 122
...) in melanoma causes activation of TGF-β signalling, thus leading to upregulation of EGFR and platelet-derived growth factor receptor-β (PDGFRB... 
Antineoplastic Agents/administration & dosage | Transforming Growth Factor beta/metabolism | Humans | Mitogen-Activated Protein Kinase Kinases/antagonists & inhibitors | Indoles/administration & dosage | Flow Cytometry | Receptor, Platelet-Derived Growth Factor beta/biosynthesis | Proto-Oncogene Proteins B-raf/antagonists & inhibitors | ErbB Receptors/biosynthesis | Melanoma/drug therapy | Female | Cell Proliferation/drug effects | Sulfonamides/administration & dosage | Gene Library | Cellular Senescence/drug effects | Receptor Protein-Tyrosine Kinases/biosynthesis | Drug Resistance, Neoplasm/drug effects | Gene Expression Regulation, Neoplastic/drug effects | SOXE Transcription Factors/deficiency | Signal Transduction/drug effects | Protein Kinase Inhibitors/administration & dosage | Vemurafenib | Animals | Mice | RNA, Small Interfering | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptor, Epidermal Growth Factor - genetics | Receptor Protein-Tyrosine Kinases - biosynthesis | Cellular Senescence - drug effects | Melanoma - enzymology | Antineoplastic Agents - administration & dosage | Receptor, Platelet-Derived Growth Factor beta - genetics | Indoles - administration & dosage | Mitogen-Activated Protein Kinase Kinases - metabolism | Receptor, Epidermal Growth Factor - metabolism | Melanoma - genetics | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | SOXE Transcription Factors - deficiency | Proto-Oncogene Proteins B-raf - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Epidermal Growth Factor - biosynthesis | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Melanoma - pathology | Receptor Protein-Tyrosine Kinases - metabolism | Sulfonamides - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Protein Kinase Inhibitors - administration & dosage | Transforming Growth Factor beta - pharmacology | Drug Resistance, Neoplasm - genetics | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Transforming Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor beta - biosynthesis | Sulfonamides - administration & dosage | Drug Resistance, Neoplasm - drug effects | SOXE Transcription Factors - genetics | Proteins | Biopsy | Rodents | Genes | Melanoma | Mutation | Kinases | Drug resistance | Patients | Tumors | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 12/2006, Volume 12, Issue 23, pp. 6920 - 6928
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 02/2009, Volume 284, Issue 7, pp. 4626 - 4634
Journal Article
Journal Article
Journal Article
Blood, ISSN 1528-0020, 02/2017, Volume 129, Issue 6, pp. 704 - 714
Molecular diagnostics has generated substantial dividends in dissecting the genetic basis of myeloid neoplasms with eosinophilia... 
Life Sciences & Biomedicine | Hematology | Science & Technology | Eosinophilia - therapy | Oncogene Proteins, Fusion - metabolism | Leukemia - pathology | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | fms-Like Tyrosine Kinase 3 | Antineoplastic Agents - therapeutic use | Myeloproliferative Disorders - pathology | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Myeloproliferative Disorders - genetics | Transplantation, Homologous | Eosinophilia - pathology | Hypereosinophilic Syndrome - diagnosis | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Janus Kinase 2 - metabolism | Eosinophilia - genetics | Hypereosinophilic Syndrome - pathology | Hypereosinophilic Syndrome - therapy | Eosinophilia - diagnosis | Janus Kinase 2 - antagonists & inhibitors | Leukemia - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Myeloproliferative Disorders - diagnosis | Proto-Oncogene Proteins c-abl - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Myeloproliferative Disorders - therapy | Janus Kinase 2 - genetics | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Leukemia - diagnosis | Proto-Oncogene Proteins c-abl - metabolism | Hypereosinophilic Syndrome - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Genes & development, ISSN 0890-9369, 05/2008, Volume 22, Issue 10, pp. 1276 - 1312
Platelet-derived growth factors (PDGFs) and their receptors (PDGFRs) have served as prototypes for growth factor and receptor tyrosine kinase function for more than 25 years... 
Development | PDGF receptor | Platelet-derived growth factor | Fibrosis | Cancer | Genetics & Heredity | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Ce