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The Journal of Immunology, ISSN 0022-1767, 06/2007, Volume 178, Issue 11, pp. 7405 - 7411
Journal Article
Cancer Research, ISSN 0008-5472, 09/2007, Volume 67, Issue 18, pp. 8429 - 8432
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 12/2004, Volume 110, Issue 24, pp. 3699 - 3707
Journal Article
Vascular Pharmacology, ISSN 1537-1891, 2011, Volume 55, Issue 1, pp. 26 - 33
Journal Article
British Journal of Cancer, ISSN 0007-0920, 03/2004, Volume 90, Issue 6, pp. 1216 - 1221
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159532
We have recently reported that Compound 49b, a novel beta-adrenergic receptor agonist, can significantly reduce VEGF levels in retinal endothelial cells (REC)... 
ISCHEMIA-REPERFUSION INJURY | IGFBP-3 | OXYGEN-INDUCED RETINOPATHY | BETA-ADRENERGIC-RECEPTOR | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | GROWTH | VASCULAR-PERMEABILITY | CELL APOPTOSIS | HYPOXIA | EXPRESSION | Insulin-Like Growth Factor Binding Protein 3 - genetics | Retina - drug effects | Diabetic Retinopathy - drug therapy | Vascular Endothelial Growth Factor A - biosynthesis | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Apoptosis - drug effects | Humans | Reperfusion Injury - drug therapy | Diabetic Retinopathy - pathology | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor Receptor-2 - genetics | Adrenergic beta-Agonists - administration & dosage | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Reperfusion Injury - genetics | Angiopoietin-1 - biosynthesis | Reperfusion Injury - pathology | Receptor, TIE-2 - biosynthesis | Rats | Diabetic Retinopathy - genetics | Receptor, TIE-2 - genetics | Gene Expression Regulation - drug effects | Angiopoietin-1 - genetics | Insulin-Like Growth Factor Binding Protein 3 - biosynthesis | Animals | Diabetes Mellitus, Experimental - pathology | Cell Hypoxia - genetics | Endothelial Cells - pathology | Retina - pathology | Endothelial Cells - drug effects | Diabetic retinopathy | Analysis | Influence | Research | Reperfusion (Physiology) | Vascular endothelial growth factor | Risk factors | Angiopoietin | Retina | Biology | Insulin-like growth factors | Drug overdose | Western blotting | Proteins | Reperfusion | Ischemia | Rodents | Insulin-like growth factor-binding protein 3 | Capillaries | Diabetes mellitus | Exposure | Insulin | Endothelial cells | Animal control | Studies | Ghosts | Hypoxia | Adrenergic receptors | Diabetes | Cancer | Apoptosis
Journal Article
The EMBO Journal, ISSN 0261-4189, 06/2005, Volume 24, Issue 11, pp. 1976 - 1987
The ecotropic viral integration site‐1 ( Evi1 ) is an oncogenic transcription factor in murine and human myeloid leukemia. We herein show that Evi1 is... 
Evi1 | angiopoiesis | hematopoiesis | GATA‐2 | Angiopoiesis | Hematopoiesis | GATA-2 | MYELOID-TRANSFORMING GENE | YOLK-SAC | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE EMBRYOS | PARAAORTIC SPLANCHNOPLEURA | CELL BIOLOGY | DEFINITIVE HEMATOPOIESIS | ZINC-FINGER PROTEIN | MICE | LEUKEMIA | DIFFERENTIATION | BINDING | Angiopoietin-2 - genetics | Cells, Cultured - cytology | Humans | Blood Vessels - embryology | RNA, Messenger - biosynthesis | Yolk Sac - blood supply | Proto-Oncogenes - genetics | Gene Expression Regulation, Developmental | Bone Marrow - metabolism | Cell Division | Transcription, Genetic | Hematopoietic System - embryology | Proto-Oncogenes - physiology | Angiopoietin-1 - biosynthesis | Promoter Regions, Genetic | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Neovascularization, Physiologic - genetics | Receptor, TIE-2 - biosynthesis | Angiopoietin-2 - biosynthesis | Hematopoiesis - genetics | Transcription Factors - biosynthesis | Hematopoietic Stem Cells - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, Knockout | Receptor, TIE-2 - genetics | Angiopoietin-1 - genetics | Neovascularization, Physiologic - physiology | Animals | Hematopoietic Stem Cells - cytology | Cells, Cultured - metabolism | GATA2 Transcription Factor | Mice | DNA-Binding Proteins - biosynthesis | MDS1 and EVI1 Complex Locus Protein
Journal Article
Angiogenesis, ISSN 0969-6970, 10/2013, Volume 16, Issue 4, pp. 921 - 937
Notch is an intercellular signaling pathway related mainly to sprouting neo-angiogenesis. The objective of our study was to evaluate the angiogenic mechanisms... 
Angiogenesis | Biomedicine | Notch signaling | EphrinB2/EphB4 | Cancer Research | Oncology | Ophthalmology | Molecular regulation of intussusceptive angiogenesis | SDF-1/CXCR4 | Cardiology | Biomedicine general | Cell Biology | ACTIVATION | RECEPTOR | LIGAND DELTA-LIKE-4 | SPECIFICATION | VASCULAR MORPHOGENESIS | PATHWAY | ENDOTHELIAL-CELLS | GROWTH | PERIPHERAL VASCULAR DISEASE | TIP | EXPRESSION | Liver - pathology | Male | Receptor, EphB4 - genetics | RNA, Messenger - biosynthesis | Receptor, Notch1 - deficiency | Receptors, Notch - antagonists & inhibitors | Chemokine CXCL12 - genetics | Receptor, EphB2 - biosynthesis | Pericytes - pathology | Bone Marrow Transplantation | Leukocytes, Mononuclear - transplantation | Neovascularization, Pathologic - physiopathology | Neovascularization, Pathologic - prevention & control | Receptors, CXCR4 - genetics | Receptors, Vascular Endothelial Growth Factor - genetics | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptor, TIE-2 - biosynthesis | Receptors, Vascular Endothelial Growth Factor - biosynthesis | Gene Expression Regulation | Receptors, Notch - physiology | Receptor, EphB2 - genetics | Chick Embryo | Receptor, TIE-2 - genetics | Animals | Receptors, CXCR4 - biosynthesis | Signal Transduction - drug effects | Chemokine CXCL12 - biosynthesis | Neovascularization, Pathologic - genetics | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Receptor, EphB4 - biosynthesis | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Oligopeptides - pharmacology
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