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Cell death and differentiation, ISSN 1476-5403, 06/2014, Volume 21, Issue 10, pp. 1511 - 1521
Necroptosis is a form of programmed cell death that depends on the activation of receptor interacting protein kinase-1 (RIPK1... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
Journal Article
Nature immunology, ISSN 1529-2916, 03/2013, Volume 14, Issue 5, pp. 480 - 488
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 10/2013, Volume 288, Issue 43, pp. 31268 - 31279
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 09/2018, Volume 22, Issue 9, pp. 4183 - 4196
... such as the NO cascade, the reperfusion injury salvage kinase (RISK) pathway and the survival activating factor enhancement (SAFE) have been suggested to underlie the phenotypes... 
RIP1 inhibition | ischaemia‐reperfusion injury | heart | necroptosis | ischaemia-reperfusion injury | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Protein Kinases - genetics | Oxidative Stress | Rats, Wistar | Apoptosis - drug effects | Anti-Arrhythmia Agents - pharmacology | Male | Protein Transport - drug effects | Necrosis - pathology | Myocardial Reperfusion Injury - pathology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Indoles - pharmacology | Cell Membrane - metabolism | Phosphorylation - drug effects | Myocardial Reperfusion Injury - genetics | Cell Membrane - drug effects | Organ Culture Techniques | Protein-Serine-Threonine Kinases - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Calcium-Binding Proteins - metabolism | Heart - physiopathology | Signal Transduction | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Rats | Necrosis - metabolism | Imidazoles - pharmacology | Necrosis - prevention & control | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Myocytes, Cardiac - drug effects | Myocardial Reperfusion Injury - therapy | Ischemic Preconditioning, Myocardial | Heart - drug effects | Myocytes, Cardiac - metabolism | Necrosis - genetics | Calcium-Binding Proteins - genetics | Anti-arrhythmia drugs | Heart | Translocation | Oxidative stress | Phosphorylation | Damage assessment | Oligomerization | Pharmacology | Heart function | Muscle contraction | Mitigation | Signaling | Reperfusion | Ischemia | Inhibition | Preconditioning | Peroxidation | Ca2+/calmodulin-dependent protein kinase II | Apoptosis | Index Medicus | Original
Journal Article
Journal Article