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The New England Journal of Medicine, ISSN 0028-4793, 08/2013, Volume 369, Issue 7, pp. 621 - 629
Exome sequencing in a family with autosomal dominant congenital urinary tract malformations showed a mutation in dual serine–threonine and tyrosine protein... 
MEDICINE, GENERAL & INTERNAL | ZEBRAFISH | FGF | ABNORMALITIES | GENES | CONGENITAL-ANOMALIES | KIDNEY | RENAL HYPODYSPLASIA | EMBRYOGENESIS | CHILDREN | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Humans | Molecular Sequence Data | Infant | Male | Urinary Tract - abnormalities | Gene Knockdown Techniques | Exome | Young Adult | Base Sequence | Adult | Female | Urinary Tract - metabolism | Child | Genetic Linkage | Genome-Wide Association Study | Urinary Tract - growth & development | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Kidney - abnormalities | Pedigree | Heterozygote | Mice | RNA, Small Interfering | Mutation | Urogenital Abnormalities - genetics | Child development deviations | Causes of | Genetic aspects | Research | Gene mutations | Developmental disabilities | Pediatrics | Fibroblast growth factor | Phosphorylation | Nephrology | Mesenchyme | Congenital defects | Serine | Genomes | mRNA | Urinary tract | Kinases | Proteins | Ureter | Charitable foundations | Urogenital system | Age | Linkage analysis | Tyrosine | Immunoglobulins | Congenital diseases | Kidneys | Threonine | Splicing | Extracellular signal-regulated kinase | Cell membranes | Grants | Studies | Protein kinase | Renal failure | Ligands | Diabetes | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 43, pp. 31268 - 31279
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2018, Volume 128, Issue 5, pp. 2064 - 2075
Receptor interacting protein kinase 1 (RIPK1) has important kinase-dependent and kinase-independent scaffolding functions that activate or prevent apoptosis or... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | MLKL | PHASE-III | IN-VIVO | SEPTIC SHOCK | DOUBLE-BLIND | PROGRAMMED NECROSIS | TNF-ALPHA | SEVERE SEPSIS | CELL-DEATH | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Amino Acid Chloromethyl Ketones - toxicity | Liver - pathology | Liver - enzymology | Endothelium, Vascular - injuries | Mice, 129 Strain | Endothelium, Vascular - enzymology | Tumor Necrosis Factor-alpha - toxicity | Systemic Inflammatory Response Syndrome - genetics | Necrosis | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Systemic Inflammatory Response Syndrome - pathology | Animals | Mice, Mutant Strains | Systemic Inflammatory Response Syndrome - enzymology | Endothelium, Vascular - pathology | Mice | Systemic Inflammatory Response Syndrome - chemically induced | Hematopoietic Stem Cells | Care and treatment | Tumor necrosis factor | Physiological aspects | Development and progression | Sepsis | Inflammation | Models | Protein kinases | Health aspects | Apoptosis | Shock | Phosphorylation | Liver | Lethality | Kinases | Clotting | Proteins | Intestine | Rodents | Cell survival | Bacterial infections | Cytokines | Neutrophils | MAP kinase | Permeability | Trauma | Endothelial cells | Hemopoiesis | Endothelium | Protein kinase | Chemokines | Hypothermia | Index Medicus | Abridged Index Medicus
Journal Article
Cell, ISSN 0092-8674, 05/2014, Volume 157, Issue 5, pp. 1175 - 1188
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 09/2016, Volume 23, Issue 9, pp. 1565 - 1576
Necroptosis is a caspase-independent form of cell death that is triggered by activation of the receptor interacting serine/threonine kinase 3 (RIPK3) and... 
MYOCARDIAL-INFARCTION | RECEPTOR-INTERACTING PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOMAIN-LIKE PROTEIN | REPERFUSION INJURY | MIXED LINEAGE KINASE | PROGRAMMED NECROSIS | MEDIATES NECROPTOSIS | TNF-ALPHA | NECROPTOTIC CELL-DEATH | NF-KAPPA-B | CELL BIOLOGY | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Protein Kinases - genetics | Sepsis - etiology | Apoptosis - drug effects | Colitis - pathology | Male | Inflammation - metabolism | Sepsis - pathology | Sepsis - metabolism | Colitis - chemically induced | Female | Protein Kinases - deficiency | Reperfusion Injury - metabolism | Disease Models, Animal | Systemic Inflammatory Response Syndrome - metabolism | Lipopolysaccharides - toxicity | Reperfusion Injury - pathology | Mice, Inbred C57BL | Ceruletide - toxicity | Reperfusion Injury - mortality | Pancreatitis - chemically induced | Dextran Sulfate - toxicity | Mice, Knockout | Tumor Necrosis Factor alpha-Induced Protein 3 - genetics | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Systemic Inflammatory Response Syndrome - pathology | Animals | Colitis - metabolism | Pancreatitis - pathology | Systemic Inflammatory Response Syndrome - etiology | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Pancreatitis - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor alpha-Induced Protein 3 - deficiency | Index Medicus | Original Paper
Journal Article