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Alcoholism: Clinical and Experimental Research, ISSN 0145-6008, 10/2011, Volume 35, Issue 10, pp. 1812 - 1821
Background:  Hypothalamic glial–neuronal communications are important for the activation of luteinizing hormone releasing hormone (LHRH) secretion at the time... 
Puberty | Alcohol | Receptor Protein Tyrosine Phosphatase‐β | Glia | Insulin‐Like Growth Factor‐1 | Insulin-like growth factor-1 | Receptor protein tyrosine phosphatase-β | RHESUS-MONKEYS | Insulin-Like Growth Factor-1 | RIBONUCLEIC-ACID EXPRESSION | SUBSTANCE ABUSE | HORMONE-RELEASING HORMONE | LUTEINIZING-HORMONE | FACTOR-ALPHA | Receptor Protein Tyrosine Phosphatase-beta | SEXUAL-MATURATION | MEDIAN-EMINENCE | KISS-1 GENE-EXPRESSION | GROWTH-FACTOR-I | ESTROUS-CYCLE | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - biosynthesis | Gene Expression - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - analysis | Humans | Neuroglia | Insulin-Like Growth Factor I - genetics | Sexual Maturation - physiology | Central Nervous System Depressants - pharmacology | Contactin 1 - analysis | Luteinizing Hormone - antagonists & inhibitors | Female | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - genetics | Insulin-Like Growth Factor I - biosynthesis | Ethanol - metabolism | RNA - analysis | Signal Transduction | Rats | Gonadotropin-Releasing Hormone - biosynthesis | Rats, Sprague-Dawley | Ethanol - pharmacology | Animals | Hypothalamus - metabolism | Contactin 1 - biosynthesis | Contactin 1 - genetics | Insulin-Like Growth Factor I - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - metabolism | Central Nervous System Depressants - metabolism | alcohol | RPTPβ | puberty | IGF-1 | glia
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2014, Volume 135, Issue 5, pp. 1101 - 1109
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2011, Volume 15, Issue 11, pp. 2353 - 2363
Substantial genetic evidence suggests that chromosome 11q is involved in regulating initiation and progression of malignant melanomas. Mutations of the MEN1... 
melanoma | pleiotrophin | menin | RPTP β/ζ | Melanoma | Menin | Pleiotrophin | MEDICINE, RESEARCH & EXPERIMENTAL | RPTP ss | PROTEIN | GROWTH-FACTOR PLEIOTROPHIN | TYROSINE-PHOSPHATASE BETA/ZETA | RECEPTOR | TUMOR-CELLS | ENDOCRINE NEOPLASIA TYPE-1 | CANCER | CELL BIOLOGY | ANAPLASTIC LYMPHOMA KINASE | GENE | CELL-MIGRATION | Phosphorylation | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - biosynthesis | Cell Proliferation | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Melanoma, Experimental - metabolism | Transplantation, Heterologous | Extracellular Signal-Regulated MAP Kinases - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Chromatin Immunoprecipitation | Female | Phosphatidylinositol 3-Kinase - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - antagonists & inhibitors | Melanoma - metabolism | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Phosphatidylinositol 3-Kinase - biosynthesis | Cytokines - metabolism | Signal Transduction | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Carrier Proteins - biosynthesis | Mice, Inbred C57BL | Carrier Proteins - antagonists & inhibitors | Intercellular Signaling Peptides and Proteins - genetics | Melanoma, Experimental - pathology | Melanoma - pathology | Phenotype | Animals | Carrier Proteins - metabolism | Cell Line, Tumor | CpG Islands - genetics | Mice | Cytokines - antagonists & inhibitors | Cytokines - biosynthesis | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - metabolism | Cell Movement | Cell proliferation | Leukemia | DNA damage | Lung cancer | Metastasis | Kinases | Skin cancer | Proteins | Signal transduction | Cell growth | Pathways | Multiple endocrine neoplasia | Penicillin | DNA methylation | Chromosome 11 | Inhibition | Chromosomes | Growth factors | Deoxyribonucleic acid--DNA | CpG islands | Tyrosine | Phenotypes | Immunoglobulins | Extracellular signal-regulated kinase | Gene expression | 1-Phosphatidylinositol 3-kinase | Signaling | Focal adhesion kinase | Mutation | Cell migration | Apoptosis | Protein-tyrosine-phosphatase | Tumors | RPTP β | Original
Journal Article
NeuroMolecular Medicine, ISSN 1535-1084, 6/2014, Volume 16, Issue 2, pp. 457 - 472
To address the role of the transforming growth factor beta (TGFβ)-Smad3 signaling pathway in dendrite growth and associated synaptogenesis, we used small... 
Neurology | Neurosciences | Smad3 | Biomedicine | Synaptogenesis | Dendrite growth | Internal Medicine | Astrocyte | Transforming growth factor beta | Status epilepticus | Chondroitin sulfate proteoglycans | CELLS | AMYLOID DEPOSITION | NEUROTROPHIC FACTOR | ASTROCYTES | NEUROSCIENCES | ACTIVIN | ENHANCES SYNAPTOGENESIS | GENE PROMOTER | NEURITE OUTGROWTH | TRANSFORMING GROWTH-FACTOR-BETA-1 | FACTOR BETA-1 | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - biosynthesis | Chondroitin Sulfate Proteoglycans - genetics | Culture Media, Conditioned - pharmacology | Male | Chondroitin ABC Lyase - pharmacology | Smad3 Protein - deficiency | Smad3 Protein - genetics | Neurons - ultrastructure | Protein Processing, Post-Translational - drug effects | RNA Interference | Female | Neurons - metabolism | Active Transport, Cell Nucleus | Chondroitin Sulfate Proteoglycans - biosynthesis | Transforming Growth Factor beta1 - pharmacology | Synapsins - analysis | Astrocytes - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 5 - genetics | RNA, Small Interfering - pharmacology | Cells, Cultured | Gene Expression Regulation | Synapses - ultrastructure | Smad3 Protein - antagonists & inhibitors | Transforming Growth Factor beta1 - physiology | Mice, Inbred ICR | Mice, Knockout | Astrocytes - ultrastructure | Neurocan - genetics | Animals | Signal Transduction - physiology | Mice | Status Epilepticus - metabolism | Smad3 Protein - physiology | Neurocan - biosynthesis | Astrocytes - metabolism
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Journal Article
Journal Article